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Immunity, ISSN 1074-7613, 03/2012, Volume 36, Issue 3, pp. 401 - 414
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 02/2014, Volume 50, Issue 2, pp. 270 - 280
IL-1 beta is a potent proinflammatory cytokine that is implicated in the pathogenesis of acute respiratory distress syndrome. We hypothesized that LPS and... 
Acute respiratory distress syndrome | Acute lung injury | IL-1β | Nucleotide binding domain and leucine-rich repeat pyrin domain containing 3 inflammasome | TIDAL VOLUME | ACTIVATION | IL-18 SECRETION | BIOCHEMISTRY & MOLECULAR BIOLOGY | PULMONARY INFLAMMATION | MECHANICAL VENTILATION | RECEPTOR | IL-1 beta | acute lung injury | NEUTROPHIL RECRUITMENT | CELL BIOLOGY | RESPIRATORY SYSTEM | acute respiratory distress syndrome | LIPOPOLYSACCHARIDE | ALVEOLAR MACROPHAGES | nucleotide binding domain and leucine-rich repeat pyrin domain containing 3 inflammasome | ARDS | Interleukin-1 - metabolism | Respiration, Artificial - methods | NLR Family, Pyrin Domain-Containing 3 Protein | Receptors, Interleukin-1 - immunology | Mice, Inbred C57BL | Interleukin-1beta - immunology | Caspase 1 - metabolism | Mice, Transgenic | Caspase 1 - immunology | Hypoxia - immunology | Inflammation - immunology | Lipopolysaccharides - immunology | Receptors, Interleukin-1 - metabolism | Signal Transduction - immunology | Animals | Carrier Proteins - metabolism | Respiration, Artificial - adverse effects | Interleukin-1beta - metabolism | Interleukin-1 - immunology | Acute Lung Injury - immunology | Acute Lung Injury - metabolism | Mice | Disease Models, Animal | Neutrophil Infiltration - immunology | Studies | Respiratory distress syndrome | Mortality | Rodents | Ventilation | Gene expression | Experiments | Apoptosis | Original Research
Journal Article
JOURNAL OF CLINICAL INVESTIGATION, ISSN 0021-9738, 07/2019, Volume 129, Issue 7, pp. 2888 - 2897
Journal Article
Journal Article
Frontiers in Immunology, ISSN 1664-3224, 2019, Volume 10, p. 754
Autophagy can either antagonize or promote intracellular bacterial growth, depending on the pathogen. Here, we investigated the role of autophagy during a... 
Chlamydia pneumoniae | Inflammasome | IL-1β | Macrophages | Autophagy | INTERLEUKIN-1 | CLEARANCE | ACTIVATION | BACTERIA | autophagy | PROTECTION | IL-1 beta | INDUCTION | IMMUNOLOGY | macrophages | ATG16L1 | inflammasome | PSEUDOMONAS-AERUGINOSA | DISEASE | Chlamydia infections | Autophagy (Cytology) | Interleukin-1 | Research
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 07/2019, Volume 129, Issue 7, p. 2888
Influenza A virus-specific (IAV-specific) T cell responses are important correlates of protection during primary and subsequent infections. The generation and... 
Dendritic cells | Cell death | Influenza | T cells | Comparative analysis | Health aspects
Journal Article
Journal Article
Cell Metabolism, ISSN 1550-4131, 09/2018, Volume 28, Issue 3, p. 432
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1016/j.cmet.2018.05.027 Pathogen... 
Medical research | Medical colleges | Pneumonia | Interleukins | Chlamydia | Analysis | Bacterial pneumonia | Atherosclerosis | Medicine, Experimental | Macrophages | Niacin
Journal Article
Cell metabolism, ISSN 1550-4131, 9/2018, Volume 28, Issue 3, pp. 432 - 448.e4
Pathogen burden accelerate atherosclerosis, but the mechanisms remain unresolved. Activation of the NLRP3 inflammasome is linked to atherogenesis. Here we... 
atherosclerosis | foam cells | ABCA1 | C. pneumoniae | Gpr109a | oxLDL | niacin | macrophage | Nlrp3 | cholesterol efflux | aspartate | Interleukin-1 beta
Journal Article
Immunity, ISSN 1074-7613, 04/2015, Volume 42, Issue 4, pp. 640 - 653
Acute lung injury (ALI) remains a serious health issue with little improvement in our understanding of the pathophysiology and therapeutic approaches. We... 
BACTERIAL LIPOPOLYSACCHARIDE | STERILE INFLAMMATION | SIGNALING PATHWAY | P2X RECEPTORS | ALPHA PRODUCTION | ENDOTHELIAL-CELLS | MOUSE MODEL | IN-VIVO | ACUTE LUNG INJURY | IMMUNOLOGY | INFLAMMATORY RESPONSE | Interleukin-1alpha - immunology | Macrophages, Alveolar - pathology | Calcium - metabolism | Humans | Capillary Permeability - immunology | Neutrophil Infiltration | Necrosis - pathology | Receptors, Purinergic P2X7 - genetics | Necrosis - immunology | Cadherins - immunology | Adenosine Triphosphate - metabolism | HEK293 Cells | Interleukin-1alpha - genetics | Cadherins - genetics | Receptors, Purinergic P2X7 - immunology | Macrophages, Alveolar - immunology | Neutrophils - pathology | Lung - pathology | Protein Precursors - genetics | Signal Transduction | Neutrophils - drug effects | Gene Expression Regulation | Neutrophils - immunology | Lipopolysaccharide Receptors - immunology | Mice, Transgenic | Protein Precursors - immunology | Acute Lung Injury - pathology | Necrosis - chemically induced | Endothelial Cells - immunology | Animals | Acute Lung Injury - chemically induced | Macrophages, Alveolar - drug effects | Lung - drug effects | Lipopolysaccharides - pharmacology | Lipopolysaccharide Receptors - genetics | Acute Lung Injury - immunology | Mice | Endothelial Cells - pathology | Intubation, Intratracheal | Lung - immunology | Endothelial Cells - drug effects | endothelial cells | alveolar macrophage | Ca2+ influx | MyD88 | IL-1α | neutrophil | P2X7R | lung injury | necrosis | ARDS | ALI | sepsis | lung inflammation
Journal Article
Journal of immunology (Baltimore, Md. : 1950), ISSN 0022-1767, 2/2015, Volume 194, Issue 4, pp. 1686 - 1694
We previously identified a novel alternatively spliced isoform of human myeloid differentiation protein-2 (MD-2s) that competitively inhibits binding of MD-2... 
HDM | TLR-4 | Der p 2 | innate immunity | MD-2 | acute lung injury | LPS | allergic lung inflammation
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2015, Volume 35, Issue 12, pp. 2605 - 2616
OBJECTIVE—Kawasaki disease (KD) is the most common cause of acute vasculitis and acquired cardiac disease among US children. We have previously shown that both... 
dendritic cells | interleukin-1 | mucocutaneous lymph node syndrome | MyD88 | Endothelial cells | protein | endothelial cells | ELEMENT-BINDING PROTEIN-2 | ACTIVATION | RECEPTOR | CORONARY-ARTERY ANEURYSMS | NLRP3 INFLAMMASOME | POLYMORPHISM | ACUTE-PHASE | INTRAVENOUS IMMUNOGLOBULIN | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | ASSOCIATION | Lactobacillus casei | Mucocutaneous Lymph Node Syndrome - metabolism | NLR Family, Pyrin Domain-Containing 3 Protein | Stromal Cells - pathology | Caspase 1 - metabolism | Interleukin-1alpha - metabolism | Aortitis - chemically induced | Aorta - metabolism | Coronary Vessels - metabolism | Cell Wall | DNA-Binding Proteins - metabolism | Receptors, Interleukin-1 Type I - metabolism | Interleukin-1beta - metabolism | Bone Marrow Transplantation | Coronary Artery Disease - pathology | Aortitis - pathology | Dendritic Cells - metabolism | Disease Models, Animal | Mucocutaneous Lymph Node Syndrome - pathology | Coronary Vessels - pathology | Signal Transduction | Coronary Artery Disease - chemically induced | Endothelial Cells - metabolism | Mucocutaneous Lymph Node Syndrome - chemically induced | Coronary Artery Disease - metabolism | Mice, Inbred C57BL | Stromal Cells - metabolism | Cells, Cultured | Myeloid Differentiation Factor 88 - genetics | Aortitis - genetics | DNA-Binding Proteins - genetics | Transplantation Chimera | Mice, Knockout | Aorta - pathology | Mucocutaneous Lymph Node Syndrome - genetics | Aortitis - metabolism | Macrophages - metabolism | Animals | Carrier Proteins - metabolism | Coronary Artery Disease - genetics | Receptors, Interleukin-1 Type I - genetics | Myeloid Differentiation Factor 88 - metabolism | Bone Marrow Cells - metabolism | coronary artery vasculitis | Kawasaki disease | IL-1
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2017, Volume 66, Issue 1, pp. S501 - S501
Journal Article
Journal of Hepatology, ISSN 0168-8278, 2017, Volume 66, Issue 1, pp. S501 - S501
Journal Article