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Journal Article
Neurobiology of Disease, ISSN 0969-9961, 2006, Volume 22, Issue 2, pp. 265 - 273
Parkinson disease (PD) and dementia with Lewy bodies (DLB) are characterized by the accumulation of abnormal α-synuclein and ubiquitin in protein aggregates... 
Parkinson's disease | Dementia with Lewy Bodies | UCHL | α-synuclein | Proteasome | proteasome | BODY DISEASE | GAD MUTANT MOUSE | PARKINSONS-DISEASE SUSCEPTIBILITY | PROTEASOMAL FUNCTION | alpha-synuclein | SUBSTANTIA-NIGRA | NEUROSCIENCES | NERVE-TERMINALS | GENE | PATHWAY | dementia with Lowy bodies | NEUROPATHOLOGY | Substantia Nigra - physiopathology | Neurons - pathology | Substantia Nigra - pathology | Humans | Middle Aged | Cerebral Cortex - pathology | Lewy Body Disease - enzymology | Ubiquitin - metabolism | Brain - enzymology | Male | RNA, Messenger - metabolism | Protein Subunits - metabolism | Cerebral Cortex - physiopathology | Medulla Oblongata - physiopathology | Lewy Bodies - genetics | Lewy Body Disease - genetics | Aged, 80 and over | Ubiquitin Thiolesterase - metabolism | Female | Medulla Oblongata - pathology | Protein Subunits - genetics | Substantia Nigra - enzymology | Alzheimer Disease - physiopathology | Cerebral Cortex - enzymology | Brain - physiopathology | Lewy Bodies - enzymology | Alzheimer Disease - enzymology | Parkinson Disease - genetics | Parkinson Disease - physiopathology | Ubiquitin Thiolesterase - genetics | Down-Regulation - physiology | Proteasome Endopeptidase Complex - genetics | Neurons - enzymology | Parkinson Disease - enzymology | Brain - pathology | Signal Transduction - physiology | Aged | Medulla Oblongata - enzymology | Proteasome Endopeptidase Complex - metabolism | Alzheimer Disease - genetics | Lewy Body Disease - physiopathology | Ubiquitin | Enzymes | Hydrolases | Messenger RNA | Proteolysis | Brain diseases
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2018, Volume 115, Issue 41, pp. 10481 - 10486
Journal Article
Journal of Neuropathology and Experimental Neurology, ISSN 0022-3069, 09/2005, Volume 64, Issue 9, pp. 816 - 830
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 06/2005, Volume 280, Issue 22, pp. 21522 - 21530
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 12/2005, Volume 20, Issue 3, pp. 685 - 693
The aim of the present work was to analyze the status of metabotropic glutamate receptors (mGluRs) in the frontal cortex (area 8) from ten cases with common... 
Alzheimer's disease | Metabotropic glutamate receptor | Dementia with Lewy bodies | Phospholipase C | dementia with Lewy bodies | phospholipase C | metabotropic glutamate receptor | TERMINALS | ALPHA-SYNUCLEIN | NEUROTOXICITY | NEUROSCIENCES | BODY DEMENTIA | PSYCHOSIS | RAT-BRAIN | BINDING | EXPRESSION | AGGREGATION | SPORADIC PARKINSONS-DISEASE | Type C Phospholipases - drug effects | Neurons - pathology | Guanosine Triphosphate - pharmacology | Humans | Middle Aged | Lewy Body Disease - enzymology | Type C Phospholipases - metabolism | Male | Guanosine Triphosphate - metabolism | Receptors, Metabotropic Glutamate - metabolism | Alzheimer Disease - pathology | Phospholipase C beta | Isoenzymes - metabolism | Aged, 80 and over | Female | Radioligand Assay | Alzheimer Disease - physiopathology | Binding, Competitive - physiology | Glutamic Acid - pharmacology | Frontal Lobe - physiopathology | Lewy Body Disease - pathology | Alzheimer Disease - enzymology | Enzyme Activation - drug effects | Frontal Lobe - enzymology | Disease Progression | Down-Regulation - physiology | Neurons - enzymology | Signal Transduction - physiology | Aged | Glutamic Acid - metabolism | Isoenzymes - drug effects | Enzyme Activation - physiology | Frontal Lobe - pathology | Lewy Body Disease - physiopathology | Brain | Analysis | Development and progression | Amino acids | Phospholipases | Glutamate
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 9/2018, Volume 55, Issue 9, pp. 7533 - 7552
Parkinson’s disease is associated with intracellular α-synuclein accumulation and ventral midbrain dopaminergic neuronal death in the Substantia Nigra of brain... 
Dopaminergic neurons | Neurology | Neurosciences | Biomedicine | Autophagy impairment | Neurobiology | RAC1/ced-10 | Alpha-synuclein accumulation | Parkinson’s disease | Cell Biology | SIGNALING PATHWAYS | STEM-CELLS | Parkinson's disease | CHAPERONE-MEDIATED AUTOPHAGY | ALZHEIMERS-DISEASE | NEUROSCIENCES | PARKINSONS-DISEASE MODEL | CELL-DEATH | CAENORHABDITIS-ELEGANS | IN-VIVO | C-ELEGANS | APOPTOTIC CELL | Humans | Caenorhabditis elegans Proteins - metabolism | rac GTP-Binding Proteins - metabolism | Autophagy - drug effects | Neuroprotection - drug effects | Caenorhabditis elegans - physiology | Amyloid - metabolism | Behavior, Animal - drug effects | Cell Death - drug effects | Inclusion Bodies - metabolism | Dopamine - metabolism | Neurites - drug effects | Induced Pluripotent Stem Cells - metabolism | Mesencephalon - pathology | Biomarkers - metabolism | Induced Pluripotent Stem Cells - pathology | Cell Survival - drug effects | Parkinson Disease - pathology | Induced Pluripotent Stem Cells - drug effects | alpha-Synuclein - toxicity | Neurites - metabolism | Mutation - genetics | Dopaminergic Neurons - enzymology | Animals | Caenorhabditis elegans - drug effects | Cell Line, Tumor | Caenorhabditis elegans - enzymology | rac1 GTP-Binding Protein - metabolism | Amyloidogenesis | Dopamine | Cell survival | Mesencephalon | Toxicity | Pathogenesis | Substantia nigra | Parkinsons disease | Rac1 protein | Neuroblastoma | Synuclein | Autophagy | Neuroblastoma cells | Accumulation | Cell death | Actin | LRRK2 protein | Phagocytosis | Dopamine receptors | Guanosinetriphosphatase | Index Medicus | RAC1 | ced-10
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2018, Volume 115, Issue 41, p. 10481
Parkinson’s disease (PD) is characterized by a progressive loss of dopaminergic neurons, a process that current therapeutic approaches cannot prevent. In PD,... 
Dopamine | Parkinson's disease | Neurodegenerative diseases | Neurons | Toxicity | Computational neuroscience | Fibrils | Parkinsons disease | Muscles | High-throughput screening | Disaggregation | Agglomeration | Inclusions | Synuclein | Lewy bodies | Proteins | Axons | Protein folding | Neurodegeneration | Computer applications | Amyloid | Degeneration | Movement disorders | Dopamine receptors
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 9/2018, Volume 55, Issue 9, pp. 7553 - 7554
With the author(s)’ decision to opt for Open Choice the copyright of the article changed on March 2018 to © The Author(s) 2018 and the name of one of the... 
Neurology | Neurosciences | Biomedicine | Neurobiology | Cell Biology | Dopamine | Cell survival | Toxicity | Rac1 protein | Synuclein | Dopamine receptors | Guanosinetriphosphatase
Journal Article
Neurobiology of Aging, ISSN 0197-4580, 03/2008, Volume 29, Issue 3, p. 408
Previous studies in Lewy body diseases (LBDs), including Parkinson's disease (PD) and Dementia with Lewy bodies (DLB), have shown oxidative stress damage more... 
Journal Article
Neurobiology of Aging, ISSN 0197-4580, 2006, Volume 27, Issue 6, pp. 848 - 856
Journal Article
Proceedings of the National Academy of Sciences of the United States, ISSN 0027-8424, 10/2018, Volume 115, Issue 41, p. 10481
Parkinson's disease | [alpha]-synuclein | protein aggregation | aggregation inhibition | dopaminergic degeneration 
Physiological aspects | Neural circuitry | Parkinson's disease | Dopamine | Health aspects
Journal Article
16.