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Nature Communications, ISSN 2041-1723, 04/2014, Volume 5, Issue 1, p. 3630
Journal Article
Nature Genetics, ISSN 1061-4036, 03/2017, Volume 49, Issue 3, pp. 451 - 456
Journal Article
Nature Genetics, ISSN 1061-4036, 12/2016, Volume 48, Issue 12, pp. 1551 - 1556
Journal Article
Nature Genetics, ISSN 1061-4036, 04/2015, Volume 47, Issue 4, pp. 330 - 337
Journal Article
Veterinary Pathology, ISSN 0300-9858, 11/2019, Volume 56, Issue 6, pp. 950 - 958
In humans and in mouse models, precursor B-cell lymphoblastic leukemia (B-ALL)/lymphoblastic lymphoma (B-LBL) can be classified as either the pro-B or pre-B... 
B-ALL | flow cytometry | CD25 | immunohistochemistry | B-LBL | mice | B-cell leukemia/lymphoma | IgM | Index Medicus
Journal Article
Nature Communications, ISSN 2041-1723, 12/2019, Volume 10, Issue 1, pp. 2789 - 10
IGH@ proto-oncogene translocation is a common oncogenic event in lymphoid lineage cancers such as B-ALL, lymphoma and multiple myeloma. Here, to investigate... 
ACTIVATION | ALIGNMENT | PRE-B | MULTIDISCIPLINARY SCIENCES | GENES | DUX4 | GENOME | Translocation | Breakpoints | Immunoglobulins | Profiling | Acute lymphatic leukemia | Leukemia | Multiple myeloma | Genomes | Lymphatic leukemia | Gene expression | Lymphoma | Gene sequencing | Lymphocytes B | Allelic exclusion | Heavy chains | Alleles | Epigenetics
Journal Article
Leukemia, ISSN 0887-6924, 10/2017, Volume 31, Issue 10, pp. 2228 - 2234
Acute megakaryoblastic leukemia in patients without Down syndrome is a rare malignancy with a poor prognosis. RNA sequencing of fourteen pediatric cases... 
ONCOLOGY | FLT3 | ACUTE LYMPHOBLASTIC-LEUKEMIA | DOWN-SYNDROME | ACUTE MEGAKARYOBLASTIC LEUKEMIA | GENOME-WIDE ANALYSIS | GENE-EXPRESSION | ACUTE MYELOID-LEUKEMIA | MUTATIONS | HEMATOLOGY | CHILDRENS ONCOLOGY GROUP | ACUTE PROMYELOCYTIC LEUKEMIA | Neoplasm Transplantation | RNA, Small Interfering - genetics | Humans | Hematopoietic Stem Cells - pathology | Gene Expression Profiling | Gene Expression Regulation, Leukemic - genetics | Oncogene Proteins, Fusion - physiology | Thrombopoiesis - genetics | Cell Self Renewal | Neoplastic Stem Cells - metabolism | Cell Transformation, Neoplastic - genetics | Neoplastic Stem Cells - pathology | Female | Leukemia, Megakaryoblastic, Acute - genetics | Transcription Factors - physiology | Mice, Inbred C57BL | Hematopoietic Stem Cells - metabolism | Radiation Chimera | Transcription Factors - genetics | Phenotype | Animals | Oncogene Proteins, Fusion - genetics | Bone Marrow - pathology | Mice | Models, Genetic | Myeloid Cells - pathology | Transcription factors | Care and treatment | Leukemia | Development and progression | Genetic aspects | Gene expression | Health aspects | Transformation | Animal models | Syngeneic grafts | Genes | Bone marrow transplantation | Pharmacology | Malignancy | Ribonucleic acid--RNA | Chemical compounds | Gene sequencing | Down's syndrome | Bone marrow | Mutation | Inducers | Tumors
Journal Article
Journal of Neurology, ISSN 0340-5354, 1/2009, Volume 256, Issue 1, pp. 89 - 103
Teriflunomide is an orally available anti-inflammatory drug that prevents T and B cell proliferation and function by inhibition of dihydroorotate... 
Neurology | dihydroorotate dehydrogenase | Neurosciences | experimental autoimmune encephalomyelitis | Medicine & Public Health | Dark Agouti rat | multiple sclerosis | teriflunomide | Neuroradiology | Experimental autoimmune encephalomyelitis | Teriflunomide | Multiple sclerosis | Dihydroorotate dehydrogenase | RHEUMATOID-ARTHRITIS | IMMUNOSUPPRESSIVE AGENT LEFLUNOMIDE | DE-NOVO SYNTHESIS | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | SPINAL-CORD | OVERGROUND LOCOMOTION | CLINICAL NEUROLOGY | MULTIPLE-SCLEROSIS | DEMYELINATED AXONS | CENTRAL-NERVOUS-SYSTEM | Evoked Potentials, Somatosensory - drug effects | Crotonates - pharmacology | Electrophysiology | Male | Toluidines - pharmacology | Toluidines - administration & dosage | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Behavior, Animal | Dose-Response Relationship, Drug | Crotonates - administration & dosage | Demyelinating Diseases - etiology | Demyelinating Diseases - drug therapy | Disease Models, Animal | Encephalomyelitis, Autoimmune, Experimental - physiopathology | Encephalomyelitis, Autoimmune, Experimental - pathology | Encephalomyelitis, Autoimmune, Experimental - drug therapy | Axons - drug effects | Rats | Treatment Outcome | Encephalomyelitis, Autoimmune, Experimental - psychology | Animals | Axons - pathology | Central Nervous System Agents - administration & dosage | Anti-Inflammatory Agents, Non-Steroidal - administration & dosage | Central Nervous System Agents - pharmacology | Dosage and administration | Research | Anti-inflammatory drugs | Encephalomyelitis | Drug therapy
Journal Article
Blood, ISSN 0006-4971, 12/2014, Volume 124, Issue 21, pp. 127 - 127
Abstract Pathologic germ line mutations that predispose patients to cancer are estimated to occur in 4-30% of all pediatric oncology cases. In addition to... 
Journal Article
Cancer Cell, ISSN 1535-6108, 11/2012, Volume 22, Issue 5, pp. 683 - 697
To define the mutation spectrum in non-Down syndrome acute megakaryoblastic leukemia (non-DS-AMKL), we performed transcriptome sequencing on diagnostic blasts... 
ONCOLOGY-GROUP | ONCOLOGY | ACUTE LYMPHOBLASTIC-LEUKEMIA | ZINC-FINGER PROTEIN | GLI PROTEINS | FUNCTIONAL-CHARACTERIZATION | ACUTE MYELOID-LEUKEMIA | DIFFERENTIATION | ACUTE MEGAKARYOCYTIC LEUKEMIA | CELL-LINE | GENETIC ALTERATIONS | CELL BIOLOGY | Oncogene Proteins, Fusion - metabolism | Prognosis | Humans | Leukemia, Megakaryoblastic, Acute - classification | Recombinant Fusion Proteins - physiology | Gene Expression Profiling | Chromosome Inversion | Recombinant Fusion Proteins - metabolism | Oncogene Proteins, Fusion - physiology | Bone Morphogenetic Proteins - metabolism | Tumor Suppressor Proteins - genetics | Leukemia, Megakaryoblastic, Acute - genetics | Child | Drosophila - genetics | Leukemia, Megakaryoblastic, Acute - diagnosis | Signal Transduction | Repressor Proteins - genetics | Animals | Sequence Analysis, RNA | Oncogene Proteins, Fusion - genetics | Chromosomes, Human, Pair 16 | Recombinant Fusion Proteins - genetics | Drosophila - growth & development | Mice | Kruppel-Like Transcription Factors - genetics | Pediatrics | Medical colleges | Oncology, Experimental | Analysis | Bone marrow | Transplantation | Research | Children | Health aspects | Biometry | Cancer | Leukemia, Megakaryoblastic, Acute/classification | Medical and Health Sciences | Medicin och hälsovetenskap | Klinisk medicin | Drosophila/genetics | Bone Morphogenetic Proteins/metabolism | Kruppel-Like Transcription Factors/genetics | Oncogene Proteins, Fusion/genetics | Recombinant Fusion Proteins/genetics | Basic Medicine | Repressor Proteins/genetics | Tumor Suppressor Proteins/genetics | Clinical Medicine | Medicinska och farmaceutiska grundvetenskaper | Cancer and Oncology | Cancer och onkologi
Journal Article