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Journal of Clinical Oncology, ISSN 0732-183X, 06/2009, Volume 27, Issue 16, pp. 2622 - 2629
Purpose PTEN, AKT, and KRAS are epidermal growth factor receptor (EGFR) downstream regulators. KRAS mutations confer resistance to cetuximab. This... 
CELL LUNG-CANCER | PANITUMUMAB | K-RAS MUTATIONS | THERAPY | GROWTH-FACTOR-RECEPTOR | EFFICACY | ONCOLOGY | SIGNALING PATHWAY | RESISTANCE | EGFR | CHEMOTHERAPY | Immunohistochemistry | Predictive Value of Tests | ras Proteins - genetics | Phosphorylation | Proto-Oncogene Proteins p21(ras) | Colorectal Neoplasms - genetics | Humans | Middle Aged | PTEN Phosphohydrolase - analysis | Drug Resistance, Neoplasm | Male | Patient Selection | Antibodies, Monoclonal, Humanized | Antineoplastic Agents, Phytogenic - administration & dosage | Time Factors | DNA Mutational Analysis | Colorectal Neoplasms - drug therapy | Adult | Camptothecin - administration & dosage | Female | Retrospective Studies | Cetuximab | Camptothecin - analogs & derivatives | Colorectal Neoplasms - mortality | Colorectal Neoplasms - enzymology | Risk Assessment | Kaplan-Meier Estimate | Proportional Hazards Models | Proto-Oncogene Proteins - genetics | Treatment Outcome | Receptor, Epidermal Growth Factor - analysis | Protein Kinase Inhibitors - administration & dosage | Antibodies, Monoclonal - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Proto-Oncogene Proteins c-akt - analysis | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Colorectal Neoplasms - secondary | Italy - epidemiology | Aged | Mutation | Longitudinal Studies | Index Medicus
Journal Article
Cancer Research, ISSN 0008-5472, 2010, Volume 70, Issue 11, pp. 4528 - 4538
MicroRNA-21 (miR-21) was reported to be overexpressed and contributes to invasion and gemcitabine resistance in pancreatic ductal adenocarcinoma (PDAC). The... 
SURVIVAL | CELLS | INVASION | METASTASIS | INHIBITION | ONCOLOGY | PHOSPHORYLATION | POOR-PROGNOSIS | ADENOCARCINOMA | EXPRESSION PROFILES | CHEMOTHERAPY | Vascular Endothelial Growth Factor A - biosynthesis | Pancreatic Neoplasms - metabolism | Apoptosis - drug effects | Humans | Middle Aged | Carcinoma, Pancreatic Ductal - metabolism | Apoptosis - genetics | Carcinoma, Pancreatic Ductal - genetics | Dose-Response Relationship, Drug | Pancreatic Neoplasms - drug therapy | RNA, Messenger - biosynthesis | Deoxycytidine - therapeutic use | Matrix Metalloproteinase 9 - genetics | Aged, 80 and over | Adult | Retrospective Studies | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Matrix Metalloproteinase 2 - biosynthesis | Matrix Metalloproteinase 9 - biosynthesis | Cell Growth Processes - drug effects | Pancreatic Neoplasms - pathology | RNA, Messenger - genetics | PTEN Phosphohydrolase - biosynthesis | MicroRNAs - biosynthesis | Pancreatic Neoplasms - genetics | Treatment Outcome | Carcinoma, Pancreatic Ductal - pathology | Proto-Oncogene Proteins c-akt - biosynthesis | Carcinoma, Pancreatic Ductal - drug therapy | Matrix Metalloproteinase 2 - genetics | Antimetabolites, Antineoplastic - therapeutic use | Cell Line, Tumor | Aged | MicroRNAs - genetics | Deoxycytidine - analogs & derivatives | Index Medicus
Journal Article
PLoS One, ISSN 1932-6203, 2012, Volume 7, Issue 11, pp. e49145 - e49145
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 02/2012, Volume 16, Issue 2, pp. 318 - 327
Journal Article
Cytokine, ISSN 1043-4666, 03/2012, Volume 57, Issue 3, pp. 347 - 359
We studied the expression of granulocyte–macrophage colony-stimulating factor (GM-CSF) and its receptors (GM-CSF.R) in 20 human brain gliomas with different... 
GM-CSF receptor | Human glioma | GM-CSF | Glioblastoma multiforme | Tumor progression | HUMAN MYELOID CELLS | FACTOR-RECEPTOR | FACTOR GM-CSF | TYROSINE PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | IMMUNOLOGY | CELL BIOLOGY | MOLECULAR-CLONING | RECURRENT MALIGNANT GLIOMAS | EXPRESSION | GLIOBLASTOMA CELLS | T-CELLS | Immunohistochemistry | Apoptosis - drug effects | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Humans | Antibodies, Blocking - pharmacology | Autocrine Communication - drug effects | RNA, Messenger - metabolism | Glioma - genetics | Intercellular Signaling Peptides and Proteins - metabolism | Granulocyte-Macrophage Colony-Stimulating Factor - biosynthesis | Cell Transformation, Neoplastic - genetics | Glioma - pathology | Biological Assay | Clone Cells | Gene Expression Regulation, Neoplastic - drug effects | Cell Survival - drug effects | Enzyme-Linked Immunosorbent Assay | RNA, Messenger - genetics | Immunophenotyping | Disease Progression | Xenograft Model Antitumor Assays | Blotting, Northern | Animals | Models, Biological | Culture Media - pharmacology | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | Cell Transformation, Neoplastic - pathology | Receptors, Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Proteins | Messenger RNA | Gliomas | Brain tumors | Genes | Development and progression | Nerve growth factor | Macrophage colony stimulating factor | Gene expression | Vascular endothelial growth factor | Index Medicus
Journal Article
Cancer Research, ISSN 0008-5472, 04/2006, Volume 66, Issue 7, pp. 3928 - 3935
Journal Article