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The New England Journal of Medicine, ISSN 0028-4793, 09/2015, Volume 373, Issue 10, pp. 895 - 907
Journal Article
by Claussnitzer, Melina and Dankel, Simon N and Klocke, Bernward and Grallert, Harald and Glunk, Viktoria and Berulava, Tea and Lee, Heekyoung and Oskolkov, Nikolay and Fadista, Joao and Ehlers, Kerstin and Wahl, Simone and Hoffmann, Christoph and Qian, Kun and Rönn, Tina and Riess, Helene and Müller-Nurasyid, Martina and Bretschneider, Nancy and Schroeder, Timm and Skurk, Thomas and Horsthemke, Bernhard and Voight, Benjamin F and Scott, Laura J and Steinthorsdottir, Valgerdur and Morris, Andrew D and Morris, Andrew P and Dina, Christian and Welch, Ryan P and Zeggini, Eleftheria and Huth, Cornelia and Aulchenko, Yurii S and Thorleifsson, Gudmar and McCulloch, Laura J and Ferreira, Teresa and Amin, Najaf and Wu, Guanming and Willer, Cristen J and Raychaudhuri, Soumya and McCarroll, Steve A and Langenberg, Claudia and Hofmann, Oliver M and Dupuis, Josée and Qi, Lu and Segrè, Ayellet V and van Hoek, Mandy and Navarro, Pau and Ardlie, Kristin and Balkau, Beverley and Benediktsson, Rafn and Bennett, Amanda J and Blagieva, Roza and Boerwinkle, Eric and Bonnycastle, Lori L and Boström, Kristina Bengtsson and Bravenboer, Bert and Bumpstead, Suzannah and Burtt, Noël P and Charpentier, Guillaume and Chines, Peter S and Cornelis, Marilyn and Couper, David J and Crawford, Gabe and Doney, Alex S.F and Elliott, Amanda L and Elliott, Katherine S and Erdos, Michael R and Fox, Caroline S and Franklin, Christopher S and Ganser, Martha and Gieger, Christian and Grarup, Niels and Green, Todd and Griffin, Simon and Groves, Christopher J and Guiducci, Candace and Hadjadj, Samy and Hassanali, Neelam and Herder, Christian and Isomaa, Bo and Jackson, Anne U and Johnson, Paul R.V and Jørgensen, Torben and Kao, Wen H.L and Klopp, Norman and Kong, Augustine and Kraft, Peter and Kuusisto, Johanna and Lauritzen, Torsten and Li, Man and Lieverse, Aloysius and Lindgren, Cecilia M and Lyssenko, Valeriya and Marre, Michel and Meitinger, Thomas and Midthjell, Kristian and Morken, Mario A and Narisu, Narisu and Nilsson, Peter and Owen, Katharine R and Payne, Felicity and Perry, John R.B and ... and DIAGRAM Consortium and DIAGRAM+Consortium and Diabetes - Epigenetics and Lund University and Genomics, Diabetes and Endocrinology and Genomik, diabetes och endokrinologi and Diabetes - epigenetik and Lunds universitet and EXODIAB: Excellence in Diabetes Research in Sweden
Cell, ISSN 0092-8674, 01/2014, Volume 156, Issue 1-2, pp. 343 - 358
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2019, Volume 9, Issue 1, pp. 4614 - 12
Studies have implicated the extracellular matrix (ECM) of adipose tissue in insulin resistance. The proteoglycan decorin, a component of ECM, has been... 
INSULIN-RESISTANCE | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | BIGLYCAN | COLLAGEN-VI | PPAR-GAMMA | ADULT-RATS | EXTRACELLULAR-MATRIX | BINDING | ADIPOSE-TISSUE | OBESE WOMEN | Decorin | Adipose tissue | Liver | Body weight | Glucose | Gene expression | Metabolism | Insulin | High fat diet | Glucose tolerance | Food intake | Leptin | Low fat diet | Chromium | Extracellular matrix | Feed efficiency
Journal Article
Science Translational Medicine, ISSN 1946-6234, 07/2014, Volume 6, Issue 247, pp. 247ra103 - 247ra103
Journal Article
PLoS ONE, ISSN 1932-6203, 2010, Volume 5, Issue 6, p. e11033
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2013, Volume 8, Issue 7, p. e70096
The p160/Steroid Receptor Coactivators SRC-1, SRC-2/GRIP1, and SRC-3/AIB1 are important regulators of Estrogen Receptor alpha (ER alpha) activity. However,... 
GLUCOCORTICOID-RECEPTOR | PROTEIN-KINASE | TIF2 | MULTIDISCIPLINARY SCIENCES | INTERMEDIARY FACTOR-2 | TUMOR-SUPPRESSOR | AIB1 | TRANSCRIPTIONAL COACTIVATOR | SRC FAMILY | BINDING PROTEIN | EXPRESSION | Cyclic AMP-Dependent Protein Kinases - metabolism | Oncogenes - genetics | RNA, Small Interfering - genetics | Cell Proliferation | Humans | Gene Silencing | Gene Expression Profiling | Nuclear Receptor Coactivator 2 - metabolism | Breast Neoplasms - metabolism | Gene Knockdown Techniques | Breast Neoplasms - genetics | MCF-7 Cells | RNA Interference | Proteolysis | Tumor Suppressor Proteins - genetics | Estrogen Receptor alpha - metabolism | Female | Gene Expression Regulation, Neoplastic - drug effects | Cyclic AMP - metabolism | Nuclear Receptor Coactivator 2 - genetics | RNA, Small Interfering - metabolism | Ubiquitin | RNA | Genes | Estrogen | Cancer cells | Phenols | Development and progression | Breast cancer | Genetic aspects | Cyclic adenylic acid | Genetic transcription | Cancer | Cell proliferation | Protein kinase A | Regulators | Transcription | Laboratories | Brain cancer | Genomics | Estrogens | Science | Activation | Kinases | Proteins | Ubiquitination | Cell cycle | Tumorigenesis | EGR-1 protein | Calcium channels (voltage-gated) | Src protein | Gene expression | Ribonucleic acid--RNA | CXCR4 protein | Studies | Androgens | Presenilin 2 | Plasmids | Proteasomes | Breast | Steroid receptor coactivator 1 | Tumor suppressor genes | GRIP1 protein | Prostate cancer | Tumors | Ribonucleic acid
Journal Article
International Journal of Obesity, ISSN 0307-0565, 2018
A causal obesity risk variant in the FTO locus was recently shown to inhibit adipocyte thermogenesis via increased adipose expression of the homeobox... 
Journal Article
American Journal of Clinical Nutrition, ISSN 0002-9165, 01/2017, Volume 105, Issue 1, pp. 85 - 99
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 6, p. e20481
Background: Primary hyperparathyroidism (PHPT) is characterised by increased production of parathyroid hormone (PTH) resulting in elevated serum calcium... 
DENSITY | MATRIX METALLOPROTEINASES | OBESITY | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | CARDIOVASCULAR-DISEASE | ATHEROSCLEROSIS | MATRIX-METALLOPROTEINASE-9 | PARATHYROID-HORMONE | PROTEINS | MILD PRIMARY HYPERPARATHYROIDISM | Oligonucleotide Array Sequence Analysis | Humans | Gene Expression Regulation | Male | Hyperparathyroidism, Primary - genetics | DNA Primers | Case-Control Studies | Adipose Tissue - metabolism | Base Sequence | Inflammation Mediators - metabolism | Polymerase Chain Reaction | Female | Energy Metabolism - genetics | Adipose tissues | Hypertension | Medical research | RNA | Genes | Thyroid diseases | Inflammation | Genetic transcription | Folic acid | Parathyroid hormone | Analysis | Medicine, Experimental | Hyperparathyroidism | Protein binding | Adipose tissue | Calcium | Disease | Laboratories | Gene regulation | Risk | Adipocytes | Family medical history | CD14 antigen | Immunity | Bone resorption | Calcium (blood) | Calcium-binding protein | Surgery | Atherosclerosis | Metalloproteinase | Neck | Heart diseases | Parathyroid | Thyroid | Obesity | Health risks | Homogenization | Bone turnover | Metabolism | Gene expression | Ribonucleic acid--RNA | Insulin | Patients | Low density lipoprotein | Cholesterol | Diseases | Medicine | Hospitals | Insulin resistance | Bone | Cardiovascular diseases | Binding sites | Ribonucleic acid
Journal Article
The American Journal of Clinical Nutrition, ISSN 0002-9165, 01/2017, Volume 105, Issue 1, p. 85
  Background: Different aspects of dietary pattern, including macronutrient and food profiles, may affect visceral fat mass and metabolic syndrome. Objective:... 
Diet | Energy | Clinical trials | Oils & fats | Metabolic syndrome
Journal Article