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Cancer Research, ISSN 0008-5472, 04/2013, Volume 73, Issue 8 Supplement, pp. 2931 - 2931
Journal Article
Nature Cell Biology, ISSN 1465-7392, 08/2009, Volume 11, Issue 8, pp. 973 - 979
Cellular senescence suppresses cancer by stably arresting the proliferation of damaged cells. Paradoxically, senescent cells also secrete factors that alter... 
TUMOR SUPPRESSION | HUMAN-CELLS | HUMAN FIBROBLASTS | GROWTH-FACTOR | ONCOGENE-INDUCED SENESCENCE | CELLULAR SENESCENCE | CANCER | TUMORIGENESIS | P53 | TELOMERES | CELL BIOLOGY | RNA, Small Interfering - genetics | Humans | Male | Green Fluorescent Proteins - genetics | Tumor Suppressor Protein p53 - genetics | DNA-Binding Proteins - metabolism | Telomerase - genetics | Transfection | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Telomerase - metabolism | Nuclear Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Fibroblasts - metabolism | Telomere - genetics | Cell Line | Green Fluorescent Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Enzyme-Linked Immunosorbent Assay | Cell Cycle Proteins - metabolism | Cells, Cultured | Cytokines - secretion | Protein-Serine-Threonine Kinases - genetics | Tumor Suppressor Protein p53 - metabolism | Cellular Senescence - physiology | Cyclin-Dependent Kinase Inhibitor p16 - genetics | Nuclear Proteins - metabolism | Ataxia Telangiectasia Mutated Proteins | DNA-Binding Proteins - genetics | Interleukin-6 - secretion | Blotting, Western | Fibroblasts - radiation effects | Checkpoint Kinase 2 | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Signal Transduction - physiology | Fibroblasts - cytology | DNA Damage | Microscopy, Fluorescence | Physiological aspects | Cellular signal transduction | Genetic aspects | Research | Cytokines | DNA damage | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 08/2015, Volume 17, Issue 8, pp. 1049 - 1061
The TOR (target of rapamycin) kinase limits longevity by poorly understood mechanisms. Rapamycin suppresses the mammalian TORC1 complex, which regulates... 
EPITHELIAL-CELL LINE | GROWTH ARREST | DNA-DAMAGE | IN-VIVO | INFLAMMATORY CYTOKINE SECRETION | HUMAN FIBROBLASTS | TUMOR-CELLS | CANCER-THERAPY | REPLICATIVE SENESCENCE | P53 | CELL BIOLOGY | Up-Regulation | Fibroblasts - enzymology | Cell Proliferation | TOR Serine-Threonine Kinases - metabolism | Humans | Gene Expression Regulation, Neoplastic | Interleukin-1alpha - metabolism | Male | NF-kappa B - metabolism | RNA, Messenger - metabolism | Prostatic Neoplasms - secretion | Prostatic Neoplasms - immunology | Dose-Response Relationship, Drug | TOR Serine-Threonine Kinases - antagonists & inhibitors | Prostatic Neoplasms - genetics | TOR Serine-Threonine Kinases - genetics | Transfection | RNA Interference | Time Factors | Cellular Senescence | Inflammation Mediators - metabolism | Transcription, Genetic | Antineoplastic Agents - pharmacology | Interleukin-1alpha - genetics | Interleukin-6 - metabolism | Prostatic Neoplasms - drug therapy | Prostatic Neoplasms - pathology | Anti-Inflammatory Agents - pharmacology | Interleukin-1alpha - secretion | Sirolimus - metabolism | Tumor Burden | Mice, SCID | Sirolimus - pharmacology | Xenograft Model Antitumor Assays | Phenotype | Animals | Fibroblasts - drug effects | Cell Line, Tumor | Prostatic Neoplasms - enzymology | Mitoxantrone - pharmacology | Interleukins | Aging | Genetic aspects | Properties | Phosphotransferases | Carcinogenesis | Health aspects | Cells | Index Medicus
Journal Article
Methods in molecular biology (Clifton, N.J.), ISSN 1064-3745, 2019, Volume 1896, pp. 71 - 81
Senescent cells secrete diverse array of proteins. One group of proteins, damage-associated molecular pattern (DAMP) proteins exhibit relocalization from... 
Immunohistochemistry | Western blot | Damage-associated molecular pattern (DAMP) | Protein precipitation | High Mobility Group Box 1 protein (HMGB1) | Alarmin | Immunofluorescence
Journal Article
Journal of Cell Biology, ISSN 0021-9525, 05/2013, Volume 201, Issue 4, pp. 613 - 629
Journal Article
Cancer Research, ISSN 0008-5472, 04/2012, Volume 72, Issue 8 Supplement, pp. LB-483 - LB-483
Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 2410 - 9
Processes that have been linked to aging and cancer include an inflammatory milieu driven by senescent cells. Senescent cells lose the ability to divide,... 
CELLS | ACTIVATION | NUTLIN-3A | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | IN-VIVO | GENE-EXPRESSION | P53 PATHWAY | CELLULAR SENESCENCE | CANCER-THERAPY | FIBROBLASTS | Gamma Rays | Proto-Oncogene Proteins c-mdm2 - genetics | Epithelial Cells - metabolism | Interleukin-6 - antagonists & inhibitors | Epithelial Cells - drug effects | Humans | Cellular Senescence - drug effects | Interleukin-1alpha - metabolism | Male | Lung - cytology | Tumor Suppressor Protein p53 - genetics | Tumor Suppressor Protein p53 - agonists | Cellular Senescence - radiation effects | Cell Cycle Checkpoints - genetics | Indoles - pharmacology | Interleukin-1alpha - genetics | Epithelial Cells - cytology | Interleukin-6 - metabolism | Proto-Oncogene Proteins c-mdm2 - antagonists & inhibitors | Proto-Oncogene Proteins c-mdm2 - metabolism | Fibroblasts - metabolism | Cell Line | Cellular Senescence - genetics | Epithelial Cells - radiation effects | Interleukin-6 - genetics | Enzyme Inhibitors - pharmacology | Tumor Suppressor Protein p53 - metabolism | Interleukin-1alpha - antagonists & inhibitors | Imidazoles - pharmacology | Piperazines - pharmacology | Fibroblasts - radiation effects | Foreskin - cytology | Cell Cycle Checkpoints - drug effects | Fibroblasts - drug effects | Fibroblasts - cytology | Spiro Compounds - pharmacology | Biodegradation | MDM2 protein | Phenotypes | Senescence | p53 Protein | Genotoxicity | Antagonists | Breast cancer | Inflammation | Interleukin 6 | Fibroblasts | Aging | Tumor suppressor genes | Growth factors | Cancer | Index Medicus
Journal Article
Cancer Research, ISSN 0008-5472, 07/2018, Volume 78, Issue 13 Supplement, pp. 4864 - 4864
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 9/2003, Volume 162, Issue 7, pp. 1197 - 1209
Bloom syndrome (BS) is a hereditary disorder characterized by pre- and postnatal growth retardation, genomic instability, and cancer. BLM, the gene defective... 
T lymphocytes | Bloom syndrome | Cell growth | Interphase | DNA damage | DNA | Cell cycle | Cultured cells | Cells | Apoptosis | DNA replication | S phase checkpoint | DNA repair | γH2AX | SYNDROME PROTEIN | HISTONE H2AX | WERNERS-SYNDROME GENES | SACCHAROMYCES-CEREVISIAE | DNA-REPLICATION | CELL BIOLOGY | GENOMIC INSTABILITY | gamma H2AX | S-PHASE | HUMAN FIBROBLASTS | HOMOLOGOUS RECOMBINATION | MAMMARY EPITHELIAL-CELLS | Nucleic Acid Synthesis Inhibitors - pharmacology | Fibroblasts - physiology | Humans | DNA Repair - physiology | Bloom Syndrome - physiopathology | Tumor Suppressor Protein p53 - physiology | Telomerase - genetics | RecQ Helicases | DNA-Binding Proteins | Bloom Syndrome - pathology | Cells, Cultured | BRCA1 Protein - physiology | Etoposide - pharmacology | G1 Phase - physiology | Cell Division - physiology | Phenotype | DNA Replication - physiology | S Phase - physiology | Fibroblasts - cytology | Nuclear Proteins - physiology | Apoptosis - physiology | Adenosine Triphosphatases - physiology | Cell Cycle Proteins - physiology | Cell Line, Transformed | DNA Helicases - physiology | Genetic disorders | Cytology | Research | Analysis | Fibroblasts | Physiological aspects | Causes of | Genetic aspects | Catalysis | Proteins | Cellular biology | Index Medicus | APOPTOSIS | LAWRENCE BERKELEY LABORATORY | REPAIR | BASIC BIOLOGICAL SCIENCES | 60 APPLIED LIFE SCIENCES | DNA damage; DNA repair; DNA replication; γH2AX; S phase checkpoint
Journal Article
Cancer Research, ISSN 0008-5472, 08/2015, Volume 75, Issue 15 Supplement, pp. 1271 - 1271
Journal Article
Molecular Cell, ISSN 1097-2765, 02/2016, Volume 61, Issue 4, pp. 535 - 546
Journal Article