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Journal Article
Journal Article
Current Opinion in Cell Biology, ISSN 0955-0674, 2004, Volume 16, Issue 6, pp. 647 - 652
Mitochondria play an important role in the integration and transmission of cell death signals, activating caspases and other cell death execution events by... 
Proto-Oncogene Proteins c-bcl-2 - metabolism | Animals | Caspases - metabolism | Membrane Potentials | Humans | Mitochondria - metabolism | Enzyme Activation | Mitochondria - pathology | Intracellular Membranes - metabolism | Apoptosis | Cytochrome c | Proteins | Mitochondrial DNA | Permeability | Cell death
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 08/2016, Volume 113, Issue 31, pp. E4558 - E4566
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 09/2018, Volume 22, Issue 9, pp. 4522 - 4533
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2016, Volume 113, Issue 31, pp. E4558 - E4566
Bromodomain and extraterminal domain protein inhibitors (BETi) hold great promise as a novel class of cancer therapeutics. Because acquired resistance... 
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 08/2016, Volume 113, Issue 31, p. E4558
  Bromodomain and extraterminal domain protein inhibitors (BETi) hold great promise as a novel class of cancer therapeutics. Because acquired resistance... 
Signal transduction | RNA-protein interactions | Colorectal cancer | Protein expression | Oncology | Cancer therapies | Growth factors
Journal Article
Proceedings of the National Academy of Sciences, USA, ISSN 0027-8424, 08/2016, Volume 113, Issue 31, pp. E4558 - E4558
Bromodomain and extraterminal domain protein inhibitors (BETi) hold great promise as a novel class of cancer therapeutics. Because acquired resistance... 
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 07/2015, Volume 286, Issue 1, pp. 1 - 9
Metabolic activation and oxidant stress are key events in the pathophysiology of acetaminophen (APAP) hepatotoxicity. The initial mitochondrial oxidative... 
c-Jun N-terminal kinase | Acetaminophen | Hepatotoxicity | ASK1 | Oxidant stress | C-Jun N-terminal kinase | ACTIVATION | N-TERMINAL KINASE | MOUSE HEPATOCYTES | CELL-DEATH | MITOCHONDRIAL PERMEABILITY TRANSITION | DNA FRAGMENTATION | IN-VIVO | PHARMACOLOGY & PHARMACY | MICE | TOXICOLOGY | INDUCED HEPATOTOXICITY | Liver - pathology | Acetaminophen - toxicity | Glutathione - metabolism | Liver - metabolism | Mice, Inbred C57BL | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Glutathione Disulfide - metabolism | Protective Agents - therapeutic use | Animals | Liver - drug effects | Chemical and Drug Induced Liver Injury - metabolism | Protective Agents - pharmacology | Protein Kinase Inhibitors - therapeutic use | Chemical and Drug Induced Liver Injury - drug therapy | Protein Kinase Inhibitors - pharmacology | Chemical and Drug Induced Liver Injury - pathology | MAP Kinase Kinase Kinase 5 - antagonists & inhibitors | Chemical and Drug Induced Liver Injury - etiology | Acetylcysteine | Mitochondrial DNA | Analysis | Liver | Apoptosis | Index Medicus | OXIDATION | APOPTOSIS | PATIENTS | INJURIES | SAFETY | MITOCHONDRIA | SIGNALS | METABOLIC ACTIVATION | TOXICITY | 60 APPLIED LIFE SCIENCES | MOLECULES | STRESSES | INHIBITION | NECROSIS | GLUTATHIONE | OXIDIZERS | LIVER | oxidant stress | hepatotoxicity | c-jun N-terminal kinase
Journal Article
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA, ISSN 0027-8424, 08/2016, Volume 113, Issue 31, pp. E4558 - E4566
Journal Article