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Frontiers in immunology, ISSN 1664-3224, 2018, Volume 9, p. 1946
Journal Article
Journal Article
Journal of interferon & cytokine research, ISSN 1557-7465, 2015, Volume 35, Issue 4, pp. 252 - 264
Journal Article
PLoS pathogens, ISSN 1553-7374, 2013, Volume 9, Issue 11, p. e1003773
Interferons (IFNs) are a group of cytokines with a well-established antiviral function. They can be induced by viral infection, are secreted and bind to... 
CELLS | DEFENSE | VIRUS-INFECTION | DISTINCT | RIG-I | MICROBIOLOGY | IRF-7 | ANTIVIRAL RESPONSE | VIROLOGY | GENE | SIGNALING PATHWAY | ADAPTER PROTEIN | PARASITOLOGY | Epithelial Cells - metabolism | Influenza A virus - genetics | Respiratory Mucosa - virology | Interferon Regulatory Factor-7 - genetics | Interferon Type I - immunology | Interferon Regulatory Factor-3 - genetics | Interleukins - metabolism | Orthomyxoviridae Infections - genetics | Respiratory Mucosa - pathology | Interleukins - genetics | Interleukins - immunology | Respiratory Mucosa - immunology | Adaptor Proteins, Signal Transducing - immunology | Interferon Type I - metabolism | Influenza A virus - immunology | Membrane Proteins - metabolism | Interferon Regulatory Factor-3 - immunology | Nerve Tissue Proteins - immunology | Membrane Proteins - genetics | Orthomyxoviridae Infections - metabolism | Epithelial Cells - pathology | Membrane Proteins - immunology | Interferon Regulatory Factor-7 - immunology | Interferon Regulatory Factor-7 - metabolism | Nerve Tissue Proteins - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Influenza A virus - metabolism | Epithelial Cells - immunology | Epithelial Cells - virology | Adaptor Proteins, Signal Transducing - genetics | Interferon Regulatory Factor-3 - metabolism | Interferon Type I - genetics | Mice | Respiratory Mucosa - metabolism | Adaptor Proteins, Signal Transducing - metabolism | Orthomyxoviridae Infections - immunology | Epithelial cells | Influenza | Physiological aspects | Host-parasite relationships | Interferon | Genetic aspects | Genetic transcription | Research | Health aspects | Airway (Medicine) | Interleukins/metabolism | Nerve Tissue Proteins/immunology | Orthomyxoviridae Infections/genetics | Orthomyxoviridae Infections/metabolism | Membrane Proteins/genetics | Adaptor Proteins, Signal Transducing/genetics | Interferon Regulatory Factor-7/genetics | Interleukins/immunology | Membrane Proteins/immunology | Life Sciences | Orthomyxoviridae Infections/immunology | Influenza A virus/genetics | Nerve Tissue Proteins/metabolism | Respiratory Mucosa/immunology | Immunology | Interferon Type I/immunology | Epithelial Cells/immunology | Epithelial Cells/virology | Interferon Regulatory Factor-7/immunology | Epithelial Cells/metabolism | Interferon Regulatory Factor-3/immunology | Respiratory Mucosa/pathology | Influenza A virus/immunology | Interleukins/genetics | Interferon Regulatory Factor-3/genetics | Influenza A virus/metabolism | Adaptor Proteins, Signal Transducing/metabolism | Epithelial Cells/pathology | Adaptor Proteins, Signal Transducing/immunology | Interferon Type I/genetics | Interferon Regulatory Factor-3/metabolism | Membrane Proteins/metabolism | Nerve Tissue Proteins/genetics | Interferon Regulatory Factor-7/metabolism | Interferon Type I/metabolism | Respiratory Mucosa/metabolism | Respiratory Mucosa/virology | Cytokines | Genes | Rodents | Genomics | Genomes | Kinases | Experiments | Viral infections
Journal Article
Journal Article
EMBO reports, ISSN 1469-221X, 09/2015, Volume 16, Issue 9, pp. 1203 - 1218
Streptococcus pneumoniae coinfection is a major cause of influenza‐associated mortality; however, the mechanisms underlying pathogenesis or protection remain... 
influenza | C‐C‐chemokine receptor type (CCR) 2 | TNF‐related apoptosis‐inducing ligand (TRAIL) | neutrophil | Streptococcus pneumoniae | TNF-related apoptosis-inducing ligand (TRAIL) | C-C-chemokine receptor type (CCR) 2 | VIRUS-INFECTION | DENDRITIC CELLS | PANDEMIC INFLUENZA | BIOCHEMISTRY & MOLECULAR BIOLOGY | LETHAL SYNERGISM | NECROSIS-FACTOR-ALPHA | CELL BIOLOGY | BACTERIAL PNEUMONIA | NEGATIVE REGULATION | MOUSE MODEL | PNEUMOCOCCAL PNEUMONIA | TNF-ALPHA | Receptors, CCR2 - genetics | Lung - microbiology | Streptococcus pneumoniae - growth & development | Influenza A Virus, H3N2 Subtype | Humans | Orthomyxoviridae Infections - complications | Monocytes - immunology | Coinfection - physiopathology | Streptococcus pneumoniae - immunology | Lung - virology | Pneumococcal Infections - immunology | Streptococcus pneumoniae - pathogenicity | Tumor Necrosis Factor-alpha - immunology | Cytokines - immunology | Disease Models, Animal | Pneumococcal Infections - complications | Disease Susceptibility | Coinfection - immunology | Mice, Inbred C57BL | Neutrophils - immunology | Lung - physiopathology | TNF-Related Apoptosis-Inducing Ligand - metabolism | Animals | Mice | Receptors, CCR2 - deficiency | Lung - immunology | Orthomyxoviridae Infections - immunology | Ligands | Lungs | Influenza | Streptococcus infections | Apoptosis
Journal Article
Nature chemical biology, ISSN 1552-4469, 2012, Volume 8, Issue 10, pp. 839 - 847
Journal Article
American journal of respiratory and critical care medicine, ISSN 1535-4970, 2009, Volume 179, Issue 10, pp. 883 - 893
Rationale: One of the immunopathological features of allergic inflammation is the infiltrationof helper T type 2 (Th2) cells to the site of disease. Activation... 
Innate immunity | Neutrophil | Eosinophil | Asthma | ACTIVATED PROTEIN-KINASE | DENDRITIC CELLS | OX40 LIGAND | asthma | ALLERGIC AIRWAY INFLAMMATION | neutrophil | CUTTING EDGE | innate immunity | INHALED ANTIGEN | RESPIRATORY SYSTEM | IMMUNE-RESPONSES | TOLL-LIKE RECEPTORS | ANTIGEN-PRESENTING CELLS | eosinophil | ASTHMATIC-PATIENTS | CRITICAL CARE MEDICINE
Journal Article
The Journal of clinical investigation, ISSN 1558-8238, 2019, Volume 129, Issue 10, pp. 4239 - 4244
Macrophage activation in response to LPS is coupled to profound metabolic changes, typified by accumulation of the TCA cycle intermediates citrate, itaconate,... 
MEDICINE, RESEARCH & EXPERIMENTAL | SUCCINATE-DEHYDROGENASE | INTERFERON | METABOLITE | ITACONIC ACID | Macrophages | Tricarboxylic acid cycle | Enzymes | Dehydrogenases | Gene expression | Metabolism | Carbon | Citric acid | Intermediates | Lipopolysaccharides | Proteins | Cell activation | Metabolites | Interleukin 10 | Ketoglutaric acid | Interferon | Autocrine signalling | Isocitrate dehydrogenase
Journal Article
Star (Whitehorse), 01/2018
...For the next two mont