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The New England Journal of Medicine, ISSN 0028-4793, 02/2005, Volume 352, Issue 8, pp. 786 - 792
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2015, Volume 10, Issue 2, p. e0115635
The wild-type p53-induced phosphatase 1 (WIP1) is a serine/threonine phosphatase that negatively regulates multiple proteins involved in DNA damage response... 
PROTEIN PHOSPHATASE | STEM-CELLS | PPM1D MUTATIONS | DNA-DAMAGE RESPONSE | MULTIDISCIPLINARY SCIENCES | DOWN-REGULATION | TUMOR-SUPPRESSOR | CELL-CYCLE CONTROL | TUMORIGENESIS | MDM2 ANTAGONIST NUTLIN-3 | P53 | Dipeptides - pharmacology | Humans | Neuroblastoma - genetics | Phosphoprotein Phosphatases - metabolism | Protein Phosphatase 2C | Tumor Suppressor Protein p53 - metabolism | Male | Phosphoprotein Phosphatases - antagonists & inhibitors | Genetic Loci | Tumor Suppressor Protein p53 - genetics | Phosphoprotein Phosphatases - genetics | Aminopyridines - pharmacology | Neuroblastoma - drug therapy | Alleles | Cell Line, Tumor | Female | Neuroblastoma - metabolism | Mutation | Neuroblastoma - pathology | Ionizing radiation | Chemotherapy | Phosphatases | RNA | Gliomas | Neuroblastoma | Tumor proteins | Health aspects | Cancer | Pediatrics | Biotechnology | Histone H2A | Copy number | Leukemia | Serine | p53 Protein | DNA damage | Oncology | Genomes | mRNA | Activation | Drug development | Phosphatase | Neurogenesis | Caspase-3 | Doxorubicin | Ovarian cancer | Proteins | Cell cycle | Tumorigenesis | Inhibition | Damage | Deoxyribonucleic acid--DNA | Threonine | Protein-serine/threonine phosphatase | Caspase | Breast cancer | Tumor cell lines | Gene expression | Virology | Amplification | Cell death | Medulloblastoma | Cell lines | Carboplatin | Apoptosis | Tumors | Threonine phosphatase | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
Nature Immunology, ISSN 1529-2908, 07/2006, Volume 7, Issue 7, pp. 732 - 739
During 'emergency' situations such as infections, host defense requires rapid mobilization of bone marrow granulocyte progenitors. 'Steady-state'... 
BINDING PROTEIN-ALPHA | RECEPTOR-DEFICIENT MICE | COLONY-STIMULATING FACTOR | HEPATOCYTE PROLIFERATION | MULTI-CSF | PLASMID DNA | GRANULOCYTIC DIFFERENTIATION | MYELOID-LEUKEMIA | IMMUNOLOGY | KNOCKOUT MICE | TRANSCRIPTION FACTOR
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, p. e55989
Genomic instability stemming from dysregulation of cell cycle checkpoints and DNA damage response (DDR) is a common feature of many cancers. The cancer adult T... 
TRANSCRIPTION FACTORS | TRANSACTIVATOR TAX | HUMAN-LYMPHOCYTES | RETINOBLASTOMA PROTEIN | TYPE-1 TAX | VIRUS TYPE-I | MULTIDISCIPLINARY SCIENCES | CYCLE PROGRESSION | ONCOGENE PRODUCT TAX | NF-KAPPA-B | NUCLEOTIDE EXCISION-REPAIR | RNA, Small Interfering - genetics | Phosphorylation | Immunoprecipitation | Humans | Phosphoprotein Phosphatases - metabolism | DNA Repair - physiology | DNA Repair - radiation effects | Gene Products, tax - genetics | Embryo, Mammalian - metabolism | Flow Cytometry | Ultraviolet Rays | DNA Damage - physiology | DNA Replication - radiation effects | Adult | Human T-lymphotropic virus 1 - genetics | Real-Time Polymerase Chain Reaction | Fibroblasts - metabolism | Embryo, Mammalian - radiation effects | Peptide Fragments - metabolism | RNA, Messenger - genetics | Cells, Cultured | Protein Phosphatase 2C | Rats | Phosphoprotein Phosphatases - antagonists & inhibitors | G1 Phase - physiology | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | S Phase - radiation effects | Animals | Histones - genetics | Phosphoprotein Phosphatases - genetics | Fibroblasts - radiation effects | G1 Phase - radiation effects | Embryo, Mammalian - cytology | Fluorescent Antibody Technique | S Phase - physiology | Fibroblasts - cytology | Histones - metabolism | Pyrimidine Dimers | DNA Damage - radiation effects | DNA replication | Phosphatases | RNA | Phosphoproteins | DNA damage | Leukemia | Genomics | DNA | Genetic research | T cells | DNA repair | Flow cytometry | Adult T cell leukemia | Viruses | Lymphocytes T | Genomes | mRNA | Kinases | Phosphatase | Defects | Genomic instability | Proteins | Lymphocytes | Cell cycle | Lesions | Repair | Damage | Deoxyribonucleic acid--DNA | G1 phase | Stability | Abnormalities | siRNA | Gene expression | Virology | Medicine | DNA biosynthesis | Cytometry | Cellular biology | Taxation | Mutation | Molecular biology | Cancer | Deoxyribonucleic acid
Journal Article
Blood, ISSN 0006-4971, 11/2018, Volume 132, Issue Supplement 1, pp. 746 - 746
Abstract Clonal hematopoiesis (CH) is a condition in which individual hematopoietic stem cells (HSCs) acquire a fitness advantage and contribute... 
Journal Article
Cell Stem Cell, ISSN 1934-5909, 11/2018, Volume 23, Issue 5, pp. 700 - 713.e6
Clonal hematopoiesis (CH), in which stem cell clones dominate blood production, becomes increasingly common with age and can presage malignancy development.... 
clonal hematopoiesis | CHIP | topoisomerase inhibitors | DNA damage response | etoposide | t-MDS | doxorubicin | PPM1D | cisplatin | t-AML | EVOLUTION | PHOSPHATASE | DYNAMICS | WIP1 | LEUKEMIA | CANCER | BREAST | STEM | BLOOD | P53 | CELL & TISSUE ENGINEERING | CELL BIOLOGY
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 10/2018, Volume 115, Issue 42, p. 10666
Scientific progress depends on formulating testable hypotheses informed by the literature. In many domains, however, this model is strained because the number... 
Automated reasoning | Phosphorylation | Mitosis | p53 Protein | Cell division | Kinases | Scientific papers | Domains | Biomedical materials | Hypotheses | Computer applications | Tumor suppressor genes | Molecular biology | Tumors | Pharmaceuticals
Journal Article
Journal Article
ISSN 1934-5909, 2018
Clonal hematopoiesis (CH), in which stem cell clones dominate blood production, becomes increasingly common with age and can presage malignancy development.... 
clonal hematopoiesis | CHIP | topoisomerase inhibitors | DNA damage response | etoposide | doxorubicin | t-MDS | PPM1D | cisplatin | t-AML
Journal Article
Genes and Development, ISSN 0890-9369, 08/2008, Volume 22, Issue 15, pp. 2085 - 2092
Journal Article