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Human Mutation, ISSN 1059-7794, 05/2013, Volume 34, Issue 5, pp. 785 - 791
Pathogenic A lu element insertions are rarely reported, whereas their occurrence is expected to be much higher. Alu containing alleles are usually out‐competed... 
BRCA | mutation screen | lu insertion | NGS | BRCA1 | BRCA2 | Alu insertion | Mutation screen | BREAST-CANCER | FAMILIES | GENES | GENETICS & HEREDITY | BRCA2 REARRANGEMENT | MUTATIONS | Sequence Homology, Amino Acid | Base Sequence | Humans | Polymerase Chain Reaction | Genes, BRCA2 | Molecular Sequence Data | Software | Genes, BRCA1 | Sequence Analysis, DNA - methods | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, pp. e60317 - e60317
Aberrant expression of microRNA-146a (miR-146a) has been reported to be involved in the development and progression of various types of cancers. However, its... 
SUPPRESSOR | ONCOGENESIS | RNA | MULTIDISCIPLINARY SCIENCES | CERVICAL-CANCER | INTERFERENCE | MICRORNAS | NF-KAPPA-B | FACTOR RECEPTOR | EXPRESSION | CONTRIBUTES | Lung Neoplasms - drug therapy | Humans | Lung Neoplasms - metabolism | Middle Aged | Gene Expression Regulation, Neoplastic | Lung Neoplasms - pathology | Male | MicroRNAs - metabolism | NF-kappa B - metabolism | Receptor, Epidermal Growth Factor - metabolism | Antibodies, Monoclonal, Humanized - pharmacology | Aged, 80 and over | Adult | Female | Antineoplastic Agents - pharmacology | Lung - metabolism | Cetuximab | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - genetics | Lung - pathology | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Lung - drug effects | Cell Line, Tumor | Aged | Cell Proliferation - drug effects | MicroRNAs - genetics | Carcinoma, Non-Small-Cell Lung - drug therapy | Apoptosis | Cell Movement | MicroRNA | Growth | Monoclonal antibodies | Lung cancer, Small cell | Development and progression | Lung cancer, Non-small cell | Health aspects | Cell proliferation | Laboratories | Lung | Lung cancer | Oncology | Drug delivery | Kinases | Paraffin | Cancer therapies | Metastases | Signal transduction | Cell growth | Bioindicators | Inhibition | Gefitinib | Sensitizing | NF-κB protein | Phenotypes | Cytokines | Epidermal growth factor receptors | MiRNA | Non-small cell lung carcinoma | siRNA | Breast cancer | Gene expression | Ribonucleic acid--RNA | Patients | Pathology | Signaling | Chemotherapy | Thyroid cancer | MicroRNAs | Medical prognosis | Pancreatic cancer | Cell lines | Biomarkers | Mutation | Cell migration | Cervical cancer | Tumors | Cancer | Index Medicus | Ribonucleic acid
Journal Article
Journal Article
BMC Medicine, ISSN 1741-7015, 03/2012, Volume 10, Issue 1, pp. 28 - 28
Background: The epidermal growth factor receptor (EGFR) is a validated therapeutic target in non-small cell lung cancer (NSCLC). However, current single agent... 
RNA interference | Lung cancer | Tyrosine kinase inhibitors (TKIs) | Proliferation | EGFR | Anti-EGFR monoclonal antibodies (mabs) | Apoptosis | GEFITINIB | ADVANCED SOLID TUMORS | proliferation | apoptosis | MONOCLONAL-ANTIBODY | COMBINATION | I DOSE-ESCALATION | tyrosine kinase inhibitors (TKIs) | lung cancer | PHASE-III TRIAL | MEDICINE, GENERAL & INTERNAL | MUTATION | RESISTANCE | EGFR MUTANTS | anti-EGFR monoclonal antibodies (mAbs) | ERLOTINIB | Caspase 7 - metabolism | Erlotinib Hydrochloride | Lung Neoplasms - drug therapy | RNA Interference - drug effects | Apoptosis - drug effects | Humans | Caspase 3 - metabolism | Antibodies, Monoclonal - therapeutic use | Lung Neoplasms - pathology | Molecular Targeted Therapy | Antibodies, Monoclonal, Humanized | Receptor, Epidermal Growth Factor - metabolism | Transfection | Cetuximab | Carcinoma, Non-Small-Cell Lung - pathology | Lung Neoplasms - enzymology | Antibodies, Monoclonal - pharmacology | Down-Regulation - drug effects | Phenotype | Protein Kinase Inhibitors - therapeutic use | Quinazolines - therapeutic use | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Carcinoma, Non-Small-Cell Lung - drug therapy | Carcinoma, Non-Small-Cell Lung - enzymology | Quinazolines - pharmacology | RNA, Small Interfering - metabolism | Lung cancer, Non-small cell | Research | Kinases | Experiments | Cancer therapies | Manuscripts | Proteins | Studies | Angiogenesis | Signal transduction | Ligands | Mutation | Head & neck cancer | Tumors | Index Medicus
Journal Article
Oncotarget, ISSN 1949-2553, 2017, Volume 8, Issue 36, pp. 60094 - 60108
Approximately half of BRAF-mutated Non-small cell lung cancers (NSCLCs) harbor a non-V600 BRAF mutation, accounting for similar to 40,000 annual deaths... 
Impaired-kinase | Dabrafenib | Lung cancer | Non-V600 BRAF | Trametinib | ACTIVATION | TUMOR PROGRESSION | MECHANISM | non-V600 BRAF | ACQUIRED-RESISTANCE | CRAF | CELL BIOLOGY | lung cancer | METASTATIC MELANOMA | kinase | impaired | MEK INHIBITION | PRECLINICAL MODELS | COLORECTAL-CANCER | RAF INHIBITORS
Journal Article
Biological Procedures Online, ISSN 1480-9222, 11/2010, Volume 17, Issue 1
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, pp. e59708 - e59708
Epidermal growth factor receptor (EGFR) and c-MET receptors are expressed on many non-small cell lung cancer (NSCLC) cells. Current single agent therapeutic... 
GROWTH-FACTOR RECEPTOR | CETUXIMAB | RNA | MULTIDISCIPLINARY SCIENCES | PHASE-III | TYROSINE KINASE INHIBITOR | OPEN-LABEL | MUTATIONS | MET | HEPATOCYTE GROWTH | EGFR | Erlotinib Hydrochloride | Proto-Oncogene Proteins c-met - metabolism | RNA, Small Interfering - genetics | Apoptosis - drug effects | Proto-Oncogene Proteins c-met - antagonists & inhibitors | Humans | Drug Resistance, Neoplasm | Fluorometry | Reverse Transcriptase Polymerase Chain Reaction | Sulfonamides - pharmacology | Piperazines - pharmacology | Blotting, Western | Drug Synergism | Receptor, Epidermal Growth Factor - metabolism | Analysis of Variance | RNA Interference | Signal Transduction - drug effects | Cell Line, Tumor | Receptor, Epidermal Growth Factor - antagonists & inhibitors | Indoles - pharmacology | Carcinoma, Non-Small-Cell Lung - drug therapy | Quinazolines - pharmacology | Spectrophotometry | Microscopy, Fluorescence | Tyrosine | Epidermal growth factor | Erlotinib | Monoclonal antibodies | Fluorescence | Lung cancer, Small cell | Lung cancer, Non-small cell | Gefitinib | Health aspects | Fluorescence microscopy | Cell proliferation | Biotechnology | Laboratories | Lung cancer | Cytology | Oncology | Kinases | Cancer therapies | c-Met protein | Receptors | Biological effects | Cell growth | Rodents | Cell cycle | Protein-tyrosine kinase receptors | Inhibition | Fluorimetry | Protein-tyrosine kinase | Epidermal growth factor receptors | Therapeutic applications | RNA-mediated interference | Non-small cell lung carcinoma | Caspase | siRNA | Pharmacology | Gene expression | Ribonucleic acid--RNA | Chemical compounds | Signaling | Chemotherapy | Gene amplification | Medical prognosis | Cell lines | Ligands | Mutation | Synergistic effect | Viability | Apoptosis | Tumors | Cancer | Index Medicus | Ribonucleic acid
Journal Article