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Acta clinica Belgica, 10/2018, p. 1
To document the frequency and predictors of low-density lipoprotein cholesterol (LDL-C) target value attainment among patients with coronary heart disease... 
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2017, Volume 12, Issue 1, pp. e0170307 - e0170307
Background Cytochrome oxidase IV complex regulates energy production in mitochondria. Therefore, we determined the relation of COX genes with atherosclerosis... 
OXIDATIVE STRESS | OBESITY | METABOLISM | GENE | MULTIDISCIPLINARY SCIENCES | LOW-DENSITY-LIPOPROTEIN | C-OXIDASE | EXPRESSION | Cytochrome-c Oxidase Deficiency - physiopathology | Oxidative Stress | Receptors, Leptin - genetics | Caloric Restriction | Atherosclerosis - genetics | Diabetes Mellitus, Experimental - genetics | Peroxisome Proliferator-Activated Receptors - biosynthesis | Coronary Vessels - metabolism | Atherosclerosis - etiology | Swine, Miniature - metabolism | Atherosclerosis - enzymology | Hypercholesterolemia - pathology | Hypercholesterolemia - enzymology | Swine | Receptors, LDL - deficiency | Lipoproteins, LDL - metabolism | Diabetes Mellitus, Experimental - metabolism | Plaque, Atherosclerotic - pathology | Aorta, Thoracic - pathology | Peroxisome Proliferator-Activated Receptors - genetics | Cytochrome-c Oxidase Deficiency - pathology | Receptors, LDL - genetics | Coronary Vessels - pathology | Cytochrome-c Oxidase Deficiency - complications | Macrophages - pathology | Mice, Inbred C57BL | Insulin Resistance | Aorta, Thoracic - metabolism | Leptin - genetics | Mitochondria - metabolism | Electron Transport Complex IV - genetics | Mice, Knockout | Receptors, Leptin - deficiency | Animals | Energy Metabolism | Electron Transport Complex IV - physiology | Mice, Obese | Nuclear Receptor Coactivators - biosynthesis | Mice | Leptin - deficiency | Nuclear Receptor Coactivators - genetics | Complications and side effects | Atherosclerosis | Cytochrome oxidase | Physiological aspects | Mitochondrial diseases | Research | Risk factors | Cytochrome | Lipoproteins (low density) | Oxidative stress | Genes | Oxidase | Antibodies | Mitochondrial DNA | Macrophages | Nuclei | Complexity | Proteins | Dietary restrictions | Receptors | Mitochondria | Oxidation resistance | Coding | Rodents | Aorta | Cardiology | Deoxyribonucleic acid--DNA | Plaques | Age | Stresses | Obesity | Enzymes | Diabetes mellitus | Metabolism | Insulin | Stress | Hogs | Lipoproteins | Arteriosclerosis | Leptin | Coronary vessels | Aortic arch | Diabetes | Mutation | Laboratory animals | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
PLoS ONE, ISSN 1932-6203, 01/2012, Volume 7, Issue 1, pp. e30414 - e30414
Background: Visceral obesity is associated with the rising incidence of type 2 diabetes and metabolic syndrome. Low-grade chronic inflammation and oxidative... 
IRAK-M | OXIDATIVE STRESS | MYOCARDIAL-INFARCTION | ADIPONECTIN | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | LOW-DENSITY-LIPOPROTEIN | CARDIOVASCULAR-DISEASE | MONOCYTES | RISK | EXPRESSION | Interleukin-1 Receptor-Associated Kinases - metabolism | Superoxide Dismutase - genetics | Interleukin-1 Receptor-Associated Kinases - genetics | Reactive Oxygen Species - metabolism | Oligonucleotide Array Sequence Analysis | Oxidative Stress | Diabetes Mellitus, Type 2 - genetics | Humans | Middle Aged | Male | Metabolic Syndrome - metabolism | Monocytes - metabolism | Gene Expression Profiling | Diabetes Mellitus, Type 2 - metabolism | Obesity - genetics | Inflammation - metabolism | Adult | Female | Adipogenesis - genetics | Diabetes Mellitus, Type 2 - complications | Superoxide Dismutase - metabolism | Cell Line | Obesity - complications | Cells, Cultured | Signal Transduction - genetics | Intra-Abdominal Fat - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Metabolic Syndrome - complications | Obesity - metabolism | Metabolic Syndrome - genetics | Adipocytes - metabolism | Inflammation - genetics | Interleukin-1 Receptor-Associated Kinases - blood | Cohort Studies | Type 2 diabetes | Obesity | C-reactive protein | RNA | Interleukins | Surgery | Weight loss | Inflammation | Superoxide | Glucose | Dextrose | Slopes | Adipose tissue | Cardiovascular disease | Body weight loss | Proteins | Mitochondria | Bioinformatics | Stresses | Cytokines | RNA-mediated interference | Gene expression | Low density lipoprotein | Product differentiation | Polymerase chain reaction | Inhibitors | Ribonucleic acids | Metabolic disorders | Endocrinology | Oxidative stress | Reactive oxygen species | Interleukin | Superoxide dismutase | Kinases | Metabolic syndrome | Synergism | Adiponectin | Interleukin 6 | Quality | Atherosclerosis | Interleukin 1 | Tumor necrosis factor-TNF | Chronology | Dendritic cells | Diabetes mellitus | Triglycerides | Stress | Medicine | Hogs | Sensitivity | Monocytes | Weight control | Insulin resistance | Diagnostic systems | Gastrointestinal surgery | Diabetes | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2009, Volume 29, Issue 12, pp. 2041 - 2046
Journal Article
Circulation, ISSN 0009-7322, 11/2004, Volume 110, Issue 20, pp. 3259 - 3269
Background - Weight loss in obese insulin-resistant but not in insulin-sensitive persons reduces coronary heart disease risk. To what extent changes in gene... 
Obesity | Lipoproteins | Circadian rhythm | Genes | Atherosclerosis | atherosclerosis | METABOLIC SYNDROME | CARDIAC & CARDIOVASCULAR SYSTEMS | LIPID-METABOLISM | genes | LOW-DENSITY-LIPOPROTEIN | BLOOD-PRESSURE VARIABILITY | BODY-COMPOSITION | circadian rhythm | PPAR-GAMMA | PERIPHERAL VASCULAR DISEASE | CIRCULATING OXIDIZED LDL | CORONARY-HEART-DISEASE | HEMATOLOGY | lipoproteins | obesity | TRANSGENIC MICE | ADIPOSE-TISSUE | Obesity - diet therapy | Up-Regulation | Oxidative Stress | Arteriosclerosis - genetics | Obesity - genetics | Adipose Tissue - metabolism | Myocardium - metabolism | Receptors, LDL - deficiency | Autoantibodies - analysis | Transcription, Genetic | Heart Function Tests | PPAR gamma - genetics | Arteriosclerosis - etiology | Receptors, LDL - genetics | Echocardiography | PPAR alpha - biosynthesis | Obesity - complications | Mice, Inbred C57BL | Adipose Tissue - pathology | Insulin Resistance | Lipoproteins, LDL - analysis | Aorta, Thoracic - metabolism | Gene Expression Regulation - physiology | Genotype | PPAR alpha - genetics | Inflammation | Leptin - genetics | Circadian Rhythm | PPAR gamma - biosynthesis | Mice, Knockout | Obesity - pathology | Hypertriglyceridemia - prevention & control | Arteriosclerosis - prevention & control | Animals | Weight Loss | Hypertriglyceridemia - etiology | Glucose - metabolism | Mice, Obese | Arteriosclerosis - metabolism | Mice | Leptin - deficiency | Lipoproteins, LDL - immunology | Index Medicus | Abridged Index Medicus
Journal Article
European Heart Journal, ISSN 0195-668X, 1/2008, Volume 29, Issue 1, pp. 128 - 137
Aims Statins improve atherosclerotic diseases through cholesterol-reducing effects. Whether the latter exclusively mediate similar benefits, e.g. on... 
Superoxide dismutase | Blood pressure | Statin | Nitric oxide | PPARγ | blood pressure | nitric oxide | METABOLIC SYNDROME | CARDIAC & CARDIOVASCULAR SYSTEMS | NITRIC-OXIDE SYNTHASE | ATHEROSCLEROSIS | ALPHA | MECHANISMS | PROLIFERATOR-ACTIVATED-RECEPTOR | PPAR gamma | INHIBITION | statin | SIGNALING PATHWAY | superoxide dismutase | IN-VIVO | SUPEROXIDE-DISMUTASE
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 07/2006, Volume 26, Issue 7, pp. 1558 - 1565
Journal Article
European Heart Journal, ISSN 0195-668X, 01/2008, Volume 29, Issue 1, p. 128
  Aims Statins improve atherosclerotic diseases through cholesterol-reducing effects. Whether the latter exclusively mediate similar benefits, e.g. on... 
Journal Article
Future Lipidology, ISSN 1746-0875, 2008, Volume 3, Issue 6, pp. 637 - 649
Journal Article
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 06/2007, Volume 151, Issue 3, pp. 347 - 355
Background and purpose: Our goal was to elucidate mechanisms of the inhibitory effect of rosuvastatin on the accumulation of plaque oxidized low density... 
atherosclerosis | metabolic syndrome | cholesterol | superoxide dismutase | lipoproteins | obesity | Obesity | Superoxide dismutase | Lipoproteins | Metabolic syndrome | Cholesterol | Atherosclerosis | IMPROVES INSULIN SENSITIVITY | COA REDUCTASE INHIBITORS | LOW-DENSITY-LIPOPROTEIN | ATORVASTATIN | OVEREXPRESSION | ACTIVATED-RECEPTOR-ALPHA | CHOLESTEROL EFFLUX | PPAR-GAMMA | PHARMACOLOGY & PHARMACY | TRANSGENIC MICE | Dyslipidemias - genetics | Superoxide Dismutase - genetics | Body Weight | Gene Expression - drug effects | Humans | Aorta - metabolism | Insulin - blood | Obesity - blood | Obesity - genetics | Fluorobenzenes - pharmacology | Dyslipidemias - blood | Lipids - blood | Receptors, LDL - deficiency | Lipoproteins, LDL - metabolism | Superoxide Dismutase - metabolism | PPAR gamma - genetics | Cell Line | Receptors, LDL - genetics | Atherosclerosis - pathology | Aorta - drug effects | Mice, Inbred C57BL | Rosuvastatin Calcium | Obesity - physiopathology | Leptin - genetics | Macrophages - cytology | Pyrimidines - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Sulfonamides - pharmacology | Atherosclerosis - metabolism | Mice, Knockout | Aorta - pathology | Macrophages - metabolism | Animals | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | Macrophages - drug effects | Mice, Obese | Mice | Blood Glucose - metabolism | Leptin - deficiency | Atherosclerosis - prevention & control | Dyslipidemias - physiopathology | Index Medicus | Research Papers
Journal Article
Physiologia Plantarum, ISSN 0031-9317, 06/2013, Volume 148, Issue 2, pp. 176 - 188
Journal Article
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