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The Journal of Cell Biology, ISSN 0021-9525, 10/2008, Volume 183, Issue 1, pp. 101 - 116
Although Akt is known as a survival kinase, inhibitors of the phosphatidylinositol 3-kinase (PI3K)--Akt pathway do not always induce substantial apoptosis. We... 
Protein isoforms | Cell growth | Cell death | Cell nucleus | Cell cycle | Cell lines | Fluorescence | Tumors | Apoptosis | Cancer | CANCER-CELLS | PROTEIN-KINASE B/AKT | MALIGNANT GLIOMA-CELLS | GLUCOSE-HOMEOSTASIS | ETHER LIPID ANALOGS | RAT HEPATOCYTES | SIGNALING PATHWAY | PROSTATE-CANCER | TRANSCRIPTION FACTOR | MICE LACKING | CELL BIOLOGY | Cell Cycle - genetics | RNA, Small Interfering - genetics | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Furans - pharmacology | Humans | Apoptosis - genetics | Autophagy - physiology | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Autophagy - drug effects | Proto-Oncogene Proteins c-akt - genetics | Quinoxalines - pharmacology | Chloroquine - pharmacology | Drug Interactions | Lysosomes - metabolism | Neoplasms - genetics | RNA Interference | Macrolides - pharmacology | Female | Proto-Oncogene Proteins c-akt - metabolism | Proton-Translocating ATPases - antagonists & inhibitors | Lysosomes - drug effects | PTEN Phosphohydrolase - genetics | Ubiquitin-Activating Enzymes - genetics | Mitochondria - metabolism | Mitochondria - drug effects | Pyrimidines - pharmacology | Neoplasms - drug therapy | Xenograft Model Antitumor Assays | Autophagy-Related Protein 7 | Animals | Mice, Nude | Cell Cycle - physiology | Cell Line, Tumor | Mice | Pyridines - pharmacology | Apoptosis - physiology | Mutation | Cell Cycle - drug effects | Neoplasms - pathology | Benzylamines - pharmacology | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Phosphatidylinositol | Gene silencing | Genetic aspects | Research | Properties | Index Medicus
Journal Article
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 10/2007, Volume 179, Issue 1, pp. 101 - 115
Journal Article
Scientific Reports, ISSN 2045-2322, 05/2019, Volume 9, Issue 1, pp. 6907 - 14
Cystine-knot peptides are attractive templates in drug discovery due to a number of features they possess including their 3D conformation, physicochemical... 
Journal Article | CELLS | DESIGN | MCOTI-II | MECHANISM | CYCLOTIDE KALATA B1 | MULTIDISCIPLINARY SCIENCES | MINIPROTEIN | ANTAGONISTS | SCAFFOLD | Coated pits | Clathrin | Transfection | Peptides | Microscopy | Internalization | Cell membranes | Intracellular | Drug discovery | Electron microscopy | Cytosol
Journal Article
Neuron, ISSN 0896-6273, 12/2018, Volume 100, Issue 6, pp. 1322 - 1336.e7
Synapse loss and Tau pathology are hallmarks of Alzheimer’s disease (AD) and other tauopathies, but how Tau pathology causes synapse loss is unclear. We used... 
postsynaptic density | C1q | Alzheimer’s disease | proteomics | neurodegeneration | Tau | mass spectrometry | synapse | complement | GTPase | Neuroscience(all) | Alzheimer's disease | DENDRITIC SPINES | ACTIN | MICROGLIA | AMYLOID-BETA | ALZHEIMERS-DISEASE | DYSFUNCTION | MUTATIONS | EXPRESSION | NEUROSCIENCES | COGNITIVE DEFICITS | PLASTICITY | Tauopathies - genetics | Embryo, Mammalian | Complement C1q - immunology | Humans | Tauopathies - pathology | tau Proteins - metabolism | Presenilin-2 - metabolism | Complement C1q - metabolism | Prion Proteins - metabolism | Synapses - metabolism | tau Proteins - genetics | Tauopathies - diagnostic imaging | Amyloid beta-Protein Precursor - metabolism | Female | Cell Differentiation | Post-Synaptic Density - metabolism | Prion Proteins - genetics | Animals, Newborn | Antibodies - therapeutic use | Synapses - drug effects | Induced Pluripotent Stem Cells - drug effects | Mice, Inbred C57BL | Cells, Cultured | Rats | Mice, Transgenic | Synapses - ultrastructure | Post-Synaptic Density - ultrastructure | Complement C1q - ultrastructure | Mutation - genetics | Amyloid beta-Protein Precursor - genetics | Animals | Post-Synaptic Density - pathology | Presenilin-2 - genetics | Mice | Tauopathies - drug therapy | Proteome - metabolism | Brain | Ontology | Genes | Blocking antibodies | Complement | Synaptic density | Experiments | Proteins | Dendritic spines | Neurodegeneration | Actin | Transgenic animals | Age | Statistical analysis | Neurodegenerative diseases | Therapeutic applications | Transgenic mice | Microglia | Complement component C1q | Pathology | Tau protein | Cytoskeleton | Evacuations & rescues | Regulatory proteins | Guanosinetriphosphatase
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 09/2013, Volume 123, Issue 9, pp. 3997 - 4009
Many oncology drugs are administered at their maximally tolerated dose without the knowledge of their optimal efficacious dose range. In this study, we... 
REGRESSION | MEDICINE, RESEARCH & EXPERIMENTAL | EGFL7 | THERAPY | VESSELS | ANGIOGENESIS | MODELS | BEVACIZUMAB | CANCER | EXPRESSION | Lung Neoplasms - drug therapy | Pancreatic Neoplasms - metabolism | Neoplastic Stem Cells - drug effects | Humans | Pancreatic Neoplasms - blood supply | Neoplastic Cells, Circulating - metabolism | Lung Neoplasms - pathology | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Bevacizumab | Endothelial Growth Factors - immunology | Pancreatic Neoplasms - drug therapy | Human Umbilical Vein Endothelial Cells - physiology | Neoplastic Stem Cells - metabolism | Insulinoma - blood supply | Antibodies, Monoclonal, Humanized - pharmacology | Tumor Cells, Cultured | Carcinoma, Non-Small-Cell Lung - pathology | Neoplastic Cells, Circulating - drug effects | Human Umbilical Vein Endothelial Cells - drug effects | Insulinoma - drug therapy | Angiogenesis Inhibitors - pharmacology | Mice, Transgenic | Antibodies - pharmacology | Xenograft Model Antitumor Assays | Animals | Tumor Burden - drug effects | Insulinoma - metabolism | Mice, Nude | Mice | Carcinoma, Non-Small-Cell Lung - drug therapy | Vascular Endothelial Growth Factor A - physiology | Clinical Trials, Phase I as Topic | Apoptosis | Care and treatment | Epidermal growth factor | Anti-antibodies | Physiological aspects | Models | Angiogenesis inhibitors | Research | Lung cancer, Non-small cell | Identification and classification | Proteins | Studies | Hypotheses | Biomarkers | Clinical trials | Oncology | Tumors | Cancer
Journal Article
Nature, ISSN 0028-0836, 2014, Volume 509, Issue 7499, pp. 240 - 244
Journal Article
Autophagy, ISSN 1554-8627, 04/2009, Volume 5, Issue 3, pp. 415 - 418
Journal Article
Traffic, ISSN 1398-9219, 2008, Volume 9, Issue 6, pp. 951 - 963
Journal Article