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Nature, ISSN 0028-0836, 07/2016, Volume 535, Issue 7612, pp. 382 - 387
Cells deficient in the Brca1 and Brca2 genes have reduced capacity to repair DNA double-strand breaks by homologous recombination and consequently are... 
BREAST-CANCER | RECOMBINATION | LYSINE-4 METHYLTRANSFERASE COMPLEX | MUTANT-CELLS | 53BP1 | MULTIDISCIPLINARY SCIENCES | RESISTANCE | HOMOLOGY-DIRECTED REPAIR | CONDITIONAL MOUSE MODEL | END RESECTION | DNA-REPAIR | Embryonic Stem Cells - metabolism | DNA Replication - drug effects | Homologous Recombination | DNA Repair - genetics | DNA Breaks, Double-Stranded | DNA-Binding Proteins - metabolism | MRE11 Homologue Protein | Neoplasms - genetics | Gene Deletion | Nuclear Proteins - deficiency | DNA Repair Enzymes - metabolism | DNA Damage - genetics | Genes, BRCA2 | DNA - biosynthesis | Female | DNA Repair Enzymes - antagonists & inhibitors | Genes, BRCA1 | Nuclear Proteins - genetics | DNA Helicases - genetics | DNA Damage - drug effects | DNA Repair - drug effects | DNA-Binding Proteins - antagonists & inhibitors | Cisplatin - pharmacology | DNA - metabolism | Carrier Proteins - genetics | Poly(ADP-ribose) Polymerase Inhibitors - pharmacology | Drug Resistance, Neoplasm - genetics | Animals | Poly(ADP-ribose) Polymerases - genetics | DNA Replication - physiology | Embryonic Stem Cells - drug effects | Cell Line, Tumor | Mice | Poly (ADP-Ribose) Polymerase-1 | Neoplasms - pathology | Drug Resistance, Neoplasm - drug effects | Physiological aspects | Chemotherapy | Replication fork | B cells | DNA damage | Cancer | Proteins | Lymphocytes | Stem cells | Breast cancer | Drug resistance | Deoxyribonucleic acid--DNA | Tumors
Journal Article
Cancer Cell, ISSN 1535-6108, 06/2018, Volume 33, Issue 6, pp. 1078 - 1093.e12
Journal Article
Nature Genetics, ISSN 1061-4036, 08/2017, Volume 49, Issue 8, pp. 1219 - 1230
Journal Article
BMC Bioinformatics, ISSN 1471-2105, 10/2018, Volume 19, Issue 1, pp. 366 - 366
Journal Article
Cell Reports, ISSN 2211-1247, 08/2016, Volume 16, Issue 8, pp. 2087 - 2101
Invasive lobular carcinoma (ILC) is an aggressive breast cancer subtype with poor response to chemotherapy. Besides loss of E-cadherin, a hallmark of ILC,... 
Cre-lox | metastasis | classical invasive lobular carcinoma | PTEN | BEZ235 | breast cancer | tumor microenvironment | E-cadherin | genetically engineered mouse model | BREAST-CANCER | INACTIVATION | DISSEMINATION | LEADS | RESISTANCE | MUTATIONS | INHIBITOR | EXPRESSION | TUMORS | GLAND | CELL BIOLOGY | Carcinoma, Lobular - pathology | Receptors, Estrogen - metabolism | Gene Expression Regulation, Neoplastic | Tumor Microenvironment | Gene Expression Profiling | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Carcinoma, Lobular - mortality | Mammary Neoplasms, Experimental - genetics | Quinolines - pharmacology | Carcinoma, Lobular - genetics | Mammary Neoplasms, Experimental - pathology | Female | Integrases - metabolism | Antineoplastic Agents - pharmacology | Cadherins - genetics | Mammary Neoplasms, Experimental - drug therapy | PTEN Phosphohydrolase - genetics | Mammary Neoplasms, Experimental - mortality | PTEN Phosphohydrolase - deficiency | Receptors, Estrogen - genetics | Signal Transduction | Cell Survival | Neoplasm Invasiveness | Gene Silencing | Imidazoles - pharmacology | Mice, Knockout | Phosphatidylinositol 3-Kinases - genetics | Animals | Survival Analysis | Cell Line, Tumor | Carcinoma, Lobular - drug therapy | Mice | Integrases - genetics | Cadherins - deficiency
Journal Article
Cancer Research, ISSN 0008-5472, 05/2018, Volume 78, Issue 10 Supplement, pp. A47 - A47
Journal Article
by Menden, Michael P and Wang, Dennis and Mason, Mike J and Szalai, Bence and Bulusu, Krishna C and Guan, Yuanfang and Yu, Thomas and Kang, Jaewoo and Jeon, Minji and Wolfinger, Russ and Nguyen, Tin and Zaslavskiy, Mikhail and Abante, Jordi and Abecassis, Barbara Schmitz and Aben, Nanne and Aghamirzaie, Delasa and Aittokallio, Tero and Akhtari, Farida S and Al-lazikani, Bissan and Alam, Tanvir and Allam, Amin and Allen, Chad and de Almeida, Mariana Pelicano and Altarawy, Doaa and Alves, Vinicius and Amadoz, Alicia and Anchang, Benedict and Antolin, Albert A and Ash, Jeremy R and Aznar, Victoria Romeo and Ba-alawi, Wail and Bagheri, Moeen and Bajic, Vladimir and Ball, Gordon and Ballester, Pedro J and Baptista, Delora and Bare, Christopher and Bateson, Mathilde and Bender, Andreas and Bertrand, Denis and Wijayawardena, Bhagya and Boroevich, Keith A and Bosdriesz, Evert and Bougouffa, Salim and Bounova, Gergana and Brouwer, Thomas and Bryant, Barbara and Calaza, Manuel and Calderone, Alberto and Calza, Stefano and Capuzzi, Stephen and Carbonell-Caballero, Jose and Carlin, Daniel and Carter, Hannah and Castagnoli, Luisa and Celebi, Remzi and Cesareni, Gianni and Chang, Hyeokyoon and Chen, Guocai and Chen, Haoran and Chen, Huiyuan and Cheng, Lijun and Chernomoretz, Ariel and Chicco, Davide and Cho, Kwang-Hyun and Cho, Sunghwan and Choi, Daeseon and Choi, Jaejoon and Choi, Kwanghun and Choi, Minsoo and Cock, Martine De and Coker, Elizabeth and Cortes-Ciriano, Isidro and Cserzö, Miklós and Cubuk, Cankut and Curtis, Christina and Daele, Dries Van and Dang, Cuong C and Dijkstra, Tjeerd and Dopazo, Joaquin and Draghici, Sorin and Drosou, Anastasios and Dumontier, Michel and Ehrhart, Friederike and Eid, Fatma-Elzahraa and ElHefnawi, Mahmoud and Elmarakeby, Haitham and van Engelen, Bo and Engin, Hatice Billur and de Esch, Iwan and Evelo, Chris and Falcao, Andre O and Farag, Sherif and Fernandez-Lozano, Carlos and Fisch, Kathleen and Flobak, Asmund and Fornari, Chiara and Foroushani, Amir B. K and Fotso, Donatien Chedom and Fourches, Denis and ... and AstraZeneca-Sanger Drug Combinatio and AstraZeneca-Sanger Drug Combination DREAM Consortium
Nature Communications, ISSN 2041-1723, 12/2019, Volume 10, Issue 1, pp. 2674 - 17
The effectiveness of most cancer targeted therapies is short-lived. Tumors often develop resistance that might be overcome with drug combinations. However, the... 
BREAST-CANCER | ANDROGEN RECEPTOR | INHIBITION | GENE | PATHWAY | LANDSCAPE | MULTIDISCIPLINARY SCIENCES | RESISTANCE | MUTATIONS | CELL | RESOURCE | Antagonism | Computation | Identification methods | Cell lines | Computer applications | Predictions | Biomarkers | Pharmacology | Tumor cell lines | Cancer | Tumors | 1-Phosphatidylinositol 3-kinase | High-throughput screening | Statistical methods | Systems biology | Machine learning
Journal Article