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Journal of Applied Toxicology, ISSN 0260-437X, 03/2019, Volume 39, Issue 3, pp. 498 - 509
Journal Article
Journal Article
Hepatology Research, ISSN 1386-6346, 2006, Volume 34, Issue 3, pp. 199 - 206
Reactive oxygen species (ROS) have been associated with acute ethanol-induced liver damage. N-acetylcysteine (NAC) is a glutathione (GSH) precursor and direct... 
Liver damage | Ethanol | Antioxidant | Pro-oxidant | N-acetylcysteine | antioxidant | KAPPA-B | ethanol | PREGNANE-X-RECEPTOR | FREE-RADICALS | FACTOR-ALPHA | pro-oxidant | TUMOR-NECROSIS-FACTOR | INDUCED OXIDATIVE STRESS | KUPFFER CELLS | ACETYL-L-CYSTEINE | ALCOHOLIC HEPATITIS | GASTROENTEROLOGY & HEPATOLOGY | liver damage | INTESTINAL PERMEABILITY
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2014, Volume 9, Issue 9, pp. e106786 - e106786
Lipopolysaccharide (LPS) is associated with adverse developmental outcomes including embryonic resorption, fetal death, congenital teratogenesis and fetal... 
ANDROGEN RECEPTOR | MALE-MICE LACKING | GRAM-NEGATIVE BACTEREMIA | INTRAUTERINE FETAL-DEATH | LEYDIG-CELLS | MULTIDISCIPLINARY SCIENCES | NITRIC-OXIDE | GROWTH RESTRICTION | AMNIOTIC-FLUID | PRETERM LABOR | DEFECTIVE SPERMATOGENESIS | Sperm Count | Body Weight - drug effects | Prostate - growth & development | Male | Leydig Cells - drug effects | Lipopolysaccharides - adverse effects | Testis - drug effects | Maternal Exposure - adverse effects | Prostate - drug effects | Female | Testosterone - blood | Steroids - biosynthesis | Steroids - blood | Spermatogenesis - drug effects | Luteinizing Hormone - blood | Seminal Vesicles - drug effects | Testosterone - biosynthesis | Spermatozoa - drug effects | Testis - growth & development | Fetus - drug effects | Pregnancy | Organ Size - drug effects | Animals | Seminal Vesicles - growth & development | Mice | Spermatozoa - cytology | Prenatal Exposure Delayed Effects - chemically induced | Prenatal Exposure Delayed Effects - pathology | Testosterone | Embryonic development | Fetus | Growth | Pregnant women | Germ cells | Growth rate | Seminal vesicle | Spermatogenesis | Body weight | Infections | Gestation | Males | Anogenital | Lipopolysaccharides | Teratogenesis | Toxicology | Rodents | Population | Public health | Bacterial infections | Fetuses | Tubules | Histology | Exposure | Embryos | Progeny | Androgens | Offspring | Infertility | Steroidogenesis | Laboratory animals | Testes | Females | Endocrinology | Index Medicus
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 01/2017, Volume 314, pp. 39 - 47
The farnesoid X receptor (FXR) is a ligand-activated transcription factor that plays important roles in regulating bile acid homeostasis. The aim of the... 
Inflammation | Farnesoid X receptor (FXR) | Acute liver injury | Obeticholic acid | Carbon tetrachloride (CCl4) | Carbon tetrachloride (CCl | FIBROSIS | HEPATOCYTE APOPTOSIS | STELLATE CELLS | NECROSIS-FACTOR-ALPHA | NONALCOHOLIC STEATOHEPATITIS | MECHANISMS | FAILURE | HEPATIC INFLAMMATION | FARNESOID-X-RECEPTOR | PHARMACOLOGY & PHARMACY | TOXICOLOGY | AGONIST | Chemical and Drug Induced Liver Injury - prevention & control | Apoptosis - drug effects | Male | Alanine Transaminase - blood | Carbon Tetrachloride Poisoning - complications | Extracellular Signal-Regulated MAP Kinases - metabolism | Carbon Tetrachloride Poisoning - prevention & control | Inflammation - etiology | Chenodeoxycholic Acid - analogs & derivatives | Animals | Chemical and Drug Induced Liver Injury - complications | Chenodeoxycholic Acid - pharmacology | Hepatocytes - chemistry | Mice | p38 Mitogen-Activated Protein Kinases - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Hepatocytes - drug effects | Receptors, Cytoplasmic and Nuclear - metabolism | Bile acids | Liver | Analysis | Carbon tetrachloride | Index Medicus | APOPTOSIS | TRANSLOCATION | PHOSPHORYLATION | 60 APPLIED LIFE SCIENCES | NECROSIS | LIGANDS | GENES | LIVER CELLS | MICE | BILE | TRANSCRIPTION FACTORS | HOMEOSTASIS | CARBON | INJURIES | LABELLING | NEOPLASMS | MACROPHAGES | TRANSCRIPTION | MONOCYTES | LYMPHOKINES | CARBON TETRACHLORIDE | INFLAMMATION | LIVER | PHOSPHOTRANSFERASES | RECEPTORS | BILE ACIDS
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2012, Volume 7, Issue 9, p. e45617
Aberrant activation of beta-catenin/Tcf-4 signaling has been implicated in human carcinogenesis, including colorectal cancer. In this study, we compared the... 
TRANSCRIPTION FACTORS | IN-VITRO | APC | CYCLIN D1 | COLORECTAL-CANCER | MULTIDISCIPLINARY SCIENCES | GROWTH | GENE-EXPRESSION | DOWN-REGULATION | NUCLEAR BETA-CATENIN | CATENIN SIGNALING PATHWAY | Phosphorylation | Transcription Factor 4 | Signal Transduction | Apoptosis - drug effects | Colonic Neoplasms - drug therapy | HCT116 Cells | Humans | Transcription Factors - genetics | DNA Primers | Wnt Proteins - metabolism | beta Catenin - metabolism | Colonic Neoplasms - metabolism | Organoplatinum Compounds - pharmacology | beta Catenin - genetics | Gene Knockdown Techniques | Transcription Factors - metabolism | Base Sequence | Colonic Neoplasms - pathology | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - genetics | Polymerase Chain Reaction | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Antineoplastic Agents - pharmacology | Fluorouracil - pharmacology | Genetic Vectors | Care and treatment | Transcription factors | Colon cancer | Cancer cells | Physiological aspects | Research | Apoptosis | Cell proliferation | Laboratories | Toxicity | Colorectal carcinoma | Colorectal cancer | Cytotoxicity | Kinases | Carcinogenesis | DNA repair | Proteins | β-catenin | Carcinogens | Cell growth | Oxaliplatin | Cell cycle | FOXO4 protein | Forkhead protein | Colon | Inhibition | Deoxyribonucleic acid--DNA | Gene expression | Ribonucleic acid--RNA | Signaling | Hospitals | Plasmids | Adenoviruses | Cyclin-dependent kinase inhibitor p27 | Aberration | Cancer | RNA | Deoxyribonucleic acid | Ribonucleic acid | DNA
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2014, Volume 9, Issue 12, pp. e114780 - e114780
It is increasingly recognized that intra-uterine growth restriction (IUGR) is associated with an increased risk of metabolic disorders in late life. Previous... 
PRETERM DELIVERY | INTRAUTERINE FETAL-DEATH | INCREASED SUSCEPTIBILITY | FATTY LIVER-DISEASE | MULTIDISCIPLINARY SCIENCES | CATCH-UP GROWTH | BIRTH-WEIGHT | N-ACETYLCYSTEINE | INSULIN SENSITIVITY | GLUCOSE-TOLERANCE | FOR-GESTATIONAL-AGE | Liver - pathology | Prenatal Exposure Delayed Effects | Body Weight | Lipopolysaccharides - toxicity | Liver - metabolism | Insulin Resistance | Diet, High-Fat - adverse effects | Lipid Metabolism | Male | Fetal Growth Retardation - metabolism | Insulin - blood | Maternal Exposure | Mice, Inbred ICR | Obesity - metabolism | Triglycerides - metabolism | Pregnancy | Adipose Tissue - metabolism | Insulin - metabolism | Animals | Metabolic Diseases - metabolism | Metabolic Diseases - etiology | Female | Phenotype | Blood sugar | Pregnant women | Analysis | Liver | Triglycerides | Genetic aspects | Glucose tolerance tests | Intervention | Adipose tissue | Lactation | Body fat | Laboratories | Cardiovascular disease | Glucose | Gestation | Metabolic syndrome | High fat diet | Lipopolysaccharides | Toxicology | Uterus | Rodents | Population | Obesity | Medical research | Phenotypes | Sensitivity analysis | Liver diseases | Fetuses | Exposure | Gene expression | Insulin | Glucose tolerance | Progeny | Offspring | Hospitals | Diet | Insulin resistance | Mice | Females | Metabolic disorders | Index Medicus
Journal Article
Redox Biology, ISSN 2213-2317, 04/2019, Volume 22, pp. 101148 - 101148
Mitochondria damage plays a critical role in acetaminophen (APAP)-induced necrosis and liver injury. Cells can adapt and protect themselves by removing damaged... 
Drug | Mitochondria | Cell death | Autophagy | Hepatotoxicity | ACTIVATION | UBIQUITIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | MITOCHONDRIAL-FUNCTION | MECHANISMS | INDUCTION | AUTOPHAGY PROTECTS | PATHOPHYSIOLOGICAL ROLES | PROMOTES | Index Medicus
Journal Article
Journal Article