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1. Full Text QnAs with Ronald DePinho
by Prashant Nair and Ronald DePinho
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2013, Volume 110, Issue 36, pp. 14508 - 14509
Education legislation | Developmental biology | Genes | Mortality | Melanoma | QNAS | Genetic mutation | Cancer | Sun tanning | MULTIDISCIPLINARY SCIENCES | QnAs
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2. Full Text An interview with Ronald DePinho
by Landau, Misia
Clinical Chemistry, ISSN 0009-9147, 01/2013, Volume 59, Issue 1, pp. 11 - 17
  Believing, too, that the best way to get at cancer was to study it in a living animal, he set out to learn the new and largely untried techniques of genetic... 
MEDICAL LABORATORY TECHNOLOGY | Geneticists | Science | Families & family life | Cell division | Grocery stores | Telomerase | Cancer
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3. Full Text Straight talk with ... Ronald DePinho
by Hersher, R and DePinho, R
NATURE MEDICINE, ISSN 1078-8956, 01/2012, Volume 18, Issue 1, pp. 9 - 9
  On 1 September, Ronald DePinho became the new president of the MD Anderson Cancer Center at the University of Texas in Houston. He arrives there after 14... 
MEDICINE, RESEARCH & EXPERIMENTAL | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | Officials and employees | Nonprofit organizations
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4. Full Text How stem cells age and why this makes us grow old
by Sharpless, Norman E and DePinho, Ronald A
Nature Reviews Molecular Cell Biology, ISSN 1471-0072, 09/2007, Volume 8, Issue 9, pp. 703 - 713
Recent data suggest that we age, in part, because our self-renewing stem cells grow old as a result of heritable intrinsic events, such as DNA damage, as well... 
PROGENITOR CELLS | MULTIPLE-MYELOMA | LIFE-SPAN | OXIDATIVE STRESS | TELOMERE DYSFUNCTION | BONE-MARROW-TRANSPLANTATION | MURINE HEMATOPOIETIC STEM | SELF-RENEWAL | HUMAN FIBROBLASTS | GENOME-WIDE ASSOCIATION | CELL BIOLOGY | Atherosclerosis - etiology | Animals | Aging - physiology | Humans | Tumor Suppressor Protein p14ARF - physiology | Diabetes Mellitus, Type 2 - etiology | Stem Cells - physiology | Stem cells | Aging | Causes of | Chronic diseases | Physiological aspects | Genetic aspects | Properties | Risk factors
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5. Full Text QnAs with Ronald DePinho. Interview by Prashant Nair
by DePinho, Ronald
Proceedings of the National Academy of Sciences of the United States of America, 09/2013, Volume 110, Issue 36, p. 14508
RNA-Binding Proteins - genetics | Genetic Predisposition to Disease - genetics | History, 21st Century | United States | History, 20th Century | Humans | Glioblastoma - prevention & control | RNA Splicing Factors | Brain Neoplasms - genetics | Transcription Factors - genetics | Nerve Tissue Proteins - genetics | Brain Neoplasms - prevention & control | Glioblastoma - genetics | Glioblastoma - history | Mutation | Brain Neoplasms - history
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6. Full Text Linking functional decline of telomeres, mitochondria and stem cells during ageing
by Sahin, Ergün and DePinho, Ronald A
Nature, ISSN 0028-0836, 03/2010, Volume 464, Issue 7288, pp. 520 - 528
The study of human genetic disorders and mutant mouse models has provided evidence that genome maintenance mechanisms, DNA damage signalling and metabolic... 
LIFE-SPAN | TUMOR SUPPRESSION | ATM DEFICIENCY | AGE-RELATED-CHANGES | MULTIDISCIPLINARY SCIENCES | DNA-DAMAGE | REVERSE-TRANSCRIPTASE | TERMINAL TRANSFERASE | CANCER | CHROMOSOMAL INSTABILITY | P53 | Mitochondria - enzymology | Humans | Genome - genetics | Tumor Suppressor Protein p53 - metabolism | Mitochondria - pathology | Aging - pathology | Phenotype | Telomere - pathology | Animals | Telomerase - genetics | Homeostasis - physiology | Stem Cells - pathology | Telomerase - metabolism | Hematopoietic System - pathology | Telomeres | Stem cell research | Mitochondrial DNA | Research | Tumor proteins | Proteins | Tissue | Wound healing | Medical technology | Biomarkers | Risk factors | Deoxyribonucleic acid--DNA | Cancer | Apoptosis
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7. Full Text The mighty mouse: genetically engineered mouse models in cancer drug development
by Sharpless, Norman E and DePinho, Ronald A
Nature Reviews Drug Discovery, ISSN 1474-1776, 09/2006, Volume 5, Issue 9, pp. 741 - 754
Deficiencies in the standard preclinical methods for evaluating potential anticancer drugs,such as xenograft mouse models, have been highlighted as a key... 
Reproducibility of Results | Animals | Drug Screening Assays, Antitumor - methods | Humans | Drug Design | Neoplasms, Experimental - genetics | Mice, Transgenic | Antineoplastic Agents - therapeutic use | Mice | Neoplasms, Experimental - drug therapy
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8. Full Text DeVita, Hellman, and Rosenberg's cancer
: principles & practice of oncology
by DeVita, Vincent T., Jr., 1935 and Lawrence, Theodore S and Rosenberg, Steven A
2008, 8th ed., ISBN 9780781772075
Acclaimed by the worldwide medical community as "a staple reference text in the medical oncologist's library" (JAMA), DeVita, Hellman, and Rosenberg's Cancer:... 
Neoplasms
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9. Full Text Telomeres and telomerase in cancer
by Artandi, Steven E and DePinho, Ronald A
Carcinogenesis, ISSN 0143-3334, 1/2010, Volume 31, Issue 1, pp. 9 - 18
Myriad genetic and epigenetic alterations are required to drive normal cells toward malignant transformation. These somatic events commandeer many signaling... 
CATALYTIC SUBUNIT | LIFE-SPAN | MAMMALIAN TELOMERES | ONCOLOGY | DNA-DAMAGE RESPONSE | DOMINANT DYSKERATOSIS-CONGENITA | REVERSE-TRANSCRIPTASE | DYSFUNCTIONAL TELOMERES | TUMOR-SUPPRESSOR | HUMAN BREAST-CANCER | IN-SITU | Humans | Tumor Suppressor Protein p53 - metabolism | Neoplasms - enzymology | Dyskeratosis Congenita - genetics | Telomere | Dyskeratosis Congenita - enzymology | Mice, Knockout | Animals | Telomerase - genetics | Neoplasms - genetics | Cellular Senescence | Germ-Line Mutation | Telomerase - metabolism | Mice | Index Medicus | Cancer Biology
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10. Full Text Hepatocellular carcinoma pathogenesis: from genes to environment
by Farazi, Paraskevi A and DePinho, Ronald A
Nature Reviews Cancer, ISSN 1474-175X, 09/2006, Volume 6, Issue 9, pp. 674 - 687
Hepatocellular carcinoma is among the most lethal and prevalent cancers in the human population. Despite its significance, there is only an elemental... 
LI-FRAUMENI-SYNDROME | ONCOLOGY | CHRONIC LIVER-DISEASE | MESSENGER-RNA EXPRESSION | GROWTH-FACTOR-ALPHA | ABERRANT DNA METHYLATION | HEPATITIS-C-VIRUS | COMPARATIVE FUNCTIONAL GENOMICS | COMPARATIVE GENOMIC HYBRIDIZATION | BETA-CATENIN MUTATIONS | TELOMERASE REVERSE-TRANSCRIPTASE | Liver Neoplasms - etiology | Environment | Carcinoma, Hepatocellular - etiology | Genes, Neoplasm - physiology | Humans | Care and treatment | Genetic aspects | Hepatoma | Diagnosis | Research | Health aspects | Risk factors | Cancer
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