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1995, Di 1 ban., ISBN 7211025778, 3, 2, 1, 2, 21, 548
Book
Science, ISSN 0036-8075, 12/2010, Volume 330, Issue 6009, pp. 1390 - 1393
Although the proteins BAX and BAK are required for initiation of apoptosis at the mitochondria, how BAX and BAK are activated remains unsettled. We provide in... 
T lymphocytes | Mitochondria | Cytokines | Thymocytes | Neurons | Cell death | REPORTS | Cytochromes | Mice | Potassium | Apoptosis | NEURONAL APOPTOSIS | CYTOCHROME-C | MITOCHONDRIAL APOPTOSIS | MECHANISM | MULTIDISCIPLINARY SCIENCES | BH3 DOMAINS | RELEASE | JNK PATHWAY | PROTEINS | BCL-2 FAMILY-MEMBERS | MEMBRANE PERMEABILIZATION | BH3 Interacting Domain Death Agonist Protein - deficiency | T-Lymphocytes - physiology | bcl-2-Associated X Protein - chemistry | Protein Multimerization | Stress, Physiological | bcl-2 Homologous Antagonist-Killer Protein - genetics | BH3 Interacting Domain Death Agonist Protein - genetics | bcl-2 Homologous Antagonist-Killer Protein - metabolism | Membrane Proteins - deficiency | Caspases - metabolism | Bcl-2-Like Protein 11 | Apoptosis Regulatory Proteins - deficiency | Tumor Suppressor Proteins - deficiency | Tumor Suppressor Proteins - genetics | Neurons - physiology | Apoptosis Regulatory Proteins - genetics | Membrane Proteins - metabolism | BH3 Interacting Domain Death Agonist Protein - metabolism | bcl-2-Associated X Protein - genetics | Proto-Oncogene Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Cytochromes c - metabolism | Cells, Cultured | bcl-2-Associated X Protein - metabolism | Proto-Oncogene Proteins - genetics | Mitochondria - metabolism | Permeability | Proto-Oncogene Proteins - deficiency | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Animals | Models, Biological | Cerebellum - cytology | Intracellular Membranes - metabolism | bcl-2 Homologous Antagonist-Killer Protein - chemistry | Protein research | Genetic aspects | Mitochondrial DNA | Biochemical genetics | Research | Properties | Methods | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 10/2015, Volume 17, Issue 10, pp. 1270 - 1281
Multidomain pro-apoptotic BAX and BAK, once activated, permeabilize mitochondria to trigger apoptosis, whereas anti-apoptotic BCL-2 members preserve... 
CYTOCHROME-C | MITOCHONDRIAL APOPTOSIS | ACTIVATION | CONFORMATIONAL-CHANGE | MEMBERS | PROAPOPTOTIC BAX | BH3 DOMAINS | PUMA | BH3-ONLY PROTEINS | MEMBRANE PERMEABILIZATION | CELL BIOLOGY | bcl-2 Homologous Antagonist-Killer Protein - genetics | Immunoblotting | BH3 Interacting Domain Death Agonist Protein - genetics | Cytochromes c - genetics | bcl-2 Homologous Antagonist-Killer Protein - metabolism | Embryo, Mammalian - metabolism | Proto-Oncogene Proteins c-bcl-2 - metabolism | Bcl-2-Like Protein 11 | Mitochondria - genetics | RNA Interference | Tumor Suppressor Proteins - genetics | Apoptosis Regulatory Proteins - genetics | Membrane Proteins - metabolism | BH3 Interacting Domain Death Agonist Protein - metabolism | bcl-2-Associated X Protein - genetics | Fibroblasts - metabolism | Proto-Oncogene Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Cytochromes c - metabolism | Mice, Inbred C57BL | Cells, Cultured | bcl-2-Associated X Protein - metabolism | Proto-Oncogene Proteins - genetics | Mitochondria - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Animals | Intestine, Small - cytology | Embryo, Mammalian - cytology | Models, Biological | Fibroblasts - cytology | Intestine, Small - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | Apoptosis | Genotype | Genetic aspects | Properties | Cell death | Cellular control mechanisms | Index Medicus
Journal Article
Diabetes, ISSN 0012-1797, 06/2019, Volume 68, Issue Supplement 1, p. 362
N6-methyladenosine (m6A) is the most abundant mRNA modification. However, the role of m6A in regulating β-cell function is unknown. To address the functional... 
Cell proliferation | RNA modification | Secretion | Diabetes mellitus | Methyltransferase | Transgenic mice | Glucose | Tamoxifen | Gene expression | Insulin | Feeding | Beta cells | 3' Untranslated regions | Clonal deletion | Cell death | Rodents | Diabetes | Insulin-like growth factor-binding protein 5
Journal Article
Journal of Structural Engineering, ISSN 0733-9445, 02/2016, Volume 142, Issue 2, p. 4015122
AbstractIn this study, a novel force-based frame element with delocalization is developed to address localization issues induced by material strain softening.... 
Technical Papers | Force-based frame element | Strain softening | Analysis and computation | Delocalization | Plastic hinge | Implicit gradient method | R/C FRAMES | NONLINEAR-ANALYSIS | DAMAGE | CONCRETE | ISSUES | ENGINEERING, CIVIL | CONSTRUCTION & BUILDING TECHNOLOGY | BEAM-COLUMN MODEL | PLASTICITY | Hinges | Research | Analysis | Deformations (Mechanics)
Journal Article
Oncotarget, ISSN 1949-2553, 2017, Volume 8, Issue 34, pp. 56768 - 56779
Mutations in the gene for Immediate Early Response 3 Interacting Protein 1 (IER3IP1) cause permanent neonatal diabetes mellitus in human. The mechanisms... 
IER3IP1 | Cell death and proliferation | Beta-cell | APOPTOSIS | ACTIVATION | PHOSPHORYLATION | BIM | ENDOPLASMIC-RETICULUM STRESS | cell death and proliferation | CELL BIOLOGY | UNFOLDED PROTEIN RESPONSE | beta-cell | BAX | EPILEPSY | PUMA | EXPRESSION
Journal Article
Journal of Endocrinology, ISSN 0022-0795, 08/2018, Volume 238, Issue 2, pp. 151 - 163
Journal Article
DIABETES, ISSN 0012-1797, 07/2018, Volume 67, Issue Supplement 1, p. 2103
Conference Proceeding
IEEE Transactions on Geoscience and Remote Sensing, ISSN 0196-2892, 11/2018, Volume 56, Issue 11, pp. 6537 - 6546
Journal Article
Tetrahedron, ISSN 0040-4020, 08/2014, Volume 70, Issue 34, pp. 5274 - 5282
A CuO nanoparticle-catalyzed one-pot process has engineered for the preparation of diverse quinazolinones from the readily available isatoic anhydride,... 
2,3-Dihydroquinazolin-4(1H)-ones | Quinazolin-4(3H)-ones | Ultrasound irradiation | CuO nanoparticles | ASYMMETRIC-SYNTHESIS | ONE-POT | CHEMISTRY, ORGANIC | IONIC LIQUID | ALDEHYDES | DERIVATIVES | WATER | Nanoparticles | IRRADIATION | Quinazolinones | PARTICLES | Ethanol | AMINES | Tolerances | ETHYL ALCOHOL | Ultrasound | RADIATION | CATALYSTS | COPPER OXIDE
Journal Article
Molecular Cell, ISSN 1097-2765, 11/2009, Volume 36, Issue 3, pp. 487 - 499
While activation of BAX/BAK by BH3-only molecules (BH3s) is essential for mitochondrial apoptosis, the underlying mechanisms remain unsettled. Here we... 
CELLCYCLE | CYTOCHROME-C | PROAPOPTOTIC BAX | OLIGOMERIZES BAK | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOPLASMIC-RETICULUM | SUBCELLULAR LOCATION | PROTEINS | BCL-2 FAMILY-MEMBERS | BH3 DOMAIN | CELL-DEATH | MEMBRANE PERMEABILIZATION | CELL BIOLOGY | bcl-2-Associated X Protein - chemistry | Immunoprecipitation | Apoptosis - drug effects | Protein Multimerization | bcl-2 Homologous Antagonist-Killer Protein - genetics | Immunoblotting | BH3 Interacting Domain Death Agonist Protein - genetics | Green Fluorescent Proteins - genetics | bcl-2 Homologous Antagonist-Killer Protein - metabolism | Bcl-2-Like Protein 11 | Protein Binding - drug effects | Tumor Suppressor Proteins - genetics | Apoptosis Regulatory Proteins - genetics | Membrane Proteins - metabolism | BH3 Interacting Domain Death Agonist Protein - metabolism | bcl-2-Associated X Protein - genetics | Fibroblasts - metabolism | Proto-Oncogene Proteins - metabolism | Green Fluorescent Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Cells, Cultured | bcl-2-Associated X Protein - metabolism | Etoposide - pharmacology | Proto-Oncogene Proteins - genetics | Mitochondria - metabolism | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Animals | Models, Biological | Tunicamycin - pharmacology | Fibroblasts - drug effects | Thapsigargin - pharmacology | Fibroblasts - cytology | Mice | Mutation | Microscopy, Fluorescence | Staurosporine - pharmacology | bcl-2 Homologous Antagonist-Killer Protein - chemistry | Monomers | Apoptosis | Oligomers | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2009, Volume 106, Issue 4, pp. 1093 - 1098
Journal Article
by Sun, QQ and Yuan, F and Ren, DC and Ma, GN and Yang, FP and Wu, X and He, L and He, G
NEUROPSYCHIATRIC DISEASE AND TREATMENT, ISSN 1176-6328, 2019, Volume 15, pp. 657 - 661
Purpose: Venlafaxine is one of the commonly prescribed antidepressants for major depressive disorder (MDD). Accumulated evidence revealed the involvement of... 
venlafaxine | major depressive disorder | BDNF | PSYCHIATRY | association | POLYMORPHISM | GSK-3 beta | CLINICAL NEUROLOGY | Haplotypes | Neurosciences | Laboratories | Genes | Science | Systematic review | Mental depression | Kinases | Clinical outcomes | Studies | Psychosis | Marital status | Brain-derived neurotrophic factor | Antidepressants | Education | Population | Psychiatry | Age | Polymorphism | GSK-3β
Journal Article
1976, Tai 1 ban., Zhongguo fang zhi cong shu Hua bei di fang, Volume di 365 hao, 3 v. (1108 p.)
Book
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