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Giornale Italiano di Cardiologia, ISSN 1827-6806, 09/2010, Volume 11, Issue 9, pp. 654 - 659
Journal Article
L'Endocrinologo, ISSN 1590-170X, 04/2018, Volume 19, Issue S1, pp. 1 - 2
Journal Article
The Lancet Diabetes & Endocrinology, ISSN 2213-8587, 05/2019, Volume 7, Issue 5, pp. 328 - 329
Journal Article
The Lancet Diabetes & Endocrinology, ISSN 2213-8587, 09/2019, Volume 7, Issue 9, pp. 659 - 661
Journal Article
Journal Article
Diabetes Care, ISSN 0149-5992, 08/2016, Volume 39, pp. S113 - S114
Journal Article
Diabetes, ISSN 0012-1797, 05/2018, Volume 67, Issue Supplement 1, p. 1181
Journal Article
European Endocrinology, ISSN 1758-3772, 2017, Volume 13, Issue 2, pp. 54 - 55
After many years of limited therapeutic opportunities, the treatment of type 2 diabetes has become more target and pathophysiologically driven. A typical... 
Type 2 diabetes | DPP-4 inhibitors | Vildagliptin | vildagliptin | Type 2 Diabetes
Journal Article
Lancet, The, ISSN 0140-6736, 2014, Volume 383, Issue 9922, pp. 1068 - 1083
Summary Glucose metabolism is normally regulated by a feedback loop including islet β cells and insulin-sensitive tissues, in which tissue sensitivity to... 
Internal Medicine | ALL-CAUSE MORTALITY | LIFE-STYLE INTERVENTION | MEDICINE, GENERAL & INTERNAL | DIPEPTIDYL PEPTIDASE-4 INHIBITORS | GLUCAGON-LIKE PEPTIDE-1 | 11-BETA-HYDROXYSTEROID DEHYDROGENASE TYPE-1 | IMPAIRED GLUCOSE-TOLERANCE | INSULIN-TREATED PATIENTS | FIBROBLAST GROWTH-FACTORS | ACTING BASAL INSULIN | BETA-CELL FUNCTION | Bariatric Surgery | Insulin-Secreting Cells - physiology | Obesity - surgery | Genetic Predisposition to Disease | Obesity - complications | Epigenesis, Genetic | Humans | Diabetes Mellitus, Type 2 - prevention & control | Insulin Resistance | Gene-Environment Interaction | Inflammation - complications | Insulin - metabolism | Insulin-Secreting Cells - metabolism | Glucagon-Secreting Cells - physiology | Glucose - metabolism | Diabetes Mellitus, Type 2 - etiology | Diabetes Mellitus, Type 2 - drug therapy | Type 2 diabetes | Physiological aspects | Development and progression | Insulin resistance | Pancreatic beta cells | Care and treatment | Medical research | Medical colleges | Obesity | Glucose | Fatty acids | Insulin | Dextrose | Surgery | Medicine, Experimental | Drug therapy | Diabetes therapy | Pathogenesis | Feedback | Lifestyles | Gene loci | Diabetes | treatment | α-cell | bariatric surgery | impaired glucose tolerance | β cell | insulin secretion | microbiome | environment | type 2 diabetes | genetics | impaired fasting glucose | inflammation | glucagon secretion | pathophysiology | medications | clinical trials | insulin resistance | prevention
Journal Article
Expert Opinion on Pharmacotherapy, ISSN 1465-6566, 12/2011, Volume 12, Issue 17, pp. 2759 - 2762
Dipeptidyl peptidase-4 (DPP-4) inhibitors are a new class of antidiabetic agents that have been rapidly adopted since their introduction in 2006. There are now... 
linagliptin | type 2 diabetes | DPP-IV inhibitor
Journal Article
Diabetes Research and Clinical Practice, ISSN 0168-8227, 2016, Volume 120, pp. 184 - 185
Journal Article
Diabetes Research and Clinical Practice, ISSN 0168-8227, 10/2016, Volume 120, pp. 184 - 185
Journal Article
Journal Article
Lancet, The, ISSN 0140-6736, 2011, Volume 378, Issue 9786, pp. 182 - 197
Summary The increasing prevalence, variable pathogenesis, progressive natural history, and complications of type 2 diabetes emphasise the urgent need for new... 
Internal Medicine | MEDICINE, GENERAL & INTERNAL | DIPEPTIDYL PEPTIDASE-4 INHIBITOR | GLUCAGON-LIKE PEPTIDE-1 | 11-BETA-HYDROXYSTEROID DEHYDROGENASE TYPE-1 | ALPHA-CELL DYSFUNCTION | SMALL-MOLECULE | INSULIN-RESISTANCE | DOUBLE-BLIND | BARIATRIC SURGERY | BETA-CELL | INADEQUATE GLYCEMIC CONTROL | Glucokinase - physiology | Dipeptidyl-Peptidase IV Inhibitors - therapeutic use | Humans | Allylamine - therapeutic use | Liver - physiopathology | Dipeptidyl-Peptidase IV Inhibitors - pharmacology | Peptides - administration & dosage | Hypoglycemic Agents - administration & dosage | Liver - drug effects | Insulin Resistance - physiology | Allylamine - pharmacology | Receptors, Dopamine D2 - drug effects | Glucagon-Like Peptide 1 - analysis | Venoms - administration & dosage | Indoles - pharmacology | Hyperglycemia - physiopathology | Colesevelam Hydrochloride | Bariatric Surgery | Hypoglycemic Agents - therapeutic use | Insulin-Secreting Cells - physiology | Obesity - surgery | Insulin - pharmacology | Comorbidity | Receptors, Dopamine D2 - agonists | Allylamine - analogs & derivatives | Treatment Outcome | Cardiovascular System - drug effects | 11-beta-Hydroxysteroid Dehydrogenase Type 1 - antagonists & inhibitors | Bile Acids and Salts | Sodium-Glucose Transporter 2 - antagonists & inhibitors | Hypoglycemic Agents - pharmacology | Randomized Controlled Trials as Topic | Anticholesteremic Agents - therapeutic use | Diabetes Mellitus, Type 2 - physiopathology | Insulin-Secreting Cells - drug effects | Signal Transduction - drug effects | Obesity - epidemiology | Indoles - therapeutic use | Signal Transduction - physiology | Diabetes Mellitus, Type 2 - drug therapy | Anticholesteremic Agents - pharmacology | Type 2 diabetes | Technology application | Care and treatment | Therapeutics | Diagnosis | Research | Homeopathy | Materia medica and therapeutics | Obesity | Medical research | Insulin resistance | Diabetes | Insulin
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