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Nature Cell Biology, ISSN 1465-7392, 2014, Volume 16, Issue 4, pp. 357 - 366
The YAP and TAZ mediators of the Hippo pathway ( hereafter called YAP/TAZ) promote tissue proliferation and organ growth. However, how their biological... 
ORGAN GROWTH | CHOLESTEROL | YAP/TAZ | TRANSCRIPTION | HIPPO PATHWAY | STATINS | CELL BIOLOGY | Phosphorylation - physiology | Cell Proliferation | Active Transport, Cell Nucleus - physiology | Humans | Intracellular Signaling Peptides and Proteins - metabolism | Drosophila Proteins - metabolism | Phosphoproteins - metabolism | Sterol Regulatory Element Binding Proteins - genetics | Breast Neoplasms - metabolism | Drosophila melanogaster - metabolism | RNA Interference | Tumor Suppressor Proteins - genetics | HEK293 Cells | Trans-Activators - genetics | Female | Transcription, Genetic | Hydroxymethylglutaryl-CoA Reductases, NAD-Dependent - metabolism | Nuclear Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Signal Transduction | HCT116 Cells | Protein-Serine-Threonine Kinases - genetics | Nuclear Proteins - metabolism | Sterol Regulatory Element Binding Proteins - metabolism | Transcription Factors - genetics | Mevalonic Acid - metabolism | Polyisoprenyl Phosphates - metabolism | Transcription Factors - metabolism | Animals | Hydroxymethylglutaryl-CoA Reductase Inhibitors - pharmacology | rho GTP-Binding Proteins - metabolism | Trans-Activators - metabolism | Mice | Polyisoprenyl Phosphates - biosynthesis | Pyridines - pharmacology | RNA, Small Interfering | Drosophila Proteins - genetics | Adaptor Proteins, Signal Transducing - metabolism | Cell metabolism | Genetic research | Genetic aspects | Research | Cellular control mechanisms | Index Medicus
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 01/2019, Volume 26, Issue 2, pp. 199 - 212
Forty years of research have established that the p53 tumor suppressor provides a major barrier to neoplastic transformation and tumor progression by its... 
BREAST-CANCER | WILD-TYPE | KAPPA-B ACTIVATION | ONCOGENE | RETINOIC ACID | METASTASIS | GAIN-OF-FUNCTION | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEAT-SHOCK | COMMON GAIN | EXPRESSION | CELL BIOLOGY | Transformation | Cell survival | p53 Protein | Chemoresistance | Homeostasis | Genomes | Metastases | Missense mutation | Addiction | Tumor suppressor genes | Mutation | Deoxyribonucleic acid--DNA | Cancer | Tumors | Immune system | Review
Journal Article
FEBS Letters, ISSN 0014-5793, 08/2014, Volume 588, Issue 16, pp. 2600 - 2609
The tumor suppressor p53 is a transcription factor that in response to a plethora of stress stimuli activates a complex and context-dependent cellular response... 
Mitochondria | Transcription-independent | Metabolism | Cytoplasm | p53 | Apoptosis | PROLYL-ISOMERASE PIN1 | DNA-BINDING DOMAIN | STEM-CELLS | INTRINSIC-APOPTOTIC PATHWAY | BIOCHEMISTRY & MOLECULAR BIOLOGY | INTERACTING PROTEIN KINASE-2 | TUMOR-SUPPRESSOR P53 | CELL-DEATH | CELL BIOLOGY | MITOCHONDRIAL-MEMBRANE PERMEABILIZATION | BIOPHYSICS | IN-VIVO | MUTANT P53
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 02/2015, Volume 112, Issue 6, pp. 1779 - 1784
Journal Article
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 03/2018, Volume 25, Issue 4, pp. 643 - 645
Journal Article
Journal Article
Oncotarget, ISSN 1949-2553, 04/2018, Volume 9, Issue 29, pp. 20508 - 20523
NRF2 (NFE2L2) is one of the main regulators of the antioxidant response of the cell. Here we show that in cancer cells NRF2 targets are selectively upregulated... 
Oxidative stress | Mutant p53 | NRF2 | Cancer
Journal Article
Journal of Toxicologic Pathology, ISSN 0914-9198, 2013, Volume 26, Issue 4, pp. 423 - 427
A nephroblastoma is a tumor arising from metanephric blastema occurring in childhood. Among laboratory rodents, nephroblastoma has been frequently reported in... 
kidney | mouse | pathogenesis | nephroblastoma | immunohistochemistry | Immunohistochemistry | Mouse | Nephroblastoma | Pathogenesis | Kidney | PROTEIN | RATS | PATHOLOGY | WILMS-TUMOR | FEATURES | AGGRESSIVENESS | TOXICOLOGY | INDEX | Case Report
Journal Article
by Tan, Meng How and Li, Qin and Shanmugam, Raghuvaran and Piskol, Robert and Kohler, Jennefer and Young, Amy N and Liu, Kaiwen Ivy and Zhang, Rui and Ramaswami, Gokul and Ariyoshi, Kentaro and Gupte, Ankita and Keegan, Liam P and George, Cyril X and Ramu, Avinash and Huang, Ni and Pollina, Elizabeth A and Leeman, Dena S and Rustighi, Alessandra and Goh, Y. P. Sharon and Aguet, François and Ardlie, Kristin G and Cummings, Beryl B and Gelfand, Ellen T and Getz, Gad and Hadley, Kane and Handsaker, Robert E and Huang, Katherine H and Kashin, Seva and Karczewski, Konrad J and Lek, Monkol and Li, Xiao and MacArthur, Daniel G and Nedzel, Jared L and Nguyen, Duyen T and Noble, Michael S and Segrè, Ayellet V and Trowbridge, Casandra A and Tukiainen, Taru and Abell, Nathan S and Balliu, Brunilda and Barshir, Ruth and Basha, Omer and Battle, Alexis and Bogu, Gireesh K and Brown, Andrew and Brown, Christopher D and Castel, Stephane E and Chen, Lin S and Chiang, Colby and Conrad, Donald F and Cox, Nancy J and Damani, Farhan N and Davis, Joe R and Delaneau, Olivier and Dermitzakis, Emmanouil T and Engelhardt, Barbara E and Eskin, Eleazar and Ferreira, Pedro G and Frésard, Laure and Gamazon, Eric R and Garrido-Martín, Diego and Gewirtz, Ariel D.H and Gliner, Genna and Gloudemans, Michael J and Guigo, Roderic and Hall, Ira M and Han, Buhm and He, Yuan and Hormozdiari, Farhad and Howald, Cedric and Im, Hae Kyung and Jo, Brian and Kang, Eun Yong and Kim, Yungil and Kim-Hellmuth, Sarah and Lappalainen, Tuuli and Li, Gen and Li, Xin and Liu, Boxiang and Mangul, Serghei and McCarthy, Mark I and McDowell, Ian C and Mohammadi, Pejman and Monlong, Jean and Montgomery, Stephen B and Muñoz-Aguirre, Manuel and Ndungu, Anne W and Nicolae, Dan L and Nobel, Andrew B and Oliva, Meritxell and Ongen, Halit and Palowitch, John J and Panousis, Nikolaos and Papasaikas, Panagiotis and Park, Yoson and Parsana, Princy and Payne, Anthony J and Peterson, Christine B and Quan, Jie and Reverter, Ferran and ... and Gtex Consortium and GTEx Consortium and Leidos Biomedical—Project Management and NIH/NCI and Laboratory, Data Analysis &Coordinating Center (LDACC)—Analysis Working Group and Statistical Methods groups—Analysis Working Group and Biospecimen Collection Source Site—NDRI and NIH/NHGRI and Brain Bank Repository—University of Miami Brain Endowment Bank and Genome Browser Data Integration &Visualization—UCSC Genomics Institute, University of California Santa Cruz and NIH Common Fund and Enhancing GTEx (eGTEx) groups and NIH/NIMH and Genome Browser Data Integration &Visualization—EBI and Biospecimen Collection Source Site—RPCI and Biospecimen Core Resource—VARI and ELSI Study and NIH/NIDA
Nature, ISSN 0028-0836, 10/2017, Volume 550, Issue 7675, pp. 249 - 254
Journal Article