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Nature, ISSN 0028-0836, 03/2012, Volume 483, Issue 7388, pp. 222 - 226
Cognitive decline is a debilitating feature of most neurodegenerative diseases of the central nervous system, including Alzheimer's disease(1). The causes... 
REMOTE MEMORIES | LONG-TERM-MEMORY | CDK5 | CHROMATIN | PHOSPHORYLATION | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | CLINICAL-IMPLICATIONS | SYNAPTIC PLASTICITY | EXPRESSION | TRANSGENIC MICE | Memory Disorders - physiopathology | Alzheimer Disease - complications | Memory Disorders - genetics | Peptide Fragments - toxicity | Humans | Neuronal Plasticity - drug effects | Receptors, Glucocorticoid - metabolism | Hippocampus - drug effects | RNA Polymerase II - metabolism | Memory Disorders - complications | Promoter Regions, Genetic - drug effects | Promoter Regions, Genetic - genetics | Gene Knockdown Techniques | Brain - metabolism | Neuronal Plasticity - genetics | Epigenesis, Genetic - drug effects | Phosphorylation - drug effects | Disease Models, Animal | Hydrogen Peroxide - toxicity | Histone Deacetylase 2 - genetics | Alzheimer Disease - physiopathology | Amyloid beta-Peptides - toxicity | Brain - physiopathology | Neurodegenerative Diseases - genetics | Neurodegenerative Diseases - complications | Brain - drug effects | Gene Expression Regulation - drug effects | Hippocampus - metabolism | Acetylation - drug effects | Animals | Neurodegenerative Diseases - physiopathology | Histone Deacetylase 2 - deficiency | Mice | Histone Deacetylase 2 - metabolism | Histones - metabolism | Alzheimer Disease - genetics | Epigenetic inheritance | Complications and side effects | Causes of | Nervous system | Genetic aspects | Degeneration | Cognition disorders | Health aspects | Epigenetics | Genetics | RNA polymerase | Kinases | Neurodegeneration | Cyclin-dependent kinases
Journal Article
Journal Article
The EMBO Journal, ISSN 0261-4189, 07/2007, Volume 26, Issue 13, pp. 3169 - 3179
Journal Article
Journal Article
Neuron, ISSN 0896-6273, 03/2016, Volume 89, Issue 6, pp. 1208 - 1222
Trisomy 21, or Down syndrome (DS), is the most common genetic cause of developmental delay and intellectual disability. To gain insight into the underlying... 
brain development | white matter | genomics | neocortex | neurodevelopmental disorders | glia | gene expression | Genomics | Brain development | Gene expression | Glia | Neocortex | Neurodevelopmental disorders | White matter | TS65DN MOUSE MODEL | DEVELOPING CNS | WHITE-MATTER | CORTEX | IN-VIVO | GENE-EXPRESSION | LONG-TERM POTENTIATION | HIPPOCAMPAL-FORMATION | MATURATION | COGNITIVE-DEVELOPMENT | NEUROSCIENCES | Action Potentials - genetics | Humans | Child, Preschool | Gene Expression Regulation, Developmental - genetics | Infant | Male | Down Syndrome - physiopathology | Gene Expression Profiling | Brain - growth & development | White Matter - ultrastructure | Chromosomes, Human, Pair 17 - genetics | Myelin Sheath - metabolism | Brain - metabolism | Myelin Basic Protein - genetics | Young Adult | Cell Differentiation - genetics | Adult | Female | Cell Differentiation - physiology | Child | Infant, Newborn | Disease Models, Animal | Down Syndrome - pathology | Myelin Basic Protein - metabolism | Myelin Sheath - pathology | Neural Conduction - genetics | Mice, Transgenic | White Matter - pathology | Oligodendroglia - pathology | Animals | Trisomy - genetics | Adolescent | Down Syndrome - genetics | Brain - pathology | Mosaicism | Mice | Myelin Sheath - ultrastructure | Postmortem Changes | Brain | Medical colleges | Down syndrome | Analysis | Neurophysiology | Anopheles | Genes | Development and progression | Neuropathology | Principal components analysis | Genomes | Alzheimers disease | Laboratory animals | Chromosomes
Journal Article
Journal of Alzheimer's Disease, ISSN 1387-2877, 05/2018, Volume 63, Issue 4, pp. 1433 - 1443
The pathophysiology of Alzheimer’s disease (AD) includes signaling defects mediated by the transforming growth factor β—bone morphogenetic protein—growth and... 
Immunohistochemistry | Animal models | Neurons | Therapeutic applications | Memory | Neuropil | Kinases | Proteins | Fornix | Axons | Bone growth | Neurofibrillary tangles | Pyramidal cells | Rodents | Amyloidosis | Activin | Bone morphogenetic protein 9 | Alzheimer's disease | Hippocampus | ACVRL1 | CA1 | CA3 | hippocampus | immunohistochemistry | GAD67 | ALK1
Journal Article
Journal of Alzheimer's Disease, ISSN 1387-2877, 03/2019, Volume 68, Issue 1, pp. 357 - 365
Journal Article
The EMBO Journal, ISSN 0261-4189, 10/2017, Volume 36, Issue 19, pp. 2815 - 2828
Age‐associated memory decline is due to variable combinations of genetic and environmental risk factors. How these risk factors interact to drive disease onset... 
HDAC inhibitor | aging | Formin 2 | Alzheimer's disease | post‐traumatic stress disorder | post-traumatic stress disorder | MEMORY FORMATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | HIPPOCAMPAL | TRANSCRIPTION | DIFFERENTIAL GENE-EXPRESSION | CELL BIOLOGY | FEAR EXTINCTION | GERMAN VERSION | MOUSE MODEL | INHIBITORS | BRAIN | COGNITIVE DEFICITS | Dementia - psychology | Humans | Middle Aged | Risk Factors | Stress Disorders, Post-Traumatic - epidemiology | Male | Dementia - epidemiology | Dementia - genetics | Case-Control Studies | Stress Disorders, Post-Traumatic - complications | Mice, Knockout | Neuronal Plasticity - genetics | Phenotype | Animals | Aging - genetics | Aging - physiology | Age of Onset | Adult | Mice | Nuclear Proteins - genetics | Stress Disorders, Post-Traumatic - genetics | Microfilament Proteins - genetics | Memory - physiology | Histone deacetylase | Deregulation | Transcription | Mental disorders | Memory | Homeostasis | Posttraumatic stress disorder | Risk factors | Consolidation | Actin | Rodents | Post-traumatic stress disorder | Dementia disorders | Age | Neurodegenerative diseases | Health risks | Risk analysis | Gene expression | Environmental risk | Plasticity (synaptic) | Post traumatic stress disorder | Life span | Aberration | Alzheimers disease | Dementia | PTSD | Cognitive Disorders | Predisposition | Memory Impairment | Drug Therapy | Neurobiology | Aging | Genetics | Conditioned Emotional Responses | Animal Models | Neuroscience | Molecular Biology of Disease
Journal Article