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Gut, ISSN 0017-5749, 05/2012, Volume 61, Issue 5, pp. 673 - 684
ObjectiveGastric cancer is a major gastrointestinal malignancy for which targeted therapies are emerging as treatment options. This study sought to identify... 
GROWTH-FACTOR RECEPTOR | LUNG | AMPLIFICATION | COPY-NUMBER | ACQUIRED-RESISTANCE | TYROSINE KINASE INHIBITOR | GASTROENTEROLOGY & HEPATOLOGY | TRASTUZUMAB RESISTANCE | EXPRESSION | LINEAGE-SURVIVAL ONCOGENE | GENETIC ALTERATIONS | Stomach Neoplasms - genetics | Receptor, Epidermal Growth Factor - genetics | ras Proteins - genetics | Proto-Oncogene Proteins p21(ras) | Oligonucleotide Array Sequence Analysis | Receptor, ErbB-2 - genetics | Proportional Hazards Models | Proto-Oncogene Proteins - genetics | Antineoplastic Agents - therapeutic use | Genetic Markers | Stomach Neoplasms - drug therapy | Molecular Targeted Therapy | Proto-Oncogene Proteins c-met - genetics | Gene Amplification | Gene Deletion | Polymorphism, Single Nucleotide | Stomach Neoplasms - mortality | Receptor, Fibroblast Growth Factor, Type 2 - genetics | Development and progression | Drug targeting | Genetic aspects | Research | Demographic aspects | Stomach cancer | Proteins | Studies | Chemotherapy | Medical prognosis | Mortality | Clinical trials | Breast cancer | Mutation | Cancer therapies | Patients | Tumors | Index Medicus | Abridged Index Medicus | Stomach | gastroscopy | Barrett's metaplasia | gastric cancer | targeted therapies | gene mutation | gastric pre-cancer | chemotherapy | oesophageal cancer | Barrett's carcinoma | receptor tyrosine kinases | cell signalling | gastrointestinal cancer | cancer | colorectal cancer screening | gastrointesinal endoscopy | gastric carcinoma | 1506 | molecular pathology | copy number alterations | gene expression
Journal Article
Nature Genetics, ISSN 1061-4036, 05/2012, Volume 44, Issue 5, pp. 570 - 574
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 2897 - 18
Journal Article
Gut, ISSN 0017-5749, 05/2015, Volume 64, Issue 5, pp. 707 - 719
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 7820 - 12
Docetaxel and cabazitaxel are taxane chemotherapy treatments for metastatic castration-resistant prostate cancer (CRPC). However, therapeutic resistance... 
MITOXANTRONE | CABAZITAXEL | MULTIDISCIPLINARY SCIENCES | DOWN-REGULATION | TUMOR-SUPPRESSOR | CELL-PROLIFERATION | OPEN-LABEL | OVARIAN-CANCER | MIR-217 | DOCETAXEL | EXPRESSION | Cell proliferation | MiRNA | Genomes | Metastases | Gene silencing | Signal transduction | Chemotherapy | Castration | MicroRNAs | Cell lines | Taxanes | Prostate cancer | Prostate | Apoptosis
Journal Article
Endocrine-related cancer, ISSN 1351-0088, 02/2019, Volume 26, Issue 2, pp. 251 - 264
The role of androgen receptor (AR) in endocrine-resistant breast cancer is controversial and clinical trials targeting AR with an AR antagonist (e.g.,... 
breast cancer | endocrine resistance | PDX | enzalutamide | PROSTATE | TRANSCRIPTION | CELL-PROLIFERATION | ER-ALPHA | ANDROGEN RECEPTOR EXPRESSION | THERAPY | ANTIANDROGEN | ONCOLOGY | GROWTH | ENDOCRINOLOGY & METABOLISM | INHIBITOR | ACETATE
Journal Article
Oncogene, ISSN 0950-9232, 08/2018, Volume 37, Issue 33, pp. 4518 - 4533
MASTL kinase is essential for correct progression through mitosis, with loss of MASTL causing chromosome segregation errors, mitotic collapse and failure of... 
PHOSPHORYLATION | HUMAN-CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | DNA-DAMAGE | PHENOTYPE | PROTEIN PHOSPHATASE 2A | GREATWALL KINASE | CELL BIOLOGY | EPITHELIAL-CELLS | ONCOLOGY | SUBSTRATE | DEPHOSPHORYLATION | GENETICS & HEREDITY | MITOTIC EXIT | Cell Proliferation - genetics | Microtubule-Associated Proteins - genetics | Humans | Protein-Serine-Threonine Kinases - genetics | Signal Transduction - genetics | Epithelial-Mesenchymal Transition - genetics | Mice, SCID | Proto-Oncogene Proteins c-akt - genetics | Phosphatidylinositol 3-Kinases - genetics | Chromosomal Instability - genetics | Animals | Breast Neoplasms - genetics | MAP Kinase Signaling System - genetics | TOR Serine-Threonine Kinases - genetics | Breast Neoplasms - pathology | Cell Line, Tumor | DNA Damage - genetics | Cell Cycle Checkpoints - genetics | Female | Mice, Inbred NOD | Mice | Actin Cytoskeleton - genetics | Care and treatment | Chromosome abnormalities | Development and progression | Breast cancer | Genetic aspects | Gene expression | Health aspects | Phosphotransferases | Risk factors | TOR protein | Cell proliferation | Contact inhibition | Cell culture | Mesenchyme | Interphase | Mitosis | Lung cancer | DNA damage | Desmosomes | AKT protein | Metastasis | Cytokinesis | Kinases | 1-Phosphatidylinositol 3-kinase | Metastases | Signal transduction | Actin | Micronuclei | Cytoskeleton | Mathematical models | Cell migration | Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 03/2016, Volume 6, Issue 1, pp. 22371 - 22371
Journal Article
Journal Article