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BBA - Reviews on Cancer, ISSN 0304-419X, 09/2007, Volume 1776, Issue 1, pp. 86 - 107
Journal Article
Nature Communications, ISSN 2041-1723, 04/2015, Volume 6, Issue 1, pp. 6683 - 6683
Transcriptional reprogramming of proliferative melanoma cells into a phenotypically distinct invasive cell subpopulation is a critical event at the origin of... 
TRANSCRIPTION FACTOR-BINDING | ENCYCLOPEDIA | R/BIOCONDUCTOR PACKAGE | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | PHENOTYPE | ENHANCER | MITF | DIFFERENTIATION | CHROMOSOME CONFORMATION CAPTURE | TAZ | Chromatin - metabolism | RNA, Small Interfering - genetics | Transcription Factor AP-1 - genetics | Humans | Gene Expression Regulation, Neoplastic | Transcriptome | Gene Regulatory Networks | Transcription Factor AP-1 - metabolism | DNA-Binding Proteins - metabolism | DNA Methylation | Protein Isoforms - metabolism | Cell Transformation, Neoplastic - genetics | Melanoma - genetics | Transcription, Genetic | Antineoplastic Agents - pharmacology | Nuclear Proteins - genetics | Cellular Reprogramming - genetics | Chromatin - chemistry | Melanoma - metabolism | DNA-Binding Proteins - antagonists & inhibitors | Microphthalmia-Associated Transcription Factor - metabolism | Signal Transduction | Neoplasm Invasiveness | SOXE Transcription Factors - metabolism | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Melanoma - pathology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Cell Transformation, Neoplastic - metabolism | Transcription Factors - metabolism | Histones - genetics | Melanoma - drug therapy | Nuclear Proteins - antagonists & inhibitors | Cell Line, Tumor | Protein Kinase Inhibitors - pharmacology | Histones - metabolism | Cell Transformation, Neoplastic - pathology | Microphthalmia-Associated Transcription Factor - genetics | Protein Isoforms - genetics | RNA, Small Interfering - metabolism | SOXE Transcription Factors - genetics | Index Medicus
Journal Article
Nature Medicine, ISSN 1078-8956, 08/2012, Volume 18, Issue 8, pp. 1239 - 1247
The inactivation of the p53 tumor suppressor pathway, which often occurs through mutations in TP53 (encoding tumor protein 53) is a common step in human... 
MEDICINE, RESEARCH & EXPERIMENTAL | N-RAS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TUMOR-SUPPRESSOR ACTIVITY | STAPLED P53 | BRAF | MALIGNANT-MELANOMA | P53 PATHWAY | CELL-DEATH | CELL BIOLOGY | BREAST-CANCER | METASTATIC MELANOMA | IN-VIVO | Up-Regulation | Proto-Oncogene Proteins c-mdm2 - genetics | Tumor Suppressor Protein p53 - antagonists & inhibitors | Apoptosis - drug effects | Humans | Neoplasm Proteins - physiology | Gene Expression Regulation, Neoplastic | Recombinant Fusion Proteins - physiology | Skin Neoplasms - chemistry | Male | Melanocytes - metabolism | Neoplasm Proteins - antagonists & inhibitors | Cell Line, Tumor - transplantation | Proto-Oncogene Proteins - biosynthesis | Tumor Suppressor Protein p53 - physiology | Cell-Penetrating Peptides - pharmacology | Nuclear Proteins - biosynthesis | Female | Antineoplastic Agents - pharmacology | Neoplasm Proteins - genetics | Nuclear Proteins - genetics | Cell Line, Tumor - metabolism | Melanoma - chemistry | Proto-Oncogene Proteins c-mdm2 - biosynthesis | Proto-Oncogene Proteins - antagonists & inhibitors | Tumor Stem Cell Assay | Membrane Proteins - genetics | Neoplasm Proteins - biosynthesis | Melanoma, Experimental - etiology | Mice, Inbred C57BL | Mice, Transgenic | Proto-Oncogene Proteins - genetics | Melanoma - pathology | Melanoma - secondary | Proto-Oncogene Proteins B-raf - antagonists & inhibitors | Xenograft Model Antitumor Assays | Animals | Melanoma, Experimental - genetics | GTP Phosphohydrolases - genetics | Keratinocytes - metabolism | Mice, Nude | Nuclear Proteins - antagonists & inhibitors | Proto-Oncogene Proteins - physiology | Signal Transduction - physiology | Mice | Nuclear Proteins - physiology | Drug Resistance, Neoplasm - physiology | Drug Resistance, Neoplasm - drug effects | Care and treatment | Gene mutations | Melanoma | Diagnosis | Research | Gene expression | Identification and classification | Skin cancer | Proteins | Cell growth | Mutation | Cell cycle | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 06/2018, Volume 558, Issue 7711, pp. 605 - 609
Reprogramming of mRNA translation has a key role in cancer development and drug resistance(1). However, the molecular mechanisms that are involved in this... 
CELLS | MELANOMA | INHIBITION | TRANSFER-RNAS | MULTIDISCIPLINARY SCIENCES | KINASE | IMPROVED SURVIVAL | MUTATIONS | HUMAN CANCER | VEMURAFENIB | RIBOSOME | Phosphorylation | Humans | Melanoma, Experimental - drug therapy | Male | Uridine - genetics | RNA, Messenger - metabolism | Vemurafenib - therapeutic use | Melanoma - genetics | RNA, Transfer - genetics | Carrier Proteins - chemistry | Female | Codon - genetics | Codon - drug effects | RNA, Transfer - chemistry | Uridine - chemistry | Signal Transduction | RNA, Transfer - metabolism | RNA, Messenger - genetics | Melanoma, Experimental - pathology | Melanoma - pathology | Mice, SCID | Vemurafenib - pharmacology | Zebrafish - genetics | Proto-Oncogene Proteins B-raf - antagonists & inhibitors | Mechanistic Target of Rapamycin Complex 2 - metabolism | Drug Resistance, Neoplasm - genetics | Animals | Carrier Proteins - metabolism | Melanoma, Experimental - genetics | Proto-Oncogene Proteins B-raf - genetics | Melanoma - drug therapy | Cell Line, Tumor | Mice, Inbred NOD | Protein Biosynthesis - drug effects | Uridine - metabolism | Mice | Drug Resistance, Neoplasm - drug effects | Care and treatment | Codon | Oncology, Experimental | Melanoma | Research | Genetic translation | Cancer | Enzymes | Physiological aspects | Development and progression | Protein biosynthesis | Transfer RNA | Short term | Therapy | Transformation | Genomes | mRNA | Kinases | Proteins | Signal transduction | Translation | Cell survival | tRNA | MAP kinase | Pharmacology | Decoding | Metabolism | Gene expression | Chemical compounds | Survival | Molecular chains | 1-Phosphatidylinositol 3-kinase | Signaling | Molecular modelling | Protein synthesis | Uridine | Glycolysis | Mutation | Index Medicus
Journal Article
Autophagy, ISSN 1554-8627, 12/2011, Volume 7, Issue 12, pp. 1472 - 1489
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2016, Volume 126, Issue 1, pp. 68 - 84
Journal Article
Journal of Cellular and Molecular Medicine, ISSN 1582-1838, 06/2011, Volume 15, Issue 6, pp. 1402 - 1414
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2012, Volume 7, Issue 10, pp. e37807 - e37807
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 02/2014, Volume 67, pp. 292 - 303
Emerging evidence indicates that oxidative stress instigates the formation of ubiquitin (Ub) aggregates, substrates of autophagy, through a process requiring... 
PDT | Free radicals | Proteotoxicity | ROS | NBR1 | ER stress | Photodynamic stress | p62 | Autophagy
Journal Article
2003, Multilingual matters (Series), ISBN 9781853596254, viii, 233
Book
FREE RADICAL BIOLOGY AND MEDICINE, ISSN 0891-5849, 02/2014, Volume 67, pp. 292 - 303
Journal Article