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Nature, ISSN 0028-0836, 12/2012, Volume 492, Issue 7429, pp. 382 - 386
Journal Article
Nature, ISSN 0028-0836, 12/2010, Volume 468, Issue 7327, pp. 1119 - 1123
Interaction of pathogens with cells of the immune system results in activation of inflammatory gene expression. This response, although vital for immune... 
MACHINES | BINDING MODULES | CHROMATIN | MULTIDISCIPLINARY SCIENCES | COMPLEXES | GENE-EXPRESSION | BROMODOMAIN PROTEIN BRD4 | TRANSCRIPTIONAL ELONGATION | P-TEFB | Sepsis - prevention & control | Salmonella Infections - immunology | Shock, Septic - prevention & control | Salmonella Infections - physiopathology | Salmonella typhimurium | Heterocyclic Compounds, 4 or More Rings - pharmacology | Sepsis - drug therapy | Heterocyclic Compounds, 4 or More Rings - therapeutic use | Inflammation - drug therapy | Shock, Septic - drug therapy | Anti-Inflammatory Agents - therapeutic use | Hydroxamic Acids - pharmacology | Protein-Serine-Threonine Kinases - metabolism | Protein Structure, Tertiary | Salmonella Infections - drug therapy | Genome-Wide Association Study | Epigenomics | Anti-Inflammatory Agents - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Heterocyclic Compounds, 4 or More Rings - chemistry | Kaplan-Meier Estimate | Models, Molecular | Salmonella Infections - prevention & control | Benzodiazepines | Gene Expression Regulation - drug effects | Acetylation - drug effects | Animals | Anti-Inflammatory Agents - chemistry | Lipopolysaccharides - pharmacology | Histone Deacetylase Inhibitors - pharmacology | Macrophages - drug effects | Inflammation - prevention & control | Mice | Proteins | Pathology | Eukaryotes | Chromatin | Immunology | Pharmacology | Gene expression
Journal Article
Nature, ISSN 0028-0836, 10/2010, Volume 467, Issue 7317, pp. 849 - 853
Gastrointestinal stromal tumour (GIST) is the most common human sarcoma and is primarily defined by activating mutations in the KIT or PDGFRA receptor tyrosine... 
PROFILES | INHIBITION | MULTIDISCIPLINARY SCIENCES | PROSTATE-CANCER | MOUSE MODEL | GENE-EXPRESSION | CAJAL | NETWORKS | MUTATIONS | ENHANCERS | INTERSTITIAL-CELLS | NIH 3T3 Cells | Humans | Gene Expression Profiling | Oncogenes - physiology | Proto-Oncogene Proteins c-kit - metabolism | DNA-Binding Proteins - metabolism | Interstitial Cells of Cajal - metabolism | Gastrointestinal Stromal Tumors - pathology | Biomarkers, Tumor - metabolism | Proto-Oncogene Proteins c-kit - genetics | Protein Stability | Binding Sites | Gene Expression Regulation, Neoplastic - genetics | Cell Survival - drug effects | Oncogenes - genetics | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction | Mutant Proteins - genetics | Interstitial Cells of Cajal - pathology | Mutant Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Pyrimidines - pharmacology | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Imatinib Mesylate | Piperazines - pharmacology | Disease Progression | Transcription Factors - metabolism | Cell Lineage | Animals | Cell Transformation, Neoplastic | Cell Line, Tumor | Gastrointestinal Stromal Tumors - metabolism | Biomarkers, Tumor - genetics | Enhancer Elements, Genetic - genetics | Mice | Benzamides | Mutation | Proteins | Gene expression | Rodents | Apoptosis | Tumors
Journal Article
Journal Article
Journal Article