2003, Progress in experimental cardiology, ISBN 9781402073113, Volume 8, xx, 492
Book
2003, Progress in experimental cardiology, ISBN 9781402071959, Volume 6, xvi, 541
Book
1984, ISBN 0849359961, 2 v.
Book
Circulation Research, ISSN 0009-7330, 08/2018, Volume 123, Issue 4, pp. 409 - 411
Sudden cardiac death (SCD) is a leading cause of mortality under various stressful and pathological conditions. In view of the overactivation of the...
ADRENOLUTIN | CARDIAC & CARDIOVASCULAR SYSTEMS | sympathetic nervous system | catecholamine cardiotoxicity | cardiovascular abnormalities | FAILURE | TRANSPORT | PRODUCT | heart diseases | PERIPHERAL VASCULAR DISEASE | RAT-HEART | HEMATOLOGY | CATECHOLAMINE-INDUCED CARDIOMYOPATHY | ADRENOCHROME | oxidative stress
ADRENOLUTIN | CARDIAC & CARDIOVASCULAR SYSTEMS | sympathetic nervous system | catecholamine cardiotoxicity | cardiovascular abnormalities | FAILURE | TRANSPORT | PRODUCT | heart diseases | PERIPHERAL VASCULAR DISEASE | RAT-HEART | HEMATOLOGY | CATECHOLAMINE-INDUCED CARDIOMYOPATHY | ADRENOCHROME | oxidative stress
Journal Article
2017, ISBN 9811025134
Using a multidisciplinary approach, this book describes the biochemical mechanisms associated with dysregulation of proteases and the resulting...
Medicine | Human physiology | Cancer research | Pharmaceutical technology | Molecular biology
Medicine | Human physiology | Cancer research | Pharmaceutical technology | Molecular biology
Web Resource
2017, ISBN 9811025134
Using a multidisciplinary approach, this book describes the biochemical mechanisms associated with dysregulation of proteases and the resulting...
Medicine | Human physiology | Cancer research | Pharmaceutical technology | Molecular biology
Medicine | Human physiology | Cancer research | Pharmaceutical technology | Molecular biology
Web Resource
1988, ISBN 0849368871, 245
Book
Book
1998, Progress in experimental cardiology, ISBN 079238105X, Volume 1., xliv, 575
Over the past three decades, impressive progress in the field of pathogenesis, prevention and therapy of ischemic heart disease has resulted in a marked...
Myocardial infarction | Coronary heart disease | Reperfusion injury | Cardiology
Myocardial infarction | Coronary heart disease | Reperfusion injury | Cardiology
Book
2003, Progress in experimental cardiology, ISBN 1402074514, Volume 9, xvi, 595
Book
2003, Progress in experimental cardiology, ISBN 9781402071775, Volume 5, xvi, 545
Book
2016, 1st ed. 2016., Advances in Biochemistry in Health and Disease, ISBN 3319247786, Volume 14
The biological membranes of cellular organization enfold an important group of membrane proteins called the ATPases, which are not only versatile in...
Proteins | Molecular structure | Life sciences | Cell membranes | Spectra | Molecular biology | Atomic structure
Proteins | Molecular structure | Life sciences | Cell membranes | Spectra | Molecular biology | Atomic structure
Web Resource
2016, 1st ed. 2016., Advances in Biochemistry in Health and Disease, ISBN 9783319247502, Volume 15
Na+-K+ ATPase or Na-pump ATPase, a member of “P”-type ATPase superfamily, is characterized by association of multiple isoforms mainly of it’s α- and β-...
Proteins | Molecular structure | Life sciences | Cell membranes | Spectra | Molecular biology | Atomic structure
Proteins | Molecular structure | Life sciences | Cell membranes | Spectra | Molecular biology | Atomic structure
Web Resource
2017, ISBN 9789811031618
This book bridges the gap between fundamental research and biomedical and pharmacological applications on proteases. It represents a comprehensive overview of...
Medicine | Human physiology | Cancer research | Pharmaceutical technology | Molecular biology
Medicine | Human physiology | Cancer research | Pharmaceutical technology | Molecular biology
Web Resource
2014, Advances in biochemistry in health and disease, ISBN 1461493161, Volume 9.
Web Resource
2014, Advances in Biochemistry in Health and Disease, ISBN 1461490987, Volume 8.
Web Resource
17.
Attenuation of diabetes-induced cardiac and subcellular defects by sulphur-containing amino acids
Current Medicinal Chemistry, ISSN 0929-8673, 01/2018, Volume 25, Issue 3, pp. 336 - 345
Background: Patients with diabetes mellitus have an increased risk of mortality due to cardiovascular complications. Supplementation with specific...
Diabetic cardiomyopathy | Taurine | Cysteine | Preventive nutrition | Diabetes mellitus | Sulphur-containing amino acids | ANTIOXIDANT ACTIVITY | RENIN-ANGIOTENSIN | L-CYSTEINE SUPPLEMENTATION | CHEMISTRY, MEDICINAL | PLATELET-AGGREGATION | TAURINE SUPPLEMENTATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | N-ACETYLCYSTEINE | CALCIUM PARADOX | NA+-K+-ATPASE | INTRACELLULAR CA2 | II-INDUCED HYPERTROPHY | PHARMACOLOGY & PHARMACY | Animals | Sulfur - pharmacology | Diabetic Cardiomyopathies - drug therapy | Diabetes Mellitus - physiopathology | Humans | Diabetes Mellitus - metabolism | Diabetic Cardiomyopathies - complications | Antioxidants - pharmacology | Oxidative Stress - drug effects | Diabetic Cardiomyopathies - physiopathology | Amino Acids - pharmacology | Heart | Oxidative stress | Complications | Cardiomyopathy | Pathogenesis | Abnormalities | Health risks | Amino acids | Insulin | Antioxidants | Attenuation | Diabetes | Supplementation | Cardiovascular diseases | Sulfur | Heart diseases | Metabolic disorders
Diabetic cardiomyopathy | Taurine | Cysteine | Preventive nutrition | Diabetes mellitus | Sulphur-containing amino acids | ANTIOXIDANT ACTIVITY | RENIN-ANGIOTENSIN | L-CYSTEINE SUPPLEMENTATION | CHEMISTRY, MEDICINAL | PLATELET-AGGREGATION | TAURINE SUPPLEMENTATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | N-ACETYLCYSTEINE | CALCIUM PARADOX | NA+-K+-ATPASE | INTRACELLULAR CA2 | II-INDUCED HYPERTROPHY | PHARMACOLOGY & PHARMACY | Animals | Sulfur - pharmacology | Diabetic Cardiomyopathies - drug therapy | Diabetes Mellitus - physiopathology | Humans | Diabetes Mellitus - metabolism | Diabetic Cardiomyopathies - complications | Antioxidants - pharmacology | Oxidative Stress - drug effects | Diabetic Cardiomyopathies - physiopathology | Amino Acids - pharmacology | Heart | Oxidative stress | Complications | Cardiomyopathy | Pathogenesis | Abnormalities | Health risks | Amino acids | Insulin | Antioxidants | Attenuation | Diabetes | Supplementation | Cardiovascular diseases | Sulfur | Heart diseases | Metabolic disorders
Journal Article
11/2017, 1st ed. 2017, ISBN 9789811061400, 668
This book provides a comprehensive overview of the multifaceted field of protease in the cellular environment and focuses on the recently elucidated functions...
Proteolytic enzymes-Pathophysiology | Proteolytic enzymes | Pharmaceutical Sciences/Technology | Cancer Research | Molecular Medicine | Biomedicine | Human Physiology
Proteolytic enzymes-Pathophysiology | Proteolytic enzymes | Pharmaceutical Sciences/Technology | Cancer Research | Molecular Medicine | Biomedicine | Human Physiology
eBook
Heart Failure Reviews, ISSN 1382-4147, 5/2012, Volume 17, Issue 3, pp. 395 - 409
It is believed that cardiac remodeling due to geometric and structural changes is a major mechanism for the progression of heart failure in different...
Heart failure | Calpains and cathepsins | Matrix metalloproteinases | Medicine & Public Health | Dilated cardiomyopathy | Diabetic cardiomyopathy | Cardiac caspases and apoptosis | Cardiology | INDUCTION/ACTIVATION SYSTEM EXISTS | CARDIAC & CARDIOVASCULAR SYSTEMS | CASPASE INHIBITION | MYOCYTE CELL-DEATH | NA+-K+-ATPASE | HYPERGLYCEMIA-INDUCED APOPTOSIS | ACUTE MYOCARDIAL-INFARCTION | TISSUE INHIBITOR | MATRIX-METALLOPROTEINASE ACTIVITY | LEFT-VENTRICULAR FUNCTION | OXIDATIVE STRESS INJURY | Diabetic Cardiomyopathies - metabolism | Humans | Heart Failure - physiopathology | Heart Failure - metabolism | Myocardial Infarction - metabolism | Ventricular Remodeling - physiology | Cardiomyopathy, Dilated - metabolism | Disease Progression | Hypertension - metabolism | Myocytes, Cardiac - metabolism | Apoptosis - physiology | Matrix Metalloproteinases - metabolism | Peptide Hydrolases - metabolism | Proteins | Hypertension | Enzymes | Proteases | Physiological aspects | Cathepsins | Development and progression | Calpain | Heart attack
Heart failure | Calpains and cathepsins | Matrix metalloproteinases | Medicine & Public Health | Dilated cardiomyopathy | Diabetic cardiomyopathy | Cardiac caspases and apoptosis | Cardiology | INDUCTION/ACTIVATION SYSTEM EXISTS | CARDIAC & CARDIOVASCULAR SYSTEMS | CASPASE INHIBITION | MYOCYTE CELL-DEATH | NA+-K+-ATPASE | HYPERGLYCEMIA-INDUCED APOPTOSIS | ACUTE MYOCARDIAL-INFARCTION | TISSUE INHIBITOR | MATRIX-METALLOPROTEINASE ACTIVITY | LEFT-VENTRICULAR FUNCTION | OXIDATIVE STRESS INJURY | Diabetic Cardiomyopathies - metabolism | Humans | Heart Failure - physiopathology | Heart Failure - metabolism | Myocardial Infarction - metabolism | Ventricular Remodeling - physiology | Cardiomyopathy, Dilated - metabolism | Disease Progression | Hypertension - metabolism | Myocytes, Cardiac - metabolism | Apoptosis - physiology | Matrix Metalloproteinases - metabolism | Peptide Hydrolases - metabolism | Proteins | Hypertension | Enzymes | Proteases | Physiological aspects | Cathepsins | Development and progression | Calpain | Heart attack
Journal Article
Canadian Journal of Physiology and Pharmacology, ISSN 0008-4212, 06/2019, Volume 97, Issue 6, p. 570
Journal Article
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