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Circulation Research, ISSN 0009-7330, 04/2019, Volume 124, Issue 8, pp. 1160 - 1162
Diabetic cardiomyopathy was initially described as a human pathophysiological condition in which heart failure occurred in the absence of coronary artery... 
coronary artery disease | HEART | heart failure | CARDIAC & CARDIOVASCULAR SYSTEMS | calcium | mitochondria | PERIPHERAL VASCULAR DISEASE | diabetes mellitus | GENE-THERAPY | MECHANISMS | diabetic cardiomyopathy | HEMATOLOGY
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 2015, Volume 309, Issue 9, pp. C593 - C599
Cardiovascular disease is the primary cause of morbidity and mortality in diabetes, and endothelial dysfunction is commonly seen in these patients. Increased... 
Endothelium-dependent hyperpolarization | Vascular rarefaction | GCA | Endothelial cell dysfunction | Diabetic vascular complications | N-ACETYLGLUCOSAMINE | diabetic vascular complications | ACTIVATION | PHYSIOLOGY | PROTEIN | ISOFORMS | GLCNACYLATION | PHOSPHORYLATION | NITRIC-OXIDE SYNTHASE | vascular rarefaction | endothelial cell dysfunction | CELL BIOLOGY | endothelium-dependent hyperpolarization | ROLES | Diabetes Mellitus, Experimental - enzymology | Coronary Vessels - physiopathology | Histone Acetyltransferases - biosynthesis | Humans | Antigens, Neoplasm - biosynthesis | Diabetes Mellitus, Experimental - genetics | Histone Acetyltransferases - genetics | Endothelium, Vascular - drug effects | Male | Coronary Artery Disease - enzymology | Endothelium, Vascular - enzymology | Vasodilation | Diabetic Angiopathies - physiopathology | Coronary Artery Disease - physiopathology | Diabetic Angiopathies - enzymology | beta-N-Acetylhexosaminidases - genetics | Diabetic Angiopathies - genetics | Diabetes Mellitus, Experimental - physiopathology | Diabetes Mellitus, Type 1 - enzymology | Antigens, Neoplasm - genetics | Coronary Vessels - drug effects | Coronary Vessels - enzymology | Signal Transduction | Diabetes Mellitus, Type 1 - physiopathology | Cells, Cultured | Endothelium, Vascular - physiopathology | Enzyme Inhibitors - pharmacology | Diabetes Mellitus, Type 1 - genetics | Enzyme Induction | Mice, Transgenic | beta-N-Acetylhexosaminidases - antagonists & inhibitors | Glycosylation | beta-N-Acetylhexosaminidases - biosynthesis | N-Acetylglucosaminyltransferases - metabolism | Connexins - metabolism | Animals | Histone Acetyltransferases - antagonists & inhibitors | Coronary Artery Disease - genetics | Hyaluronoglucosaminidase - antagonists & inhibitors | Protein Processing, Post-Translational | Hyaluronoglucosaminidase - biosynthesis | Endothelial Cells - enzymology | Hyaluronoglucosaminidase - genetics | Neovascularization, Physiologic | Endothelial Cells - drug effects | Complications and side effects | Care and treatment | Usage | Type 1 diabetes | Genetic engineering | Research | Cardiovascular diseases | Health aspects | Risk factors | Endothelium | Cardiovascular disease | Protein expression | Diabetes | Rodents | Coronary vessels | Cells | Index Medicus
Journal Article
Diabetologia, ISSN 0012-186X, 8/2010, Volume 53, Issue 8, pp. 1783 - 1794
Mitochondria frequently change their shapes by fusion and fission and these morphological dynamics play important roles in mitochondrial function and... 
O 2 | Medicine & Public Health | Human Physiology | OPA1 | Type 1 diabetes | Mitochondrial dynamics | Metabolic Diseases | Internal Medicine | Fusion | Fission | DRP1 | Endothelium | APOPTOSIS | OXIDATIVE STRESS | MAMMALIAN-CELLS | O-2(-) | DOMINANT OPTIC ATROPHY | ENDOCRINOLOGY & METABOLISM | HIGH GLUCOSE | HEME OXYGENASE-1 | EXPRESSION | GTPASE | DYNAMIN-RELATED PROTEIN | Oxidative Stress - physiology | Diabetes Mellitus, Type 1 - metabolism | Endothelium, Vascular - drug effects | Spin Labels | Isoprostanes - metabolism | Cyclic N-Oxides - pharmacology | Myocardium - metabolism | Superoxides - metabolism | Membrane Fusion - drug effects | Endothelial Cells - metabolism | Cells, Cultured | Diabetes Mellitus, Type 1 - pathology | Myocardium - pathology | Glucose - pharmacology | Mitochondria - metabolism | Antioxidants - pharmacology | Mitochondria - drug effects | Mitochondria - pathology | Blotting, Western | Dinoprostone - metabolism | Animals | Endothelium, Vascular - metabolism | Dinoprostone - analogs & derivatives | Endothelium, Vascular - pathology | Mice | Oxidative Stress - drug effects | Endothelial Cells - pathology | Membrane Fusion - physiology | Endothelial Cells - drug effects | Cell research | Hyperglycemia | Cell death | Utrophin | Physiological aspects | Mitochondrial DNA | Superoxide | Ophthalmology | Index Medicus | O2
Journal Article
Journal Article
Journal Article
Journal Article
American Journal of Physiology - Cell Physiology, ISSN 0363-6143, 02/2014, Volume 306, Issue 3, pp. C221 - C229
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 04/2004, Volume 13, Issue 7, pp. 703 - 714
Journal Article
Circulation Research, ISSN 0009-7330, 05/2005, Volume 96, Issue 9, pp. 1006 - 1013
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 11/2003, Volume 278, Issue 45, pp. 44230 - 44237
Diabetic cardiomyopathy is characterized by impaired cardiac contractility leading to poor myocardial performance. We investigated the role that the hexosamine... 
HEART | CONTRACTILITY | CA2 | INSULIN-RESISTANCE | BIOCHEMISTRY & MOLECULAR BIOLOGY | CARDIAC MYOCYTES | GENE-EXPRESSION | GLYCOSYLATION | PROTEINS | MUSCLE-CELLS | LINKED N-ACETYLGLUCOSAMINE | Histone Acetyltransferases | Multienzyme Complexes | Calcium - metabolism | Uridine Diphosphate - analysis | beta-N-Acetylhexosaminidases | N-Acetylglucosaminyltransferases - genetics | RNA, Messenger - analysis | DNA-Binding Proteins - analysis | Calcium-Transporting ATPases - genetics | Acetylglucosaminidase - genetics | Cardiomyopathies - etiology | Hexosamines - metabolism | Promoter Regions, Genetic - genetics | Glucosamine - metabolism | Sp1 Transcription Factor - analysis | Cell Nucleus - metabolism | Transfection | Acetylglucosaminidase - metabolism | Myocardium - metabolism | Adenoviridae - genetics | Calcium-Transporting ATPases - analysis | Diabetes Mellitus, Experimental - complications | Glucosamine - pharmacology | Indoles | Diabetes Mellitus, Experimental - metabolism | MEF2 Transcription Factors | Acylation | Myocardium - ultrastructure | Animals, Newborn | Gene Expression | Cells, Cultured | Rats | Glucose - pharmacology | Glycosylation | Myogenic Regulatory Factors | Sarcoplasmic Reticulum Calcium-Transporting ATPases | N-Acetylglucosaminyltransferases - metabolism | Hyperglycemia - metabolism | Animals | Cardiomyopathies - metabolism | Transcription Factors - analysis | Genetic Vectors | Fluorescent Dyes | Index Medicus
Journal Article
American Journal of Physiology - Regulatory, Integrative and Comparative Physiology, ISSN 0363-6119, 06/2011, Volume 300, Issue 6, pp. 1296 - 1302
Mitochondria are crucial organelles in cell life serving as a source of energy production and as regulators of Ca2+ homeostasis, apoptosis, and development.... 
Hyperglycemia | Mitochondrial fission and fusion | Membrane potential | Glcnacase | Mitochondrial dynamics | mitochondrial fission and fusion | PHYSIOLOGY | FUSION | PROTEIN | INNER MEMBRANE | DIABETIC CARDIOMYOPATHY | MAMMALIAN-CELLS | mitochondrial dynamics | hyperglycemia |