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Blood, ISSN 0006-4971, 12/2016, Volume 128, Issue 22, pp. 1569 - 1569
Abstract Class III receptor tyrosine kinases (RTK), which include c-fms, c-kit, FMS-like tyrosine kinase receptor-3(FLT3) and platelet-derived growth factor... 
Journal Article
Blood, ISSN 0006-4971, 11/2016, Volume 128, Issue 18, pp. 2266 - 2270
Journal Article
Blood, ISSN 0006-4971, 11/2016, Volume 128, Issue 18, pp. 2266 - 2270
Journal Article
Journal of Molecular Medicine, ISSN 0946-2716, 5/2016, Volume 94, Issue 5, pp. 597 - 608
Journal Article
Blood, ISSN 0006-4971, 11/2018, Volume 132, Issue Supplement 1, pp. 1028 - 1028
Abstract Obstructive sleep apnea (OSA), characterized by intermittent hypoxia, causes cardiovascular, metabolic, neurocognitive and cancer complications.... 
Journal Article
HUMAN MUTATION, ISSN 1059-7794, 11/2016, Volume 37, Issue 11, pp. 1153 - 1156
We report an infant with sickle cell disease phenotype by biochemical analysis whose -globin gene (HBB) sequencing showed sickle cell mutation (HBBS)... 
LOCUS-CONTROL REGION | ELEMENTS | ACTIVATION | regulation | globin genes | HBB duplication | TRANSCRIPTION | GENETICS & HEREDITY | LCR | sickle cell disease | DELETION | Nucleases | Corporate sponsorship | Sickle cell anemia | Analysis | Genes | Genetic research | Genetic engineering | Genetic transcription | DNA binding proteins | Enzymes | Genotype & phenotype | Mutation | Gene expression | Sickle cell disease
Journal Article
Stem Cells and Development, ISSN 1547-3287, 02/2010, Volume 19, Issue 2, pp. 181 - 193
Journal Article
Blood, ISSN 0006-4971, 12/2015, Volume 126, Issue 23, pp. 3335 - 3335
Abstract Gain-of-function mutations of erythropoietin receptor (EPOR) resulting from truncation of the cytoplasmic domain of EPOR are associated with primary... 
Journal Article
Journal Article
Blood, ISSN 0006-4971, 11/2012, Volume 120, Issue 21, pp. 991 - 991
Abstract Abstract 991 Introduction: Erythroid differentiation and iron metabolism are interconnected processes in order to produce sufficient numbers of... 
Journal Article
Blood, ISSN 0006-4971, 12/2014, Volume 124, Issue 21, pp. 4008 - 4008
Abstract Until now, disease-causing JAK2 mutations have been associated with clonal myeloproliferative neoplasms (acquired mutations) or with polyclonal... 
Journal Article
Human Mutation, ISSN 1059-7794, 10/2013, Volume 34, Issue 10, p. 1361
We describe the molecular etiology of [beta].sub.+-thalassemia that is caused by the insertion of the full-length transposable element LINE-1 (L1) into the... 
Methylation | Thalassemia
Journal Article
Blood, ISSN 0006-4971, 11/2018, Volume 132, Issue Supplement 1, pp. 3066 - 3066
Abstract The clinical consequences of the highly prevalent JAK2 V617F mutation in Ph-negative myeloproliferative neoplasms (MPNs) are well studied. However,... 
Journal Article
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