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American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 06/2012, Volume 185, Issue 11, pp. 1225 - 1234
Journal Article
Journal Article
Journal Article
Physiological Genomics, ISSN 1094-8341, 06/2006, Volume 26, Issue 1, pp. 68 - 75
In the lungs, high-pressure mechanical ventilation induces an inflammatory response similar to that observed in acute respiratory distress syndrome. To further... 
Acute respiratory distress syndrome | Overventilation | Gene expression analysis | APOPTOSIS | ACTIVATION | CYTOKINES | PHYSIOLOGY | gene expression analysis | MECHANICAL VENTILATION | MODEL | overventilation | RESPIRATORY-DISTRESS-SYNDROME | TRANSPLANTATION | CELL BIOLOGY | AMPHIREGULIN | EPITHELIAL-CELLS | GENETICS & HEREDITY | acute respiratory distress syndrome | LIPOPOLYSACCHARIDE | Immunohistochemistry | Receptors, Steroid - metabolism | Oligonucleotide Array Sequence Analysis | Glycoproteins - metabolism | Nuclear Receptor Subfamily 4, Group A, Member 1 | Male | Amphiregulin | RNA, Messenger - metabolism | Lipopolysaccharides | DNA-Binding Proteins - metabolism | Immediate-Early Proteins - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | EGF Family of Proteins | Respiration, Artificial - adverse effects | Cell Cycle Proteins - genetics | Lung - metabolism | Lung Injury | Glycoproteins - genetics | Reproducibility of Results | Cell Cycle Proteins - metabolism | Gene Expression Regulation | Intercellular Signaling Peptides and Proteins - genetics | Gene Expression Profiling - methods | Interleukin-11 - genetics | Interleukin-11 - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Cysteine-Rich Protein 61 | Transcription Factors - metabolism | Animals | Immediate-Early Proteins - genetics | Receptors, Steroid - genetics | Lung - drug effects | A Kinase Anchor Proteins | Mice | Mice, Inbred BALB C | Cluster Analysis | Receptors, Cytoplasmic and Nuclear - metabolism | Index Medicus
Journal Article
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 06/2005, Volume 171, Issue 11, pp. 1318 - 1319
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 08/2017, Volume 57, Issue 2, pp. 193 - 203
Ventilator-induced lung injury (VILI) is a severe complication of mechanical ventilation that can lead to acute respiratory distress syndrome. VILI is... 
Integrated stress response | Alveolar epithelium | Ventilator-induced lung injury | CELLS | STRETCH | ER STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | BARRIER FUNCTION | ACUTE LUNG INJURY | ENDOPLASMIC-RETICULUM STRESS | NLRP3 INFLAMMASOME | PERK | CELL BIOLOGY | alveolar epithelium | UNFOLDED PROTEIN RESPONSE | RESPIRATORY SYSTEM | ventilator-induced lung injury | integrated stress response | Transcription Factor CHOP - genetics | Ventilator-Induced Lung Injury - physiopathology | Cell Membrane Permeability | Stress, Mechanical | Pulmonary Edema - etiology | Protease Inhibitors - pharmacology | Alveolar Epithelial Cells - pathology | RNA Interference | Adenine - therapeutic use | Cell Death | Indoles - pharmacology | Drug Evaluation, Preclinical | Protease Inhibitors - therapeutic use | eIF-2 Kinase - genetics | Stress, Physiological - physiology | Activating Transcription Factor 4 - antagonists & inhibitors | Adenine - analogs & derivatives | Signal Transduction | Cells, Cultured | Cytokines - secretion | Gene Expression Regulation | Activating Transcription Factor 4 - genetics | Rats | Transcription Factor CHOP - antagonists & inhibitors | Adenine - pharmacology | Alveolar Epithelial Cells - metabolism | Unfolded Protein Response | Pulmonary Edema - drug therapy | Rats, Sprague-Dawley | eIF-2 Kinase - antagonists & inhibitors | Animals | eIF-2 Kinase - physiology | Transcription Factor CHOP - physiology | Indoles - therapeutic use | Enzyme Activation | Activating Transcription Factor 4 - physiology | Transcription factors | Lung | Infections | Kinases | Proteins | Signal transduction | Rodents | Stress response | Alveoli | Ventilators | Edema | Cytokines | Inflammation | Permeability | Epithelium | Ribonucleic acid--RNA | Studies | Pathology | Ostomy | Respiratory distress syndrome | Protein kinase | Cell death | Ventilation | Hypoxia | Endoplasmic reticulum | Apoptosis | Index Medicus | Original Research
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2012, Volume 122, Issue 6, pp. 1973 - 1990
EC activation and dysfunction have been linked to a variety of vascular inflammatory disease states. The function of microRNAs (miRNAs) in vascular EC... 
CELL-ADHESION MOLECULE-1 | TRANSCRIPTION FACTORS | MEDICINE, RESEARCH & EXPERIMENTAL | TUMOR-NECROSIS-FACTOR | DEPENDENT INDUCTION | LEUKOCYTE RECRUITMENT | HUMAN-ENDOTHELIAL-CELLS | ACUTE MYELOID-LEUKEMIA | PROGNOSTIC-SIGNIFICANCE | EXPRESSION PROFILES | SIGNAL-TRANSDUCTION PATHWAY | alpha Karyopherins - metabolism | Endotoxemia - therapy | Leukocytes - pathology | Vasculitis - metabolism | Acute Lung Injury - therapy | Endotoxemia - genetics | Humans | NF-kappa B - immunology | Critical Illness | Acute Lung Injury - genetics | MicroRNAs - metabolism | NF-kappa B - metabolism | Endotoxemia - pathology | Vasculitis - immunology | Leukocytes - immunology | Vasculitis - genetics | alpha Karyopherins - immunology | E-Selectin - genetics | Endotoxemia - immunology | Vascular Cell Adhesion Molecule-1 - genetics | Acute Lung Injury - metabolism | Vascular Cell Adhesion Molecule-1 - immunology | Vasculitis - therapy | Lipopolysaccharides - toxicity | Endothelial Cells - metabolism | E-Selectin - metabolism | E-Selectin - immunology | MicroRNAs - immunology | Acute Lung Injury - pathology | Endothelial Cells - immunology | Animals | NF-kappa B - genetics | alpha Karyopherins - genetics | Acute Lung Injury - immunology | Mice | MicroRNAs - genetics | Endothelial Cells - pathology | Endotoxemia - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Leukocytes - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
Cell Reports, ISSN 2211-1247, 02/2019, Volume 26, Issue 7, pp. 1880 - 1892.e6
Circadian rhythms are a hallmark of physiology, but how such daily rhythms organize cellular catabolism is poorly understood. Here, we used proteomics to map... 
proteasome | chaperone-mediated autophagy | autophagy | inflammation | macroautophagy | circadian rhythm | clock | lipopolysaccharide | endotoxin | CIRCADIAN CLOCK | ORCHESTRATION | PROTEIN | CHAPERONE-MEDIATED AUTOPHAGY | MEMBRANE | GUIDELINES | DEFICIENT | DEGRADATION | PROTEASOME ACTIVITY | STATISTICAL-MODEL | CELL BIOLOGY
Journal Article