2009, Chu ban., ISBN 9628931431, 2, 4, 214
Book
Geology in China, ISSN 1000-3657, 08/2015, Volume 42, Issue 4, pp. 948 - 959
Journal Article
Pediatric Pulmonology, ISSN 8755-6863, 04/2017, Volume 52, Issue 4, pp. 423 - 429
Summary Background As for the association of vitamin D receptor (VDR) gene polymorphisms with susceptibility to pediatric asthma, results of published studies...
gene polymorphism | vitamin D | pediatric asthma | VARIANTS | RESPIRATORY SYSTEM | MARKERS | RISK | PEDIATRICS | CHILDREN | Homozygote | Asian Continental Ancestry Group | Genetic Predisposition to Disease | Humans | European Continental Ancestry Group | Risk | Odds Ratio | Child | Receptors, Calcitriol - genetics | Asthma - genetics | Polymorphism, Genetic | Vitamin D | Asthma in children | Genes | Calcifediol | Genetic aspects | Disease susceptibility | Alfacalcidol | Genetic polymorphisms
gene polymorphism | vitamin D | pediatric asthma | VARIANTS | RESPIRATORY SYSTEM | MARKERS | RISK | PEDIATRICS | CHILDREN | Homozygote | Asian Continental Ancestry Group | Genetic Predisposition to Disease | Humans | European Continental Ancestry Group | Risk | Odds Ratio | Child | Receptors, Calcitriol - genetics | Asthma - genetics | Polymorphism, Genetic | Vitamin D | Asthma in children | Genes | Calcifediol | Genetic aspects | Disease susceptibility | Alfacalcidol | Genetic polymorphisms
Journal Article
Hepatology, ISSN 0270-9139, 01/2012, Volume 55, Issue 1, pp. 108 - 120
Hepatitis B virus X (HBx) protein is implicated in hepatitis B virus (HBV)‐associated liver carcinogenesis. However, it remains unclear whether HBx‐expressing...
LIVER-CANCER | HEPATOCELLULAR-CARCINOMA CELLS | PROTEIN | RAT | DISEASE | CATENIN | STEM/PROGENITOR CELLS | MOUSE-LIVER | OVAL CELLS | GASTROENTEROLOGY & HEPATOLOGY | CANCER STEM-CELLS | Bile Duct Neoplasms - chemically induced | Carcinoma, Hepatocellular - chemically induced | Cholangiocarcinoma - chemically induced | Humans | Middle Aged | Liver Neoplasms, Experimental - chemically induced | Male | Cell Transformation, Neoplastic - chemically induced | Cholangiocarcinoma - virology | Bile Duct Neoplasms - physiopathology | Liver Neoplasms, Experimental - physiopathology | Trans-Activators - genetics | Bile Ducts, Intrahepatic | Female | Gene Expression Regulation, Neoplastic - drug effects | Gene Expression Regulation, Neoplastic - physiology | Disease Models, Animal | Stem Cells - virology | Bile Duct Neoplasms - virology | Carcinoma, Hepatocellular - physiopathology | Cholangiocarcinoma - physiopathology | Liver Neoplasms, Experimental - virology | Mice, Inbred C57BL | Mice, Transgenic | Carcinoma, Hepatocellular - virology | Mice, SCID | Animals | Pyridines - toxicity | Signal Transduction - drug effects | Stem Cells - drug effects | Mice, Inbred NOD | Signal Transduction - physiology | Stem Cells - physiology | Trans-Activators - metabolism | Mice | Hepatitis | Liver cancer | Transgenic animals | Rodents | Tumors | Index Medicus
LIVER-CANCER | HEPATOCELLULAR-CARCINOMA CELLS | PROTEIN | RAT | DISEASE | CATENIN | STEM/PROGENITOR CELLS | MOUSE-LIVER | OVAL CELLS | GASTROENTEROLOGY & HEPATOLOGY | CANCER STEM-CELLS | Bile Duct Neoplasms - chemically induced | Carcinoma, Hepatocellular - chemically induced | Cholangiocarcinoma - chemically induced | Humans | Middle Aged | Liver Neoplasms, Experimental - chemically induced | Male | Cell Transformation, Neoplastic - chemically induced | Cholangiocarcinoma - virology | Bile Duct Neoplasms - physiopathology | Liver Neoplasms, Experimental - physiopathology | Trans-Activators - genetics | Bile Ducts, Intrahepatic | Female | Gene Expression Regulation, Neoplastic - drug effects | Gene Expression Regulation, Neoplastic - physiology | Disease Models, Animal | Stem Cells - virology | Bile Duct Neoplasms - virology | Carcinoma, Hepatocellular - physiopathology | Cholangiocarcinoma - physiopathology | Liver Neoplasms, Experimental - virology | Mice, Inbred C57BL | Mice, Transgenic | Carcinoma, Hepatocellular - virology | Mice, SCID | Animals | Pyridines - toxicity | Signal Transduction - drug effects | Stem Cells - drug effects | Mice, Inbred NOD | Signal Transduction - physiology | Stem Cells - physiology | Trans-Activators - metabolism | Mice | Hepatitis | Liver cancer | Transgenic animals | Rodents | Tumors | Index Medicus
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2017, Volume 7, Issue 1, pp. 2736 - 10
Glucocorticoids have been used to treat hearing loss and vestibular dysfunction for many years. However, some reports have indicated that a subset of patients...
HISTONE DEACETYLASE 2 | OBSTRUCTIVE PULMONARY-DISEASE | INDUCED OTITIS-MEDIA | INDUCED AIRWAY INFLAMMATION | MULTIDISCIPLINARY SCIENCES | INNER-EAR DISEASE | ENDOTOXIN | GENE-EXPRESSION | MIDDLE-EAR | THEOPHYLLINE | NF-KAPPA-B | Aminophylline - pharmacology | Guinea Pigs | Hearing Loss, Sensorineural - metabolism | Cochlea - physiopathology | Lipopolysaccharides - adverse effects | Drug Synergism | Hearing Loss, Sensorineural - diagnosis | Animals | Cochlea - metabolism | Protective Agents - pharmacology | Hearing Loss, Sensorineural - etiology | Fluorescent Antibody Technique | Glucocorticoids - pharmacology | Histone Deacetylase 2 - metabolism | Hearing Loss, Sensorineural - drug therapy | Histone deacetylase | Dexamethasone | Glucocorticoids | Cochlea | Vestibular system | Hearing protection | HDAC2 protein | Hearing impairment | Enzyme-linked immunosorbent assay | Hearing loss | Lipopolysaccharides
HISTONE DEACETYLASE 2 | OBSTRUCTIVE PULMONARY-DISEASE | INDUCED OTITIS-MEDIA | INDUCED AIRWAY INFLAMMATION | MULTIDISCIPLINARY SCIENCES | INNER-EAR DISEASE | ENDOTOXIN | GENE-EXPRESSION | MIDDLE-EAR | THEOPHYLLINE | NF-KAPPA-B | Aminophylline - pharmacology | Guinea Pigs | Hearing Loss, Sensorineural - metabolism | Cochlea - physiopathology | Lipopolysaccharides - adverse effects | Drug Synergism | Hearing Loss, Sensorineural - diagnosis | Animals | Cochlea - metabolism | Protective Agents - pharmacology | Hearing Loss, Sensorineural - etiology | Fluorescent Antibody Technique | Glucocorticoids - pharmacology | Histone Deacetylase 2 - metabolism | Hearing Loss, Sensorineural - drug therapy | Histone deacetylase | Dexamethasone | Glucocorticoids | Cochlea | Vestibular system | Hearing protection | HDAC2 protein | Hearing impairment | Enzyme-linked immunosorbent assay | Hearing loss | Lipopolysaccharides
Journal Article
Electrochemistry Communications, ISSN 1388-2481, 03/2014, Volume 40, pp. 35 - 37
In this communication, a coaxial pH microelectrode consisting of a working electrode made of IrO and a Ag/AgCl reference electrode has been developed. The...
Iridium oxide | Electrodeposition | Coaxial pH ultramicroelectrode | ELECTROCHEMISTRY | ARRAY | FILM | OXIDATIVE BURST | SENSORS | OILSEED RAPE | ELECTRODE | MICROELECTRODES | FABRICATION | CARBON-FIBER NANOELECTRODES | Hydrogen-ion concentration
Iridium oxide | Electrodeposition | Coaxial pH ultramicroelectrode | ELECTROCHEMISTRY | ARRAY | FILM | OXIDATIVE BURST | SENSORS | OILSEED RAPE | ELECTRODE | MICROELECTRODES | FABRICATION | CARBON-FIBER NANOELECTRODES | Hydrogen-ion concentration
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, p. e90528
We previously reported that the intronic tagSNP +357G/C in the metastasis suppressor HTPAP is associated with metastasis and prognosis of hepatocellular...
CANCER-CELLS | SUPPRESSOR | VARIANTS | GENE | MULTIDISCIPLINARY SCIENCES | RISK | CHROMOSOME 8P DELETION | EXPRESSION | ASSOCIATION | Prognosis | Liver Neoplasms - genetics | Carcinoma, Hepatocellular - diagnosis | Neoplasm Invasiveness | Genomics | Humans | Middle Aged | Phosphatidate Phosphatase - metabolism | Male | Phosphatidate Phosphatase - genetics | Promoter Regions, Genetic - genetics | Haplotypes - genetics | Neoplasm Metastasis | Liver Neoplasms - diagnosis | Carcinoma, Hepatocellular - genetics | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Line, Tumor | Female | Liver Neoplasms - pathology | Polymorphism, Single Nucleotide | Transcription, Genetic - genetics | Gene Expression Regulation, Neoplastic - genetics | Carcinoma, Hepatocellular - metabolism | Genetic aspects | Metastasis | Hepatoma | RNA | Luciferase | Cancer | Haplotypes | Transcription | Laboratories | Linkage disequilibrium | Science | Hepatocellular carcinoma | Genomes | Single-nucleotide polymorphism | Multivariate analysis | Western blotting | Metastases | Liver cancer | Education | Hepatology | Surgery | Deoxyribonucleic acid--DNA | Regression analysis | Gene expression | Patients | Promoters | Genetic variance | DNA microarrays | Plasmids | Medical prognosis | Cell lines | Mutation | Genetic testing | Aberration | Deoxyribonucleic acid | DNA
CANCER-CELLS | SUPPRESSOR | VARIANTS | GENE | MULTIDISCIPLINARY SCIENCES | RISK | CHROMOSOME 8P DELETION | EXPRESSION | ASSOCIATION | Prognosis | Liver Neoplasms - genetics | Carcinoma, Hepatocellular - diagnosis | Neoplasm Invasiveness | Genomics | Humans | Middle Aged | Phosphatidate Phosphatase - metabolism | Male | Phosphatidate Phosphatase - genetics | Promoter Regions, Genetic - genetics | Haplotypes - genetics | Neoplasm Metastasis | Liver Neoplasms - diagnosis | Carcinoma, Hepatocellular - genetics | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Line, Tumor | Female | Liver Neoplasms - pathology | Polymorphism, Single Nucleotide | Transcription, Genetic - genetics | Gene Expression Regulation, Neoplastic - genetics | Carcinoma, Hepatocellular - metabolism | Genetic aspects | Metastasis | Hepatoma | RNA | Luciferase | Cancer | Haplotypes | Transcription | Laboratories | Linkage disequilibrium | Science | Hepatocellular carcinoma | Genomes | Single-nucleotide polymorphism | Multivariate analysis | Western blotting | Metastases | Liver cancer | Education | Hepatology | Surgery | Deoxyribonucleic acid--DNA | Regression analysis | Gene expression | Patients | Promoters | Genetic variance | DNA microarrays | Plasmids | Medical prognosis | Cell lines | Mutation | Genetic testing | Aberration | Deoxyribonucleic acid | DNA
Journal Article
Hepatology, ISSN 0270-9139, 05/2014, Volume 59, Issue 5, pp. 1874 - 1885
MicroRNA (miR)‐26a can suppress tumor growth and metastasis of hepatocellular carcinoma (HCC). Since angiogenesis is important for tumor growth and metastasis,...
CELLS | INVASION | METASTASIS | VEGF | TUMOR ANGIOGENESIS | MIR-26A | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | CANCER | CONTRIBUTES | EZH2 | Proto-Oncogene Proteins c-met - metabolism | Cell Proliferation | Prognosis | Liver Neoplasms - genetics | Signal Transduction | Humans | Vascular Endothelial Growth Factor A - analysis | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Proto-Oncogene Proteins c-met - genetics | Animals | Liver Neoplasms - blood supply | Carcinoma, Hepatocellular - genetics | Carcinoma, Hepatocellular - pathology | Hepatocyte Growth Factor - genetics | Carcinoma, Hepatocellular - blood supply | Liver Neoplasms - pathology | Neovascularization, Pathologic - prevention & control | Mice | Mice, Inbred BALB C | MicroRNAs - physiology | Cell Movement | Angiogenesis | Liver cancer | Rodents | Medical prognosis | Hepatology | MicroRNAs | Multivariate analysis | Index Medicus
CELLS | INVASION | METASTASIS | VEGF | TUMOR ANGIOGENESIS | MIR-26A | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | CANCER | CONTRIBUTES | EZH2 | Proto-Oncogene Proteins c-met - metabolism | Cell Proliferation | Prognosis | Liver Neoplasms - genetics | Signal Transduction | Humans | Vascular Endothelial Growth Factor A - analysis | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Proto-Oncogene Proteins c-met - genetics | Animals | Liver Neoplasms - blood supply | Carcinoma, Hepatocellular - genetics | Carcinoma, Hepatocellular - pathology | Hepatocyte Growth Factor - genetics | Carcinoma, Hepatocellular - blood supply | Liver Neoplasms - pathology | Neovascularization, Pathologic - prevention & control | Mice | Mice, Inbred BALB C | MicroRNAs - physiology | Cell Movement | Angiogenesis | Liver cancer | Rodents | Medical prognosis | Hepatology | MicroRNAs | Multivariate analysis | Index Medicus
Journal Article
Hepatology, ISSN 0270-9139, 07/2013, Volume 58, Issue 1, pp. 158 - 170
Down‐regulation of microRNA‐26a (miR‐26a) is associated with poor prognosis of hepatocellular carcinoma (HCC), but its functional mechanism in HCC remains...
APOPTOSIS | STEM-CELLS | ACTIVATION | IN-VIVO | STAT3 | HUMAN LIVER-CANCER | CELL-GROWTH | MIR-26A | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | EZH2 | MicroRNAs - therapeutic use | Down-Regulation | Humans | Middle Aged | Liver Neoplasms - drug therapy | Male | Carcinoma, Hepatocellular - secondary | Animals | Carcinoma, Hepatocellular - drug therapy | STAT3 Transcription Factor - physiology | Interleukin-6 - pharmacology | Signal Transduction - drug effects | Mice, Nude | Carcinoma, Hepatocellular - pathology | Female | Liver Neoplasms - pathology | Mice | Liver Neoplasms - secondary | Interleukin-6 - metabolism | Liver cancer | Cytokines | Multivariate analysis | Hepatology | Rodents | Tumors | Index Medicus
APOPTOSIS | STEM-CELLS | ACTIVATION | IN-VIVO | STAT3 | HUMAN LIVER-CANCER | CELL-GROWTH | MIR-26A | GASTROENTEROLOGY & HEPATOLOGY | EXPRESSION | EZH2 | MicroRNAs - therapeutic use | Down-Regulation | Humans | Middle Aged | Liver Neoplasms - drug therapy | Male | Carcinoma, Hepatocellular - secondary | Animals | Carcinoma, Hepatocellular - drug therapy | STAT3 Transcription Factor - physiology | Interleukin-6 - pharmacology | Signal Transduction - drug effects | Mice, Nude | Carcinoma, Hepatocellular - pathology | Female | Liver Neoplasms - pathology | Mice | Liver Neoplasms - secondary | Interleukin-6 - metabolism | Liver cancer | Cytokines | Multivariate analysis | Hepatology | Rodents | Tumors | Index Medicus
Journal Article
LEUKEMIA, ISSN 0887-6924, 10/2019, Volume 33, Issue 10, pp. 2454 - 2465
New prognostic factors are needed to establish indications for haematopoietic stem cell transplantation (HSCT) in first complete remission (CR1) for T-cell...
SURVIVAL | APOPTOSIS | HYPER-CVAD | THERAPY | ONCOLOGY | PROLIFERATION | MARROW-TRANSPLANTATION | LEUKEMIA | HEMATOLOGY | UP-REGULATION | EXPRESSION | Genetic aspects | Prognosis | MicroRNA | Health aspects | T cell lymphoma | Classifiers | Fetuses | MiRNA | Health risks | Stem cell transplantation | Transplantation | Lymphocytes T | Ribonucleic acid--RNA | Lymphoma | Survival | Cdc4 protein | Medical prognosis | MicroRNAs | Stem cells | Biomarkers | Remission | Lymphomas | Adults
SURVIVAL | APOPTOSIS | HYPER-CVAD | THERAPY | ONCOLOGY | PROLIFERATION | MARROW-TRANSPLANTATION | LEUKEMIA | HEMATOLOGY | UP-REGULATION | EXPRESSION | Genetic aspects | Prognosis | MicroRNA | Health aspects | T cell lymphoma | Classifiers | Fetuses | MiRNA | Health risks | Stem cell transplantation | Transplantation | Lymphocytes T | Ribonucleic acid--RNA | Lymphoma | Survival | Cdc4 protein | Medical prognosis | MicroRNAs | Stem cells | Biomarkers | Remission | Lymphomas | Adults
Journal Article
Frontiers in Pharmacology, ISSN 1663-9812, 05/2019, Volume 10, p. 441
Gubenfangxiao decoction (GBFXD) is a traditional Chinese medicine based on a combination of Yu-Ping-Feng-San and Erchen decoctions. GBFXD has been widely used...
iTRAQ | IPA | Gu-Ben-Fang-Xiao decoction | Chronic persistent asthma | Macrophage | AIRWAY EPITHELIAL-CELLS | LUNG | GU-BEN-FANG-XIAO decoction | GENETIC POLYMORPHISMS | MACROPHAGES | TISSUE | macrophage | RISK | CHILDREN | FIZZ1 | INFLAMMATION | chronic persistent asthma | PHARMACOLOGY & PHARMACY | STRESS | Models | Health aspects | Analysis | Asthma
iTRAQ | IPA | Gu-Ben-Fang-Xiao decoction | Chronic persistent asthma | Macrophage | AIRWAY EPITHELIAL-CELLS | LUNG | GU-BEN-FANG-XIAO decoction | GENETIC POLYMORPHISMS | MACROPHAGES | TISSUE | macrophage | RISK | CHILDREN | FIZZ1 | INFLAMMATION | chronic persistent asthma | PHARMACOLOGY & PHARMACY | STRESS | Models | Health aspects | Analysis | Asthma
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2014, Volume 9, Issue 3, p. e92020
Tuberculosis (TB) is the leading cause of death due to an infectious disease worldwide, particularly in developing countries. A series of candidate genes have...
SOCS PROTEINS | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | PULMONARY TUBERCULOSIS | SOUTHEAST IRAN | CYTOKINE | INTERLEUKIN-10 | ASSOCIATION | EXPRESSION | T-CELLS | SH2-CONTAINING PROTEIN | Genetic Predisposition to Disease - genetics | Humans | Asian Continental Ancestry Group - genetics | Gene Expression Regulation | Child, Preschool | Suppressor of Cytokine Signaling Proteins - genetics | Male | Promoter Regions, Genetic - genetics | Asian Continental Ancestry Group - ethnology | Haplotypes - genetics | Ethnic Groups - genetics | Tuberculosis - genetics | Mycobacterium tuberculosis - physiology | Female | Polymorphism, Single Nucleotide | Tuberculosis - blood | Child | Cytokines - blood | Pediatrics | Medical research | Cytokines | Communicable diseases | Genes | Developing countries | Development and progression | Genetic transcription | T cells | Tuberculosis | Medicine, Experimental | Genetic aspects | Single nucleotide polymorphisms | Transcription | Laboratories | Heterozygotes | Homology | Lymphocytes T | Infections | Single-nucleotide polymorphism | Kinases | Developing countries--LDCs | Proteins | Cell activation | Lymphocytes | Education | Children | Discipline | Genotypes | Health risks | Interleukin 12 | Inflammation | Gene expression | Minority & ethnic groups | Homozygotes | Infectious diseases | Hospitals | Cell lines | Interleukin 10 | Viral infections | LDCs
SOCS PROTEINS | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | PULMONARY TUBERCULOSIS | SOUTHEAST IRAN | CYTOKINE | INTERLEUKIN-10 | ASSOCIATION | EXPRESSION | T-CELLS | SH2-CONTAINING PROTEIN | Genetic Predisposition to Disease - genetics | Humans | Asian Continental Ancestry Group - genetics | Gene Expression Regulation | Child, Preschool | Suppressor of Cytokine Signaling Proteins - genetics | Male | Promoter Regions, Genetic - genetics | Asian Continental Ancestry Group - ethnology | Haplotypes - genetics | Ethnic Groups - genetics | Tuberculosis - genetics | Mycobacterium tuberculosis - physiology | Female | Polymorphism, Single Nucleotide | Tuberculosis - blood | Child | Cytokines - blood | Pediatrics | Medical research | Cytokines | Communicable diseases | Genes | Developing countries | Development and progression | Genetic transcription | T cells | Tuberculosis | Medicine, Experimental | Genetic aspects | Single nucleotide polymorphisms | Transcription | Laboratories | Heterozygotes | Homology | Lymphocytes T | Infections | Single-nucleotide polymorphism | Kinases | Developing countries--LDCs | Proteins | Cell activation | Lymphocytes | Education | Children | Discipline | Genotypes | Health risks | Interleukin 12 | Inflammation | Gene expression | Minority & ethnic groups | Homozygotes | Infectious diseases | Hospitals | Cell lines | Interleukin 10 | Viral infections | LDCs
Journal Article
Viruses, ISSN 1999-4915, 03/2016, Volume 8, Issue 4, pp. 1 - 11
A Pseudorabies virus (PRV) variant has emerged in China since 2011 that is not protected by commercial vaccines, and has not been well studied. The PRV genome...
Cyclosporine A | CRISPR | Firefly luciferase | Pseudorabies virus | Cas9 | Chloroquine | CRISPR/Cas9 | CHINA | BARTHA-K61-VACCINATED SWINE POPULATION | VARIANT | firefly luciferase | chloroquine | PATHOGENESIS | CYCLOSPORINE-A | REPLICATION | INHIBITION | VIROLOGY | pseudorabies virus | CONSTRUCTION | GENOMIC CHARACTERIZATION | TYPE-1 | Cell Line | Drug Evaluation, Preclinical - methods | Virus Replication - drug effects | Antiviral Agents - pharmacology | Gene Expression | Genetic Vectors - genetics | Animals | CRISPR-Cas Systems | Luciferases, Firefly - genetics | RNA, Guide | Gene Order | Herpesvirus 1, Suid - drug effects | Herpesvirus 1, Suid - genetics | Genes, Reporter
Cyclosporine A | CRISPR | Firefly luciferase | Pseudorabies virus | Cas9 | Chloroquine | CRISPR/Cas9 | CHINA | BARTHA-K61-VACCINATED SWINE POPULATION | VARIANT | firefly luciferase | chloroquine | PATHOGENESIS | CYCLOSPORINE-A | REPLICATION | INHIBITION | VIROLOGY | pseudorabies virus | CONSTRUCTION | GENOMIC CHARACTERIZATION | TYPE-1 | Cell Line | Drug Evaluation, Preclinical - methods | Virus Replication - drug effects | Antiviral Agents - pharmacology | Gene Expression | Genetic Vectors - genetics | Animals | CRISPR-Cas Systems | Luciferases, Firefly - genetics | RNA, Guide | Gene Order | Herpesvirus 1, Suid - drug effects | Herpesvirus 1, Suid - genetics | Genes, Reporter
Journal Article
International Journal of Cancer, ISSN 0020-7136, 08/2014, Volume 135, Issue 3, pp. 541 - 550
Our previous microarray data showed that microRNA‐224 (miR‐224) was downregulated in human prostate cancer (PCa) tissues compared with adjacent benign tissues....
microRNA‐224 | prostate cancer | biochemical recurrence‐free survival | TRIB1 | clinicopathological feature | MicroRNA-224 | Biochemical recurrence-free survival | Clinicopathological feature | Prostate cancer | SIGNATURES | DYSREGULATED MIRNAS | PROLIFERATION | EXPRESSION PROFILES | TUMORS | PREDICTION | biochemical recurrence-free survival | METABOLISM | ONCOLOGY | GENES | TARGETS | TRIBBLES | microRNA-224 | Cell Proliferation | Prognosis | Tissue Array Analysis | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Intracellular Signaling Peptides and Proteins - metabolism | Immunoenzyme Techniques | Prostate - metabolism | Prostate - pathology | Flow Cytometry | In Situ Hybridization | Neoplasm Grading | Prostatic Neoplasms - genetics | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Aged, 80 and over | Biomarkers, Tumor - metabolism | Adult | Tumor Cells, Cultured | Intracellular Signaling Peptides and Proteins - genetics | Real-Time Polymerase Chain Reaction | Protein-Serine-Threonine Kinases - metabolism | Prostatic Neoplasms - pathology | RNA, Messenger - genetics | Protein-Serine-Threonine Kinases - genetics | Survival Rate | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Disease Progression | Prostatic Neoplasms - mortality | Aged | Biomarkers, Tumor - genetics | MicroRNAs - genetics | Neoplasm Staging | Apoptosis | Cell Movement | Development and progression | MicroRNA | Biomarkers | Medical research | Metastasis | Medical prognosis | MicroRNAs
microRNA‐224 | prostate cancer | biochemical recurrence‐free survival | TRIB1 | clinicopathological feature | MicroRNA-224 | Biochemical recurrence-free survival | Clinicopathological feature | Prostate cancer | SIGNATURES | DYSREGULATED MIRNAS | PROLIFERATION | EXPRESSION PROFILES | TUMORS | PREDICTION | biochemical recurrence-free survival | METABOLISM | ONCOLOGY | GENES | TARGETS | TRIBBLES | microRNA-224 | Cell Proliferation | Prognosis | Tissue Array Analysis | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Intracellular Signaling Peptides and Proteins - metabolism | Immunoenzyme Techniques | Prostate - metabolism | Prostate - pathology | Flow Cytometry | In Situ Hybridization | Neoplasm Grading | Prostatic Neoplasms - genetics | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Aged, 80 and over | Biomarkers, Tumor - metabolism | Adult | Tumor Cells, Cultured | Intracellular Signaling Peptides and Proteins - genetics | Real-Time Polymerase Chain Reaction | Protein-Serine-Threonine Kinases - metabolism | Prostatic Neoplasms - pathology | RNA, Messenger - genetics | Protein-Serine-Threonine Kinases - genetics | Survival Rate | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Disease Progression | Prostatic Neoplasms - mortality | Aged | Biomarkers, Tumor - genetics | MicroRNAs - genetics | Neoplasm Staging | Apoptosis | Cell Movement | Development and progression | MicroRNA | Biomarkers | Medical research | Metastasis | Medical prognosis | MicroRNAs
Journal Article