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Nature communications, ISSN 2041-1723, 11/2018, Volume 9, Issue 1, pp. 4848 - 14
X-linked myotubular myopathy (XLMTM, also known as XLCNM) is a severe congenital muscular disorder due to mutations in the myotubularin gene, MTM1. It is... 
BREAST-CANCER | SKELETAL-MUSCLE | INHIBITION | PHOSPHATIDYLINOSITOL 3-KINASE | MULTIDISCIPLINARY SCIENCES | MEMBRANE TUBULATION | MOUSE MODEL | DEFICIENT | DYNAMIN 2 | GENE-THERAPY | ESTROGEN-RECEPTOR | Class II Phosphatidylinositol 3-Kinases - metabolism | Electric Stimulation | Gene Expression - drug effects | Longevity - drug effects | Humans | Motor Activity - drug effects | Male | Muscle, Skeletal - metabolism | Myopathies, Structural, Congenital - genetics | Genes, Lethal | Myopathies, Structural, Congenital - drug therapy | Myofibrils - ultrastructure | Protective Agents - pharmacology | Muscle, Skeletal - drug effects | Protein Tyrosine Phosphatases, Non-Receptor - genetics | Class II Phosphatidylinositol 3-Kinases - genetics | Female | Dynamin II - metabolism | Disease Models, Animal | Excitation Contraction Coupling - drug effects | Myofibrils - drug effects | Protein Tyrosine Phosphatases, Non-Receptor - deficiency | Dynamin II - genetics | Disease Progression | Mice, Knockout | Animals | Myopathies, Structural, Congenital - pathology | Tamoxifen - pharmacology | Mice | Myofibrils - metabolism | Myopathies, Structural, Congenital - metabolism | Muscle, Skeletal - pathology | Neonates | Phenotypes | Breast cancer | Tamoxifen | Patients | Survival | Contraction | Coupling (molecular) | Hypotonia | Life span | Excitation-contraction coupling | Rodents | Paralysis | Mutation | Myopathy
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2014, Volume 9, Issue 10, p. e110708
Elevation of intracellular Ca2+, excessive ROS production and increased phospholipase A(2) activity contribute to the pathology in dystrophin-deficient muscle.... 
CELLS | FIBERS | SUPEROXIDE | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | (-)-EPIGALLOCATECHIN GALLATE | DUCHENNE MUSCULAR-DYSTROPHY | PHOSPHOLIPASE A | MDX MICE | GREEN TEA EXTRACT | PATHOPHYSIOLOGY | Reactive Oxygen Species - metabolism | Phospholipases A2 - biosynthesis | Muscular Dystrophy, Duchenne - drug therapy | Antioxidants - metabolism | NADPH Oxidases - antagonists & inhibitors | NADPH Oxidases - metabolism | Muscle, Skeletal - metabolism | Muscular Dystrophy, Duchenne - pathology | Phospholipases A2 - metabolism | Animals | Acetophenones - administration & dosage | Dystrophin - genetics | Calcium Signaling - drug effects | Muscle Contraction - drug effects | Dystrophin - deficiency | Antioxidants - administration & dosage | Biphenyl Compounds - administration & dosage | Mice, Inbred mdx | Mice | Muscular Dystrophy, Duchenne - metabolism | Muscle, Skeletal - pathology | Oxidases | Dystrophin | Utrophin | Oxidative stress | Reactive oxygen species | Eccentricity | Lipid peroxidation | Feedback loops | Kinases | Positive feedback | Muscular dystrophy | NAD(P)H oxidase | Calcium influx | Rodents | Procion | Gangrene | Damage | Pharmaceutical sciences | Enzymes | Calcium (intracellular) | Myotubes | Phospholipase A2 | Muscles | Muscle contraction | Pathology | Musculoskeletal system | Phospholipase | Inhibitors | Muscle function | Dystrophy | Calcium ions
Journal Article
Nature Communications, ISSN 2041-1723, 2015, Volume 6, Issue 1, p. 7629
Bile acids are signalling molecules, which activate the transmembrane receptor TGR5 and the nuclear receptor FXR. BA sequestrants (BAS) complex bile acids in... 
GLP-1 SECRETION | FXR | BILE-ACID RECEPTORS | PROTEIN | GLUCOSE | MULTIDISCIPLINARY SCIENCES | INTESTINE | METABOLIC-RATE | EXPRESSION | Colon - cytology | Intestinal Mucosa - metabolism | Sequestering Agents - pharmacology | Humans | Ileum - metabolism | RNA, Messenger - metabolism | Colesevelam Hydrochloride - pharmacology | Obesity - genetics | Glucagon-Like Peptide 1 - genetics | Jejunum - metabolism | Insulin-Secreting Cells - metabolism | Proglucagon - drug effects | Diet, High-Fat | Jejunum - cytology | Enteroendocrine Cells - metabolism | Ileum - cytology | Proglucagon - metabolism | Insulin Secretion | Glucagon-Like Peptide 1 - metabolism | Signal Transduction | Bile Acids and Salts - metabolism | Nuclear Proteins - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Colon - metabolism | Mice, Knockout | Obesity - metabolism | Transcription Factors - metabolism | Insulin - metabolism | Animals | Glycolysis | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice, Obese | Mice | Proglucagon - genetics | Receptors, G-Protein-Coupled - genetics | Blood Glucose - metabolism | Anticholesteremic Agents - pharmacology | Intestines - cytology | Carbohydrates | Glucose | Gene expression | Insulin | Cell and Molecular Biology | RAT SMALL-INTESTINE | GLUCOSE-HOMEOSTASIS | OBESITY | Endokrinologi och diabetes | MICE | Multidisciplinary Sciences | Cell- och molekylärbiologi | TYPE-2 DIABETES-MELLITUS | Endocrinology and Diabetes
Journal Article
Human molecular genetics, ISSN 0964-6906, 06/2019, Volume 28, Issue 12, pp. 1931 - 1946
Journal Article
Journal Article
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2013, Volume 182, Issue 2, pp. 485 - 504
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 06/2013, Volume 33, Issue 11, pp. 2202 - 2211
Journal Article