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Canadian Journal of Physiology and Pharmacology, ISSN 0008-4212, 2014, Volume 92, Issue 2, pp. 132 - 139
Journal Article
2016, ISBN 7507546292, 1 v.
Book
2017, Yong yuan de chang zheng, ISBN 7541749974
Book
PLoS ONE, ISSN 1932-6203, 06/2017, Volume 12, Issue 7, p. e0180464
...Author(s): Shida Zhou 1, Xinxing Du 2, Junxia Xie 1,*, Jun Wang 1,* Introduction Parkinson's disease (PD) is a common neurodegenerative disorder characterized... 
SIGNALING PATHWAYS | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | INTRACEREBRAL HEMORRHAGE | NEURONS | NEURODEGENERATION | MODEL | STRESS | EXPRESSION | PARKINSONS-DISEASE | FACTOR-ALPHA | Microglia - cytology | Cell Line | Microglia - metabolism | Interleukin-6 - genetics | Microglia - drug effects | RNA, Messenger - genetics | Microglia - enzymology | Ferric Compounds - pharmacology | JNK Mitogen-Activated Protein Kinases - metabolism | Blotting, Western | Animals | Iron Regulatory Protein 1 - metabolism | Cation Transport Proteins - metabolism | Interleukin-6 - physiology | Mice | Enzyme Activation | Real-Time Polymerase Chain Reaction | Quaternary Ammonium Compounds - pharmacology | Physiological aspects | Research | c-Jun N-terminal kinases | Interleukin-6 | Iron regulatory protein | Biotechnology | Neurosciences | Transcription factors | Parkinson's disease | Pathogenesis | Substantia nigra | Interleukin | Activation | Iron | Kinases | Accumulation | Interleukin 6 | Proteins | Cell activation | Pathways | Neurodegeneration | Neostriatum | Microglial cells | Attenuation | Pretreatment | Movement disorders | Activation analysis | Dopamine receptors | Dopamine | Deposits | Neurodegenerative diseases | Neurons | c-Jun protein | Divalent metal transporter-1 | Metabolism | Gene expression | Microglia | Cell lines | Transporter
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2014, Volume 9, Issue 12, p. e112532
Background: The therapeutic potential of mesenchymal stem cells (MSCs) has been highlighted recently for treatment of acute or chronic liver injury, by... 
FIBROSIS | IN-VITRO | THERAPY | HEPATOCYTE PROLIFERATION | HEPATECTOMY | MULTIDISCIPLINARY SCIENCES | INJURY | MECHANISMS | ISCHEMIA-REPERFUSION | TRANSPLANTATION | Inflammation - pathology | Reperfusion Injury - therapy | Liver Transplantation | Humans | JNK Mitogen-Activated Protein Kinases - biosynthesis | Rats | Hepatocytes - pathology | Cyclin D1 - biosynthesis | Cell- and Tissue-Based Therapy | Cell Differentiation - genetics | Liver Regeneration - genetics | Animals | Cyclin D1 - genetics | Mesenchymal Stromal Cells | NF-kappa B - genetics | NF-kappa B - biosynthesis | Inflammation - genetics | JNK Mitogen-Activated Protein Kinases - genetics | Reperfusion Injury - genetics | Immunohistochemistry | Therapy | Transcription factors | Bcl-2 protein | Syngeneic grafts | Liver | Adipocytes | Cyclin D1 | Grafting | Osteoblasts | Ischemia | Bcl-x protein | Surgery | Fibroblasts | Bone marrow | Biocompatibility | Growth factors | Liver diseases | Cytokines | CXCR2 protein | Activator protein 1 | JNK protein | Survival | Polymerase chain reaction | Hospitals | Injury prevention | Hepatocytes | Stem cells | Interleukin 10 | Bile | Liver transplantation | Transplants & implants | Mesenchyme | Bone marrow transplantation | Transplantation | Kinases | Interleukin 6 | Biomedical materials | Reperfusion | Allografts | Hepatology | Rodents | IP-10 protein | Hypertension | Cell survival | c-Jun protein | Implantation | Inflammation | Medicine | Regeneration | Grafts | Laboratory animals | Apoptosis
Journal Article
Toxicology and Applied Pharmacology, ISSN 0041-008X, 11/2014, Volume 281, Issue 1, pp. 58 - 66
Acetaminophen (APAP) overdose causes severe hepatotoxicity in animals and humans. However, the mechanisms underlying the gender differences in susceptibility... 
Protein adducts | c-jun N-terminal kinase | Oxidant stress | Acetaminophen hepatotoxicity | Gender difference | Glutathione | C-jun N-terminal kinase | PEROXYNITRITE | MECHANISMS | INDUCED LIVER-INJURY | CELL-DEATH | PERMEABILITY TRANSITION | REACTIVE NITROGEN | DNA FRAGMENTATION | IN-VIVO | GENDER | PHARMACOLOGY & PHARMACY | TOXICOLOGY | Chemical and Drug Induced Liver Injury - prevention & control | Disease Susceptibility | JNK Mitogen-Activated Protein Kinases - antagonists & inhibitors | Acetaminophen - toxicity | Glutathione - metabolism | Mitochondria, Liver - metabolism | Mice, Inbred C57BL | Oxidative Stress - physiology | JNK Mitogen-Activated Protein Kinases - metabolism | Male | Sex Characteristics | Anthracenes - pharmacology | Anthracenes - therapeutic use | Mitochondria, Liver - drug effects | Animals | Analgesics, Non-Narcotic - toxicity | Chemical and Drug Induced Liver Injury - metabolism | Female | Mice | Oxidative Stress - drug effects | Cysteine | Mitochondrial DNA | Acetaminophen | Estrogen | HUMAN POPULATIONS | INJURIES | DMSO | MATERIALS RECOVERY | MITOCHONDRIA | FEMALES | METABOLIC ACTIVATION | 60 APPLIED LIFE SCIENCES | BIOLOGICAL RECOVERY | GLUTATHIONE | ESTRADIOL | LIVER | LIGASES | MICE | MALES | protein adducts | oxidant stress | gender difference | acetaminophen hepatotoxicity | glutathione
Journal Article
Oncotarget, ISSN 1949-2553, 2017, Volume 8, Issue 43, pp. 74406 - 74422
.... In addition, western blot and RT-qPCR assays were used to examine potential TNC metastasis-associated targets, such as JNK/c-Jun, Paxillin/FAK, E-cadherin, N-cadherin, Vimentin, and MMP9/2... 
TNC | JNK/c-Jun | EMT | Pancreatic cancer | PAXILLIN | ACTIVATION | METASTASIS | DUCTAL ADENOCARCINOMA | PHOSPHORYLATION | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | pancreatic cancer | CELL-MIGRATION | EXPRESSION | CARCINOMA
Journal Article
1974, Chu ban, he ding ben., Du shi cong kan. Di 3 ji, Volume 21-22, 1 v. (302, 93 p.)
Book
中国药理学报:英文版, ISSN 1671-4083, 2011, Volume 32, Issue 4, pp. 519 - 527
.... Caspase 3 activity was analyzed to detect apoptosis at different time points. Protein levels of phospho-c-Jun, phosphor-c-Jun N-termi- nal kinase (p-JNK... 
细胞凋亡 | K562细胞 | M期 | Fas配体 | 激酶 | 诱导形成 | Bax蛋白 | 白血病 | ACTIVATION | cell cycle | genipin | JNK | apoptosis | dicoumarol | DEATH | p63 | CANCER | CHEMISTRY, MULTIDISCIPLINARY | PROTEIN-KINASES | K562 cells | p-JNK | PATHWAY | Bax | NITRIC-OXIDE | GENIPOSIDE | GARDENIA | PHARMACOLOGY & PHARMACY | cytochrome c | Fas-L
Journal Article
1991, Di 1 ban., Zhang shi fu zi yu Feng xi li shi yan jiu cong shu, ISBN 9787205018634, 4, 3, 4, 538 p., [17] p. of plates
Book
1976, Jin dai Zhongguo shi liao cong kan xu ji, Volume 284-285, 2 v. (514 p.).
Book
Diabetes, ISSN 0012-1797, 01/2009, Volume 58, Issue 1, pp. 104 - 115
Journal Article