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Physiology (Bethesda, Md.), ISSN 1548-9213, 07/2019, Volume 34, Issue 4, pp. 232 - 239
Skeletal muscle atrophy proceeds through a complex molecular signaling network that is just beginning to be understood. Here, we discuss examples of recently... 
Journal Article
by Garrett-Bakelman, Francine E and Darshi, Manjula and Green, Stefan J and Gur, Ruben C and Lin, Ling and Macias, Brandon R and McKenna, Miles J and Meydan, Cem and Mishra, Tejaswini and Nasrini, Jad and Piening, Brian D and Rizzardi, Lindsay F and Sharma, Kumar and Siamwala, Jamila H and Taylor, Lynn and Vitaterna, Martha Hotz and Afkarian, Maryam and Afshinnekoo, Ebrahim and Ahadi, Sara and Ambati, Aditya and Arya, Maneesh and Bezdan, Daniela and Callahan, Colin M and Chen, Songjie and Choi, Augustine M. K and Chlipala, George E and Contrepois, Kévin and Covington, Marisa and Crucian, Brian E and De Vivo, Immaculata and Dinges, David F and Ebert, Douglas J and Feinberg, Jason I and Gandara, Jorge A and George, Kerry A and Goutsias, John and Grills, George S and Hargens, Alan R and Heer, Martina and Hillary, Ryan P and Hoofnagle, Andrew N and Hook, Vivian Y. H and Jenkinson, Garrett and Jiang, Peng and Keshavarzian, Ali and Laurie, Steven S and Lee-McMullen, B and Lumpkins, Sarah B and MacKay, Matthew and Maienschein-Cline, Mark G and Melnick, Ari M and Moore, Tyler M and Nakahira, Kiichi and Patel, Hemal H and Pietrzyk, Robert and Rao, Varsha and Saito, Rintaro and Salins, Denis N and Schilling, Jan M and Sears, Dorothy D and Sheridan, Caroline K and Stenger, Michael B and Tryggvadottir, Rakel and Urban, Alexander E and Vaisar, Tomas and Van Espen, Benjamin and Zhang, Jing and Ziegler, Michael G and Zwart, Sara R and Charles, John B and Kundrot, Craig E and Scott, Graham B. I and Bailey, Susan M and Basner, Mathias and Feinberg, Andrew P and Lee, Stuart M. C and Mason, Christopher E and Mignot, Emmanuel and Rana, Brinda K and Smith, Scott M and Snyder, Michael P and Turek, Fred W
Science, ISSN 0036-8075, 2019, Volume 364, Issue 6436, pp. 144 - 144
Journal Article
Cell Metabolism, ISSN 1550-4131, 2011, Volume 13, Issue 6, pp. 627 - 638
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2012, Volume 7, Issue 6, p. e39332
Journal Article
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 10/2013, Volume 305, Issue 7, pp. E907 - E915
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2012, Volume 287, Issue 33, pp. 27290 - 27301
Diverse stresses including starvation and muscle disuse cause skeletal muscle atrophy. However, the molecular mechanisms of muscle atrophy are complex and not... 
ACTIVATION | PROTEIN | PATHWAY | PGC-1-ALPHA | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENE-EXPRESSION | DEGRADATION | GENERATION | FOXO TRANSCRIPTION FACTORS | UBIQUITIN LIGASES | COACTIVATORS | Stress, Physiological | Muscle, Skeletal - metabolism | Proto-Oncogene Proteins c-akt - genetics | RNA, Messenger - biosynthesis | Cell Nucleus - metabolism | Cell Nucleus - pathology | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Muscle Proteins - metabolism | Trans-Activators - genetics | Cell Cycle Proteins - genetics | Protein Biosynthesis - genetics | Energy Metabolism - genetics | Nuclear Proteins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Cell Line | Muscular Atrophy - metabolism | Muscular Atrophy - pathology | RNA, Messenger - genetics | Cell Cycle Proteins - metabolism | Gene Expression Regulation | Activating Transcription Factor 4 - genetics | Muscular Atrophy - genetics | Nuclear Proteins - metabolism | Signal Transduction - genetics | Mice, Knockout | Muscle Proteins - genetics | Animals | Activating Transcription Factor 4 - metabolism | Cell Nucleus - genetics | Trans-Activators - metabolism | Mice | Transcription Factors | Muscle, Skeletal - pathology | Molecular Bases of Disease | Transcription Regulation | Skeletal Muscle | Skeletal Muscle Metabolism | Stress Response | Gadd45a | Muscle Atrophy | ATF4
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 1782, Volume 289, Issue 21, pp. 14913 - 14924
Skeletal muscle atrophy is a common and debilitating condition that lacks an effective therapy. To address this problem, we used a systems-based discovery... 
HYPERTROPHY | ALPHA-TOMATINE | ACTIVATION | MANAGEMENT | BIOCHEMISTRY & MOLECULAR BIOLOGY | TOMATOES | SARCOPENIA | DEFICIENT MICE | EXPRESSION SIGNATURES | FAT MASS | PROTECTS | Gene Expression - drug effects | TOR Serine-Threonine Kinases - metabolism | Humans | Mitochondria, Muscle - metabolism | Drug Discovery - methods | Immunoblotting | Male | Multiprotein Complexes - genetics | Muscle, Skeletal - metabolism | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | Tomatine - pharmacology | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - metabolism | Myoblasts, Skeletal - cytology | TOR Serine-Threonine Kinases - genetics | MCF-7 Cells | Muscle, Skeletal - drug effects | Muscle Proteins - metabolism | Muscular Atrophy - prevention & control | Mitochondria, Muscle - genetics | Myoblasts, Skeletal - metabolism | Cell Line | Muscular Atrophy - metabolism | Mice, Inbred C57BL | Cells, Cultured | Muscular Atrophy - genetics | Tomatine - analogs & derivatives | Reverse Transcriptase Polymerase Chain Reaction | Muscle Proteins - genetics | Animals | Signal Transduction - drug effects | Cell Line, Tumor | HL-60 Cells | Muscle Fibers, Skeletal - cytology | Mice | Mitochondria, Muscle - drug effects | Muscle, Skeletal - pathology | Myoblasts, Skeletal - drug effects | Skeletal Muscle | Skeletal Muscle Metabolism | Muscle Atrophy | Tomatidine | Metabolism | Systems Biology | mTOR complex (mTORC)
Journal Article
Journal Article
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 08/2014, Volume 307, Issue 3, pp. E245 - E261
Immobilization causes skeletal muscle atrophy via complex signaling pathways that are not well understood. To better understand these pathways, we investigated... 
Skeletal muscle atrophy | Immobilization | p21 | Skeletal muscle | Activating transcription factor 4 | p53 | ACTIVATION | PHYSIOLOGY | immobilization | PROTEIN | skeletal muscle atrophy | PHOSPHORYLATION | DNA-DAMAGE | INDUCTION | TRANSLATION | TOTAL KNEE ARTHROPLASTY | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | skeletal muscle | STRESS | activating transcription factor 4 | Up-Regulation | Male | Muscle, Skeletal - metabolism | Tumor Suppressor Protein p53 - genetics | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Muscular Atrophy - etiology | Recombinant Proteins - metabolism | Promoter Regions, Genetic | Genome-Wide Association Study | Disease Resistance | Muscular Atrophy - metabolism | Mice, Inbred C57BL | Muscular Atrophy - pathology | Tumor Suppressor Protein p53 - metabolism | Activating Transcription Factor 4 - genetics | Cyclin-Dependent Kinase Inhibitor p21 - antagonists & inhibitors | Mice, Transgenic | Mutant Proteins - metabolism | Immobilization - adverse effects | Hindlimb Suspension | Mice, Knockout | Animals | Activating Transcription Factor 4 - metabolism | MicroRNAs | Mice | Muscle, Skeletal - pathology | Amino Acid Substitution | Crosses, Genetic | Muscles | Genetic research | Genetic aspects | Research | Gene expression | Atrophy, Muscular | Health aspects
Journal Article
Journal of applied physiology (Bethesda, Md. : 1985), ISSN 8750-7587, 08/2019
Age-related skeletal muscle atrophy is a very common and serious condition that remains poorly understood at the molecular level. Several lines of evidence... 
Journal Article