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Journal of the American College of Surgeons, ISSN 1072-7515, 10/2019, Volume 229, Issue 4, pp. S131 - S132
Journal Article
Biochemical Journal, ISSN 0264-6021, 03/2016, Volume 473, Issue 5, pp. 593 - 603
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AMERICAN SURGEON, ISSN 0003-1348, 09/2016, Volume 82, Issue 9, pp. E233 - E235
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Cancer Cell, ISSN 1535-6108, 05/2013, Volume 23, Issue 5, pp. 603 - 617
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JOURNAL OF IMMUNOLOGY, ISSN 0022-1767, 07/2019, Volume 203, Issue 1, pp. 137 - 147
PI3K is one of the most frequently mutated genes in cancers and has been the target of numerous anticancer therapies. With the additional development of... 
RAPAMYCIN | TARGET | NATURAL-KILLER-CELLS | ACTIVATION | 3-KINASE | PHOSPHORYLATION | KINASE | RIBOSOMAL-PROTEIN S6 | IMMUNOLOGY | IDENTIFICATION | CANCER
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Cancer Cell, ISSN 1535-6108, 2010, Volume 17, Issue 4, pp. 362 - 375
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Molecular Cancer Therapeutics, ISSN 1535-7163, 07/2015, Volume 14, Issue 7 Supplement, pp. B37 - B37
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Molecular Cancer Therapeutics, ISSN 1535-7163, 12/2011, Volume 10, Issue 12, pp. 2426 - 2436
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Cancer Research, ISSN 0008-5472, 07/2017, Volume 77, Issue 13 Supplement, pp. 146 - 146
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Cancer Research, ISSN 0008-5472, 07/2016, Volume 76, Issue 14 Supplement, pp. 370 - 370
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Journal of Medicinal Chemistry, ISSN 0022-2623, 06/2013, Volume 56, Issue 11, pp. 4597 - 4610
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The American surgeon, 09/2016, Volume 82, Issue 9, p. e233
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PLoS ONE, ISSN 1932-6203, 05/2012, Volume 7, Issue 5, p. e36402
The PTEN/PI3K pathway is commonly mutated in cancer and therefore represents an attractive target for therapeutic intervention. To investigate the primary... 
EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER CELLS | I PHOSPHATIDYLINOSITIDE 3-KINASES | POTENT | PIK3CA MUTATIONS | KINASE | BIOLOGY | GENE-EXPRESSION | INHIBITOR | ONCOGENIC PATHWAYS | LINES | Neoplasms - metabolism | Humans | Gene Silencing | Cell Survival - genetics | Gene Expression Profiling | Phosphatidylinositol 3-Kinases - metabolism | Epithelial-Mesenchymal Transition - genetics | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Sulfonamides - pharmacology | Cell Movement - genetics | Indazoles - pharmacology | Phosphatidylinositol 3-Kinases - genetics | Phenotype | Class I Phosphatidylinositol 3-Kinases | Neoplasms - genetics | RNA Interference | Signal Transduction - drug effects | Protein Interaction Domains and Motifs - genetics | Cell Line, Tumor | Mutation | Enzyme Activation - genetics | Cluster Analysis | Medicine, Experimental | Medical research | Genetic aspects | Metastasis | Health aspects | TOR protein | Cell culture | Mesenchyme | Laboratories | Epithelial cells | Reversing | Oncology | AKT protein | Kinases | Positive feedback | Metastases | Genotype & phenotype | Cell growth | Penicillin | Phosphatidylinositol 3,4,5-triphosphate | Invasiveness | Pharmacology | Breast cancer | Gene expression | Chemical compounds | Insulin | 1-Phosphatidylinositol 3-kinase | Signaling | Alleles | PTEN protein | Cancer
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Cancer Research, ISSN 0008-5472, 04/2013, Volume 73, Issue 8 Supplement, pp. 4470 - 4470
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Cancer Research, ISSN 0008-5472, 08/2015, Volume 75, Issue 15 Supplement, pp. 2672 - 2672
Journal Article
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