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1. Full Text The consensus molecular subtypes of colorectal cancer
by Guinney, Justin and Dienstmann, Roigo and Wang, Xin and de Reyniès, Aurélien and Schlicker, Aneas and Soneson, Charlotte and Marisa, Laetitia and Roepman, Paul and Nyamundanda, Gift and Angelino, Paolo and Bot, Brian M and Morris, Jeffrey S and Simon, Iris M and Gerster, Sarah and Fessler, Evelyn and de Sousa E Melo, Felipe and Missiaglia, Edoardo and Ramay, Hena and Barras, David and Homicsko, Krisztian and Maru, Dipen and Manyam, Ganiraju C and Broom, Bradley and Boige, Valerie and Perez-Villamil, Beatriz and Laderas, Ted and Salazar, Ramon and Gray, Joe W and Hanahan, Douglas and Tabernero, Josep and Bernards, Rene and Friend, Stephen H and Laurent-Puig, Pierre and Medema, Jan Paul and Sadanandam, Anguraj and Wessels, Lodewyk and Delorenzi, Mauro and Kopetz, Scott and Vermeulen, Louis and Tejpar, Sabine
Nature medicine, ISSN 1078-8956, 2015, Volume 21, Issue 11, pp. 1350 - 1356
Colorectal cancer (CRC) is a frequently lethal disease with heterogeneous outcomes and drug responses. To resolve inconsistencies among the reported gene... 
MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | III COLON-CANCER | MICROSATELLITE INSTABILITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | PHENOTYPE | CLASSIFICATION | TUMORS | CELL BIOLOGY | RESPONSES | RECTAL-CANCER | MUTATION | GROWTH | ras Proteins - genetics | Microsatellite Instability | Proto-Oncogene Proteins p21(ras) | Colorectal Neoplasms - genetics | Humans | Gene Expression Regulation, Neoplastic | Gene Expression Profiling | Information Dissemination | Neovascularization, Pathologic - pathology | DNA Methylation | Carcinoma - pathology | Proto-Oncogene Proteins - genetics | Colorectal Neoplasms - classification | DNA Copy Number Variations - genetics | Mutation - genetics | Consensus | Phenotype | Transforming Growth Factor beta - genetics | Wnt Signaling Pathway - genetics | Proto-Oncogene Proteins B-raf - genetics | Genes, myc - genetics | CpG Islands | Carcinoma - genetics | Neovascularization, Pathologic - genetics | Carcinoma - classification | Colorectal Neoplasms - pathology | Care and treatment | Usage | MicroRNA | Colorectal cancer | Liquid chromatography | Research | Gene expression | Transforming growth factors | Genotype & phenotype | Genomics | Classificació de tumors | Oncologia | Oncology | Càncer colorectal | Tumors classification
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2. Full Text Pancreatic Endocrine Tumors: Expression Profiling Evidences a Role for AKT-mTOR Pathway
by Edoardo Missiaglia and Irene Dalai and Stefano Barbi and Stefania Beghelli and Massimo Falconi and Marco della Peruta and Lorenzo Piemonti and Gabriele Capurso and Alessia Di Florio and Gianfranco delle Fave and Paolo Pederzoli and Carlo M. Croce and Aldo Scarpa
Journal of Clinical Oncology, ISSN 0732-183X, 01/2010, Volume 28, Issue 2, pp. 245 - 255
Purpose We investigated the global gene expression in a large panel of pancreatic endocrine tumors (PETs) aimed at identifying new potential targets for... 
RAD001 EVEROLIMUS | HOMOLOGOUS FACTORS | SOMATOSTATIN ANALOG | SUPPRESSOR GENE | ONCOLOGY | CYCLIN D1 | PROTEIN-KINASE | GASTROENTEROPANCREATIC NEUROENDOCRINE TUMORS | GENE-EXPRESSION | THERAPEUTIC TARGETS | TUBEROUS SCLEROSIS | PTEN Phosphohydrolase - genetics | Signal Transduction | Down-Regulation | Humans | Middle Aged | Male | Pancreatic Neoplasms - genetics | Gene Expression Profiling | Proto-Oncogene Proteins c-akt - genetics | Neoplasm Metastasis | Tumor Suppressor Proteins - genetics | Adolescent | Carcinoma, Islet Cell - genetics | Cell Line, Tumor | Adult | Female | Aged | To4 | Endo7 | ORIGINAL REPORTS | Gic28
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3. Full Text Fusion Gene–Negative Alveolar Rhabdomyosarcoma Is Clinically and Molecularly Indistinguishable From Embryonal Rhabdomyosarcoma
by Daniel Williamson and Edoardo Missiaglia and Aurélien de Reyniès and Gaëlle Pierron and Benedicte Thuille and Gilles Palenzuela and Khin Thway and Daniel Orbach and Marick Laé and Paul Fréneaux and Kathy Pritchard-Jones and Odile Oberlin and Janet Shipley and Olivier Delattre
Journal of Clinical Oncology, ISSN 0732-183X, 05/2010, Volume 28, Issue 13, pp. 2151 - 2158
Purpose To determine whether the clinical and molecular biologic characteristics of the alveolar rhabdomyosarcoma (ARMS) and embryonal rhabdomyosarcoma (ERMS)... 
INTERNATIONAL-SOCIETY | PROGNOSTIC-FACTORS | ONCOLOGY | PAX3-FKHR | ARRAY-CGH | NONMETASTATIC RHABDOMYOSARCOMA | INTERGROUP-RHABDOMYOSARCOMA | CLASSIFICATION | CHILDHOOD | CHILDRENS ONCOLOGY GROUP | PEDIATRIC-ONCOLOGY | Paired Box Transcription Factors - genetics | Predictive Value of Tests | Genetic Testing | Humans | Gene Expression Regulation, Neoplastic | Child, Preschool | PAX7 Transcription Factor - genetics | Male | Rhabdomyosarcoma, Embryonal - diagnosis | Gene Expression Profiling | Rhabdomyosarcoma, Embryonal - genetics | Nuclear Receptor Coactivator 1 - genetics | Rhabdomyosarcoma, Alveolar - diagnosis | Rhabdomyosarcoma, Alveolar - therapy | Time Factors | Rhabdomyosarcoma, Alveolar - mortality | Rhabdomyosarcoma, Embryonal - therapy | Female | Rhabdomyosarcoma, Alveolar - pathology | France | Child | Rhabdomyosarcoma, Embryonal - pathology | Diagnosis, Differential | PAX3 Transcription Factor | Rhabdomyosarcoma, Alveolar - genetics | Risk Assessment | Risk Factors | Kaplan-Meier Estimate | Proportional Hazards Models | Treatment Outcome | United Kingdom | Chromosomes, Human, Pair 8 | Rhabdomyosarcoma, Embryonal - mortality | Reverse Transcriptase Polymerase Chain Reaction | Disease-Free Survival | Oncogene Proteins, Fusion - genetics
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4. Full Text PAX3/FOXO1 Fusion Gene Status Is the Key Prognostic Molecular Marker in Rhabdomyosarcoma and Significantly Improves Current Risk Stratification
by Edoardo Missiaglia and Dan Williamson and Julia Chisholm and Pratyaksha Wirapati and Gaëlle Pierron and Fabien Petel and Jean-Paul Concordet and Khin Thway and Odile Oberlin and Kathy Pritchard-Jones and Olivier Delattre and Mauro Delorenzi and Janet Shipley
Journal of Clinical Oncology, ISSN 0732-183X, 05/2012, Volume 30, Issue 14, pp. 1670 - 1677
Purpose To improve the risk stratification of patients with rhabdomyosarcoma (RMS) through the use of clinical and molecular biologic data. Patients and... 
STUDY-IV | ONCOLOGY | PAX | NONMETASTATIC RHABDOMYOSARCOMA | CLASSIFICATION | ADVERSE PROGNOSIS | STUDY-III | EXPRESSION | NEGATIVE ALVEOLAR RHABDOMYOSARCOMA | PREDICTION | Paired Box Transcription Factors - genetics | Multivariate Analysis | Translocation, Genetic | Prognosis | Humans | Gene Expression Regulation, Neoplastic | Child, Preschool | Male | Gene Expression Profiling | Risk Management | Forkhead Transcription Factors - metabolism | Neoplasm Invasiveness - pathology | Sensitivity and Specificity | Female | Retrospective Studies | Child | Rhabdomyosarcoma - mortality | Databases, Factual | PAX3 Transcription Factor | Kaplan-Meier Estimate | Proportional Hazards Models | United Kingdom | Forkhead Transcription Factors - genetics | Gene Fusion - genetics | Adolescent | Survival Analysis | Biomarkers, Tumor - genetics | Forkhead Box Protein O1 | Rhabdomyosarcoma - genetics | Neoplasm Staging | Rhabdomyosarcoma - therapy | Paired Box Transcription Factors - metabolism | Cohort Studies
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5. Full Text MicroRNA Expression Abnormalities in Pancreatic Endocrine and Acinar Tumors Are Associated With Distinctive Pathologic Features and Clinical Behavior
by Claudia Roldo and Edoardo Missiaglia and John P. Hagan and Massimo Falconi and Paola Capelli and Samantha Bersani and George Adrian Calin and Stefano Volinia and Chang-Gong Liu and Aldo Scarpa and Carlo M. Croce
Journal of Clinical Oncology, ISSN 0732-183X, 10/2006, Volume 24, Issue 29, pp. 4677 - 4684
Purpose We investigated the global microRNA expression patterns in normal pancreas, pancreatic endocrine tumors and acinar carcinomas to evaluate their... 
MOLECULAR-GENETICS | APOPTOSIS | GLIOBLASTOMA | ONCOLOGY | B-CELL LYMPHOMAS | MALIGNANT INSULINOMA | MIRNAS | HUMAN CANCERS | ACCUMULATION | CARCINOMA | PROGRESSION | Prognosis | Pancreatic Neoplasms - diagnosis | Humans | Pancreatic Neoplasms - pathology | Carcinoma, Acinar Cell - pathology | Insulinoma - genetics | MicroRNAs - metabolism | Pancreatic Neoplasms - genetics | Carcinoma, Acinar Cell - genetics | Gene Expression Profiling | Pancreas - metabolism | Survival | Insulinoma - diagnosis | Carcinoma, Acinar Cell - diagnosis | Cell Transformation, Neoplastic | Insulinoma - pathology | Liver Neoplasms - secondary
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6. Full Text Type II enteropathy-associated T-cell lymphoma features a unique genomic profile with highly recurrent SETD2 alterations
by Roberti, Annalisa and Dobay, Maria Pamela and Bisig, Bettina and Vallois, David and Boéchat, Cloé and Lanitis, Evripidis and Bouchindhomme, Brigitte and Parrens, Marie-Cécile and Bossard, Céline and Quintanilla-Martinez, Leticia and Missiaglia, Edoardo and Gaulard, Philippe and De Leval, Laurence
Nature Communications, ISSN 2041-1723, 09/2016, Volume 7, Issue 1, p. 12602
Enteropathy-associated T-cell lymphoma (EATL), a rare and aggressive intestinal malignancy of intraepithelial T lymphocytes, comprises two disease variants... 
EPIGENETIC REGULATORS | STAT3 MUTATIONS | PROTEIN STABILITY | NK CELLS | MULTIDISCIPLINARY SCIENCES | ACUTE LYMPHOBLASTIC-LEUKEMIA | GENES | GAMMA-DELTA-T | CANCER | SOMATIC MUTATIONS | WEB SERVER | Genetic Predisposition to Disease | Histone-Lysine N-Methyltransferase - genetics | Enteropathy-Associated T-Cell Lymphoma - genetics | Intestinal Neoplasms - classification | Genomics | Humans | Mutation | Enteropathy-Associated T-Cell Lymphoma - classification | Gene Expression Regulation, Neoplastic - physiology | Intestinal Neoplasms - genetics
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7. Full Text BRAF V600E mutant colorectal cancer subtypes based on gene expression
by Barras, David and Missiaglia, Edoardo and Wirapati, Pratyaksha and Sieber, Oliver M and Jorissen, Robert N and Love, Chris and Molloy, Peter L and Jones, Ian T and McLaughlin, Stephen and Gibbs, Peter and Guinney, Justin and Simon, Iris M and Roth, Arnaud D and Bosman, Fred T and Tejpar, Sabine and Delorenzi, Mauro
Clinical Cancer Research, ISSN 1078-0432, 01/2017, Volume 23, Issue 1, pp. 104 - 115
Purpose: Mutation of BRAF at the valine 600 residue occurs in approximately 10% of colorectal cancers, a group with particularly poor prognosis. The response... 
COLON-CANCER | MOLECULAR SUBTYPES | CETUXIMAB | INHIBITION | ONCOLOGY | BRAF MUTATION | MICROSATELLITE INSTABILITY | IRINOTECAN | CLASSIFICATION | KRAS WILD-TYPE | BRAF(V600E) | Prognosis | Colorectal Neoplasms - genetics | Humans | Gene Expression Regulation, Neoplastic | Gene Expression Profiling | Biomarkers, Tumor | Colorectal Neoplasms - diagnosis | DNA Methylation | Neoplasm Metastasis | Codon | Proteomics - methods | Colorectal Neoplasms - metabolism | Proto-Oncogene Proteins B-raf - metabolism | Colorectal Neoplasms - mortality | Computational Biology - methods | Gene Expression | Signal Transduction | Kaplan-Meier Estimate | Workflow | Models, Biological | Proto-Oncogene Proteins B-raf - genetics | Mutation | Neoplasm Staging | Amino Acid Substitution | Cluster Analysis | Cohort Studies | TOR protein | Phosphorylation | Deregulation | Epidermal growth factor receptors | Colorectal carcinoma | Colorectal cancer | AKT protein | Biology | Cyclin D1 | Gene expression | Clustering | Patients | Subgroups | K-Ras protein | 1-Phosphatidylinositol 3-kinase | Experimental design | Valine | Proteomics | Cell cycle | Cancer
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8. Full Text Dual JAK1 and STAT3 mutations in a breast implant-associated anaplastic large cell lymphoma
by Letourneau, Audrey and Maerevoet, Marie and Milowich, Dina and Dewind, Roland and Bisig, Bettina and Missiaglia, Edoardo and de Leval, Laurence
Virchows Archiv, ISSN 0945-6317, 10/2018, Volume 473, Issue 4, pp. 505 - 511
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1007/s00428-018-2352-y 
Medicine & Public Health | Pathology | LEUKEMIA | PATHOLOGY | Immunohistochemistry | Lymphoma, Large-Cell, Anaplastic - therapy | Phosphorylation | Biomarkers, Tumor - analysis | Breast Implantation - instrumentation | Breast Implants - adverse effects | Humans | In Situ Hybridization, Fluorescence | STAT3 Transcription Factor - analysis | Lymphoma, Large-Cell, Anaplastic - enzymology | Positron Emission Tomography Computed Tomography | Breast Implantation - adverse effects | Janus Kinase 1 - genetics | Female | Aged | Biomarkers, Tumor - genetics | Lymphoma, Large-Cell, Anaplastic - pathology | Mutation | STAT3 Transcription Factor - genetics | Lymphoma, Large-Cell, Anaplastic - genetics | Lymphomas | Genetic aspects | Implants, Artificial | Prosthesis | Janus kinase | Anaplastic large-cell lymphoma | Lymphoma | Stat3 protein
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9. Full Text JARID2 is a direct target of the PAX3-FOXO1 fusion protein and inhibits myogenic differentiation of rhabdomyosarcoma cells
by Walters, Z.S and Villarejo-Balcells, B and Olmos, D and Buist, T.W.S and Missiaglia, E and Allen, R and Al-Lazikani, B and Garrett, M.D and Blagg, J and Shipley, J
Oncogene, ISSN 0950-9232, 02/2014, Volume 33, Issue 9, pp. 1148 - 1157
Rhabdomyosarcomas (RMS) are the most frequent soft-tissue sarcoma in children and characteristically show features of developing skeletal muscle. The alveolar... 
histone methylation | polycomb repressive complex 2 | PAX3-FOXO1 | JARID2 | rhabdomyosarcoma | myogenic differentiation | BIOCHEMISTRY & MOLECULAR BIOLOGY | 13-CIS-RETINOIC ACID | HISTONE DEMETHYLASES | CHILDRENS ONCOLOGY GROUP | CARDIAC DEVELOPMENT | CELL BIOLOGY | ONCOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | ALVEOLAR RHABDOMYOSARCOMA | HIGH-RISK NEUROBLASTOMA | TRANSCRIPTION FACTOR | REPRESSIVE COMPLEX 2 | MUSCLE STEM-CELLS | Paired Box Transcription Factors - genetics | Oncogene Proteins, Fusion - metabolism | Cell Proliferation | Polycomb Repressive Complex 2 - genetics | Humans | Rhabdomyosarcoma - pathology | Myogenin - genetics | Histone Demethylases - genetics | Promoter Regions, Genetic - genetics | Cell Differentiation - genetics | Tumor Suppressor Proteins - genetics | Nuclear Proteins - genetics | Gene Expression Regulation, Neoplastic - genetics | Tumor Suppressor Proteins - metabolism | Nuclear Proteins - metabolism | Transcription Factors - genetics | Enhancer of Zeste Homolog 2 Protein | Transcription Factors - metabolism | Histone Demethylases - metabolism | Oncogene Proteins, Fusion - genetics | Cell Line, Tumor | Muscle Development - genetics | Rhabdomyosarcoma - genetics | Transcription, Genetic - genetics | Polycomb Repressive Complex 2 - metabolism | Myogenin - metabolism | Paired Box Transcription Factors - metabolism | Promyelocytic Leukemia Protein | Transcriptional coactivators | Care and treatment | Oncology, Experimental | Rhabdomyosarcoma | Physiological aspects | Histones | Development and progression | Research | Cell differentiation | Carcinogenesis | Cancer | DNA methylation | Pediatrics | Chromatin | Gene expression | Cancer therapies | Polycomb Repressive Complex 2
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10. Full Text MicroRNA and gene co-expression networks characterize biological and clinical behavior of rhabdomyosarcomas
by Missiaglia, Edoardo and Shepherd, Chris J and Aladowicz, Ewa and Olmos, David and Selfe, Joanna and Pierron, Gaëlle and Delattre, Olivier and Walters, Zoe and Shipley, Janet
Cancer Letters, ISSN 0304-3835, 2016, Volume 385, pp. 251 - 260
Abstract Rhabdomyosarcomas (RMS) in children and adolescents are heterogeneous sarcomas broadly defined by skeletal muscle features and the presence/absence of... 
Hematology, Oncology and Palliative Medicine | Co-expression modules | Fusion protein | Next generation sequencing | MicroRNAs | Rhabdomyosarcoma | PRIMARY TUMORS | MUSCLE-SPECIFIC MICRORNA | DOWN-REGULATION | FUSION GENE | PEDIATRIC RHABDOMYOSARCOMA | SKELETAL-MUSCLE | ONCOLOGY | EMBRYONAL RHABDOMYOSARCOMA | ALVEOLAR RHABDOMYOSARCOMA | BINDING-SITES | EXPRESSION SIGNATURE | Paired Box Transcription Factors - genetics | Oncogene Proteins, Fusion - metabolism | Humans | Gene Expression Regulation, Neoplastic | Databases, Genetic | MicroRNAs - metabolism | Gene Regulatory Networks | Rhabdomyosarcoma, Embryonal - genetics | Transfection | Rhabdomyosarcoma, Alveolar - metabolism | Biomarkers, Tumor - metabolism | Rhabdomyosarcoma, Alveolar - pathology | Rhabdomyosarcoma, Embryonal - pathology | Genetic Predisposition to Disease | Reproducibility of Results | Rhabdomyosarcoma, Alveolar - genetics | Neoplasm Invasiveness | Computational Biology | Gene Expression Profiling - methods | Gene Fusion | Phenotype | Rhabdomyosarcoma, Embryonal - metabolism | Oncogene Proteins, Fusion - genetics | Cell Line, Tumor | Biomarkers, Tumor - genetics | High-Throughput Nucleotide Sequencing | MicroRNAs - genetics | Paired Box Transcription Factors - metabolism | Cell Movement | MicroRNA | Sarcoma | Genes | Genetic research | Muscles | Metastasis | Gene expression | Cytokines | Leukemia | Genomes | Roles | Inflammation | Metabolism | Muscle contraction | Cell adhesion & migration | Children & youth | Proteins | Morphogenesis | Signal transduction | Musculoskeletal system | Cell cycle | Cytoskeleton | Extracellular matrix | Software | Localization | Systems development | Deoxyribonucleic acid--DNA | Cancer | RMS, rhabdomyosarcoma | MiRNAs, microRNAs | Original
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11. Full Text Acinar cell carcinoma of the pancreas with thyroid-like follicular features: first description of a new diagnostic challenging subtype
by Saglietti, Chiara and Schneider, Vanessa and Bongiovanni, Massimo and Missiaglia, Edoardo and Bisig, Bettina and Dorta, Gian and Demartines, Nicolas and Sempoux, Christine and La Rosa, Stefano
Virchows Archiv, ISSN 0945-6317, 12/2019, Volume 475, Issue 6, pp. 789 - 794
Acinar cell carcinomas (ACCs) of the pancreas are a heterogeneous group of neoplasms showing a wide spectrum of morphological features including acinar, solid,... 
Pathology | Acinar cell carcinoma | Thyroid-like | Medicine & Public Health | Morphology | Differential diagnosis | Pancreas | β-catenin | Phenotypes | Pancreatic carcinoma | Molecular modelling | Diagnostic systems | Thyroid gland | Mutation | Neoplasms | Thyroid | Tumors
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12. Full Text The Hippo effector TAZ (WWTR1) transforms myoblasts and TAZ abundance is associated with reduced survival in embryonal rhabdomyosarcoma
by Mohamed, Abdalla and Sun, Congshan and De Mello, Vanessa and Selfe, Joanna and Missiaglia, Edoardo and Shipley, Janet and Murray, Graeme I and Zammit, Pete S and Wackerhage, Henning
The Journal of Pathology, ISSN 0022-3417, 09/2016, Volume 240, Issue 1, pp. 3 - 14
The Hippo effector YAP has recently been identified as a potent driver of embryonal rhabdomyosarcoma (ERMS). Most reports suggest that the YAP paralogue TAZ... 
WWTR1 | myoblasts | Hippo pathway | TAZ | embryonal rhabdomyosarcoma | TRANSCRIPTION FACTORS | STEM-CELLS | SOFT-TISSUE SARCOMAS | PROTEIN | YAP | PATHOLOGY | BREAST-CANCER | MUSCLE SATELLITE CELLS | ONCOLOGY | PATHWAY | ALVEOLAR RHABDOMYOSARCOMA | SKELETAL | Prognosis | Tissue Array Analysis | Humans | Rhabdomyosarcoma - metabolism | Cell Proliferation - physiology | Rhabdomyosarcoma - pathology | Survival Rate | Intracellular Signaling Peptides and Proteins - metabolism | Myoblasts - pathology | Cell Transformation, Neoplastic - metabolism | Myoblasts - metabolism | Animals | Cell Transformation, Neoplastic - genetics | Cell Line, Tumor | Mice | Rhabdomyosarcoma - genetics | Cell Transformation, Neoplastic - pathology | Intracellular Signaling Peptides and Proteins - genetics | Rhabdomyosarcoma - mortality | Immunohistochemistry | Muscles | Physiological aspects | Analysis | Stem cells | Original Papers | Original Paper
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13. Full Text Mpn1, Mutated in Poikiloderma with Neutropenia Protein 1, Is a Conserved 3′-to-5′ RNA Exonuclease Processing U6 Small Nuclear RNA
by Shchepachev, Vadim and Wischnewski, Harry and Missiaglia, Edoardo and Soneson, Charlotte and Azzalin, Claus M
Cell Reports, ISSN 2211-1247, 10/2012, Volume 2, Issue 4, pp. 855 - 865
Clericuzio-type poikiloderma with neutropenia (PN) is a rare genodermatosis associated with mutations in the C16orf57 gene, which codes for the uncharacterized... 
IN-VITRO | 3' END | POLYMERASE-III TRANSCRIPTS | LUPUS ANTIGEN-LA | CLERICUZIO-TYPE POIKILODERMA | SIGNAL RECOGNITION PARTICLE | RECYCLING FACTOR | SM-LIKE PROTEINS | RIBONUCLEOPROTEIN-PARTICLES | SCHIZOSACCHAROMYCES-POMBE | CELL BIOLOGY | Recombinant Proteins - metabolism | Cell Line | Phosphoric Diester Hydrolases - metabolism | Neutropenia - metabolism | Humans | Exonucleases - deficiency | Phosphoric Diester Hydrolases - genetics | Recombinant Proteins - genetics | Ribonucleoprotein, U4-U6 Small Nuclear - metabolism | Schizosaccharomyces - metabolism | RNA Splicing | Skin Abnormalities - metabolism | Exonucleases - genetics | Neutropenia - pathology | Schizosaccharomyces - enzymology | RNA, Small Nuclear - metabolism | Skin Abnormalities - pathology | Exonucleases - metabolism
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