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Glia, ISSN 0894-1491, 05/2018, Volume 66, Issue 5, pp. 1098 - 1117
To elucidate mechanisms contributing to cortical pathology in multiple sclerosis (MS), we investigated neurovascular aberrations, in particular the association... 
neurovascular connections | experimental autoimmune encephalomyelitis (EAE) | inflammation | multiple sclerosis | astrocytes | glatiramer acetate (GA) | cortical damage | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | NEUROTROPHIC FACTOR | PATHOLOGY | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | CEREBRAL-CORTEX | FLOW | NEUROPROTECTION | MYELIN BASIC-PROTEIN | DISEASE | GLATIRAMER ACETATE | Capillary Permeability - physiology | Neurons - pathology | Peptide Fragments | Cerebral Cortex - blood supply | Encephalomyelitis, Autoimmune, Experimental - metabolism | Astrocytes - pathology | Cerebral Cortex - pathology | Capillary Permeability - drug effects | Cerebral Cortex - metabolism | Female | Neurons - metabolism | Cerebral Cortex - drug effects | Neurons - drug effects | Neurovascular Coupling - drug effects | Immunosuppressive Agents - pharmacology | Astrocytes - drug effects | Glatiramer Acetate - pharmacology | Encephalomyelitis, Autoimmune, Experimental - pathology | Specific Pathogen-Free Organisms | Encephalomyelitis, Autoimmune, Experimental - drug therapy | Mice, Inbred C57BL | Myelin-Oligodendrocyte Glycoprotein | Neurovascular Coupling - physiology | Blood-Brain Barrier - drug effects | Blood-Brain Barrier - metabolism | Blood-Brain Barrier - pathology | Animals | Astrocytes - metabolism | Multiple sclerosis | T cells | Neurons | Analysis | Brain | Copolymer 1 | Encephalomyelitis | Cognitive ability | Boundaries | Blood | Blood-brain barrier | Rodents | Meninges | Immunomodulation | Astrocytes | Cortex | Blood vessels | Inflammation | Substantia alba | Experimental allergic encephalomyelitis | Desmin | Endothelial cells | Cores | Detachment | Sodium | Neurological complications | Fluorescein | Pericytes | Mice | Synapses
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2005, Volume 102, Issue 52, pp. 19045 - 19050
Neurotrophins (NTs) such as BDNF, NT-3, and NT-4 are important modulators of neuronal function and survival. Their expression in the CNS after various insults... 
T lymphocytes | Biological Sciences | Nervous system diseases | Messenger RNA | Cytokines | Neurons | Central nervous system | Cell lines | Central nervous system diseases | Chronic diseases | Mice | NT-3 | NT-4 | Neurotrophins | BDNF | Immunomodulation | immunomodulation | THERAPY | MULTIPLE-SCLEROSIS | MULTIDISCIPLINARY SCIENCES | COPOLYMER 1 | NEUROGENESIS | neurotrophins | T-CELLS | NEUROPROTECTION | Immunohistochemistry | Central Nervous System - metabolism | Encephalomyelitis, Autoimmune, Experimental - immunology | Glatiramer Acetate | Neurons - cytology | Nerve Growth Factors - metabolism | Stem Cells - metabolism | Brain - metabolism | In Situ Hybridization | Neuroprotective Agents - pharmacology | Time Factors | Female | Brain-Derived Neurotrophic Factor - metabolism | Neurons - metabolism | Immunosuppressive Agents - pharmacology | Disease Models, Animal | Astrocytes - cytology | Anti-Inflammatory Agents - pharmacology | Cell Survival | Mice, Inbred C57BL | Myelin-Oligodendrocyte Glycoprotein | Reverse Transcriptase Polymerase Chain Reaction | Peptides - pharmacology | Animals | Neurotrophin 3 - metabolism | T-Lymphocytes - cytology | Myelin-Associated Glycoprotein - chemistry | Brain - pathology | Myelin Proteins | Polymers - chemistry | Mice, Inbred BALB C | Immunosuppressive Agents - chemistry | Encephalomyelitis | Health aspects | Rodents as pets
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2010, Volume 107, Issue 29, pp. 13111 - 13116
Journal Article
The EMBO Journal, ISSN 0261-4189, 11/2015, Volume 34, Issue 21, pp. 2633 - 2651
Journal Article
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 07/2008, Volume 106, Issue 2, pp. 662 - 671
Journal Article
Scientific Reports, ISSN 2045-2322, 12/2019, Volume 9, Issue 1, pp. 4140 - 15
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2003, Volume 100, Issue 24, pp. 14157 - 14162
The ability of a remedy to modulate the pathological process in the target organ is crucial for its therapeutic activity. Glatiramer acetate (GA, Copaxone,... 
T lymphocytes | Biological Sciences | Cell growth | Cytokines | Spleen cells | Neurons | Medical treatment | Central nervous system | Cell lines | Cellular immunity | Cells | IMMUNE | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | TH2 CELLS | THERAPY | MULTIPLE-SCLEROSIS | MULTIDISCIPLINARY SCIENCES | COPOLYMER 1 | CENTRAL-NERVOUS-SYSTEM | MICE | INTERLEUKIN-10 | LESIONS | Immunohistochemistry | T-Lymphocyte Subsets - immunology | Encephalomyelitis, Autoimmune, Experimental - metabolism | Humans | Encephalomyelitis, Autoimmune, Experimental - immunology | Glatiramer Acetate | Th2 Cells - immunology | Adoptive Transfer | Interferon-gamma - metabolism | Th2 Cells - drug effects | Brain - metabolism | T-Lymphocytes, Helper-Inducer - drug effects | T-Lymphocyte Subsets - drug effects | T-Lymphocytes, Helper-Inducer - immunology | Interleukin-10 - metabolism | Female | Brain-Derived Neurotrophic Factor - metabolism | Immunosuppressive Agents - pharmacology | Multiple Sclerosis - metabolism | T-Lymphocytes, Helper-Inducer - metabolism | Cytokines - metabolism | Encephalomyelitis, Autoimmune, Experimental - drug therapy | Th2 Cells - metabolism | Peptides - pharmacology | Brain - drug effects | Animals | T-Lymphocyte Subsets - metabolism | Multiple Sclerosis - immunology | Mice | Mice, Inbred BALB C | Brain - immunology | Transforming Growth Factor beta - metabolism | Multiple Sclerosis - drug therapy | T cells | Brain | Research
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2008, Volume 105, Issue 32, pp. 11358 - 11363
The interplay between demyelination and remyelination is critical in the progress of multiple sclerosis (MS) and its animal model, experimental autoimmune... 
T lymphocytes | Pathology | Nervous system diseases | Demyelinating diseases | Myelin | Oligodendroglia | Chronic diseases | Preventive medicine | Lesions | Progenitor cells | Neuroprotection | Myelin repair | Multiple sclerosis | Oligodendrocyte | multiple sclerosis | MULTIDISCIPLINARY SCIENCES | oligodendrocyte | myelin repair | MODEL | BETA | MYELIN | MULTIPLE-SCLEROSIS | neuroprotection | OLIGODENDROCYTE PROGENITORS | BIOLOGY | CENTRAL-NERVOUS-SYSTEM | MICE | T-CELLS | Oligodendroglia - metabolism | Spinal Cord - metabolism | Encephalomyelitis, Autoimmune, Experimental - metabolism | Recovery of Function - drug effects | Glatiramer Acetate | Stem Cells - metabolism | Spinal Cord - ultrastructure | Multiple Sclerosis - chemically induced | Oligodendroglia - ultrastructure | Myelin Sheath - metabolism | Encephalomyelitis, Autoimmune, Experimental - chemically induced | Axons - ultrastructure | Immunosuppressive Agents - pharmacology | Multiple Sclerosis - metabolism | Microscopy, Electron, Scanning | Encephalomyelitis, Autoimmune, Experimental - pathology | Encephalomyelitis, Autoimmune, Experimental - drug therapy | Axons - metabolism | Proteoglycans - biosynthesis | Stem Cells - ultrastructure | Peptides - pharmacology | Antigens, Differentiation - biosynthesis | Animals | Encephalomyelitis, Autoimmune, Experimental - prevention & control | Multiple Sclerosis - pathology | Antigens - biosynthesis | Cell Proliferation - drug effects | Mice | Myelin Sheath - ultrastructure | Chronic Disease | Multiple Sclerosis - drug therapy | Glatiramer acetate | Evaluation | Myelination | Encephalomyelitis | Physiological aspects | Drug therapy | Health aspects | Index Medicus | Biological Sciences
Journal Article