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Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2010, Volume 107, Issue 12, pp. 5375 - 5380
Journal Article
PLoS Pathogens, ISSN 1553-7366, 09/2016, Volume 12, Issue 9, pp. e1005857 - e1005857
Journal Article
BMC Cancer, ISSN 1471-2407, 06/2018, Volume 18, Issue 1, pp. 663 - 663
Journal Article
Journal Article
Journal Article
Journal Article
Nature Medicine, ISSN 1078-8956, 09/2014, Volume 20, Issue 10, pp. 1138 - 1146
In solid tumors, resistance to therapy inevitably develops upon treatment with cytotoxic drugs or molecularly targeted therapies. Here, we describe a system... 
PATHWAYS | MEDICINE, RESEARCH & EXPERIMENTAL | HEPATOCELLULAR-CARCINOMA | INTEGRATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENES | SENSITIVITY | KINASE INHIBITORS | MAP KINASES | JAK/STAT | STRATEGIES | CELL BIOLOGY | Niacinamide - analogs & derivatives | RNA, Small Interfering - genetics | Mitogen-Activated Protein Kinase 14 - antagonists & inhibitors | Humans | Male | Liver Neoplasms, Experimental - metabolism | Carcinoma, Hepatocellular - drug therapy | Mitogen-Activated Protein Kinase 14 - metabolism | RNA Interference | Carcinoma, Hepatocellular - genetics | Female | Antineoplastic Agents - pharmacology | Liver Neoplasms, Experimental - genetics | Liver Neoplasms - genetics | Signal Transduction | Mice, Inbred C57BL | Liver Neoplasms - drug therapy | Mitogen-Activated Protein Kinase 14 - genetics | Activating Transcription Factor 2 - metabolism | Xenograft Model Antitumor Assays | Drug Resistance, Neoplasm - genetics | Animals | MAP Kinase Signaling System - drug effects | Liver Neoplasms, Experimental - drug therapy | Liver Neoplasms - metabolism | Mice | Phenylurea Compounds - pharmacology | Protein Kinase Inhibitors - pharmacology | Niacinamide - pharmacology | Carcinoma, Hepatocellular - metabolism | Liver cancer | Research | Drug therapy | Drug resistance | Analysis | Biomarkers | Signal transduction | Protein expression | Pharmacology | Ribonucleic acid--RNA | Index Medicus
Journal Article
Journal Article
Nature, ISSN 0028-0836, 03/2012, Volume 483, Issue 7391, pp. 608 - 612
Deregulated expression of the MYC oncoprotein contributes to the genesis of many human tumours, yet strategies to exploit this for a rational tumour therapy... 
CANCER-CELLS | METABOLISM | GLUTAMINE | MITOCHONDRIAL BIOGENESIS | MULTIDISCIPLINARY SCIENCES | GROWTH | SYNTHETIC LETHAL INTERACTION | C-MYC | B KINASE | SUPPRESSION | ADDICTION | Protein Kinases - metabolism | AMP-Activated Protein Kinases - metabolism | Electron Transport | Protein Biosynthesis | Protein Kinases - genetics | TOR Serine-Threonine Kinases - metabolism | Humans | Multiprotein Complexes | Gene Expression Regulation, Neoplastic | Glutamine - metabolism | Homeostasis | Repressor Proteins - antagonists & inhibitors | Mechanistic Target of Rapamycin Complex 1 | Oncogene Protein p55(v-myc) - genetics | Carcinoma, Hepatocellular - drug therapy | Cell Respiration | RNA Interference | Repressor Proteins - deficiency | Cell Transformation, Neoplastic - genetics | Adenosine Triphosphate - metabolism | Carcinoma, Hepatocellular - genetics | Liver Neoplasms - pathology | Protein Kinases - deficiency | Repressor Proteins - metabolism | Disease Models, Animal | Oncogene Protein p55(v-myc) - metabolism | Liver Neoplasms - genetics | Signal Transduction | Cell Survival | Liver Neoplasms - drug therapy | Repressor Proteins - genetics | Mitochondria - metabolism | Animals | Proteins - metabolism | Genes, myc - genetics | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Line, Tumor | Doxycycline - pharmacology | Mice | Proteins - antagonists & inhibitors | Apoptosis | Carcinoma, Hepatocellular - metabolism | Physiological aspects | Cellular signal transduction | Genetic aspects | Research | Drug therapy | Protein kinases | Tumors | Proteins | Liver cancer | Cell growth | Phosphorylation | Automation | Protein synthesis | Dependence | Glucose | Kinases | Autophagy | Index Medicus
Journal Article