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Annals of the American Thoracic Society, ISSN 2325-6621, 02/2019, Volume 16, Issue 2, pp. 175 - 181
Journal Article
JAMA, ISSN 0098-7484, 02/2016, Volume 315, Issue 7, pp. 672 - 681
Journal Article
Clinics in Chest Medicine, ISSN 0272-5231, 09/2016, Volume 37, Issue 3, pp. 505 - 511
Hermansky-Pudlak syndrome (HPS) is an autosomal recessive disorder that is associated with oculocutaneous albinism, bleeding diatheses, granulomatous colitis,... 
Biogenesis of lysosome-related organelle complex | Pulmonary fibrosis | Adaptor protein 3 | Albinism | Hermansky-Pudlak syndrome | Interstitial lung disease | MUTATION SCREEN REVEALS | FORM | SUSCEPTIBILITY | COLITIS | RESPIRATORY SYSTEM | INFLAMMATION | MOUSE MODEL | SYNDROME TYPE-2 | LUNG-DISEASE | PULMONARY-FIBROSIS | IMMUNODEFICIENCY | Epistaxis - etiology | Hemorrhagic Disorders - physiopathology | Pulmonary Veins - abnormalities | Gastrointestinal Diseases - physiopathology | Humans | Gastrointestinal Diseases - etiology | Hypertension, Pulmonary - physiopathology | Telangiectasis - physiopathology | Epistaxis - physiopathology | Arteriovenous Malformations - physiopathology | Intracranial Arteriovenous Malformations - physiopathology | Albinism, Oculocutaneous - physiopathology | Hemorrhagic Disorders - complications | Liver Diseases - etiology | Intracranial Arteriovenous Malformations - etiology | Crohn Disease - etiology | Pulmonary Fibrosis - etiology | Albinism - physiopathology | Blood Coagulation Disorders - etiology | Arteriovenous Malformations - etiology | Pulmonary Artery - abnormalities | Albinism - complications | Albinism, Oculocutaneous - etiology | Telangiectasis - etiology | Hermanski-Pudlak Syndrome - complications | Pulmonary Fibrosis - physiopathology | Hermanski-Pudlak Syndrome - physiopathology | Crohn Disease - physiopathology | Liver Diseases - physiopathology | Hypertension, Pulmonary - etiology | Blood Coagulation Disorders - physiopathology
Journal Article
Journal of Heart and Lung Transplantation, ISSN 1053-2498, 2017, Volume 36, Issue 8, pp. 833 - 834
Journal Article
PLoS ONE, ISSN 1932-6203, 03/2015, Volume 10, Issue 3, p. e0121246
Background Epithelial cell death is a major contributor to fibrogenesis in the lung. In this study, we sought to determine the function of mitochondria and... 
PARKIN | RECRUITMENT | ACTIVATION | DISEASES | MULTIDISCIPLINARY SCIENCES | DAMAGED MITOCHONDRIA | DEGRADATION | MITOPHAGY | AUTOPHAGY | PINK1-DEFICIENT MICE | PROMOTES | Protein Kinases - metabolism | Cell Line | Gene Expression Regulation, Enzymologic - drug effects | Pulmonary Alveoli - pathology | Reactive Oxygen Species - metabolism | Epithelial Cells - drug effects | Humans | Epithelial Cells - pathology | Male | Pulmonary Fibrosis - pathology | Bleomycin - adverse effects | Mitochondria - drug effects | Mitochondria - pathology | Membrane Potential, Mitochondrial - drug effects | Animals | Mitochondrial Degradation - drug effects | Pulmonary Fibrosis - enzymology | Pulmonary Fibrosis - chemically induced | Mice | Cell Death - drug effects | Transforming Growth Factor beta1 - pharmacology | Cell death | Analysis | Fibrosis | Bone morphogenetic proteins | Respiratory tract diseases | Mitochondrial DNA | B cells | Transforming growth factors | Oxidative stress | Flow cytometry | Epithelial cells | Homeostasis | Critical care | Kinases | Autophagy | Experiments | Medical schools | Western blotting | Depolarization | Mitochondria | Bleomycin | Rodents | Animal tissues | Alveoli | Growth factors | Transforming growth factor-b1 | Mortality | Lung diseases | Gene expression | Electron microscopy | Medicine | Cytometry | Hospitals | Transmission electron microscopy | Pulmonary fibrosis | Lungs | Womens health | PTEN-induced putative kinase | Laboratory animals | Alzheimers disease | Immunofluorescence | PTEN protein | Apoptosis
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 09/2017, Volume 57, Issue 3, pp. 272 - 279
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 11/2015, Volume 125, Issue 11, pp. 4255 - 4268
Lung transplantation is the only viable option for patients suffering from otherwise incurable end-stage pulmonary diseases such as chronic obstructive... 
GRAFT-REJECTION | MEDICINE, RESEARCH & EXPERIMENTAL | BRONCHIOLITIS OBLITERANS SYNDROME | IN-VIVO | GROWTH FACTOR-C | GENE-THERAPY | HYALURONAN RECEPTOR LYVE-1 | TRANSPLANT RECIPIENTS | LYMPHATIC ENDOTHELIAL-CELLS | PULMONARY-FIBROSIS | T-CELLS | Molecular Weight | Humans | Immunosuppressive Agents - therapeutic use | Glycoproteins - metabolism | Male | Forced Expiratory Volume | Lung Transplantation | Allografts | Lung - metabolism | Vascular Endothelial Growth Factor Receptor-3 - drug effects | Vascular Endothelial Growth Factor C - therapeutic use | Graft Rejection - drug therapy | Acute Disease | Endothelial Cells - metabolism | Graft Rejection - physiopathology | Graft Rejection - therapy | Mice, Inbred C57BL | Hyaluronic Acid - chemistry | Lung - physiopathology | Vascular Endothelial Growth Factor Receptor-3 - metabolism | Vascular Endothelial Growth Factor C - genetics | Lymphangiogenesis - drug effects | Animals | Homeodomain Proteins - analysis | Hyaluronic Acid - metabolism | Lung - drug effects | Lymphatic Vessels - pathology | Protein Binding | Mice | Mice, Inbred BALB C | Lymphatic Vessels - surgery | Mutation | Prednisone - therapeutic use | Tumor Suppressor Proteins - analysis | Vascular Endothelial Growth Factor C - pharmacology | Prevention | Complications and side effects | Lungs | Transplantation | Neovascularization | Health aspects | Graft rejection | Organ transplant recipients | Hyaluronic acid | Vascular endothelial growth factor | Immunotherapy | Lung diseases
Journal Article
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 08/2016, Volume 55, Issue 2, pp. 252 - 263
Aging has been implicated in the development of pulmonary fibrosis, which has seen a sharp increase in incidence in those older than 50 years. Recent studies... 
Aging; bleomycin; mitochondrial oxidative stress; NLRP3 inflammasome; pulmonary fibrosis | bleomycin | LUNG FIBROSIS | OXIDATIVE STRESS | COLONY-STIMULATING FACTOR | DENDRITIC CELLS | mitochondrial oxidative stress | BIOCHEMISTRY & MOLECULAR BIOLOGY | INJURY | NLRP3 inflammasome | NALP3 INFLAMMASOME | CELL BIOLOGY | RESPIRATORY SYSTEM | pulmonary fibrosis | ALVEOLAR MACROPHAGES | MICE | aging | GROWTH-FACTOR | Lung - pathology | Lung Injury - pathology | Disease Susceptibility | Inflammasomes - metabolism | Instillation, Drug | NLR Family, Pyrin Domain-Containing 3 Protein - metabolism | Pulmonary Fibrosis - complications | Mice, Inbred C57BL | Male | Mitochondria - metabolism | Pulmonary Fibrosis - pathology | Mitochondria - drug effects | Aging - pathology | NLR Family, Pyrin Domain-Containing 3 Protein - deficiency | Transforming Growth Factor beta - pharmacology | Macrophages - metabolism | Animals | Bleomycin | Interleukin-1beta - metabolism | Pulmonary Fibrosis - prevention & control | Lung Injury - complications | Lung Injury - prevention & control | Lung - metabolism | Interleukin-18 - metabolism | Oxidative stress | Laboratories | Pathogenesis | Neutrophils | Inflammation | Mitochondrial DNA | Gene expression | Studies | Pulmonary fibrosis | Rodents | Aging | Bone marrow | Extracellular matrix | Growth factors | Age | Apoptosis | Original Research
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 06/2019, Volume 60, Issue 6, pp. 659 - 666
Journal Article
The New England Journal of Medicine, ISSN 0028-4793, 03/2018, Volume 378, Issue 13, p. e18
This interactive case features a 43-year-old woman who presented with a 2-week history of chest pain, cough, and dyspnea. Test your diagnostic and therapeutic... 
Journal Article
Journal of Heart and Lung Transplantation, ISSN 1053-2498, 2017, Volume 36, Issue 7, pp. 763 - 769
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 02/2018, Volume 58, Issue 2, pp. 208 - 215
Radiation-induced pulmonary fibrosis is a severe complication of patients treated with thoracic irradiation. We have previously shown that syndecan-2 reduces... 
Pulmonary fibrosis | Fibroblast | Myofibroblast | Syndecan-2 | Radiation | TRANSCRIPTION FACTORS | LUNG FIBROSIS | ROCK | myofibroblast | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | MECHANISMS | syndecan-2 | BETA | CELL BIOLOGY | IN-VITRO | fibroblast | radiation | RESPIRATORY SYSTEM | pulmonary fibrosis | SERUM RESPONSE FACTOR | RHO | MYOFIBROBLAST DIFFERENTIATION | Syndecan-2 - metabolism | Radiation Injuries - mortality | Mice, Inbred C57BL | Thorax - radiation effects | Mice, Transgenic | Phosphatidylinositol 3-Kinases - metabolism | Pulmonary Fibrosis - pathology | Cell Movement - physiology | Myofibroblasts - cytology | Myofibroblasts - metabolism | Animals | rho-Associated Kinases - metabolism | Receptor-Like Protein Tyrosine Phosphatases, Class 3 - metabolism | Syndecan-2 - genetics | Female | Radiation Injuries - pathology | Cell Proliferation - drug effects | Mice | Proto-Oncogene Proteins c-akt - metabolism | Transforming Growth Factor beta - metabolism | Cell Proliferation - radiation effects | Cell proliferation | Transcription factors | Pathogenesis | Transforming growth factor | Smooth muscle | AKT protein | Kinases | Macrophages | Contraction | Cell growth | Actin | Transgenic animals | Rodents | Fibroblasts | Alveoli | Growth factors | Threonine | Mortality | Lung diseases | Transgenic mice | Thorax | Radiation therapy | Gene expression | 1-Phosphatidylinositol 3-kinase | Cell death | Collagen | Fibrosis | Serum response factor | Protein-serine/threonine kinase | Cell migration | Syndecan | Apoptosis | Original Research
Journal Article