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Nephrology Dialysis Transplantation, ISSN 0931-0509, 12/2010, Volume 25, Issue 12, pp. 3890 - 3897
Background. Patients with IgA nephropathy (IgAN) have an increased amount of abnormally O -glycosylated IgA1 in circulation, in glomerular deposits and... 
the surface IgA1-positive human B-cell line | IgA1 hinge region | Th2 response | T-cell cytokines | Original
Journal Article
Antimicrobial Agents and Chemotherapy, ISSN 0066-4804, 08/2017, Volume 61, Issue 8
Antibiotics excreted into the intestinal tract may disrupt the microbiota that provide colonization resistance against enteric pathogens and alter normal... 
Intestinal microbiota | intestinal microbiota | CLOSTRIDIUM-DIFFICILE INFECTION | ENTEROLACTONE | BACTERIA | METABOLISM | MOUSE | HUMANS | MICROBIOLOGY | PHARMACOLOGY & PHARMACY | PATHOGENS | Index Medicus
Journal Article
Kidney International, ISSN 0085-2538, 05/2010, Volume 77, Issue 10, pp. 921 - 927
Journal Article
NEPHROLOGY DIALYSIS TRANSPLANTATION, ISSN 0931-0509, 12/2010, Volume 25, Issue 12, pp. 3890 - 3897
Background. Patients with IgA nephropathy (IgAN) have an increased amount of abnormally O-glycosylated IgA1 in circulation, in glomerular deposits and produced... 
the surface IgA1-positive human B-cell line | IMMUNE-COMPLEXES | Th2 response | NEPHROPATHY | BLOOD MONONUCLEAR-CELLS | MASS-SPECTROMETRY | TRANSPLANTATION | B-CELLS | GALACTOSYLTRANSFERASE ACTIVITY | SELF-AGGREGATION | MESSENGER-RNA | HINGE REGION | MESANGIAL CELLS | UROLOGY & NEPHROLOGY | IgA1 hinge region | T-cell cytokines
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2013, Volume 8, Issue 11, pp. e78736 - e78736
The link between glomerular IgA nephropathy (IgAN) and T helper 2 (Th2) response has been implicated, however, the mechanisms are poorly defined because of the... 
IMMUNE-COMPLEXES | GLOMERULONEPHRITIS | COMPLEMENT | ACTIVATION | TGF-BETA | MULTIDISCIPLINARY SCIENCES | O-GLYCOSYLATION | RENAL-DISEASE | DIFFERENTIATION | KNOCKOUT MICE | MURINE B-CELLS | Glomerulonephritis, IGA - genetics | Galactosyltransferases - genetics | Mice, 129 Strain | Smad4 Protein - genetics | T-Lymphocytes - metabolism | Glomerular Mesangium - metabolism | Female | Cytokines - blood | Immunoglobulin A - metabolism | Cytokines - metabolism | Enzyme-Linked Immunosorbent Assay | Isoenzymes - genetics | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Signal Transduction - genetics | Glomerular Mesangium - ultrastructure | Microscopy, Electron | Reverse Transcriptase Polymerase Chain Reaction | Smad4 Protein - metabolism | Th2 Cells - metabolism | Mice, Knockout | Gene Expression Regulation, Enzymologic | Glomerular Mesangium - pathology | Animals | Immunoglobulin A - blood | Glomerulonephritis, IGA - metabolism | Mice | Glomerulonephritis, IGA - blood | Cytokines | RNA | Analysis | Immunoglobulin G | Albumin | B cells | Kidney diseases | T cells | Immunoglobulin A | Pediatrics | Animal models | Nephrology | Pathogenesis | Homology | Lymphocytes T | mRNA | Gene deletion | Smad4 protein | IgA nephropathy | Clonal deletion | Lymphocytes | Rodents | Deletion | Creatinine | Phenotypes | Immunoglobulins | Internal medicine | Medicine | Pathology | Signaling | Lymphocytes B | Lck protein | University faculty | Index Medicus
Journal Article
Biology Direct, ISSN 1745-6150, 01/2012, Volume 7, Issue 1, pp. 3 - 3
Journal Article
Journal Article
Journal of Innate Immunity, ISSN 1662-811X, 04/2013, Volume 5, Issue 3, pp. 231 - 241
Journal Article
Journal Article