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Molecular medicine (Cambridge, Mass.), ISSN 1076-1551, 05/2018, Volume 24, Issue 1, pp. 19 - 10
High mobility group box 1 (HMGB1) protein is a central endogenous inflammatory mediator contributing to the pathogenesis of several inflammatory disorders.... 
Alarmin | Inflammation | HMGB1 | Receptor | Protein-protein interactions | TLR2 | Cell Line | Leukocytes, Mononuclear - metabolism | Animals | Cytokines - metabolism | HMGB1 Protein - metabolism | Humans | Protein Domains | Ligands | Mice | Toll-Like Receptor 2 - metabolism
Journal Article
Journal of Innate Immunity, ISSN 1662-811X, 06/2018, Volume 10, Issue 3, pp. 215 - 227
Background: Neuroinflammation triggered by infection or trauma is the cause of central nervous system dysfunction. High-mobility group box 1 protein (HMGB1),... 
Research Article | Redox | Brain | Neuroinflammation | HMGB1 | Blood-brain barrier | NERVOUS-SYSTEM | MOBILITY | GROUP BOX-1 HMGB1 | ALARMIN HMGB1 | INJURY | NLRP3 INFLAMMASOME | IMMUNOLOGY | RECEPTOR 4 | PROINFLAMMATORY CYTOKINE | DANGER SIGNALS | ISCHEMIC BRAIN-DAMAGE
Journal Article
Science Translational Medicine, ISSN 1946-6234, 03/2018, Volume 10, Issue 432, p. eaao1313
Stroke induces a multiphasic systemic immune response, but the consequences of this response on atherosclerosis-a major source of recurrent vascular... 
MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | RECRUITMENT | APOPTOSIS | MYOCARDIAL-INFARCTION | INFLAMMATION | BONE-MARROW | HMGB1 | E-DEFICIENT MICE | ISCHEMIC-STROKE | HEMATOPOIETIC STEM | CELL BIOLOGY
Journal Article
by Olga Lomakina and Ekaterina Alekseeva and Sania Valieva and Tatiana Bzarova and Irina Nikishina and Elena Zholobova and Svetlana Rodionovskaya and Maria Kaleda and Yasuo Nakagishi and Masaki Shimizu and Mao Mizuta and Akihiro Yachie and Yuko Sugita and Nami Okamoto and Kousuke Shabana and Takuji Murata and Hiroshi Tamai and Eve M. Smith and Peng Yin and Andrea L. Jorgensen and Michael W. Beresford and on behalf of On behalf of the UK JSLE Cohort Study and Eve M. Smith and Antonio Eleuteri and Beatrice Goilav and Laura Lewandowski and Angel Phuti and Dawn Wahezi and Tamar Rubinstein and Caroline Jones and Paul Newland and Stephen Marks and Rachel Corkhill and Diana Ekdawy and Clarissa Pilkington and Kjell Tullus and Chaim Putterman and Chris Scott and Antony C. Fisher and Michael W. Beresford and Eve M. Smith and Laura Lewandowski and Angel Phuti and Andrea Jorgensen and Chris Scott and Michael W. Beresford and Ezgi Deniz Batu and Can Kosukcu and Ekim Taskiran and Sema Akman and Kubra Ozturk and Betul Sozeri and Erbil Unsal and Zelal Ekinci and Yelda Bilginer and Mehmet Alikasifoglu and Seza Ozen and Hanna Lythgoe and Michael W. Beresford and Hermine I. Brunner and Gaurav Gulati and Jordan T. Jones and Mekibib Altaye and Jamie Eaton and Mark Difrancesco and Joo Guan Yeo and Jingyao Leong and Loshinidevi D/O Thana Bathi and Thaschawee Arkachaisri and Salvatore Albani and Nagla Abdelrahman and Michael W Beresford and Valentina Leone and UK JSLE study group supported by the National Institute of Health Research Clinical Research Network and Noortje Groot and D. Shaikhani and I. E. M. Bultink and M. Bijl and R. J. E. M. Dolhain and Y. K. O. Teng and E. Zirkzee and K. de Leeuw and R. Fritsch-Stork and S. S. M. Kamphuis and Rachael D. Wright and Eve M. Smith and Michael W. Beresford and Reem Abdawani and Laila Al Shaqshi and Ibrahim Al Zakwani and Natali W. Gormezano and David Kern and Oriany L. Pereira and Gladys C. C. Esteves and Adriana M. Sallum and Nadia E. Aikawa and Rosa M. Pereira and Clovis A. Silva and Eloisa Bonfa and Jessica Beckmann and ...
PEDIATRIC RHEUMATOLOGY, ISSN 1546-0096, 05/2017, Volume 15, Issue S1, pp. 105 - 201
Journal Article
by Lomakina, Olga and Alekseeva, Ekaterina and Valieva, Sania and Bzarova, Tatiana and Nikishina, Irina and Zholobova, Elena and Rodionovskaya, Svetlana and Kaleda, Maria and Nakagishi, Yasuo and Shimizu, Masaki and Mizuta, Mao and Yachie, Akihiro and Sugita, Yuko and Okamoto, Nami and Shabana, Kousuke and Murata, Takuji and Tamai, Hiroshi and Smith, Eve M and Yin, Peng and Jorgensen, Andrea L and Beresford, Michael W and Eleuteri, Antonio and Goilav, Beatrice and Lewandowski, Laura and Phuti, Angel and Wahezi, Dawn and Rubinstein, Tamar and Jones, Caroline and Newland, Paul and Marks, Stephen and Corkhill, Rachel and Ekdawy, Diana and Pilkington, Clarissa and Tullus, Kjell and Putterman, Chaim and Scott, Chris and Fisher, Antony C and Jorgensen, Andrea and Batu, Ezgi Deniz and Kosukcu, Can and Taskiran, Ekim and Akman, Sema and Ozturk, Kubra and Sozeri, Betul and Unsal, Erbil and Ekinci, Zelal and Bilginer, Yelda and Alikasifoglu, Mehmet and Ozen, Seza and Lythgoe, Hanna and Brunner, Hermine I and Gulati, Gaurav and Jones, Jordan T and Altaye, Mekibib and Eaton, Jamie and Difrancesco, Mark and Yeo, Joo Guan and Leong, Jingyao and Bathi, Loshinidevi D/O Thana and Arkachaisri, Thaschawee and Albani, Salvatore and Abdelrahman, Nagla and Beresford, Michael W and Leone, Valentina and Groot, Noortje and Shaikhani, D and Bultink, I. E. M and Bijl, M and Dolhain, R. J. E. M and Teng, Y. K. O and Zirkzee, E and de Leeuw, K and Fritsch-Stork, R and Kamphuis, S. S. M and Wright, Rachael D and Abdawani, Reem and Al Shaqshi, Laila and Al Zakwani, Ibrahim and Gormezano, Natali W and Kern, David and Pereira, Oriany L and Esteves, Gladys C. C and Sallum, Adriana M and Aikawa, Nadia E and Pereira, Rosa M and Silva, Clovis A and Bonfa, Eloisa and Beckmann, Jessica and Bartholomä, Nora and Venhoff, Nils and Henneke, Philipp and Salzer, Ulrich and Janda, Ales and Boteanu, Alina Lucica and Corral, Sandra Garrote and Giraldo, Alberto Sifuentes and Gámir, Mariluz Gámir and Mendoza, Antonio Zea and Adrovic, Amra and Dedeoglu, Reyhan and ... and Single Hub and Access point for paediatric Rheumatology in Europe (SHARE) and Juvenile Dermatomyositis Research Group and European Federation of Immunological Societies and UK JSLE study group supported by the National Institute of Health Research Clinical Research Network and on behalf of Juvenile Dermatomyositis Research Group (JDRG) and on behalf of PRINTO and Eurofever Project and on behalf of Italian Pediatric Rheumatology Study Group and PANLAR Pediatric Rheumatology Study Group and ICON study group and on behalf of On behalf of the UK JSLE Cohort Study and the Nordic Study Group of Pediatric Rheumatology (NoSPeR) and on behalf of PRINTO and PRCSG and on behalf of the UK Juvenile Dermatomyositis Research Group (JDRG) and BIKER collaborative group and on behalf of PANLAR Pediatric Rheumatology Study Group and GRIP study group and Juvenile Inflammatory Rheumatism (JIR) Cohort and CAPS and GRANT Medical University, Sofia 68/2015 and Drug Delivery Group and for the CARRA Registry Investigators and on behalf of PRINTO and Eurofever Registry and Juvenile Dermatomyositis Research Group (JDRG) and Nordic Study Group of Pediatric Rheumatology (NoSPeR)
Pediatric Rheumatology, ISSN 1546-0096, 05/2017, Volume 15, Issue S1
Journal Article
Annals of Neurology, ISSN 0364-5134, 04/2017, Volume 81, Issue 4, pp. 572 - 582
Objective Surgery launches a systemic inflammatory reaction that reaches the brain and associates with immune activation and cognitive decline. Although... 
POSITRON-EMISSION-TOMOGRAPHY | MICROGLIAL ACTIVATION | PROTEIN 18 KDA | TRANSLOCATOR PROTEIN | IN-VIVO | POSTOPERATIVE COGNITIVE DYSFUNCTION | BENZODIAZEPINE-RECEPTORS | NEUROFILAMENT LIGHT | CEREBROSPINAL-FLUID | NEUROSCIENCES | VIVO RADIOLIGAND BINDING | CLINICAL NEUROLOGY | Biomarkers - metabolism | Brain - diagnostic imaging | Gray Matter - immunology | Follow-Up Studies | Brain - physiopathology | Down-Regulation | Humans | Middle Aged | Male | Positron-Emission Tomography - methods | Prostatectomy - adverse effects | Cognitive Dysfunction - etiology | Cognitive Dysfunction - physiopathology | Abdomen - surgery | Gray Matter - diagnostic imaging | Brain - metabolism | Gray Matter - metabolism | Gray Matter - physiopathology | Aged | Brain - immunology | Neuroimaging | Brain | Correlation | Cognitive ability | Emission | Inflammatory response | Stimulation | Activation | Males | Substantia grisea | Blood | Lipopolysaccharides | Signal transduction | Cell activation | Surgery | Imaging | Tomography | Anesthesia | Standard deviation | Bioindicators | Activation analysis | Immune system | Binding | Abdominal surgery | Immune response | Color | Positron emission | T cell receptors | Inflammation | Patients | Tumor necrosis factor | Launches | Biomarkers | Immunoreactivity | Positron emission tomography | in-vivo | Neurosciences | postoperative cognitive dysfunction | Neurosciences & Neurology | neurofilament light | cerebrospinal-fluid | protein 18 kda | translocator protein | positron-emission-tomography | benzodiazepine-receptors | vivo radioligand binding | Neurovetenskaper | microglial activation
Journal Article
International Journal of Rheumatic Diseases, ISSN 1756-1841, 01/2017, Volume 20, Issue 1, pp. 25 - 32
Aims In rheumatoid arthritis (RA), pain and inflammation are initial symptoms followed by various degrees of bone and cartilage destruction. Previously, we... 
ubiquitin proteasome system | pain | spinal cord | substance P | rheumatoid arthritis | RHEUMATOID-ARTHRITIS | SYSTEM | ACTIVATION | GENE-RELATED PEPTIDE | TISSUE | SPINAL-CORD | RHEUMATOLOGY | SUBSTANCE-P | NF-KAPPA-B | TRANSCRIPTION FACTOR | EXPRESSION | Arthritis, Experimental - drug therapy | Arthritis, Experimental - genetics | Spinal Cord - drug effects | Rats, Inbred Lew | NF-kappa B - metabolism | Arthritis, Experimental - microbiology | Pain - microbiology | Ganglia, Spinal - enzymology | Proteasome Endopeptidase Complex - drug effects | Pain - enzymology | Female | Binding Sites | Mycobacterium | Pain - prevention & control | Proteasome Inhibitors - pharmacology | Anti-Inflammatory Agents - pharmacology | Spinal Cord - enzymology | DNA - metabolism | Leupeptins - pharmacology | Animals | Substance P - genetics | Substance P - metabolism | Arthritis, Experimental - enzymology | Proteasome Endopeptidase Complex - metabolism | Ganglia, Spinal - drug effects | Immunohistochemistry | Ubiquitin | Rheumatoid factor | RNA | Analysis | Arthritis | Gene expression | DNA structure | NF-κB protein | Spinal cord | Substance P | Reverse transcription | mRNA | Inflammation | Ganglia | Polymerase chain reaction | Cartilage | Dorsal root ganglia | Pain | Rheumatoid arthritis | Rodents | Medical and Health Sciences | Medicin och hälsovetenskap
Journal Article
Journal Article
Frontiers in Immunology, ISSN 1664-3224, 10/2016, Volume 7, p. 441
Postoperative neurocognitive disorders are common complications in elderly patients following surgery or critical illness. High mobility group box 1 protein... 
Aging | Inflammation | HMGB1 | Memory | Surgery | Microglia | memory | STERILE INFLAMMATION | MURINE SEPSIS SURVIVORS | RELEASE | IMMUNOLOGY | BLOOD-BRAIN-BARRIER | RECEPTOR 4 | COGNITIVE DECLINE | inflammation | MOBILITY GROUP BOX-1 | MICE | aging | PROTEIN HMGB1 | microglia | surgery | Monoclonal antibodies | Aged patients | Chromosomal proteins | Health aspects | Ageing
Journal Article
Arthritis Research and Therapy, ISSN 1478-6354, 11/2015, Volume 17, Issue 1, p. 338
Journal Article