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Circulation Research, ISSN 0009-7330, 03/2015, Volume 116, Issue 6, pp. 960 - 975
Blockers of the renin–angiotensin–aldosterone system (RAAS), that is, renin inhibitors, angiotensin (Ang)-converting enzyme (ACE) inhibitors, Ang II type 1... 
remodeling | gender | aldosterone | AT2 receptor | hyperkalemia | resistant hypertension | receptor | renin | primary aldosteronism | reactive oxygen species | angiotensin | angiotensin-(1-7) | prorenin | angiotensin-(1–7) | CARDIAC & CARDIOVASCULAR SYSTEMS | BLOOD-PRESSURE RESPONSES | PLASMA-RENIN | CONVERTING ENZYME-INHIBITORS | CORONARY VASCULAR BED | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | II TYPE-2 RECEPTOR | HEMATOLOGY | MYOCARDIAL-INFARCTION | AT 2 receptor | CELL MINERALOCORTICOID RECEPTORS | SEX-DIFFERENCES | CONGESTIVE-HEART-FAILURE | Antihypertensive Agents - pharmacology | Drugs, Investigational - pharmacology | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Humans | Drugs, Investigational - therapeutic use | Endothelium, Vascular - drug effects | Hypertension - drug therapy | Male | Ion Channels - physiology | Sex Chromosomes | Molecular Targeted Therapy | Gonadal Steroid Hormones - physiology | Vasoconstriction - physiology | Drug Interactions | Angiotensin-Converting Enzyme Inhibitors - therapeutic use | Hyperaldosteronism - physiopathology | Female | Hypertension - genetics | Drug Evaluation, Preclinical | Drug Resistance | Precision Medicine | Aldosterone - physiology | Endothelium, Vascular - physiopathology | Hyperaldosteronism - genetics | Mice, Transgenic | Renin-Angiotensin System - genetics | Renin-Angiotensin System - physiology | Clinical Trials as Topic | Sex Characteristics | Antihypertensive Agents - therapeutic use | Vasoconstriction - drug effects | Hyperaldosteronism - drug therapy | Hypertension - physiopathology | Mice, Knockout | Mineralocorticoid Receptor Antagonists - therapeutic use | Animals | Renin-Angiotensin System - drug effects | Mice | Therapeutic Equivalency
Journal Article
Journal Article
Journal Article
Hypertension, ISSN 0194-911X, 10/2010, Volume 56, Issue 4, pp. 675 - 681
Journal Article
Journal Article
Journal Article
Hypertension, ISSN 0194-911X, 08/2011, Volume 58, Issue 2, pp. 295 - 302
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2011, Volume 6, Issue 8, p. e23411
textabstractMedial degeneration is a key feature of aneurysm disease and aortic dissection. In a murine aneurysm model we investigated the structural and... 
PATHOGENESIS | ARTERIAL | GROWTH-FACTOR-BETA | RENIN | MULTIDISCIPLINARY SCIENCES | MARFAN-SYNDROME | MOUSE MODEL | SMOOTH-MUSCLE-CELLS | MECHANISMS | MEDIAL DEGENERATION | EXPRESSION | Immunohistochemistry | Angiotensin II - genetics | Receptor, Angiotensin, Type 1 - physiology | Oligonucleotide Array Sequence Analysis | Extracellular Matrix Proteins - deficiency | Humans | Transcriptome | Male | Mice, 129 Strain | Angiotensin II Type 1 Receptor Blockers - pharmacology | Vasoconstriction - physiology | Phenylephrine - pharmacology | Smad2 Protein - genetics | Female | Aorta, Thoracic - pathology | Aortic Aneurysm - prevention & control | Animals, Newborn | Vasoconstriction - genetics | Vasoconstrictor Agents - pharmacology | Angiotensin II - pharmacology | Angiotensin II - metabolism | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Aortic Aneurysm - physiopathology | Losartan - pharmacology | Smad2 Protein - metabolism | Aorta, Thoracic - metabolism | Aorta, Thoracic - physiopathology | Vasoconstriction - drug effects | Mice, Knockout | Pregnancy | Animals | Transforming Growth Factor beta - genetics | Mice | Aortic Aneurysm - genetics | In Vitro Techniques | Transforming Growth Factor beta - metabolism | Analysis | Elastin | Angiotensin | Physiological aspects | Aneurysms | Bone morphogenetic proteins | Transforming growth factors | Health aspects | Medicine, Preventive | Preventive health services | Phosphorylation | Marfan's syndrome | Disease | Pathogenesis | Genomics | Medical services | Aneurysm | Smooth muscle | Biology | Insulin-like growth factors | Genomes | Marfan syndrome | Fragmentation | Renin | Rodents | Smad2 protein | Network analysis | Angiotensin AT1 receptors | Extracellular matrix | Aorta | Degeneration | Angiotensin II | Cardiology | Dissection | Bioinformatics | Hypertension | Vascular surgery | Internal medicine | Medical treatment | Contractility | Muscles | Media | Pharmacology | Gene expression | Muscle contraction | Medicine | Pathology | Signaling | Life span | Protein synthesis | Coronary vessels | Mutation | Laboratory animals | Cancer | Structure-function relationships
Journal Article
Journal of Hypertension, ISSN 0263-6352, 12/2009, Volume 27, Issue 12, pp. 2297 - 2309
Inhibition of angiogenesis with humanized monoclonal antibodies to vascular endothelial growth factor (VEGF) or with tyrosine kinase inhibitors targeting VEGF... 
Hypertension | Vvascular endothelial growth factor | Renal disease | Angiogenesis inhibition | Cardiac disease | Proteinuria | SUNITINIB TREATMENT | DEPENDENT RELAXATION | proteinuria | COHERENCE TOMOGRAPHY FINDINGS | cardiac disease | BEVACIZUMAB AVASTIN(R) | TYROSINE-KINASE INHIBITOR | renal disease | BLOOD-PRESSURE | INTRAVITREAL INJECTION | angiogenesis inhibition | PHASE-I TRIAL | vascular endothelial growth factor | PERIPHERAL VASCULAR DISEASE | hypertension | ENDOTHELIAL GROWTH-FACTOR | MOLECULAR-MECHANISMS | Kidney Diseases - physiopathology | Humans | Antibodies, Monoclonal - adverse effects | Endothelium, Vascular - drug effects | Vascular Endothelial Growth Factor A - metabolism | Protein Kinase Inhibitors - adverse effects | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Vascular Endothelial Growth Factor A - genetics | Hypertension - physiopathology | Hypertension - metabolism | Gene Expression Regulation - drug effects | Receptors, Vascular Endothelial Growth Factor - metabolism | Signal Transduction - drug effects | Endothelium, Vascular - metabolism | Hypertension - chemically induced | Kidney Diseases - chemically induced | Receptors, Vascular Endothelial Growth Factor - antagonists & inhibitors | Angiogenesis Inhibitors - adverse effects | Kidney Diseases - metabolism | Index Medicus
Journal Article
American Journal of Physiology - Renal Physiology, ISSN 0363-6127, 2014, Volume 306, Issue 10, pp. F1179 - F1189
Journal Article
Hypertension, ISSN 0194-911X, 09/2012, Volume 60, Issue 3, pp. 722 - 729
Angiotensin II type 2 (AT2) receptor stimulation has been linked to vasodilation. Yet, AT2 receptor-independent hypertension and hypotension (or no effect on... 
coronary circulation | mouse | compound 21 | AT | receptor | rat | vasorelaxation | angiotensin II | Langendorff model | METABOLITES | STIMULATION | RELAXATION | AT receptor | MEDIATED VASODILATION | CORONARY VASCULAR BED | NATRIURESIS | AT RECEPTORS | NONPEPTIDE AGONIST | PERIPHERAL VASCULAR DISEASE | VASOCONSTRICTION | Mesenteric Arteries - physiology | Tetrazoles - pharmacology | Iliac Artery - drug effects | Rats, Wistar | Calcium - metabolism | Humans | Middle Aged | Receptor, Angiotensin, Type 2 - physiology | Receptor, Angiotensin, Type 2 - drug effects | Endothelium, Vascular - drug effects | Male | Mesenteric Arteries - drug effects | Angiotensin II Type 2 Receptor Blockers - pharmacology | Endothelium, Vascular - physiology | Phenylephrine - pharmacology | Biphenyl Compounds - pharmacology | Adult | Calcium Ionophores - pharmacology | Female | Models, Animal | Vasodilation - physiology | Rats, Inbred SHR | Vasoconstrictor Agents - pharmacology | Receptor, Angiotensin, Type 2 - genetics | Coronary Vessels - drug effects | Coronary Vessels - physiology | Mice, Inbred C57BL | Rats | Thiophenes - pharmacology | Imidazoles - pharmacology | Sulfonamides - pharmacology | Iliac Artery - physiology | Mice, Knockout | Animals | 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid - pharmacology | Mice | Pyridines - pharmacology | Vasodilation - drug effects | In Vitro Techniques
Journal Article