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Journal Article
Neurology, ISSN 0028-3878, 12/2014, Volume 83, Issue 24, pp. 2239 - 2246
Journal Article
Journal Article
Cell Reports, 03/2018, Volume 22, Issue 12, p. 3375
(Cell Reports 22, 1522–1530; February 6, 2018) In the originally published version of this article, ALKs were incorrectly defined as “anaplastic lymphoma... 
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2013, Volume 8, Issue 8, p. e71084
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2017, Volume 114, Issue 47, pp. 12448 - 12453
The TGF-beta family ligands myostatin, GDF11, and activins are negative regulators of skeletal muscle mass, which have been reported to primarily signal via... 
Myostatin | ActRII | Activin | Dual antibody | Hypertrophy | CANCER CACHEXIA | GDF11 | COMPLEX | activin | hypertrophy | MULTIDISCIPLINARY SCIENCES | INCREASES | TGF-BETA SUPERFAMILY | myostatin | PROOF-OF-CONCEPT | MASS | MICE | FOLLISTATIN | dual antibody | Prevention | Activins | Cellular signal transduction | Research | Atrophy, Muscular | Biological Sciences
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2017, Volume 114, Issue 47, p. 12448
The TGF-β family ligands myostatin, GDF11, and activins are negative regulators of skeletal muscle mass, which have been reported to primarily signal via the... 
Immunoglobulins | Regulators | Antibodies | Muscles | Blocking | Cachexia | Gene expression | X ray analysis | Skeletal muscle | Eutrophication | Mode of action | Signal transduction | Signaling | Receptors | Myostatin | Ligands | Skeletal system | Mice | Transduction | Activin | Binding sites | Hypertrophy | Neutralization
Journal Article
Journal of the American College of Cardiology, ISSN 0735-1097, 03/2018, Volume 71, Issue 11, pp. A2663 - A2663
Methods Targeted proteomic analyses of plasma samples from the Framingham Heart Study (n=899) and older adults with severe aortic stenosis (n=50) revealed... 
Heart failure | Aging | Ligands | Activin | Heart diseases | Older people
Journal Article
Journal of cachexia, sarcopenia and muscle, ISSN 2190-5991, 08/2019
The majority of patients with advanced cancer develop cachexia, a weight loss syndrome that severely reduces quality of life and limits survival. Our... 
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 02/2014, Volume 34, Issue 4, pp. 573 - 573
Journal Article
Science Translational Medicine, ISSN 1946-6234, 2019, Volume 11, Issue 482, p. eaau8680
Activin type II receptor (ActRII) ligands have been implicated in muscle wasting in aging and disease. However, the role of these ligands and ActRII signaling... 
MEDICINE, RESEARCH & EXPERIMENTAL | GDF11 | HYPERTROPHY | INHIBITION | MYOSTATIN | TRANSCRIPTION ACTIVATION | DYSFUNCTION | SERCA2A | FOLLISTATIN | BINDING | AGE | CELL BIOLOGY
Journal Article
Cell Reports, ISSN 2211-1247, 02/2018, Volume 22, Issue 6, pp. 1522 - 1530
The age-related effects of GDF11 have been a subject of controversy. Here, we find that elevated GDF11 causes signs of cachexia in mice: reduced food intake,... 
GDF11 | Activin A | ActRII | GDF15 | cachexia | atrophy | skeletal muscle | myostatin | TGFbeta | SKELETAL-MUSCLE | HYPERTROPHY | MASS | MYOSTATIN | DIFFERENTIATION FACTOR 11 | WEIGHT-LOSS | TGF-BETA SUPERFAMILY | RECEPTOR | IDENTIFICATION | AGE | CELL BIOLOGY
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 07/2012, Volume 32, Issue 14, pp. 2871 - 2879
Journal Article
Skeletal Muscle, ISSN 2044-5040, 07/2016, Volume 6, Issue 1, p. 26
Journal Article