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animals (6) 6
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cells (3) 3
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article (2) 2
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genes (2) 2
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animals; base sequence; bone neoplasms/genetics; bone neoplasms/metabolism; cell line, tumor; chromatin assembly and disassembly; enhancer elements, genetic; gene expression regulation, neoplastic; humans; mice, inbred nod; mice, scid; neoplasm transplantation; oncogene proteins, fusion/physiology; protein binding; proto-oncogene protein c-fli-1/physiology; rna-binding protein ews/physiology; sarcoma, ewing/genetics; sarcoma, ewing/metabolism (1) 1
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1. Full Text Transient expression of Bcl6 is sufficient for oncogenic function and induction of mature B-cell lymphoma
by Green, Michael R and Vicente-Dueñas, Carolina and Romero-Camarero, Isabel and Long Liu, Chih and Dai, Bo and González-Herrero, Inés and García-Ramírez, Idoia and Alonso-Escudero, Esther and Iqbal, Javeed and Chan, Wing C and Campos-Sanchez, Elena and Orfao, Alberto and Pintado, Belén and Flores, Teresa and Blanco, Oscar and Jiménez, Rafael and Martínez-Climent, Jose Angel and Criado, Francisco Javier García and Cenador, María Begoña García and Zhao, Shuchun and Natkunam, Yasodha and Lossos, Izidore S and Majeti, Ravindra and Melnick, Ari and Cobaleda, César and Alizadeh, Ash A and Sánchez-García, Isidro
Nature Communications, ISSN 2041-1723, 06/2014, Volume 5, Issue 1, p. 3904
Diffuse large B-cell lymphoma (DLBCL) is the most common lymphoma and can be separated into two subtypes based upon molecular features with similarities to... 
MOLECULAR SUBTYPES | INTEGRATIVE ANALYSIS REVEALS | STEM-CELLS | DNA METHYLATION | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | CHRONIC LYMPHOCYTIC-LEUKEMIA | NON-HODGKIN-LYMPHOMA | MYELOID-LEUKEMIA | SOMATIC MUTATION | COPY-NUMBER ALTERATION | Doxorubicin - therapeutic use | Prognosis | Epigenesis, Genetic | Humans | Gene Expression Regulation, Neoplastic | Male | Lymphoma, Large B-Cell, Diffuse - metabolism | Cyclophosphamide - therapeutic use | DNA Copy Number Variations | DNA-Binding Proteins - metabolism | DNA Methylation | Female | B-Lymphocytes - metabolism | Lymphoma, Large B-Cell, Diffuse - drug therapy | Mice, Transgenic | Hematopoietic Stem Cells - metabolism | DNA-Binding Proteins - genetics | Phenotype | Animals | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Vincristine - therapeutic use | Mice | Lymphoma, Large B-Cell, Diffuse - genetics | Prednisone - therapeutic use | Antibodies, Monoclonal, Murine-Derived - therapeutic use | Proto-Oncogene Proteins c-bcl-6
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2. Full Text A novel molecular mechanism involved in multiple myeloma development revealed by targeting MafB to haematopoietic progenitors
by Vicente‐Dueñas, Carolina and Romero‐Camarero, Isabel and González‐Herrero, Inés and Alonso‐Escudero, Esther and Abollo‐Jiménez, Fernando and Jiang, Xiaoyu and Gutierrez, Norma C and Orfao, Alberto and Marín, Nieves and Villar, Luisa María and Criado, Ma Carmen Fernández and Pintado, Belén and Flores, Teresa and Alonso‐López, Diego and De Las Rivas, Javier and Jiménez, Rafael and Criado, Francisco Javier García and Cenador, María Begoña García and Lossos, Izidore S and Cobaleda, César and Sánchez‐García, Isidro
The EMBO Journal, ISSN 0261-4189, 09/2012, Volume 31, Issue 18, pp. 3704 - 3717
Understanding the cellular origin of cancer can help to improve disease prevention and therapeutics. Human plasma cell neoplasias are thought to develop from... 
oncogenes | cancer therapy | reprogramming stem cells | MafB | multiple myeloma mouse model | STEM-CELLS | SIGNALING PATHWAYS | TUMOR-DEVELOPMENT | BIOCHEMISTRY & MOLECULAR BIOLOGY | BONE-MARROW | C-MAF | PLASMA-CELLS | CELL BIOLOGY | IN-VIVO | EXPRESSION | CHROMOSOMAL TRANSLOCATION | LYMPHOBLASTIC-LEUKEMIA | Translocation, Genetic | MafB Transcription Factor - metabolism | Gene Library | Antigens, CD34 - biosynthesis | Epigenesis, Genetic | Humans | Gene Expression Regulation, Neoplastic | In Situ Hybridization, Fluorescence | Mice, Transgenic | Gene Expression Profiling | Multiple Myeloma - metabolism | DNA Methylation | Animals | Hematopoietic Stem Cells - cytology | Antigens, Ly - metabolism | Membrane Proteins - metabolism | Mice | DNA, Complementary - metabolism | B-Lymphocytes - metabolism | Multiple Myeloma - genetics | Bone marrow | Oncology | Cellular biology | Molecular biology | Cancer
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3. Full Text Expression of MALT1 oncogene in hematopoietic stem/progenitor cells recapitulates the pathogenesis of human lymphoma in mice
by Carolina Vicente-Dueñas and Lorena Fontán and Ines Gonzalez-Herrero and Isabel Romero-Camarero and Victor Segura and M. Angela Aznar and Esther Alonso-Escudero and Elena Campos-Sanchez and Lucía Ruiz-Roca and Marcos Barajas-Diego and Ainara Sagardoy and Jose I. Martinez-Ferrandis and Fernando Abollo-Jimenez and Cristina Bertolo and Ivan Peñuelas and Francisco J. Garcia-Criado and María B. García-Cenador and Thomas Tousseyn and Xabier Agirre and Felipe Prosper and Federico Garcia-Bragado and Ellen D. McPhail and Izidore S. Lossos and Ming-Qing Du and Teresa Flores and Jesus M. Hernandez-Rivas and Marcos Gonzalez and Antonio Salar and Beatriz Bellosillo and Eulogio Conde and Reiner Siebert and Xavier Sagaert and Cesar Cobaleda and Isidro Sanchez-Garcia and Jose A. Martinez-Climent
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2012, Volume 109, Issue 26, pp. 10534 - 10539
Chromosomal translocations involving the MALT1 gene are hallmarks of mucosa-associated lymphoid tissue (MALT) lymphoma. To date, targeting these translocations... 
Lymphocytes | B lymphocytes | Genes | B cell lymphoma | Stem cells | Mucosa associated lymphoid tissue lymphoma | Signatures | Lymphoma | Hematopoietic stem cells | Tumors | Tumor reprogramming | Molecular-directed therapy | Cancer stem cells | Transgenic mice | MOLECULAR SUBTYPES | transgenic mice | GENE | TISSUE LYMPHOMAS | ABNORMALITIES | MULTIDISCIPLINARY SCIENCES | molecular-directed therapy | STEM-CELL | cancer stem cells | tumor reprogramming | Animals | Caspases - genetics | Humans | Lymphoma - pathology | Mice, Transgenic | NF-kappa B - metabolism | Transcription, Genetic | Hematopoietic Stem Cells - metabolism | Mice | Mucosa-Associated Lymphoid Tissue Lymphoma Translocation 1 Protein | Neoplasm Proteins - genetics | Oncogenes | Development and progression | Lymphomas | Genetic aspects | Research | Properties | Cancer | Biological Sciences
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4. Full Text Lineage-specific function of Engrailed-2 in the progression of chronic myelogenous leukemia to T-cell blast crisis
by Abollo-Jiménez, Fernando and Campos-Sánchez, Elena and Toboso-Navasa, Amparo and Vicente-Dueñas, Carolina and González-Herrero, Inés and Alonso-Escudero, Esther and González, Marcos and Segura, Víctor and Blanco, Óscar and Martínez-Climent, José Ángel and Sánchez-García, Isidro and Cobaleda, César
Cell Cycle, ISSN 1538-4101, 06/2014, Volume 13, Issue 11, pp. 1717 - 1726
In hematopoietic malignancies, oncogenic alterations interfere with cellular differentiation and lead to tumoral development. Identification of the proteins... 
DNA methylation | chronic myelogenous leukemia | T-cell development | engrailed-2 | blast crisis | Chronic myelogenous leukemia | Engrailed-2 | Blast crisis | METHYLATION | EN-2 | SCHLAFEN | DELETION | FAMILY | CELL BIOLOGY | ONCOGENE | HEMATOPOIESIS | GENES | CPG ISLANDS | EXPRESSION | Homeodomain Proteins - metabolism | Humans | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - genetics | Mice, Transgenic | DNA Methylation - genetics | DNA Primers - genetics | Reverse Transcriptase Polymerase Chain Reaction | Nerve Tissue Proteins - genetics | Disease Progression | Homeodomain Proteins - genetics | Nerve Tissue Proteins - metabolism | Cell Differentiation - immunology | Animals | Flow Cytometry | Microarray Analysis | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | CpG Islands - genetics | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | T-Lymphocytes - immunology | Mice | Blast Crisis - metabolism | Gene Expression Regulation, Neoplastic - physiology | Gene Expression Regulation, Neoplastic - genetics | Report
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5. Full Text A novel molecular mechanism involved in multiple myeloma development revealed by targeting MafB to haematopoietic progenitors
: MafB in MM pathogenesis
by Vicente-Dueñas, Carolina and Romero-Camarero, Isabel and González-Herrero, Inés and Alonso-Escudero, Esther and Abollo-Jiménez, Fernando and Jiang, Xiaoyu and Gutierrez, Norma C and Orfao, Alberto and Marín, Nieves and Villar, Luisa María and Criado, Ma Carmen Fernández and Pintado, Belén and Flores, Teresa and Alonso-López, Diego and De Las Rivas, Javier and Jiménez, Rafael and Criado, Francisco Javier García and Cenador, María Begoña García and Lossos, Izidore S and Cobaleda, César and Sánchez-García, Isidro
The EMBO Journal, ISSN 0261-4189, 09/2012, Volume 31, Issue 18, pp. 3704 - 3717
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6. Expression of MALT1 oncogene in hematopoietic stem/progenitor cells recapitulates the pathogenesis of human lymphoma in mice
by Carolina Vicente-Dueñas and Lorena Fontán and Ines Gonzalez-Herrero and Isabel Romero-Camarero and Victor Segura and M Angela Aznar and Esther Alonso-Escudero and Elena Campos-Sanchez and Lucía Ruiz-Roca and Marcos Barajas-Diego and Ainara Sagardoy and Jose I Martinez-Ferrandis and Fernando Abollo-Jimenez and Cristina Bertolo and Ivan Peñuelas and Francisco J Garcia-Criado and María B García-Cenador and Thomas Tousseyn and Xabier Agirre and Felipe Prosper and Federico Garcia-Bragado and Ellen D McPhail and Izidore S Lossos and Ming-Qing Du and Teresa Flores and Jesus M Hernandez-Rivas and Marcos Gonzalez and Antonio Salar and Beatriz Bellosillo and Eulogio Conde and Reiner Siebert and Xavier Sagaert and Cesar Cobaleda and Isidro Sanchez-Garcia and Jose A Martinez-Climent
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 06/2012, Volume 109, Issue 26, p. 10534
Chromosomal translocations involving the MALT1 gene are hallmarks of mucosa-associated lymphoid tissue (MALT) lymphoma. To date, targeting these translocations... 
Proteins | Pathogenesis | Rodents | Lymphomas | Gene expression | Cells
Journal Article
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7. Full Text The age of the target cell affects B-cell leukaemia malignancy
by Vicente-Dueñas, Carolina and Abollo-Jiménez, Fernando and Ruiz-Roca, Lucía and Alonso-Escudero, Esther and Jiménez, Rafael and Cenador, María Begoña García and Criado, Francisco Javier García and Cobaleda, César and Sánchez-García, Isidro
Aging, ISSN 1945-4589, 2010, Volume 2, Issue 12, pp. 908 - 913
The incidence, malignancy and treatment resistance of many types of human B-cell leukaemias (B-ALL) are directly related to patient age. A major obstacle to... 
Target cell | B-cell leukaemia | Age | BCR-ABL | target cell | ACUTE LYMPHOBLASTIC-LEUKEMIA | HEMATOPOIETIC STEM-CELLS | CHILDHOOD LEUKEMIA | MICE | PRECURSORS | age | CELL BIOLOGY | Leukemia, Experimental - pathology | Cell Line | Leukemia, B-Cell - metabolism | Age Factors | Cell Survival | Neoplasm Invasiveness | Humans | Leukemia, B-Cell - pathology | Mice, Transgenic | Fusion Proteins, bcr-abl - genetics | Animals | Leukemia, B-Cell - genetics | Neoplastic Stem Cells - metabolism | Cellular Senescence | Bone Marrow Transplantation | Genes, abl | Neoplastic Stem Cells - pathology | Mice | Fusion Proteins, bcr-abl - metabolism | Leukemia, Experimental - metabolism | Leukemia, Experimental - genetics
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8. Full Text EWS-FLI1 Utilizes Divergent Chromatin Remodeling Mechanisms to Directly Activate or Repress Enhancer Elements in Ewing Sarcoma
by Riggi, Nicolò and Knoechel, Birgit and Gillespie, Shawn M and Rheinbay, Esther and Boulay, Gaylor and Suvà, Mario L and Rossetti, Nikki E and Boonseng, Wannaporn E and Oksuz, Ozgur and Cook, Edward B and Formey, Aurélie and Patel, Anoop and Gymrek, Melissa and Thapar, Vishal and Deshpande, Vikram and Ting, David T and Hornicek, Francis J and Nielsen, G. Petur and Stamenkovic, Ivan and Aryee, Martin J and Bernstein, Bradley E and Rivera, Miguel N
Cancer Cell, ISSN 1535-6108, 11/2014, Volume 26, Issue 5, pp. 668 - 681
The aberrant transcription factor EWS-FLI1 drives Ewing sarcoma, but its molecular function is not completely understood. We find that EWS-FLI1 reprograms gene... 
CELLS | IN-VITRO | ONCOGENE | FUSION | GENE | ONCOLOGY | TRANSCRIPTION | MICROSATELLITES | DIFFERENTIATION | LINEAGE | CANCER | CELL BIOLOGY | Neoplasm Transplantation | Sarcoma, Ewing - metabolism | Humans | Gene Expression Regulation, Neoplastic | Chromatin Assembly and Disassembly | Proto-Oncogene Protein c-fli-1 - physiology | Mice, SCID | Bone Neoplasms - metabolism | Oncogene Proteins, Fusion - physiology | Animals | Enhancer Elements, Genetic | Base Sequence | RNA-Binding Protein EWS - physiology | Cell Line, Tumor | Protein Binding | Mice, Inbred NOD | Bone Neoplasms - genetics | Sarcoma, Ewing - genetics | Medical colleges | Chromatin | Sport-utility vehicles | Sarcoma | Population genetics | Stem cells
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9. Full Text Coexpression of p-IGF-1R and MMP-7 Modulates Panitumumab and Cetuximab Efficacy in RAS Wild-Type Metastatic Colorectal Cancer Patients
by Alonso, Vicente and Escudero, Pilar and Fernández-Martos, Carlos and Salud, Antonia and Méndez, José-Carlos and Méndez, Miguel and Gallego, Javier and Rodriguez, Jose-R and Martín-Richard, Marta and Fernández-Plana, Julen and Manzano, Hermini and Zanui, Monserrat and Falcó, Esther and Gil-Raga, Mireia and Rojo, Federico and Cuatrecasas, Miriam and Feliu, Jaime and García-Albéniz, Xabier and Maurel, Joan
Neoplasia, ISSN 1476-5586, 07/2018, Volume 20, Issue 7, pp. 678 - 686
The coexpression of pIGF-1R and MMP-7 (double-positive phenotype, DP) correlates with poor overall survival (OS) in wild-type (WT) (exon 2) metastatic... 
MOLECULAR SUBTYPES | FACTOR BIOAVAILABILITY | INSULIN | CELLS | PROTEINASE ACTIVITY | ONCOLOGY | STAT3 | GROWTH-FACTOR BINDING-PROTEIN-3 | FACTOR-I RECEPTOR | IGF-1 RECEPTOR | MATRIX METALLOPROTEINASE-7 | Immunohistochemistry | Irinotecan | Genotype & phenotype | Phenotypes | Colorectal carcinoma | Colorectal cancer | Clinical trials | Insulin-like growth factors | Metastasis | Survival | Matrilysin | Metastases
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10. Full Text Microvesicles in Atherosclerosis and Angiogenesis: From Bench to Bedside and Reverse
by Badimon, Lina and Suades, Rosa and Arderiu, Gemma and Peña, Esther and Chiva-Blanch, Gemma and Padró, Teresa
Frontiers in cardiovascular medicine, ISSN 2297-055X, 2017, Volume 4, p. 77
Atherosclerosis (AT) is a progressive chronic disease involving lipid accumulation, fibrosis, and inflammation in medium and large-sized arteries, and it is... 
Chronic diseases | Causes of | Development and progression | Care and treatment | Cardiovascular diseases | Atherosclerosis | atherosclerosis | cell-derived microvesicles | cardiovascular diseases | inflammation | endothelial dysfunction | angiogenesis
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11. Full Text Oral chemotherapy in colorectal cancer treatment: review of the literature
by Kurtz, Jean-Emmanuel and Andrès, Emmanuel and Natarajan-Amé, Shanti and Noel, Esther and Dufour, Patrick
2003, Volume 14, Issue 1
Recent advances in anticancer treatment have focused on the development of oral anticancer agents with the intention of improving the patients' quality of life... 
Oral chemotherapy | Treatment | Colorectal cancer
Book Review
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