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1. Full Text Dullard/Ctdnep1 Regulates Endochondral Ossification via Suppression of TGF‐β Signaling
by Hayata, Tadayoshi and Yoichi and Ezura and Asashima, Makoto and Nishinakamura, Ryuichi and Noda, Masaki
Journal of Bone and Mineral Research, ISSN 0884-0431, 02/2015, Volume 30, Issue 2, pp. 318 - 329
ABSTRACT Transforming growth factor (TGF)‐β signaling plays critical roles during skeletal development and its excessive signaling causes genetic diseases of... 
ENDOCHONDRAL OSSIFICATION | DULLARD/CTDNEP1 | TGF‐β | CHONDROCYTES | PRX1‐CRE | TGF-β | Dullard/Ctdnep1 | PRX1-CRE | Endochondral ossification | Chondrocytes | OSTEOBLAST DIFFERENTIATION | DULLARD | AORTIC-ANEURYSM | WEILL-MARCHESANI-SYNDROME | CHONDROCYTE DIFFERENTIATION | CARTILAGE | CTDNEP1 | TGF | GROWTH-FACTOR-BETA | IN-VITRO | BONE-FORMATION | ENDOCRINOLOGY & METABOLISM | MUTATIONS | SKELETAL DEVELOPMENT | Signal Transduction | Cartilage - pathology | Homeodomain Proteins - metabolism | Phosphoprotein Phosphatases - metabolism | Male | Mesoderm - cytology | Cartilage - metabolism | Mice, Knockout | Animals | Limb Buds - metabolism | Calcification, Physiologic | Sternum - pathology | Integrases - metabolism | Cell Differentiation | Mesoderm - metabolism | Chondrogenesis | Transforming Growth Factor beta - metabolism | Osteogenesis
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2. Full Text Interleukin-1[beta] Suppresses the Transporter Genes Ank and Ent1 Expression in Stromal Progenitor Cells Retaining Mineralization
by Ezura, Yoichi and Lin, Xin and Hatta, Arina and Izu, Yayoi and Noda, Masaki
Calcified Tissue International, ISSN 0171-967X, 08/2016, Volume 99, Issue 2, p. 199
To access, purchase, authenticate, or subscribe to the full-text of this article, please visit this link: http://dx.doi.org/10.1007/s00223-016-0139-1 
Phosphates | Anopheles | Interleukins | Genes | Stem cells | Genetic research | Nucleosides | Gene expression | Mineralization | Cells
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3. Full Text Dullard/Ctdnep1 Regulates Endochondral Ossification via Suppression of TGF-[beta] Signaling
by Hayata, Tadayoshi and Ezura, Yoichi and Asashima, Makoto and Nishinakamura, Ryuichi and Noda, Masaki
Journal of Bone and Mineral Research, ISSN 0884-0431, 05/2015, Volume 30, Issue 5, p. 947
Transforming growth factors | Bone morphogenetic proteins
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4. Full Text Collagens VI and XII form complexes mediating osteoblast interactions during osteogenesis
by Izu, Yayoi and Ezura, Yoichi and Koch, Manuel and Birk, David E and Noda, Masaki
Cell and Tissue Research, ISSN 0302-766X, 6/2016, Volume 364, Issue 3, pp. 623 - 635
Bone formation is precisely regulated by cell-cell communication in osteoblasts. We have previously demonstrated that genetic deletion of Col6a1 or Col12a1... 
Human Genetics | Primary osteoblasts | Biomedicine | Proteomics | Collagen VI | Molecular Medicine | Communicating cell network | Collagen XII | Osteogenesis | APOPTOSIS | BETHLEM MYOPATHY | SPLICE VARIANTS | DEFICIENCY | CELL BIOLOGY | MUSCULAR-DYSTROPHIES | MICE | MUTATIONS | DYSFUNCTION | EXPRESSION | COMMUNICATION | Collagen Type I - metabolism | Cells, Cultured | Collagen Type VI - metabolism | Cell Communication | Extracellular Matrix - metabolism | Animals | Culture Media - pharmacology | Protein Binding | Collagen Type XII - metabolism | Mice | Osteoblasts - cytology | Osteoblasts - metabolism | Bones | Density | Collagen | Analysis | Studies | Genes | Mutation | Cells | Muscular dystrophy | Regular
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5. Full Text Beta Adrenergic Receptor Stimulation Suppresses Cell Migration in Association with Cell Cycle Transition in Osteoblasts—Live Imaging Analyses Based on FUCCI System
by Katsumura, Sakie and Ezura, Yoichi and Izu, Yayoi and Shirakawa, Jumpei and Miyawaki, Atsushi and Harada, Kiyoshi and Noda, Masaki
Journal of Cellular Physiology, ISSN 0021-9541, 02/2016, Volume 231, Issue 2, pp. 496 - 504
Osteoporosis affects over 20 million patients in the United States. Among those, disuse osteoporosis is serious as it is induced by bed‐ridden conditions in... 
Recombinant Proteins - metabolism | Osteoblasts - drug effects | Cells, Cultured | Adrenergic beta-Agonists - pharmacology | Mice, Transgenic | Parathyroid Hormone - pharmacology | Recombinant Proteins - genetics | Cell Movement - drug effects | Cell Cycle Checkpoints | Animals | Isoproterenol - pharmacology | Luminescent Proteins - genetics | Mice | Osteoblasts - cytology | Osteoblasts - metabolism | Receptors, Adrenergic, beta - metabolism | Single-Cell Analysis | Luminescent Proteins - metabolism | Ubiquitin | Osteoporosis | Image processing | Parathyroid hormone | Cell cycle | Imaging systems | Equipment and supplies | Genetic engineering
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6. Full Text PTH-Induced Osteoblast Proliferation Requires Upregulation of the Ubiquitin-Specific Peptidase 2 (Usp2) Expression
by Shirakawa, Jumpei and Shirakawa, Jumpei and Harada, Hiroyuki and Harada, Hiroyuki and Noda, Masaki and Noda, Masaki and Ezura, Yoichi and Ezura, Yoichi
Calcified Tissue International, ISSN 0171-967X, 3/2016, Volume 98, Issue 3, pp. 306 - 315
Osteoporosis is a common disease that increases individual’s fragility fracture risk. PTH is the only therapeutic agent for severe osteoporosis that requires... 
Life Sciences | Biochemistry, general | PTH | Orthopedics | Osteoblast | Usp2 | Proliferation | Endocrinology | Cell Biology | IGF-I | RECEPTOR | INTERLEUKIN-11 | OSTEOPOROSIS | MECHANICAL-STRESS | ENDOCRINOLOGY & METABOLISM | CELL-CYCLE | GROWTH-FACTOR | DIFFERENTIATION | BONE | PROTEASE | Cyclin D1 - metabolism | Up-Regulation | Cell Proliferation | Alternative Splicing | Signal Transduction | Gene Expression Regulation | Mice, Transgenic | Animals | Parathyroid Hormone - metabolism | Bone and Bones - metabolism | Mice | Osteoblasts - cytology | Ubiquitin-Specific Proteases - metabolism | 3T3 Cells | RNA, Small Interfering - metabolism | Ubiquitin | Enzymes | Osteoporosis | Parathyroid hormone | Genetic research | Genetic engineering | Gene expression | Health aspects | Therapy | Fractures | Peptides
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7. Full Text Lgr4 Expression in Osteoblastic Cells Is Suppressed by Hydrogen Peroxide Treatment
by Pawaputanon Na Mahasarakham, Chantida and Izu, Yayoi and Nishimori, Katsuhiko and Izumi, Yuichi and Noda, Masaki and Ezura, Yoichi
Journal of Cellular Physiology, ISSN 0021-9541, 07/2017, Volume 232, Issue 7, pp. 1761 - 1766
LGR4 is expressed in bone and has been shown to be involved in bone metabolism. Oxidative stress is one of the key issues in pathophysiology of osteoporosis.... 
OXIDATIVE STRESS | PHYSIOLOGY | TGF-BETA | INHIBITION | PROTEIN | GENE | INVOLVEMENT | CATENIN | RECEPTOR | CELL BIOLOGY | Hydrogen Peroxide - toxicity | Cell Line | Transcription, Genetic - drug effects | Receptors, G-Protein-Coupled - metabolism | Osteoblasts - drug effects | RNA, Messenger - genetics | Alkaline Phosphatase - metabolism | Down-Regulation - drug effects | RNA, Messenger - metabolism | Animals | Time Factors | Mice | Receptors, G-Protein-Coupled - genetics | Protein Synthesis Inhibitors - pharmacology | Osteoblasts - metabolism | Cytokines - pharmacology | RNA, Small Interfering - metabolism | Bone morphogenetic proteins | Protein biosynthesis | Hydrogen peroxide | Index Medicus
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8. Full Text Angiotensin II Type 2 Receptor Blockade Increases Bone Mass
by Yayoi Izu and Fumitaka Mizoguchi and Aya Kawamata and Tadayoshi Hayata and Testuya Nakamoto and Kazuhisa Nakashima and Tadashi Inagami and Yoichi Ezura and Masaki Noda
Journal of Biological Chemistry, ISSN 0021-9258, 02/2009, Volume 284, Issue 8, pp. 4857 - 4864
Renin angiotensin system (RAS) regulates circulating blood volume and blood pressure systemically, whereas RAS also plays a role in the local milieu. Previous... 
CELLS | OSTEOPONTIN | RESORPTION | MARROW | SYMPATHETIC-NERVOUS-SYSTEM | BIOCHEMISTRY & MOLECULAR BIOLOGY | OSTEOBLASTS | PARATHYROID-HORMONE | DIFFERENTIATION | EXPRESSION | OSTEOPOROSIS | Vasoconstrictor Agents - pharmacology | Bone Development - drug effects | Bone Development - physiology | Humans | Male | Organ Size - physiology | Renin-Angiotensin System - physiology | Imidazoles - pharmacology | Osteoclasts - cytology | Osteoclasts - metabolism | Mice, Knockout | Organ Size - drug effects | Animals | Receptor, Angiotensin, Type 2 - metabolism | Bone and Bones - metabolism | Angiotensin II Type 2 Receptor Blockers | Renin-Angiotensin System - drug effects | Blood Pressure - drug effects | Blood Pressure - physiology | Mice | Pyridines - pharmacology | Osteoblasts - cytology | Osteoblasts - metabolism | Bone and Bones - cytology
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9. Full Text Erratum to: Collagens VI and XII form complexes mediating osteoblast interactions during osteogenesis
by Izu, Yayoi and Ezura, Yoichi and Koch, Manuel and Birk, David E and Noda, Masaki
Cell and Tissue Research, ISSN 0302-766X, 6/2016, Volume 364, Issue 3, pp. 677 - 679
Human Genetics | Molecular Medicine | Biomedicine | Proteomics | Erratum
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10. Full Text TGF- Suppresses Ift88 Expression in Chondrocytic ATDC5 Cells
by Kawasaki, M and Ezura, Y and Hayata, T and Notomi, T and Izu, Y and Noda, M
JOURNAL OF CELLULAR PHYSIOLOGY, ISSN 0021-9541, 11/2015, Volume 230, Issue 11, pp. 2788 - 2795
Ift88 is an intraflagella transport protein, critical for the cilium, and has been shown to be required for the maintenance of chondrocytes and cartilage.... 
PHYSIOLOGY | ARTICULAR-CARTILAGE | SIGNALS | DIFFERENTIATION | INTRAFLAGELLAR TRANSPORT | PRIMARY CILIUM | CHONDROGENESIS | CELL BIOLOGY
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11. Full Text Dullard/Ctdnep1 Regulates Endochondral Ossification via Suppression of TGF-[beta] Signaling
by Hayata, Tadayoshi and Yoichi, Tadayoshi and Ezura, Tadayoshi and Asashima, Makoto and Nishinakamura, Ryuichi and Noda, Masaki
Journal of Bone and Mineral Research, ISSN 0884-0431, 02/2015, Volume 30, Issue 2, p. 318
Transforming growth factor (TGF)-[beta] signaling plays critical roles during skeletal development and its excessive signaling causes genetic diseases of... 
Bone morphogenetic proteins | Dysplasia | RNA | Transforming growth factors
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12. Full Text FGF Suppresses Poldip2 Expression in Osteoblasts
by Katsumura, Sakie and Izu, Yayoi and Yamada, Takayuki and Griendling, Kathy and Harada, Kiyoshi and Noda, Masaki and Ezura, Yoichi
Journal of Cellular Biochemistry, ISSN 0730-2312, 07/2017, Volume 118, Issue 7, pp. 1670 - 1677
ABSTRACT Osteoporosis is one of the most prevalent ageing‐associated diseases that are soaring in the modern world. Although various aspects of the disease... 
POLDIP2 | FGF | OSTEOBLAST | PATHWAYS | MIGRATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL-CYCLE | NADPH OXIDASES | OSTEOPOROSIS | CELL BIOLOGY | Cell Line | Mice, Inbred C57BL | Osteoblasts - drug effects | RNA, Messenger - genetics | Nuclear Proteins - metabolism | Fibroblast Growth Factors - pharmacology | Mitochondrial Proteins - genetics | Cell Movement - genetics | Cell Movement - drug effects | Animals | Mitochondrial Proteins - metabolism | Mice | Nuclear Proteins - genetics | Osteoblasts - metabolism | Real-Time Polymerase Chain Reaction | Fibroblast growth factor | Femur | Dexamethasone | Transcription | Leukocyte migration | Cultures | Protein biosynthesis | siRNA | Hormones | Gene expression | Osteoblasts | Cell adhesion & migration | Osteoporosis | Angiogenesis | Protein synthesis | Rodents | Bone | Bone loss | Cell migration
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13. Full Text Unloading Induces Osteoblastic Cell Suppression and Osteoclastic Cell Activation to Lead to Bone Loss via Sympathetic Nervous System
by Hisataka Kondo and Akira Nifuji and Shu Takeda and Yoichi Ezura and Susan R. Rittling and David T. Denhardt and Kazuhisa Nakashima and Gerard Karsenty and Masaki Noda
Journal of Biological Chemistry, ISSN 0021-9258, 08/2005, Volume 280, Issue 34, pp. 30192 - 30200
Osteoporosis is one of the major health problems in our modern world. Especially, disuse (unloading) osteoporosis occurs commonly in bedridden patients, a... 
RESPONSES | OSTEOPONTIN | POSTMENOPAUSAL OSTEOPOROSIS | THERAPY | BIOCHEMISTRY & MOLECULAR BIOLOGY | Biomarkers - metabolism | Body Weight | Bone Marrow Cells - cytology | Mice, Inbred C57BL | Male | Tomography, X-Ray Computed | Osteoclasts - metabolism | Sympathetic Nervous System - metabolism | Amino Acids - pharmacology | Propranolol - pharmacology | Animals | Osteoporosis - pathology | Time Factors | Bone and Bones - metabolism | Isoproterenol - pharmacology | Dopamine beta-Hydroxylase - metabolism | Sympatholytics - pharmacology | Mice | Osteoblasts - metabolism | Guanethidine - pharmacology
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14. Full Text Nck influences preosteoblastic/osteoblastic migration and bone mass
by Smriti Aryal A. C and Kentaro Miyai and Yayoi Izu and Tadayoshi Hayata and Takuya Notomi and Masaki Noda and Yoichi Ezura
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2015, Volume 112, Issue 50, pp. 15432 - 15437
Migration of the cells in osteoblastic lineage, including preosteoblasts and osteoblasts, has been postulated to influence bone formation. However, the... 
MULTIDISCIPLINARY SCIENCES | Nck | migration | bone remodeling | osteoblast | MESENCHYMAL STEM-CELLS | bone repair | Insulin-Like Growth Factor I - pharmacology | Insulin Receptor Substrate Proteins - metabolism | Bone and Bones - drug effects | Gene Knockdown Techniques | Bone and Bones - diagnostic imaging | Protein Binding - drug effects | Bone Resorption - metabolism | Osteoblasts - cytology | Skull - drug effects | Wound Healing - drug effects | Bone and Bones - cytology | Skull - metabolism | Osteoblasts - drug effects | Osteogenesis - drug effects | Cells, Cultured | Oncogene Proteins - metabolism | Organ Size | Mice, Knockout | Cell Shape - drug effects | Radiography | Cell Movement - drug effects | Animals | Adaptor Proteins, Signal Transducing - deficiency | Bone Resorption - pathology | Oncogene Proteins - deficiency | Adaptor Proteins, Signal Transducing - metabolism | Osteoblasts - metabolism | Cell research | Physiological aspects | Bones | Research | Osteoblasts | Protein kinases | Density | Cell migration | Biological Sciences
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15. Full Text Dullard deficiency causes hemorrhage in the adult ovarian follicles
by Hayata, Tadayoshi and Chiga, Masahiko and Ezura, Yoichi and Asashima, Makoto and Katabuchi, Hidetaka and Nishinakamura, Ryuichi and Noda, Masaki
Genes to Cells, ISSN 1356-9597, 05/2018, Volume 23, Issue 5, pp. 345 - 356
In mammals, the ovarian follicles are regulated at least in part by bone morphogenetic protein (BMP) family members. Dullard (also known as Ctdnep1) gene... 
Dullard | ovary | BMP | hemorrhagic cysts | CELLS | FEMALE FERTILITY | MOUSE | NEURAL INDUCTION | MORPHOGENETIC PROTEIN SYSTEM | CELL BIOLOGY | DISRUPTION | GENE | GENETICS & HEREDITY | MICE | OSSIFICATION | EXPRESSION | Bone Morphogenetic Proteins - antagonists & inhibitors | Bone and Bones - pathology | Phosphorylation | Testis - metabolism | Male | Collagen Type I - genetics | Bone and Bones - metabolism | Ovarian Cysts - pathology | Female | Ovarian Follicle - metabolism | Pyrazoles - pharmacology | Collagen Type I - metabolism | Signal Transduction | Hemorrhage - metabolism | Pyrimidines - pharmacology | Phosphoprotein Phosphatases - physiology | Infertility, Female - metabolism | Mice, Knockout | Gene Expression Regulation - drug effects | Infertility, Female - pathology | Animals | Testis - pathology | Ovarian Cysts - metabolism | Phosphoprotein Phosphatases - deficiency | Mice | Ovarian Follicle - pathology | Hemorrhage - pathology | Smad Proteins - metabolism | Bone morphogenetic proteins | Hemorrhage | Steroid hormones | Cytochrome P-450 | Collagen (type I) | Blood cells | Erythrocytes | Homeostasis | Ovaries | Tamoxifen | Follicles | Kinases | Sertoli cells | Cysts | Clonal deletion | Infertility | Granulosa cells | Smad7 protein | Health risk assessment
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16. Full Text BMP‐2 Enhances Lgr4 Gene Expression in Osteoblastic Cells
by Pawaputanon Na Mahasarakham, Chantida and Ezura, Yoichi and Kawasaki, Makiri and Smriti, Arayal and Moriya, Shuichi and Yamada, Takayuki and Izu, Yayoi and Nifuji, Akira and Nishimori, Katsuhiko and Izumi, Yuichi and Noda, Masaki
Journal of Cellular Physiology, ISSN 0021-9541, 04/2016, Volume 231, Issue 4, pp. 887 - 895
Osteoporosis is one of the most prevalent diseases and the number of patients suffering from this disease is soaring due to the increase in the aged population... 
SIGNALING PATHWAYS | PHYSIOLOGY | PROTEIN | GPR48 | GLUCOCORTICOIDS | MANAGEMENT | BONE-FORMATION | MINERALIZATION | DIFFERENTIATION | WNT | OSTEOPOROSIS | CELL BIOLOGY | Bone Morphogenetic Protein 2 - pharmacology | Cell Line | Transcription, Genetic - drug effects | Alkaline Phosphatase - genetics | Osteoblasts - enzymology | Receptors, G-Protein-Coupled - metabolism | Osteoblasts - drug effects | RNA, Messenger - genetics | Alkaline Phosphatase - metabolism | RNA, Messenger - metabolism | Bone and Bones - drug effects | Gene Knockdown Techniques | Gene Expression Regulation - drug effects | Phenotype | Animals | Time Factors | Bone and Bones - metabolism | Mice | Receptors, G-Protein-Coupled - genetics | Osteoblasts - cytology | Osteoblasts - metabolism | Bones | Bone morphogenetic proteins | Gene expression | Density | Genes
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17. Full Text Annexin A5 Involvement in Bone Overgrowth at the Enthesis
by Shimada, Akemi and Ideno, Hisashi and Arai, Yoshinori and Komatsu, Koichiro and Wada, Satoshi and Yamashita, Teruhito and Amizuka, Norio and Pöschl, Ernst and Brachvogel, Bent and Nakamura, Yoshiki and Nakashima, Kazuhisa and Mizukami, Hiroaki and Ezura, Yoichi and Nifuji, Akira
Journal of Bone and Mineral Research, ISSN 0884-0431, 08/2018, Volume 33, Issue 8, pp. 1532 - 1543
ABSTRACT Little is known about the molecular mechanisms of enthesis formation in mature animals. Here, we report that annexin A5 (Anxa5) plays a critical role... 
ENTHESIS | TENDON/LIGAMENT | BONE MORPHOGENESIS | LACZ KNOCK‐IN MOUSE | ANNEXIN A5 | LACZ KNOCK-IN MOUSE | FIBROCARTILAGE CELLS | PROGENITORS | INSERTION | COLLAGEN | OSTEOBLASTIC DIFFERENTIATION | JUNCTION | FUNCTIONAL TENDON ENTHESIS | MINERALIZATION | ENDOCRINOLOGY & METABOLISM | GROWTH-PLATE CARTILAGE | EXPRESSION | Annexins | Femur | Molecular modelling | Clonal deletion | Mineralization | Mechanical unloading | Rodents | Contractility | Chondrocytes | Unloading | Calcein | Muscle contraction | Osteoblasts
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18. Full Text Erratum: Dullard/ctdnep1 regulates endochondral ossification via suppression of TGF-β signaling (Journal of Bone and Mineral Research (2015) 30:2 (318-29))
by Hayata, Tadayoshi and Ezura, Yoichi and Asashima, Makoto and Nishinakamura, Ryuichi and Noda, Masaki
Journal of Bone and Mineral Research, ISSN 0884-0431, 05/2015, Volume 30, Issue 5, p. 947
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