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Chinese Geographical Science, ISSN 1002-0063, 12/2005, Volume 15, Issue 4, pp. 355 - 360
Surface sink is a main geological calamity of gold mining areas and a main factor to restrict economic sustainable development of mining zone. Based on former... 
Geography | geographic information system(GIS) | P642.26 | Geography (general) | exploration vacancy | remote sensing(RS) | surface sink | ENVIRONMENTAL SCIENCES | Discovery and exploration | Gold industry | Analysis | Mineral industry | Mining industry | Sustainable development | Geographic information systems | Gold mines & mining
Journal Article
Chinese Geographical Science, ISSN 1002-0063, 2005, Volume 15, Issue 4, pp. 355 - 360
Journal Article
2012, 1st ed., Gu dong pai mai jing hua, ISBN 9787535652294, 160
Book
Journal Article
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 02/2014, Volume 5, Issue 1, pp. 3261 - 3261
Lineage transition in adenocarcinoma (ADC) and squamous cell carcinoma (SCC) of non-small cell lung cancer, as implicated by clinical observation of mixed ADC... 
LYSYL OXIDASE | SOX2 | GROWTH-FACTOR RECEPTOR | STEM-CELLS | RAS | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | JEGHERS-SYNDROME GENE | CANCER | ADENOSQUAMOUS CARCINOMA | SOMATIC MUTATIONS | Index Medicus
Journal Article
Nature Communications, ISSN 2041-1723, 08/2014, Volume 5, Issue 1, pp. 4629 - 4629
Whether the Hippo pathway contributes to cell lineage transition under pathological conditions, especially tumorigenesis, remains largely unknown. Here we show... 
YES-ASSOCIATED PROTEIN | ORGAN SIZE | HEPATOCELLULAR-CARCINOMA | STEM-LIKE CELLS | MULTIDISCIPLINARY SCIENCES | HIPPO PATHWAY | SIZE-CONTROL | DIFFERENTIATION | CANCER | ADENOSQUAMOUS CARCINOMA | TUMOR-SUPPRESSOR PATHWAY | Protein-Serine-Threonine Kinases - deficiency | Adenocarcinoma - pathology | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Lung Neoplasms - pathology | Male | Cell Transdifferentiation - genetics | Lung Neoplasms - physiopathology | Zinc Finger E-box Binding Homeobox 2 | Trans-Activators - physiology | Repressor Proteins - physiology | Cell Transdifferentiation - physiology | Adenocarcinoma - physiopathology | Tumor Suppressor Proteins - genetics | Up-Regulation - physiology | Trans-Activators - genetics | Female | Adenocarcinoma - genetics | Phosphoproteins - physiology | Disease Models, Animal | Lung Neoplasms - genetics | Transcription Factors - physiology | Protein-Serine-Threonine Kinases - genetics | Repressor Proteins - genetics | Mice, Transgenic | Phosphoproteins - genetics | Transcription Factors - genetics | Carcinoma, Squamous Cell - physiopathology | Homeodomain Proteins - genetics | Down-Regulation - physiology | Tumor Suppressor Proteins - physiology | Adaptor Proteins, Signal Transducing - physiology | Animals | Adaptor Proteins, Signal Transducing - genetics | Cell Line, Tumor | Mice | Homeodomain Proteins - physiology | Index Medicus
Journal Article
Nature Communications, ISSN 2041-1723, 2015, Volume 6, Issue 1, pp. 7639 - 7639
Peripherally derived regulatory T (pT(reg)) cell generation requires T-cell receptor (TCR) signalling and the cytokines TGF-beta 1 and IL-2. Here we show that... 
FOXP3 EXPRESSION | TGF-BETA | MULTIPLE-SCLEROSIS | TOLERANCE | MULTIDISCIPLINARY SCIENCES | IN-VIVO | CENTRAL-NERVOUS-SYSTEM | MECHANISMS | DIFFERENTIATION | INDUCTION | AUTOIMMUNE ENCEPHALOMYELITIS | Receptors, G-Protein-Coupled - metabolism | Encephalomyelitis, Autoimmune, Experimental - immunology | Receptors, G-Protein-Coupled - immunology | T-Lymphocytes, Regulatory - immunology | Cell Differentiation - genetics | Flow Cytometry | Chromatin Immunoprecipitation | Spinal Cord - pathology | T-Lymphocytes, Regulatory - cytology | Encephalomyelitis, Autoimmune, Experimental - genetics | Real-Time Polymerase Chain Reaction | Cytokines - immunology | Encephalomyelitis, Autoimmune, Experimental - pathology | Cytokines - metabolism | Receptors, Antigen, T-Cell - metabolism | Signal Transduction | MicroRNAs - immunology | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Freund's Adjuvant | Mice, Knockout | Spinal Cord - immunology | Cell Differentiation - immunology | Animals | Mice | MicroRNAs - genetics | Repressing | Regulators | Target recognition | Encephalomyelitis | Cytokines | MiRNA | Lymphocytes T | Protein deficiency | Ribonucleic acid--RNA | Experimental allergic encephalomyelitis | Proteins | T-cell receptor | Signaling | Clonal deletion | Acids | Interleukin 2 | MicroRNAs | Rodents | Deletion | Autoimmune diseases | Retinoic acid | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2010, Volume 107, Issue 44, pp. 18892 - 18897
LKB1 loss-of-function mutations, observed in ∼30% of human lung adenocarcinomas, contribute significantly to lung cancer malignancy progression. We show that... 
Oxidases | Cell growth | Lungs | Collagens | Metastasis | Renovations | Lung neoplasms | Tumors | Cancer | Integrins | BREAST-CANCER | PATHWAYS | TARGETED THERAPIES | METASTASIS | SPECIFICITY | PEUTZ-JEGHERS-SYNDROME | ADENOCARCINOMA | MULTIDISCIPLINARY SCIENCES | PHENOTYPE | TUMOR-SUPPRESSOR | MUTATIONS | Adenocarcinoma - pathology | TOR Serine-Threonine Kinases - metabolism | Humans | Lung Neoplasms - metabolism | Extracellular Matrix - metabolism | Lung Neoplasms - pathology | Extracellular Matrix - genetics | Neoplasm Metastasis | TOR Serine-Threonine Kinases - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Mice, Mutant Strains | Adenocarcinoma - genetics | Extracellular Matrix Proteins - metabolism | Protein-Serine-Threonine Kinases - metabolism | Lung Neoplasms - enzymology | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Neoplasm Invasiveness | Extracellular Matrix Proteins - genetics | Adenocarcinoma - enzymology | Protein-Serine-Threonine Kinases - genetics | Lung Neoplasms - therapy | Animals | Adenocarcinoma - therapy | Protein-Lysine 6-Oxidase - metabolism | Mice | Protein-Lysine 6-Oxidase - genetics | Prevention | Lung cancer | Development and progression | Extracellular matrix | Research | Health aspects | Studies | Enzymes | Signal transduction | Mutation | Cells | Index Medicus | Biological Sciences
Journal Article
International Journal of Cancer, ISSN 0020-7136, 05/2014, Volume 134, Issue 10, pp. 2294 - 2304
Journal Article