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1. Full Text Loss-of-Function Mutations in APOC3, Triglycerides, and Coronary Disease
by Crosby, Jacy and Peloso, Gina M and Auer, Paul L and Crosslin, David R and Stitziel, Nathan O and Lange, Leslie A and Lu, Yingchang and Tang, Zheng-Zheng and Zhang, He and Hindy, George and Masca, Nicholas and Stirrups, Kathleen and Kanoni, Stavroula and Do, Ron and Jun, Goo and Hu, Youna and Kang, Hyun Min and Xue, Chenyi and Goel, Anuj and Farrall, Martin and Duga, Stefano and Merlini, Pier Angelica and Asselta, Rosanna and Girelli, Domenico and Olivieri, Oliviero and Martinelli, Nicola and Yin, Wu and Reilly, Dermot and Speliotes, Elizabeth and Fox, Caroline S and Hveem, Kristian and Holmen, Oddgeir L and Nikpay, Majid and Farlow, Deborah N and Assimes, Themistocles L and Franceschini, Nora and Robinson, Jennifer and North, Kari E and Martin, Lisa W and DePristo, Mark and Gupta, Namrata and Escher, Stefan A and Jansson, Jan-Håkan and Van Zuydam, Natalie and Palmer, Colin N. A and Wareham, Nicholas and Koch, Werner and Meitinger, Thomas and Peters, Annette and Lieb, Wolfgang and Erbel, Raimund and Konig, Inke R and Kruppa, Jochen and Degenhardt, Franziska and Gottesman, Omri and Bottinger, Erwin P and O'Donnell, Christopher J and Psaty, Bruce M and Ballantyne, Christie M and Abecasis, Goncalo and Ordovas, Jose M and Melander, Olle and Watkins, Hugh and Orho-Melander, Marju and Ardissino, Diego and Loos, Ruth J. F and McPherson, Ruth and Willer, Cristen J and Erdmann, Jeanette and Hall, Alistair S and Samani, Nilesh J and Deloukas, Panos and Schunkert, Heribert and Wilson, James G and Kooperberg, Charles and Rich, Stephen S and Tracy, Russell P and Lin, Dan-Yu and Altshuler, David and Gabriel, Stacey and Nickerson, Deborah A and Jarvik, Gail P and Cupples, L. Adrienne and Reiner, Alex P and Boerwinkle, Eric and Kathiresan, Sekar and The TG and HDL Working Group of the Exome Sequencing Project, National Heart, Lung, and Blood Institute and Natl Heart Lung Blood Inst and Exome Sequencing Project and TG and HDL Working Group of the Exome Sequencing Project, National Heart, Lung, and Blood Institute and Medicinska fakulteten and Institutionen för folkhälsa och klinisk medicin and Umeå universitet and Medicin
The New England Journal of Medicine, ISSN 0028-4793, 07/2014, Volume 371, Issue 1, pp. 22 - 31
Whole-exome sequencing revealed an association between four rare loss-of-function mutations in the apoliprotein C3 gene ( APOC3 ) and low plasma triglyceride... 
APOLIPOPROTEIN C-III | RICH LIPOPROTEINS | MEDICINE, GENERAL & INTERNAL | DESIGN | ASSOCIATIONS | INHIBITION | RARE VARIANTS | CARDIOVASCULAR-DISEASE | PCSK9 | CAPTURE | PLASMA TRIGLYCERIDES | Coronary Disease - blood | European Continental Ancestry Group - genetics | Liver - pathology | Humans | Risk Factors | Apolipoprotein C-III - blood | Genotype | Apolipoprotein C-III - genetics | Sequence Analysis, DNA | Exome | Coronary Disease - genetics | Triglycerides - blood | Heterozygote | African Continental Ancestry Group - genetics | Mutation | Usage | Phenotype | Analysis and chemistry | Gene mutations | Analysis | Nucleotide sequencing | Research | Coronary heart disease | Risk factors | Blood | DNA sequencing | Phenotypes | Missense mutation | Nonsense mutation | Genes | Cardiovascular disease | Triglycerides | Genomes | Apolipoproteins | Heart diseases | Coronary artery disease | Medical and Health Sciences | Medicin och hälsovetenskap
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2. Full Text Exome sequencing identifies rare LDLR and APOA5 alleles conferring risk for myocardial infarction
by Do, Ron and Stitziel, Nathan O and Won, Hong-Hee and Jørgensen, Anders Berg and Duga, Stefano and Angelica Merlini, Pier and Kiezun, Adam and Farrall, Martin and Goel, Anuj and Zuk, Or and Guella, Illaria and Asselta, Rosanna and Lange, Leslie A and Peloso, Gina M and Auer, Paul L and Girelli, Domenico and Martinelli, Nicola and Farlow, Deborah N and DePristo, Mark A and Roberts, Robert and Stewart, Alexander F. R and Saleheen, Danish and Danesh, John and Epstein, Stephen E and Sivapalaratnam, Suthesh and Hovingh, G. Kees and Kastelein, John J and Samani, Nilesh J and Schunkert, Heribert and Erdmann, Jeanette and Shah, Svati H and Kraus, William E and Davies, Robert and Nikpay, Majid and Johansen, Christopher T and Wang, Jian and Hegele, Robert A and Hechter, Eliana and Marz, Winfried and Kleber, Marcus E and Huang, Jie and Johnson, Anew D and Li, Mingyao and Burke, Greg L and Gross, Myron and Liu, Yongmei and Assimes, Themistocles L and Heiss, Gerardo and Lange, Ethan M and Folsom, Aaron R and Taylor, Herman A and Olivieri, Oliviero and Hamsten, Anders and Clarke, Robert and Reilly, Dermot F and Yin, Wu and Rivas, Manuel A and Donnelly, Peter and Rossouw, Jacques E and Psaty, Bruce M and Herrington, David M and Wilson, James G and Rich, Stephen S and Bamshad, Michael J and Tracy, Russell P and Cupples, L. Aienne and Rader, Daniel J and Reilly, Muredach P and Spertus, John A and Cresci, Sharon and Hartiala, Jaana and Tang, W. H. Wilson and Hazen, Stanley L and Allayee, Hooman and Reiner, Alex P and Carlson, Christopher S and Kooperberg, Charles and Jackson, Rebecca D and Boerwinkle, Eric and Lander, Eric S and Schwartz, Stephen M and Siscovick, David S and McPherson, Ruth and Tybjaerg-Hansen, Anne and Abecasis, Goncalo R and Watkins, Hugh and Nickerson, Deborah A and Ardissino, Diego and Sunyaev, Shamil R and O'Donnell, Christopher J and Altshuler, David and Gabriel, Stacey and Kathiresan, Sekar and Gabriel, Stacey B and Altshuler, David M and Abecasis, Gonçalo R and Daly, Mark J and de Bakker, Paul I. W and Fennell, Tim and Garimella, Kiran and ... and NHLBI Exome Sequencing Project
Nature, ISSN 0028-0836, 2015, Volume 518, Issue 7537, pp. 102 - +
Myocardial infarction (MI), a leading cause of death around the world, displays a complex pattern of inheritance(1,2). When MI occurs early in life, genetic... 
HEART-DISEASE | VARIANTS | CHOLESTEROL | MULTIDISCIPLINARY SCIENCES | RECEPTOR | TRIGLYCERIDES | OF-FUNCTION MUTATIONS | HYPERCHOLESTEROLEMIA | CORONARY-ARTERY-DISEASE | GENOME-WIDE ASSOCIATION | SINGLE NUCLEOTIDE POLYMORPHISMS | Genetics, Population | Myocardial Infarction - blood | Myocardial Infarction - genetics | Receptors, LDL - genetics | Apolipoprotein A-V | Genetic Predisposition to Disease - genetics | Apolipoproteins A - genetics | National Heart, Lung, and Blood Institute (U.S.) | Age Factors | United States | Humans | Middle Aged | Male | Mutation - genetics | Case-Control Studies | Exome - genetics | Age of Onset | Alleles | Coronary Artery Disease - genetics | Triglycerides - blood | Cholesterol, LDL - blood | Female | Heterozygote | Usage | Low density lipoproteins | Exome sequencing | Physiological aspects | Genetic aspects | Research | Diagnosis | Heart attack | Risk factors | Proteins | Studies | Algorithms | Heart attacks | Genes | Cardiovascular disease | Triglycerides | Mutation | Cholesterol
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3. Full Text MAP kinase pathway alterations in BRAF -mutant melanoma patients with acquired resistance to combined RAF/MEK inhibition
by Wagle, Nikhil and Van Allen, Eliezer M and Treacy, Daniel J and Frederick, Dennie T and Cooper, Zachary A and Taylor-Weiner, Amaro and Rosenberg, Mara and Goetz, Eva M and Sullivan, Ryan J and Farlow, Deborah N and Friedrich, Dennis C and Anderka, Kristin and Perrin, Danielle and Johannessen, Cory M and McKenna, Aaron and Cibulskis, Kristian and Kryukov, Gregory and Hodis, Eran and Lawrence, Donald P and Fisher, Sheila and Getz, Gad and Gabriel, Stacey B and Carter, Scott L and Flaherty, Keith T and Wargo, Jennifer A and Garraway, Levi A
Cancer Discovery, ISSN 2159-8274, 01/2014, Volume 4, Issue 1, pp. 61 - 68
Treatment of BRAF-mutant melanoma with combined dabrafenib and trametinib, which target RAF and the downstream MAP-ERK kinase (MEK)1 and MEK2 kinases,... 
RAF INHIBITION | METASTATIC MELANOMA | AMPLIFICATION | PROTEIN | MEK INHIBITION | ONCOLOGY | IMPROVED SURVIVAL | V600E MUTATION | CANCER | VEMURAFENIB | Mitogen-Activated Protein Kinase Kinases - genetics | Skin Neoplasms - drug therapy | raf Kinases - antagonists & inhibitors | Humans | Middle Aged | Oximes - therapeutic use | Male | Antineoplastic Agents - therapeutic use | Melanoma - genetics | Imidazoles - therapeutic use | raf Kinases - genetics | Melanoma - metabolism | Signal Transduction | Pyrimidinones - therapeutic use | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | Skin Neoplasms - metabolism | Proto-Oncogene Proteins B-raf - antagonists & inhibitors | Protein Kinase Inhibitors - therapeutic use | Proto-Oncogene Proteins B-raf - genetics | Melanoma - drug therapy | Skin Neoplasms - genetics | Mitogen-Activated Protein Kinases - genetics | Aged | Pyridones - therapeutic use | Mutation | Drug Resistance, Neoplasm - physiology | Mitogen-Activated Protein Kinases - metabolism
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4. Full Text Whole-Exome Sequencing Identifies Rare and Low-Frequency Coding Variants Associated with LDL Cholesterol
by Lange, Leslie A and Lange, Ethan M and Lange, Christoph and Hu, Youna and Zhang, Qunyuan and Zhang, Ji and Zhang, He and Xue, Chenyi and Schmidt, Ellen M and Tang, Hua and Tang, Zheng-Zheng and Bizon, Chris and Smith, Nicholas L and Smith, Joshua D and Smith, Albert V and Turner, Emily H and Jun, Goo and Kang, Hyun Min and Peloso, Gina and Auer, Paul L and Auer, Paul and Li, Kuo-ping and Li, Mingyao and Li, Yun and Li, Bingshan and Li, Dalin and Flannick, Jason and Fuchsberger, Christian and Gaulton, Kyle and Lindgren, Cecilia and Locke, Adam and Manning, Alisa and Sim, Xueling and Rivas, Manuel A and Holmen, Oddgeir L and Gottesman, Omri and Lu, Yingchang and Ruderfer, Douglas and Stahl, Eli A and Duan, Qing and Durda, Peter and Durda, J. Peter and Jiao, Shuo and Isaacs, Aaron and Hofman, Albert and Bis, Joshua C and Bis, Joshua and Correa, Adolfo and Griswold, Michael E and Jakobsdottir, Johanna and Schreiner, Pamela and Schreiner, Pamela J and Feitosa, Mary F and Huffman, Jennifer E and Crosby, Jacy and Wassel, Chrstina L and Wassel, Christina L and Do, Ron and Franceschini, Nora and Martin, Lisa and Martin, Lisa W and Martin, Thomas R and Robinson, Jennifer G and Assimes, Themistocles L and Crosslin, David R and Rosenthal, Elisabeth A and Tsai, Michael Y and Tsai, Michael and Rieder, Mark J and Farlow, Deborah N and Folsom, Aaron R and Lumley, Thomas and Fox, Ervin R and Fox, Caroline S and Fox, P. Keolu and Fox, Ervin and Carlson, Christopher S and Peters, Ulrike and Jackson, Rebecca D and Jackson, Rebecca and van Duijn, Cornelia M and Uitterlinden, André G and Levy, Daniel and Rotter, Jerome I and Taylor, Herman A and Taylor, Kira C and Taylor, Kent and Gudnason, Vilmundur and Siscovick, David S and Fornage, Myriam and Borecki, Ingrid B and Hayward, Caroline and Rudan, Igor and Chen, Wei-Min and Chen, Y. Eugene and Chen, Donna T and Chen, Ida Y and Bottinger, Erwin P and Loos, Ruth J.F and Sætrom, Pål and ... and NHLBI Grand Opportunity Exome Sequ and NHLBI Grand Opportunity Exome Sequencing Project and Genomik, diabetes och endokrinologi and Lund University and Genomics, Diabetes and Endocrinology and Lunds universitet and EXODIAB: Excellence in Diabetes Research in Sweden
The American Journal of Human Genetics, ISSN 0002-9297, 02/2014, Volume 94, Issue 2, pp. 233 - 245
Elevated low-density lipoprotein cholesterol (LDL-C) is a treatable, heritable risk factor for cardiovascular disease. Genome-wide association studies (GWASs)... 
OBJECTIVES | HEART-DISEASE | PLASMA | GENE | SUSCEPTIBILITY | GENETICS & HEREDITY | DENSITY-LIPOPROTEIN-CHOLESTEROL | Dyslipidemias - genetics | Genetic Code | Follow-Up Studies | Humans | Middle Aged | Male | Proprotein Convertases - genetics | Exome | Dyslipidemias - blood | Serine Endopeptidases - genetics | Adult | Female | Receptors, LDL - genetics | Genome-Wide Association Study | Gene Frequency | Genotype | Sequence Analysis, DNA | Cholesterol, LDL - genetics | Phenotype | Apolipoproteins E - blood | Apolipoproteins E - genetics | Aged | Polymorphism, Single Nucleotide | Cohort Studies | Lipase - genetics | Proprotein Convertase 9 | Genetic code | Genetic variation | Physiological aspects | Genetic research | Cholesterol, LDL | Genetic aspects | Research | Nucleotide sequencing | DNA sequencing | Medicinsk genetik | Basic Medicine | Medical Genetics | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper
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5. Full Text Genome-Wide association study of coronary heart disease and its risk factors in 8,090 african americans: The nhlbi CARe project
by Lettre, Guillaume and Palmer, Cameron D and Young, Taylor and Ejebe, Kenechi G and Allayee, Hooman and Benjamin, Emelia J and Bennett, Franklyn and Bowden, Donald W and Chakravarti, Aravinda and Dreisbach, Al and Farlow, Deborah N and Folsom, Aaron R and Fornage, Myriam and Forrester, Terrence and Fox, Ervin and Haiman, Christopher A and Hartiala, Jaana and Harris, Tamara B and Hazen, Stanley L and Heckbert, Susan R and Henderson, Brian E and Hirschhorn, Joel N and Keating, Brendan J and Kritchevsky, Stephen B and Larkin, Emma and Li, Mingyao and Rudock, Megan E and McKenzie, Colin A and Meigs, James B and Meng, Yang A and Mosley Jr, Tom H and Newman, Anne B and Newton-Cheh, Christopher H and Paltoo, Dina N and Papanicolaou, George J and Patterson, Nick and Post, Wendy S and Psaty, Bruce M and Qasim, Atif N and Qu, Liming and Rader, Daniel J and Redline, Susan and Reilly, Muredach P and Reiner, Alexander P and Rich, Stephen S and Rotter, Jerome I and Liu, Yongmei and Shrader, Peter and Siscovick, David S and Tang, W. H. Wilson and Taylor Jr, Herman A and Tracy, Russell P and Vasan, Ramachandran S and Waters, Kevin M and Wilks, Rainford and Wilson, James G and Fabsitz, Richard R and Gabriel, Stacey B and Kathiresan, Sekar and Boerwinkle, Eric
PLoS Genetics, ISSN 1553-7390, 02/2011, Volume 7, Issue 2, p. e1001300
Coronary heart disease (CHD) is the leading cause of mortality in African Americans. To identify common genetic polymorphisms associated with CHD and its risk... 
METAANALYSIS | MYOCARDIAL-INFARCTION | VARIANTS | GENETICS & HEREDITY | LOCI | TRAITS | ARTERY-DISEASE | Cholesterol, LDL - genetics | Genome-Wide Association Study | Coronary Disease - genetics | United States | Humans | Risk Factors | African Americans - genetics | Polymorphism, Single Nucleotide - genetics | European Continental Ancestry Group | Hypertension - genetics | Cholesterol, HDL - genetics | Genome, Human | Demographic aspects | Genetic susceptibility | Genetic aspects | Research | Coronary heart disease | Risk factors | Genetic polymorphisms | Studies | African Americans | Cardiovascular disease | Genetics | Cholesterol
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6. Full Text Concept, design and implementation of a cardiovascular gene-centric 50 K SNP array for large-scale genomic association studies
by Keating, Brendan J and Tischfield, Sam and Murray, Sarah S and Bhangale, Tushar and Price, Thomas S and Glessner, Joseph T and Galver, Luana and Barrett, Jeffrey C and Grant, Struan F.A and Farlow, Deborah N and Chandrupatla, Hareesh R and Hansen, Mark and Ajmal, Saad and Papanicolaou, George J and Guo, Yiran and Li, Mingyao and DerOhannessian, Stephanie and De Bakker, Paul I.W and Bailey, Swneke D and Montpetit, Alexandre and Edmondson, Andrew C and Taylor, Kent and Gai, Xiaowu and Wang, Susanna S and Fornage, Myriam and Shaikh, Tamim and Groop, Leif and Boehnke, Michael and Hall, Alistair S and Hattersley, Andrew T and Frackelton, Edward and Patterson, Nick and Chiang, Charleston W.K and Kim, Cecelia E and Fabsitz, Richard R and Ouwehand, Willem and Price, Alkes L and Munroe, Patricia and Caulfield, Mark and Drake, Thomas and Boerwinkle, Eric and Reich, David and Whitehead, A. Stephen and Cappola, Thomas P and Samani, Nilesh J and Lusis, A. Jake and Schadt, Eric and Wilson, James G and Koenig, Wolfgang and McCarthy, Mark I and Kathiresan, Sekar and Gabriel, Stacey B and Hakonarson, Hakon and Anand, Sonia S and Reilly, Muredach and Engert, James C and Nickerson, Deborah A and Rader, Daniel J and Hirschhorn, Joel N and FitzGerald, Garret A
PLoS ONE, ISSN 1932-6203, 10/2008, Volume 3, Issue 10, p. e3583
A wealth of genetic associations for cardiovascular and metabolic phenotypes in humans has been accumulating over the last decade, in particular a large number... 
MULTIDISCIPLINARY SCIENCES | Cardiovascular Diseases - ethnology | Genome-Wide Association Study - instrumentation | Oligonucleotide Array Sequence Analysis - methods | Gene Frequency | Humans | Population Groups - genetics | Genotype | Cardiovascular Diseases - genetics | Polymorphism, Single Nucleotide | Genome-Wide Association Study - methods | Research Design | Concept Formation | Quality Control | Type 2 diabetes | Quantitative genetics | Medical research | Genes | Genomics | Atherosclerosis | Medicine, Experimental | Genetic aspects | Genomes | Single nucleotide polymorphisms | Pediatrics | Populations | Encyclopedias | Science | Cardiovascular disease | Epistasis | Single-nucleotide polymorphism | Gene polymorphism | Medical schools | Interrogation | Genetics | Deoxyribonucleic acid--DNA | Saturation | Genetic diversity | Gene expression | Loci | Quantitative trait loci | Medicine | Hospitals | Arteriosclerosis | Coronary vessels | Environment models | Tagging | Diabetes | Endocrinology | Polymorphism | Deoxyribonucleic acid | DNA
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7. Full Text Association of genetic variation with systolic and diastolic blood pressure among African Americans: the Candidate Gene Association Resource study
by Fox, Ervin R and Young, J. Hunter and Li, Yali and isbach, Albert W and Keating, Brendan J and Musani, Solomon K and Liu, Kiang and Morrison, Alanna C and Ganesh, Santhi and Kutlar, Abdullah and Ramachanan, Vasan S and Polak, Josef F and Fabsitz, Richard R and es, Daniel L and Farlow, Deborah N and Redline, Susan and Adeyemo, Adebowale and Hirschorn, Joel N and Sun, Yan V and Wyatt, Sharon B and Penman, Alan D and Palmas, Walter and Rotter, Jerome I and Townsend, Raymond R and Doumatey, Ayo P and Tayo, Bamidele O and Mosley, Thomas H., Jr and Lyon, Helen N and Kang, Sun J and Rotimi, Charles N and Cooper, Richard S and Franceschini, Nora and Curb, J. David and Martin, Lisa W and Eaton, Charles B and Kardia, Sharon L. R and Taylor, Herman A., Jr and Caulfield, Mark J and Ehret, Georg B and Johnson, Toby and Chakravarti, Aravinda and Zhu, Xiaofeng and Staessen, Jan and Levy, Daniel and Int Consortium Blood Pressure Geno and International Consortium for Blood Pressure Genome-wide Association Studies (ICBP-GWAS) and KidneyGen consortium and EchoGen consortium and The International Consortium for Blood Pressure Genome-wide Association Studies (ICBP-GWAS) and CHARGE-HF consortium and CKDGen consortium and CARDIoGRAM consortium and Sahlgrenska akademin and Institutionen för medicin, avdelningen för akut och kardiovaskulär medicin and Institute of Medicine, Department of Emergeny and Cardiovascular Medicine and Göteborgs universitet and Gothenburg University and Sahlgrenska Academy
Human Molecular Genetics, ISSN 0964-6906, 06/2011, Volume 20, Issue 11, pp. 2273 - 2284
The prevalence of hypertension in African Americans (AAs) is higher than in other US groups; yet, few have performed genome-wide association studies (GWASs) in... 
HYPERTENSION PREVALENCE | DESIGN | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENETICS & HEREDITY | RECEPTOR | RISK | NA+ | LOCI | AWARENESS | TRAITS | GENOME-WIDE ASSOCIATION | FAMILY | Blood Pressure | European Continental Ancestry Group - genetics | Genome-Wide Association Study | Diastole | Humans | Middle Aged | African Americans - genetics | Genotype | Male | Genetic Loci | Systole | Phenotype | Adult | Female | Aged | Polymorphism, Single Nucleotide | Hypertension - epidemiology | Hypertension - genetics | Cohort Studies | Association Studies | Hypertension/genetics | African Americans/genetics | European Continental Ancestry | Group/genetics | Hypertension/epidemiology | MEDICIN OCH HÄLSOVETENSKAP | Female Genetic Loci Genome-Wide Association Study Genotype | Single Nucleotide | MEDICAL AND HEALTH SCIENCES | Polymorphism
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