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Glia, ISSN 0894-1491, 03/2016, Volume 64, Issue 3, pp. 407 - 424
Journal Article
Epilepsia, ISSN 0013-9580, 05/2019, Volume 60, Issue 5, pp. 830 - 844
Journal Article
Journal Article
Journal Article
Journal of Lipid Research, ISSN 0022-2275, 10/2012, Volume 53, Issue 10, pp. 2162 - 2174
Journal Article
Journal of Neuroscience Methods, ISSN 0165-0270, 2006, Volume 156, Issue 1, pp. 101 - 110
Reverse transcription of extracted cellular RNA combined with real-time PCR is now an established method for sensitive detection and quantification of specific... 
PCR efficiency | Perforant path lesion | Permanent middle cerebral artery occlusion | Reference gene | Real-time PCR | Gene expression | QUANTIFICATION | FOCAL CEREBRAL-ISCHEMIA | BIOCHEMICAL RESEARCH METHODS | reference gene | real-time PCR | QUANTITATIVE RT-PCR | NEUROSCIENCES | TUMOR-NECROSIS-FACTOR | ENDOGENOUS REFERENCE GENES | permanent middle cerebral artery occlusion | NORMALIZATION | HOUSEKEEPING GENES | EXPRESSION | gene expression | perforant path lesion | LASER MICRODISSECTION | RNA, Messenger - isolation & purification | Male | Axons - physiology | Nervous System Diseases - genetics | Lysophosphatidylcholines - administration & dosage | RNA, Messenger - biosynthesis | Spinal Cord - pathology | Infarction, Middle Cerebral Artery - genetics | Encephalomyelitis, Autoimmune, Experimental - genetics | Nervous System Diseases - pathology | Wallerian Degeneration - genetics | Data Interpretation, Statistical | Lysophosphatidylcholines - pharmacology | Reproducibility of Results | Encephalomyelitis, Autoimmune, Experimental - pathology | Corpus Callosum | Microinjections | Mice, Inbred C57BL | RNA, Messenger - genetics | Wallerian Degeneration - physiopathology | Perforant Pathway - physiology | Glyceraldehyde-3-Phosphate Dehydrogenases - genetics | Infarction, Middle Cerebral Artery - pathology | Reverse Transcriptase Polymerase Chain Reaction | Animals | Hypoxanthine Phosphoribosyltransferase - genetics | Mice | Hippocampus - physiology | Denervation | Phosphates | Purines | Medical research | Messenger RNA | Analysis | Medicine, Experimental | Models | Index Medicus
Journal Article
Neurobiology of Disease, ISSN 0969-9961, 12/2019, Volume 132, pp. 104581 - 104581
Mutations in parkin, encoded by the gene, causes early-onset familial Parkinson's disease (PD), but dysfunctional parkin has also been implicated in sporadic... 
iPSCs | Isogenic | Parkinson's disease | Post-translational modifications | RhoA signalling | Proteomics | Neurite outgrowth | Cell migration | Index Medicus
Journal Article
Journal of Neuropathology & Experimental Neurology, ISSN 0022-3069, 08/2009, Volume 68, Issue 8, pp. 845 - 856
Journal Article
Glia, ISSN 0894-1491, 11/2006, Volume 54, Issue 6, pp. 591 - 605
Tumor necrosis factor (TNF) is a potent pro‐inflammatory and neuromodulatory cytokine. In the CNS it is produced primarily by microglia and considered to... 
myelin | Wallerian degeneration | perforant path | microglia | knock out mice | Myelin | Knock out mice | Microglia | Perforant path | CNS REMYELINATION | FOCAL CEREBRAL-ISCHEMIA | MACROPHAGE SYNTHESIS | DEFICIENT MICE | NEUROSCIENCES | FACTOR-ALPHA | LABELING TECHNIQUE | TERMINAL DEGENERATION | CENTRAL-NERVOUS-SYSTEM | ASTROGLIAL REACTIONS | Perforant Pathway - physiopathology | Tumor Necrosis Factor-alpha - metabolism | Microglia - metabolism | Tumor Necrosis Factor-alpha - genetics | Male | Gliosis - physiopathology | Receptors, Tumor Necrosis Factor, Type I - metabolism | CD11 Antigens - metabolism | RNA, Messenger - metabolism | Dentate Gyrus - physiopathology | Antigens, CD - metabolism | Perforant Pathway - surgery | Axotomy | Encephalitis - physiopathology | Receptors, Tumor Necrosis Factor, Type II - metabolism | Encephalitis - metabolism | Up-Regulation - physiology | Receptors, Tumor Necrosis Factor - genetics | Receptors, Tumor Necrosis Factor - metabolism | Dentate Gyrus - metabolism | Perforant Pathway - injuries | Wallerian Degeneration - metabolism | Gliosis - metabolism | Mice, Inbred C57BL | Axons - metabolism | Receptors, Tumor Necrosis Factor, Type I - genetics | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Animals | Antigens, Differentiation, Myelomonocytic - metabolism | Axons - pathology | Mice | Transcriptional Activation - physiology | Index Medicus
Journal Article
Journal of Neuroscience Research, ISSN 0360-4012, 01/2004, Volume 75, Issue 2, pp. 203 - 217
The inflammatory cytokine tumour necrosis factor (TNF) can both induce oligodendrocyte and myelin pathology and promote proliferation of oligodendrocyte... 
myelin gene expression | progenitor cell proliferation | axonal sprouting | inflammatory cytokines | transgenic mice | Inflammatory cytokines | Progenitor cell proliferation | Axonal sprouting | Myelin gene expression | Transgenic mice | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | FOCAL CEREBRAL-ISCHEMIA | NEUROSCIENCES | MYELIN BASIC-PROTEIN | TUMOR-NECROSIS-FACTOR | MESSENGER-RNA | MULTIPLE-SCLEROSIS | GENE-EXPRESSION | CENTRAL-NERVOUS-SYSTEM | PERFORANT PATH | DEAFFERENTED HIPPOCAMPUS | Perforant Pathway - physiopathology | Oligodendroglia - metabolism | Wallerian Degeneration - pathology | Tumor Necrosis Factor-alpha - genetics | Nerve Growth Factors - metabolism | Axons - secretion | RNA, Messenger - metabolism | Perforant Pathway - surgery | Axotomy | Neuronal Plasticity - genetics | Nerve Fibers, Myelinated - pathology | Oligodendroglia - cytology | Hippocampus - growth & development | Cell Division - genetics | Nerve Fibers, Myelinated - metabolism | Nerve Regeneration - genetics | Nerve Fibers, Myelinated - secretion | Perforant Pathway - injuries | Myelin Basic Protein - metabolism | Nerve Growth Factors - secretion | Wallerian Degeneration - metabolism | Wallerian Degeneration - physiopathology | Axons - metabolism | Mice, Transgenic | Up-Regulation - genetics | Reaction Time - physiology | Hippocampus - cytology | Hippocampus - metabolism | Animals | Axons - pathology | Mice | Index Medicus
Journal Article