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Journal of Molecular Endocrinology, ISSN 0952-5041, 07/2017, Volume 59, Issue 1, p. R47
Primary aldosteronism (PA), the most common form of secondary hypertension, is caused in the majority of cases by unilateral aldosterone-producing adenoma... 
Cell proliferation | Hyperplasia | Adenoma | Membrane potential | Mutation | Aldosterone | Age
Journal Article
La Presse Médicale, ISSN 0755-4982, 07/2018, Volume 47, Issue 7-8, pp. e151 - e158
Aldosterone-producing adenomas (APA) are a major cause of primary aldosteronism (PA), the most common form of secondary hypertension. Exome analysis of APA has... 
ALDOSTERONE-PRODUCING ADENOMA | MEDICINE, GENERAL & INTERNAL | HYPERALDOSTERONISM | K+ CHANNEL MUTATIONS | CLINICAL-PRACTICE GUIDELINE | ADRENOCORTICAL ADENOMAS | KCNJ5 MUTATIONS | PRKACA SOMATIC MUTATIONS | HYPERTENSION | ADRENAL CUSHINGS-SYNDROME | BLOOD-PRESSURE
Journal Article
Molecular and Cellular Endocrinology, ISSN 0303-7207, 05/2013, Volume 371, Issue 1-2, pp. 221 - 227
Somatic mutations of KCNJ5, coding for the potassium channel GIRK4, have recently been implicated in the formation of aldosterone producing adenoma (APA).... 
Aldosterone
Journal Article
Molecular and Cellular Endocrinology, ISSN 0303-7207, 05/2013, Volume 371, Issue 1-2, pp. 221 - 227
Somatic mutations of KCNJ5, coding for the potassium channel GIRK4, have recently been implicated in the formation of aldosterone producing adenoma (APA).... 
Mutations | Cortexes | Correlation | Correlation analysis | Remodeling | Aldosterone | Channels | Endocrinology
Journal Article
Hypertension, ISSN 0194-911X, 08/2014, Volume 64, Issue 2, pp. 354 - 361
Journal Article
Journal of Molecular Endocrinology, ISSN 0952-5041, 07/2017, Volume 59, Issue 1, pp. R47 - R63
Primary aldosteronism (PA), the most common form of secondary hypertension, is caused in the majority of cases by unilateral aldosterone-producing adenoma... 
Calcium channels | Familial hyperaldosteronism | Primary aldosteronism | Somatic mutations | Aldosterone-producing adenoma | ATPase | Germline mutations | Potassium channels | Wnt/β-catenin pathway | aldosterone-producing adenoma | HYPERALDOSTERONISM TYPE-II | MEMBRANE CA2+ ATPASE | familial hyperaldosteronism | calcium channels | Wnt/beta-catenin pathway | germline mutations | PCR-SSCP ANALYSIS | somatic mutations | MOUSE ADRENAL-CORTEX | GLUCOCORTICOID-REMEDIABLE ALDOSTERONISM | potassium channels | K+ CHANNEL MUTATIONS | primary aldosteronism | ENDOCRINOLOGY & METABOLISM | CLINICAL-PRACTICE GUIDELINE | KCNJ5 MUTATIONS | POTASSIUM CHANNEL | Adrenal Glands - pathology | Sodium-Potassium-Exchanging ATPase - genetics | Plasma Membrane Calcium-Transporting ATPases - genetics | Adenoma - genetics | Humans | Hyperaldosteronism - pathology | Hyperaldosteronism - metabolism | Adrenal Hyperplasia, Congenital - genetics | Adenoma - metabolism | Plasma Membrane Calcium-Transporting ATPases - metabolism | Hyperaldosteronism - diagnosis | Adrenal Hyperplasia, Congenital - diagnosis | Hypokalemia - metabolism | Adrenal Hyperplasia, Congenital - pathology | G Protein-Coupled Inwardly-Rectifying Potassium Channels - metabolism | Hypertension - genetics | Hypertension - diagnosis | Hypokalemia - pathology | Adenoma - diagnosis | Gene Expression | Genetic Predisposition to Disease | Adrenal Hyperplasia, Congenital - metabolism | Hyperaldosteronism - genetics | G Protein-Coupled Inwardly-Rectifying Potassium Channels - genetics | beta Catenin - metabolism | Hypertension - pathology | beta Catenin - genetics | Hypertension - metabolism | Hypokalemia - diagnosis | Sodium-Potassium-Exchanging ATPase - metabolism | Calcium Channels, L-Type - genetics | Adrenal Glands - metabolism | Adenoma - pathology | Calcium Channels, L-Type - metabolism | Mutation | Hypokalemia - genetics
Journal Article
Hypertension, ISSN 0194-911X, 11/2015, Volume 66, Issue 5, pp. 1014 - 1022
Journal Article
Frontiers in Endocrinology, ISSN 1664-2392, 2015, Volume 6, Issue MAY, p. 95
Primary aldosteronism (PA) is the most common form of secondary hypertension with an estimated prevalence of ~10% in referred patients. PA occurs as a result... 
Shh signaling pathway | Calcium channels | Atpase | Primary aldosteronism | Somatic mutations | Aldosterone producing adenoma | Potassium channels | Wnt/β-catenin pathway | Potassium Channels | Wnt/beta-catenin signalling | somatic mutations | Calcium Channels | ATPase | primary aldosteronism
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