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1. Practical management of bipolar disorder
by Young, A. H. (Allan H.) and Ferrier, I. N. (Ian Nicol) and Michalak, Erin E
2010, ISBN 0521734894, vii, 218
"Bipolar disorder, or manic depression, is characterised by episodes of pathological mood states. The two poles are mania (with a predominant elated or... 
therapy | Bipolar Disorder | Other branches of medicine | Manic-depressive illness
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2. Full Text Analysis of shared heritability in common disorders of the brain
by Anttila, Verneri and Bulik-Sullivan, Brendan and Finucane, Hilary K and Walters, Raymond K and Bras, Jose and Duncan, Laramie and Escott-Price, Valentina and Falcone, Guido J and Gormley, Padhraig and Malik, Rainer and Patsopoulos, Nikolaos A and Ripke, Stephan and Wei, Zhi and Yu, Dongmei and Lee, Phil H and Turley, Patrick and Grenier-Boley, Benjamin and Chouraki, Vincent and Kamatani, Yoichiro and Berr, Claudine and Letenneur, Luc and Hannequin, Didier and Amouyel, Philippe and Boland, Anne and Deleuze, Jean-François and Duron, Emmanuelle and Vardarajan, Bai N and Reitz, Christiane and Goate, Alison M and Huentelman, Matthew J and Ilyas Kamboh, M and Larson, Eric B and Rogaeva, Ekaterina and George-Hyslop, Peter St and Hakonarson, Hakon and Kukull, Walter A and Farrer, Lindsay A and Barnes, Lisa L and Beach, Thomas G and Yesim Demirci, F and Head, Elizabeth and Hulette, Christine M and Jicha, Gregory A and Kauwe, John S. K and Kaye, Jeffrey A and Leverenz, James B and Levey, Allan I and Denys, Damiaan and Derks, Eske M and Figee, Martijn and de Haan, Lieuwe and Huyser, Chaim and Brainstorm Consortium and The Brainstorm Consortium
Science, ISSN 0036-8075, 2018, Volume 360, Issue 6395, pp. 1313 - 1313
Disorders of the brain can exhibit considerable epidemiological comorbidity and often share symptoms, provoking debate about their etiologic overlap. We... 
ANOREXIA-NERVOSA | POPULATION-BASED TWIN | DEFICIT HYPERACTIVITY DISORDER | LONG-TERM SURVIVAL | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | GENETIC CORRELATIONS | BODY-MASS INDEX | MAJOR DEPRESSIVE DISORDER | BIPOLAR DISORDER | GENOME-WIDE ASSOCIATION | Genetic Variation | Mental Disorders - genetics | Genome-Wide Association Study | Phenotype | Brain Diseases - classification | Quantitative Trait, Heritable | Brain Diseases - diagnosis | Humans | Mental Disorders - classification | Risk Factors | Brain Diseases - genetics | Mental Disorders - diagnosis | Headache | Anorexia | Mental disorders | Medical services | Disorders | Cognitive ability | Schizophrenia | Risk sharing | Bipolar disorder | Genomes | Posttraumatic stress disorder | Mental depression | Epidemiology | Risk factors | Consortia | Proteins | Heterogeneity | Biological effects | Ischemia | Etiology | Risk assessment | Classification | Gilles de la Tourette syndrome | Alzheimer's disease | Neurological disorders | Stroke | Phenotypes | Neurodegenerative diseases | Comorbidity | Migraine | Attention deficit hyperactivity disorder | Eating disorders | Personality | Genetic diversity | Behavior disorders | Anorexia nervosa | Risk analysis | Patients | Statistics | Activity patterns | Neurological diseases | Crohn's Disease | Population (statistical) | Scaffolding | Correlation analysis | Heritability | Diagnostic systems | Alzheimers disease | Life Sciences | Human health and pathology | Mental Disorders | Neurons and Cognition | Brain Diseases | Genetics
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3. Full Text Collaborative genome-wide association analysis supports a role for ANK3 and CACNA1C in bipolar disorder
by McGhee, Kevin A and Owen, Michael J and Corvin, Aiden P and Nikolov, Ivan and Jones, Ian R and Gurling, Hugh M and Nimgaonkar, Vishwajit L and Fan, Jinbo and Collier, David A and Holmans, Peter A and O'Donovan, Michael C and Farmer, Anne and Smoller, Jordan W and Caesar, Sian and Kirov, George and Williamson, Richard and Kandaswamy, Radhika and Purcell, Shaun M and Sklar, Pamela and Moskvina, Valentina and Thase, Michael E and Anjorin, Adebayo and Blackwood, Douglas H and Perlis, Roy H and Curtis, David and Daly, Mark J and Gordon-Smith, Katherine and Meng, Yan A and Grozeva, Detelina and Hamshere, Marian L and Bass, Nicholas J and Franklin, Jennifer and McQuillin, Andrew and Scolnick, Edward M and Jones, Lisa and Stone, Jennifer and Pereira, Ana C P and Craddock, Nick and Gill, Michael and Green, Elaine K and Blumenstiel, Brendan and Defelice, Matthew and Gabriel, Stacey B and Lawrence, Jacob and Chambert, Kimberly and MacIntyre, Donald J and Ruderfer, Douglas M and MacLean, Alan W and Sachs, Gary S and Fraser, Christine and McGuffin, Peter and St Clair, David and Nicol Ferrier, I and Elkin, Amanda and Muir, Walter J and Breen, Gerome and Young, Allan H and Ferreira, Manuel A R and Ardlie, Kristin G and Van Beck, Margaret and Robinson, Michelle and Morris, Derek W and Wellcome Trust Case Control Consor and Wellcome Trust Case Control Consortium
Nature Genetics, ISSN 1061-4036, 09/2008, Volume 40, Issue 9, pp. 1056 - 1058
To identify susceptibility loci for bipolar disorder, we tested 1.8 million variants in 4,387 cases and 6,209 controls and identified a region of strong... 
RANVIER | LOCI | DISEASES | GENES | GENETICS & HEREDITY | Genetic Predisposition to Disease | Genome-Wide Association Study | Calcium Channels, L-Type - genetics | Ankyrins - genetics | Chromosomes, Human, Pair 15 | Humans | Logistic Models | Polymorphism, Single Nucleotide | Chromosomes, Human, Pair 12 | Bipolar Disorder - genetics | Chromosomes, Human, Pair 10 | Calcium channels | Genetic variation | Bipolar disorder | Genetic aspects | Research | Health aspects | Risk factors | Studies | Blood transfusions | Medical research | Inflation | Trinity | Colleges & universities
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4. Full Text Improvements in Neurocognitive Function and Mood Following Adjunctive Treatment with Mifepristone (RU-486) in Bipolar Disorder
by Young, Allan H and Gallagher, Peter and Watson, Stuart and Del-Estal, Dolores and Owen, Bruce M and Ferrier, I. Nicol
Neuropsychopharmacology, ISSN 0893-133X, 08/2004, Volume 29, Issue 8, pp. 1538 - 1545
High cortisol levels are found in severe mood disorders, particularly bipolar disorder. Hypercortisolaemia may cause or exacerbate both neurocognitive... 
Bipolar disorder | Cortisol | Mood | Antiglucocorticoid | Memory | Glucocorticoid receptor | DEPRESSION | glucocorticoid receptor | memory | mood | PSYCHIATRY | antiglucocorticoid | NATURAL-HISTORY | NEUROSCIENCES | TEMPORAL-LOBE | IMPAIRMENT | cortisol | bipolar disorder | CUSHINGS-SYNDROME | DECLARATIVE MEMORY | GLUCOCORTICOID-RECEPTOR | WEEKLY SYMPTOMATIC STATUS | PHARMACOLOGY & PHARMACY | RATING-SCALE | HORMONE TEST | Verbal Learning - drug effects | Affect - drug effects | Receptors, Glucocorticoid - antagonists & inhibitors | Double-Blind Method | Learning - drug effects | Humans | Middle Aged | Male | Memory, Short-Term - drug effects | Treatment Outcome | Psychomotor Performance - drug effects | Cognition - drug effects | Bipolar Disorder - drug therapy | Bipolar Disorder - psychology | Cross-Over Studies | Psychiatric Status Rating Scales | Space Perception - drug effects | Attention - drug effects | Adult | Female | Hormone Antagonists - therapeutic use | Hydrocortisone - blood | Mifepristone - therapeutic use
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5. Full Text Large-scale genome-wide association analysis of bipolar disorder identifies a new susceptibility locus near ODZ4
by Sklar, Pamela and Ripke, Stephan and Scott, Laura J and Andreassen, Ole A and Cichon, Sven and Craddock, Nick and Edenberg, Howard J and Nurnberger Jr, John I and Rietschel, Marcella and Blackwood, Douglas and Corvin, Aiden and Flickinger, Matthew and Guan, Weihua and Mattingsdal, Morten and McQuillin, Andrew and Kwan, Phoenix and Wienker, Thomas F and Daly, Mark and Dudbridge, Frank and Holmans, Peter A and Lin, Danyu and Burmeister, Margit and Greenwood, Tiffany A and Hamshere, Marian L and Muglia, Pierandrea and Smith, Erin N and Zandi, Peter P and Nievergelt, Caroline M and McKinney, Rebecca and Shilling, Paul D and Schork, Nicholas J and Bloss, Cinnamon S and Foroud, Tatiana and Koller, Daniel L and Gershon, Elliot S and Liu, Chunyu and Badner, Judith A and Scheftner, William A and Lawson, William B and Nwulia, Evaristus A and Hipolito, Maria and Coryell, William and Rice, John and Byerley, William and McMahon, Francis J and Schulze, Thomas G and Berrettini, Wade and Lohoff, Falk W and Potash, James B and Mahon, Pamela B and McInnis, Melvin G and Zöllner, Sebastian and Zhang, Peng and Craig, David W and Szelinger, Szabocls and Barrett, Thomas B and Breuer, René and Meier, Sandra and Strohmaier, Jana and Witt, Stephanie H and Tozzi, Federica and Farmer, Anne and McGuffin, Peter and Strauss, John and Xu, Wei and Kennedy, James L and Vincent, John B and Matthews, Keith and Day, Richard and Ferreira, Manuel A and O'Dushlaine, Colm and Perlis, Roy and Raychaudhuri, Soumya and Ruderfer, Douglas and Lee, Phil H and Smoller, Jordan W and Li, Jun and Absher, Devin and Bunney, William E and Barchas, Jack D and Schatzberg, Alan F and Jones, Edward G and Meng, Fan and Thompson, Robert C and Watson, Stanley J and Myers, Richard M and Akil, Huda and Boehnke, Michael and Chambert, Kim and Moran, Jennifer and Scolnick, Ed and Djurovic, Srdjan and Melle, Ingrid and Morken, Gunnar and Gill, Michael and Morris, Derek and Quinn, Emma and Mühleisen, Thomas W and Degenhardt, Franziska A and Mattheisen, Manuel and ... and Psychiat GWAS Consortium Bipolar D and Psychiatric GWAS Consortium Bipolar Disorder Working Group and Sahlgrenska akademin and Institutionen för neurovetenskap och fysiologi, sektionen för psykiatri och neurokemi and Göteborgs universitet and Gothenburg University and Institute of Neuroscience and Physiology, Department of Psychiatry and Neurochemistry and Sahlgrenska Academy
Nature Genetics, ISSN 1061-4036, 10/2011, Volume 43, Issue 10, pp. 977 - 985
We conducted a combined genome-wide association study (GWAS) of 7,481 individuals with bipolar disorder (cases) and 9,250 controls as part of the Psychiatric... 
RISK | SCHIZOPHRENIA | CACNA1C | ANK3 | GENETICS | GENETICS & HEREDITY | Genetic Predisposition to Disease | Genome-Wide Association Study | Humans | Databases, Genetic | Nuclear Proteins - metabolism | Genetic Loci | Bipolar Disorder - genetics | Case-Control Studies | Linkage Disequilibrium | Calcium Channels, L-Type - genetics | Schizophrenia - genetics | Alleles | Polymorphism, Single Nucleotide | Calcium Channels, L-Type - metabolism | Genome, Human | Nuclear Proteins - genetics | Studies | Hospitals | Schizophrenia | Bipolar disorder | Genetics | Genomes | Independent sample | Biochemistry, Molecular Biology | Calcium Channels, L-Type | Nuclear Proteins | Life Sciences | Bipolar Disorder | genetics | metabolism | Psykiatri | Psychiatry
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6. Full Text Neurocognitive impairment in euthymic patients with bipolar affective disorder
by Thompson, Jill M and Gallagher, Peter and Hughes, John H and Watson, Stuart and Gray, John M and Ferrier, I. Nicol and Young, Allan H
The British Journal of Psychiatry, ISSN 0007-1250, 01/2005, Volume 186, Issue 1, pp. 32 - 40
Background Persistent impairments in neurocognitive function have been described in patients with bipolar disorder whose disease is in remission. However,... 
DEPRESSION | FOLLOW | COGNITIVE IMPAIRMENT | MANIA | RATING-SCALE | PSYCHIATRY | Psychomotor Performance | Bipolar Disorder - metabolism | Multivariate Analysis | Humans | Middle Aged | Attention | Male | Saliva - chemistry | Bipolar Disorder - drug therapy | Case-Control Studies | Bipolar Disorder - psychology | Antidepressive Agents - therapeutic use | Neuropsychological Tests | Hydrocortisone - analysis | Adult | Female | Memory, Short-Term | Neurosciences | Emotional disorders | Secretion | Neurobiology | Cognitive ability | Bipolar disorder | Alcohol | Cognition | Cortisol | Mental depression | Hydrocortisone | Patients | Residual symptoms | Studies | Illnesses | Mood | Neurocognitive functioning | Dysfunction | Remission | Saliva | Symptoms
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7. Full Text Genome-wide association study of CNVs in 16,000 cases of eight common diseases and 3,000 shared controls
by Craddock, Nick and Hurles, Matthew E and Cardin, Niall and Pearson, Richard D and Plagnol, Vincent and Robson, Samuel and Vukcevic, Damjan and Barnes, Chris and Conrad, Donald F and Giannoulatou, Eleni and Holmes, Chris and Marchini, Jonathan L and Stirrups, Kathy and Tobin, Martin D and Wain, Louise V and Yau, Chris and Aerts, Jan and Ahmad, Tariq and Andrews, T. Daniel and Arbury, Hazel and Attwood, Anthony and Auton, Adam and Ball, Stephen G and Balmforth, Anthony J and Barrett, Jeffrey C and Barroso, Inês and Barton, Anne and Bennett, Amanda J and Bhaskar, Sanjeev and Blaszczyk, Katarzyna and Bowes, John and Brand, Oliver J and Braund, Peter S and Bredin, Francesca and Breen, Gerome and Brown, Morris J and Bruce, Ian N and Bull, Jaswinder and Burren, Oliver S and Burton, John and Byrnes, Jake and Caesar, Sian and Clee, Chris M and Coffey, Alison J and Connell, John M. C and Cooper, Jason D and Dominiczak, Anna F and Downes, Kate and Drummond, Hazel E and Dudakia, Darshna and Dunham, Andrew and Ebbs, Bernadette and Eccles, Diana and Edkins, Sarah and Edwards, Cathryn and Elliot, Anna and Emery, Paul and Evans, David M and Evans, Gareth and Eyre, Steve and Farmer, Anne and Ferrier, I. Nicol and Feuk, Lars and Fitzgerald, Tomas and Flynn, Edward and Forbes, Alistair and Forty, Liz and Franklyn, Jayne A and Freathy, Rachel M and Gibbs, Polly and Gilbert, Paul and Gokumen, Omer and Gordon-Smith, Katherine and Gray, Emma and Green, Elaine and Groves, Chris J and Grozeva, Detelina and Gwilliam, Rhian and Hall, Anita and Hammond, Naomi and Hardy, Matt and Harrison, Pile and Hassanali, Neelam and Hebaishi, Husam and Hines, Sarah and Hinks, Anne and Hitman, Graham A and Hocking, Lynne and Howard, Eleanor and Howard, Philip and Howson, Joanna M. M and Hughes, Debbie and Hunt, Sarah and Isaacs, John D and Jain, Mahim and Jewell, Derek P and Johnson, Toby and Jolley, Jennifer D and Jones, Ian R and Jones, Lisa A and ... and Wellcome Trust Case Control and Wellcome Trust Case Control Consortium and The Wellcome Trust Case Control Consortium and Medicinska fakulteten and Medicinska och farmaceutiska vetenskapsområdet and Institutionen för genetik och patologi and Uppsala universitet
Nature, ISSN 0028-0836, 04/2010, Volume 464, Issue 7289, pp. 713 - 720
Copy number variants (CNVs) account for a major proportion of human genetic polymorphism and have been predicted to have an important role in genetic... 
POLYMORPHISMS | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | LARGE-SCALE | HEALTH | HERITABILITY | COPY-NUMBER VARIATION | DELETION | Crohn Disease - genetics | Gene Frequency - genetics | Genetic Predisposition to Disease - genetics | Genome-Wide Association Study | Nucleic Acid Hybridization | Disease | Oligonucleotide Array Sequence Analysis | Diabetes Mellitus - genetics | Humans | DNA Copy Number Variations - genetics | Case-Control Studies | Arthritis, Rheumatoid - genetics | Pilot Projects | Polymorphism, Single Nucleotide - genetics | Quality Control | Genetic variation | Chronic diseases | Genetic aspects | Research | Single nucleotide polymorphisms | Health aspects | Risk factors | Studies | Design | Rheumatoid arthritis | Genetics | Cardiovascular disease | Principal components analysis | Statistical methods | Diabetes | Cancer | Human | Crohn's Disease | Reproduction | Arrays | Loci | Polymorphism | Diseases | Life Sciences | Medical and Health Sciences | MEDICINE | Medicin och hälsovetenskap | MEDICIN
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8. Full Text Hypothalamic-pituitary-adrenal axis function in patients with bipolar disorder
by Watson, Stuart and Gallagher, Peter and Ritchie, James C and Ferrier, I. Nicol and Young, Allan H
The British Journal of Psychiatry, ISSN 0007-1250, 06/2004, Volume 184, Issue 6, pp. 496 - 502
Background Hypothalamic-pituitary-adrenal (HPA) axis function, as variously measured by the responses to the combined dexamethasone/corticotrophin-releasing... 
HALF-LIFE | PSYCHIATRY | ADRENOCORTICAL REGULATION | REACTIVITY | PSYCHIATRIC-DISORDERS | DEXAMETHASONE SUPPRESSION TEST | MAJOR DEPRESSION | RATING-SCALE | CRH TEST | HORMONE TEST | CORTISOL | Pituitary-Adrenal System - physiopathology | Antimanic Agents - therapeutic use | Dexamethasone | Cross-Sectional Studies | Humans | Middle Aged | Bipolar Disorder - physiopathology | Carbamazepine - therapeutic use | Male | Saliva - chemistry | Bipolar Disorder - drug therapy | Corticotropin-Releasing Hormone | Pituitary-Adrenal Function Tests - methods | Antidepressive Agents - therapeutic use | Hydrocortisone - analysis | Adult | Female | Hydrocortisone - blood | Hypothalamo-Hypophyseal System - physiopathology | Menstruation | Mental disorders | Suppression | Hypothalamic-pituitary-adrenal axis | Bipolar disorder | Cortisol | Hypothalamus | Hormones | Hydrocortisone | Corticotropin-releasing hormone | Salivary cortisol | Studies | Dysfunction | Pituitary | Serum | Remission | Age
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9. Full Text Neurocognitive function in unaffected first-degree relatives of patients with bipolar disorder: a preliminary report
by Nicol Ferrier I and Chowdhury R and Thompson J.M and Watson S and Young A.H
Bipolar Disorders, ISSN 1398-5647, 08/2004, Volume 6, Issue 4, pp. 319 - 322
Ferrier IN, Chowdhury R, Thompson JM, Watson S, Young AH. Neurocognitive function in unaffected first-degree relatives of patients with bipolar disorder: a... 
first-degree relatives | high-risk paradigm | bipolar disorder | neuropsychological function | first‐degree relatives | high‐risk paradigm | Neuropsychological function | Bipolar disorder | First-degree relatives | High-risk paradigm | MEMORY | PSYCHIATRY | COGNITIVE IMPAIRMENT | RATING-SCALE | NEUROSCIENCES | CLINICAL NEUROLOGY | Neuropsychological Tests | Cross-Sectional Studies | Humans | Cognition - physiology | Adult | Female | Male | Bipolar Disorder - diagnosis | Bipolar Disorder - genetics | Remission Induction | Bipolar Disorder - epidemiology
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10. Full Text A meta-analysis of cognitive deficits in euthymic patients with bipolar disorder
by Robinson, Lucy J and Thompson, Jill M and Gallagher, Peter and Goswami, Utpal and Young, Allan H and Ferrier, I. Nicol and Moore, P. Brian
Journal of Affective Disorders, ISSN 0165-0327, 07/2006, Volume 93, Issue 1-3, pp. 105 - 115
A number of studies have reported evidence of cognitive deficits in euthymic bipolar patients. Qualitative reviews of the literature have indicated impairments... 
Bipolar disorder | Neuropsychology | Cognitive function | Memory | Euthymia | Meta-analysis | cognitive function | I DISORDER | SUBGENUAL PREFRONTAL CORTEX | memory | euthymia | ABNORMALITIES | PSYCHIATRY | SUSTAINED ATTENTION-DEFICIT | MEMORY IMPAIRMENT | neuropsychology | meta-analysis | 1ST-DEGREE RELATIVES | SCHIZOPHRENIA | CLINICAL NEUROLOGY | EXECUTIVE FUNCTION | bipolar disorder | NEUROCOGNITIVE FUNCTION | UNIPOLAR DEPRESSION | Neuropsychological Tests | Cognition Disorders - diagnosis | Humans | Affect | Bipolar Disorder - diagnosis | Bipolar Disorder - psychology | Cognition Disorders - psychology | Analysis
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