Psychopharmacology, ISSN 0033-3158, 9/2016, Volume 233, Issue 18, pp. 3437 - 3448
Chronic exposure to drugs of abuse changes glutamatergic transmission in human addicts and animal models. N-acetylcysteine (NAC) is a cysteine prodrug that...
Extinction training | Substance use disorder | N-acetylcysteine (NAC) | Neurosciences | Cue | NAC-amide, reinstatement | Biomedicine | Cocaine | Pharmacology/Toxicology | Psychiatry | OXIDATIVE STRESS | THIOL ANTIOXIDANT | PSYCHIATRY | BEHAVIORAL SENSITIZATION | RATS | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | NUCLEUS-ACCUMBENS | GLUTAMATE | INDUCED DRUG-SEEKING | PHARMACOLOGY & PHARMACY | EXTINCTION | SYNAPTIC POTENTIATION | Cues | Rats, Wistar | Extinction, Psychological - drug effects | Rats | Depression | Male | Cocaine-Related Disorders | Drug-Seeking Behavior - drug effects | Dopamine Uptake Inhibitors - administration & dosage | Cocaine - administration & dosage | Self Administration | Animals | Acetylcysteine - analogs & derivatives | Olfactory Bulb - surgery | Acetylcysteine - pharmacology | Behavior, Animal - drug effects | Reward | Psychological aspects | Drug abuse | Amides | Cysteine | Health aspects | Psychopharmacology
Extinction training | Substance use disorder | N-acetylcysteine (NAC) | Neurosciences | Cue | NAC-amide, reinstatement | Biomedicine | Cocaine | Pharmacology/Toxicology | Psychiatry | OXIDATIVE STRESS | THIOL ANTIOXIDANT | PSYCHIATRY | BEHAVIORAL SENSITIZATION | RATS | BLOOD-BRAIN-BARRIER | NEUROSCIENCES | NUCLEUS-ACCUMBENS | GLUTAMATE | INDUCED DRUG-SEEKING | PHARMACOLOGY & PHARMACY | EXTINCTION | SYNAPTIC POTENTIATION | Cues | Rats, Wistar | Extinction, Psychological - drug effects | Rats | Depression | Male | Cocaine-Related Disorders | Drug-Seeking Behavior - drug effects | Dopamine Uptake Inhibitors - administration & dosage | Cocaine - administration & dosage | Self Administration | Animals | Acetylcysteine - analogs & derivatives | Olfactory Bulb - surgery | Acetylcysteine - pharmacology | Behavior, Animal - drug effects | Reward | Psychological aspects | Drug abuse | Amides | Cysteine | Health aspects | Psychopharmacology
Journal Article
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2333-PUB: CPL207-280, a Potent and Safe GPR40 Agonist for the treatment of Type 2 Diabetes
Diabetes, ISSN 0012-1797, 06/2019, Volume 68, Issue Supplement 1, p. 2333
Journal Article
Journal of Strength and Conditioning Research, ISSN 1064-8011, 05/2018, Volume 32, Issue 5, pp. 1311 - 1315
ABSTRACTGinszt, M, Michalak-Wojnowska, M, Gawda, P, Wojcierowska-Litwin, M, Korszeń-Pilecka, I, Kusztelak, M, Muda, R, Filip, AA, and Majcher, P. ACTN3...
sport climbing | ROCK CLIMBERS | SPORT SCIENCES | PHYSIOLOGY | ENDURANCE | rock climbing | ATHLETIC PERFORMANCE | R577X | STRENGTH | POLYMORPHISM | VARIANT | genetics | GENE | sport performance | ALPHA-ACTININ-3 | Polymerase chain reaction | Sprinting | Analysis | Runners (Sports) | Genotype | Performances | Research | Genotype & phenotype | Restriction fragment length polymorphism | Gene frequency | Professional sports | Climbing | Alleles | α-Actinin | Gene polymorphism | Genotypes | Polymorphism | Original Research
sport climbing | ROCK CLIMBERS | SPORT SCIENCES | PHYSIOLOGY | ENDURANCE | rock climbing | ATHLETIC PERFORMANCE | R577X | STRENGTH | POLYMORPHISM | VARIANT | genetics | GENE | sport performance | ALPHA-ACTININ-3 | Polymerase chain reaction | Sprinting | Analysis | Runners (Sports) | Genotype | Performances | Research | Genotype & phenotype | Restriction fragment length polymorphism | Gene frequency | Professional sports | Climbing | Alleles | α-Actinin | Gene polymorphism | Genotypes | Polymorphism | Original Research
Journal Article
Addiction Biology, ISSN 1355-6215, 05/2013, Volume 18, Issue 3, pp. 455 - 466
ABSTRACT Striatal adenosine (A)2‐dopamine (D)2 receptor (R) heteromers exist with antagonistic interactions. We have studied these Rs and their interactions...
dorsla striatum | ‘yoked’ controls | cocaine self‐administration | dopamine D2 receptor | Adenosine A2A receptor | ventral striatum | cocaine self-administration | receptor | dopamine D | 'yoked' controls | Adenosine A | DOPAMINE-D-2 RECEPTOR | "yoked' controls | ANTAGONIST | BIOCHEMISTRY & MOLECULAR BIOLOGY | SUBSTANCE ABUSE | NEUROCHEMICAL CHANGES | DECREASES | ADENOSINE A(2A) RECEPTORS | AFFINITY | INCREASE | BINDING | GTP-Binding Protein gamma Subunits - metabolism | Phenethylamines - pharmacology | Dopamine Uptake Inhibitors - pharmacology | Extinction, Psychological - drug effects | Rats | Male | Adenosine - pharmacology | Receptor, Adenosine A2A - metabolism | Receptors, Dopamine D2 - metabolism | Receptor, Adenosine A2A - drug effects | Cocaine - administration & dosage | Cocaine - pharmacology | Reinforcement Schedule | Self Administration | Animals | Adenosine A2 Receptor Agonists - pharmacology | Adenosine - analogs & derivatives | Receptors, Dopamine D2 - drug effects | Corpus Striatum - drug effects | Infusions, Intravenous | Drugstores | Adenosine | Neurosciences | Cocaine | Dopamine receptors | Index Medicus
dorsla striatum | ‘yoked’ controls | cocaine self‐administration | dopamine D2 receptor | Adenosine A2A receptor | ventral striatum | cocaine self-administration | receptor | dopamine D | 'yoked' controls | Adenosine A | DOPAMINE-D-2 RECEPTOR | "yoked' controls | ANTAGONIST | BIOCHEMISTRY & MOLECULAR BIOLOGY | SUBSTANCE ABUSE | NEUROCHEMICAL CHANGES | DECREASES | ADENOSINE A(2A) RECEPTORS | AFFINITY | INCREASE | BINDING | GTP-Binding Protein gamma Subunits - metabolism | Phenethylamines - pharmacology | Dopamine Uptake Inhibitors - pharmacology | Extinction, Psychological - drug effects | Rats | Male | Adenosine - pharmacology | Receptor, Adenosine A2A - metabolism | Receptors, Dopamine D2 - metabolism | Receptor, Adenosine A2A - drug effects | Cocaine - administration & dosage | Cocaine - pharmacology | Reinforcement Schedule | Self Administration | Animals | Adenosine A2 Receptor Agonists - pharmacology | Adenosine - analogs & derivatives | Receptors, Dopamine D2 - drug effects | Corpus Striatum - drug effects | Infusions, Intravenous | Drugstores | Adenosine | Neurosciences | Cocaine | Dopamine receptors | Index Medicus
Journal Article
Genetics in medicine, ISSN 1098-3600, 2015, Volume 17, Issue 6, pp. 460 - 466
Submicroscopic deletions of chromosome band 2p25.3 are associated with intellectual disability and/or central obesity. Although MYT1L is believed to be a...
FINGER TRANSCRIPTION FACTORS | NERVOUS-SYSTEM | MYT1L | ZINC-FINGER | DIRECT CONVERSION | SCHIZOPHRENIA | 2p25.3 microdeletion | GENES | GENETICS & HEREDITY | 2p25.3 microduplication | NEURONS | WIDE ASSOCIATION | COMPARATIVE GENOMIC HYBRIDIZATION | COPY NUMBER VARIANTS | intellectual disability | obesity | Chromosome Deletion | Gene Duplication | Gene Expression | Genetic Association Studies | Humans | Middle Aged | Child, Preschool | Male | Zebrafish | Chromosome Mapping | Transcription Factors - genetics | Nerve Tissue Proteins - genetics | Obesity - genetics | Intellectual Disability - genetics | Chromosomes, Human, Pair 2 | Young Adult | Point Mutation | Animals | Adolescent | Facies | Adult | Female | Child | Cohort Studies
FINGER TRANSCRIPTION FACTORS | NERVOUS-SYSTEM | MYT1L | ZINC-FINGER | DIRECT CONVERSION | SCHIZOPHRENIA | 2p25.3 microdeletion | GENES | GENETICS & HEREDITY | 2p25.3 microduplication | NEURONS | WIDE ASSOCIATION | COMPARATIVE GENOMIC HYBRIDIZATION | COPY NUMBER VARIANTS | intellectual disability | obesity | Chromosome Deletion | Gene Duplication | Gene Expression | Genetic Association Studies | Humans | Middle Aged | Child, Preschool | Male | Zebrafish | Chromosome Mapping | Transcription Factors - genetics | Nerve Tissue Proteins - genetics | Obesity - genetics | Intellectual Disability - genetics | Chromosomes, Human, Pair 2 | Young Adult | Point Mutation | Animals | Adolescent | Facies | Adult | Female | Child | Cohort Studies
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2015, Volume 10, Issue 9, p. e0139197
Assessment of non-HLA variants alongside standard HLA testing was previously shown to improve the identification of potential coeliac disease (CD) patients. We...
DIAGNOSIS | HMSH5 | PREVALENCE | EUROPEAN GENETICS CLUSTER | MULTIDISCIPLINARY SCIENCES | Care and treatment | Genomics | Development and progression | Genetic aspects | Research | Single nucleotide polymorphisms | Autoimmune diseases | Risk factors | Haplotypes | Pediatrics | DQA1 protein | Pathogenesis | Association analysis | Oncology | Risk | Genomes | Single-nucleotide polymorphism | Gene polymorphism | Genetic screening | Consortia | Hepatology | Gastroenterology | Population | Genetics | Genotypes | Deoxyribonucleic acid--DNA | Immunoglobulins | Sensitivity analysis | Health risks | Marking | Patients | Genetic variance | Pathology | Celiac disease | Sensitivity | Major histocompatibility complex | Predictions | Tagging | Gene loci | Histocompatibility antigen HLA | Genetic testing | Tissue typing | Histocompatibility | Cancer | Polymorphism | Deoxyribonucleic acid | DNA
DIAGNOSIS | HMSH5 | PREVALENCE | EUROPEAN GENETICS CLUSTER | MULTIDISCIPLINARY SCIENCES | Care and treatment | Genomics | Development and progression | Genetic aspects | Research | Single nucleotide polymorphisms | Autoimmune diseases | Risk factors | Haplotypes | Pediatrics | DQA1 protein | Pathogenesis | Association analysis | Oncology | Risk | Genomes | Single-nucleotide polymorphism | Gene polymorphism | Genetic screening | Consortia | Hepatology | Gastroenterology | Population | Genetics | Genotypes | Deoxyribonucleic acid--DNA | Immunoglobulins | Sensitivity analysis | Health risks | Marking | Patients | Genetic variance | Pathology | Celiac disease | Sensitivity | Major histocompatibility complex | Predictions | Tagging | Gene loci | Histocompatibility antigen HLA | Genetic testing | Tissue typing | Histocompatibility | Cancer | Polymorphism | Deoxyribonucleic acid | DNA
Journal Article
Journal of Pharmacology and Experimental Therapeutics, ISSN 0022-3565, 08/2003, Volume 306, Issue 2, pp. 734 - 743
Journal Article
Brain Research, ISSN 0006-8993, 2016, Volume 1655, pp. 66 - 76
Abstract Several behavioral findings highlight the importance of glutamatergic transmission and its metabotropic receptor type 5 (mGlu5 ) in the controlling of...
Neurology | Extinction training | Drug-induced relapse | MGlu5 receptor immunohistochemistry | MGlu5 receptor binding | Cocaine self-administration | receptor binding | receptor immunohistochemistry | MGlu | INDUCED REINSTATEMENT | SUBUNIT EXPRESSION | TIME-COURSE | NEUROSCIENCES | PREFRONTAL CORTEX | ADDICTION | NUCLEUS-ACCUMBENS | PRIMED REINSTATEMENT | MGlu receptor immunohistochemistry | PROTEIN EXPRESSION | DORSAL STRIATUM | MGlu receptor binding | RAT-BRAIN | Pyridines | Recurrence | Rats, Wistar | Cocaine-Related Disorders - pathology | Male | Tritium | Receptor, Metabotropic Glutamate 5 - metabolism | Dopamine Uptake Inhibitors - administration & dosage | Brain - metabolism | Cocaine - administration & dosage | Self Administration | Animals | Cocaine-Related Disorders - metabolism | Analysis of Variance | Fluorescent Antibody Technique | Brain - pathology | Radioligand Assay | Radiopharmaceuticals | Excitatory Amino Acid Antagonists | Extinction, Psychological - physiology | Disease Models, Animal | Immunohistochemistry | Drug abuse | Cocaine | Glutamate | Analysis | Index Medicus
Neurology | Extinction training | Drug-induced relapse | MGlu5 receptor immunohistochemistry | MGlu5 receptor binding | Cocaine self-administration | receptor binding | receptor immunohistochemistry | MGlu | INDUCED REINSTATEMENT | SUBUNIT EXPRESSION | TIME-COURSE | NEUROSCIENCES | PREFRONTAL CORTEX | ADDICTION | NUCLEUS-ACCUMBENS | PRIMED REINSTATEMENT | MGlu receptor immunohistochemistry | PROTEIN EXPRESSION | DORSAL STRIATUM | MGlu receptor binding | RAT-BRAIN | Pyridines | Recurrence | Rats, Wistar | Cocaine-Related Disorders - pathology | Male | Tritium | Receptor, Metabotropic Glutamate 5 - metabolism | Dopamine Uptake Inhibitors - administration & dosage | Brain - metabolism | Cocaine - administration & dosage | Self Administration | Animals | Cocaine-Related Disorders - metabolism | Analysis of Variance | Fluorescent Antibody Technique | Brain - pathology | Radioligand Assay | Radiopharmaceuticals | Excitatory Amino Acid Antagonists | Extinction, Psychological - physiology | Disease Models, Animal | Immunohistochemistry | Drug abuse | Cocaine | Glutamate | Analysis | Index Medicus
Journal Article
Talanta, ISSN 0039-9140, 08/2017, Volume 170, pp. 158 - 164
In this work it was demonstrated for the first time that Independent Components Analysis non-deterministic multivariate strategy enables estimation of the...
Voltammetry | Faradaic current | Capacitive current | ICA
Voltammetry | Faradaic current | Capacitive current | ICA
Journal Article
Folia Histochemica et Cytobiologica, ISSN 0239-8508, 2017, Volume 55, Issue 4, pp. 203 - 211
Introduction. Significant and accessible predictive factors for bortezomib treatment in plasma cell myeloma (PCM) are still lacking. TP53 codon 72 polymorphism...
Bortezomib | TP53 gene | Plasma cell myeloma | Apoptosis | Necrosis | Polymorphism | MULTIPLE-MYELOMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | apoptosis | RISK | polymorphism | CELL BIOLOGY | plasma cell myeloma | LUNG-CANCER | P53 GENE | necrosis | PROTEASOME INHIBITOR BORTEZOMIB | PROGNOSTIC-SIGNIFICANCE | MUTATIONS | LEUKEMIA | bortezomib | CODON 72 POLYMORPHISM | HUMAN CANCER | Plasma | Cell culture | Biotechnology | p53 Protein | Multiple myeloma | Proline | Amino acids | Myeloma | Gene polymorphism | Cancer therapies | DNA repair | Subgroups | Proteins | Genotype & phenotype | Signal transduction | Cell growth | Arginine | Cell cycle | Gangrene | Bone marrow | Genotypes | Deoxyribonucleic acid--DNA | Sensitivity analysis | Hematology | Cultures | Gene expression | Patients | Blood donors | Medical prognosis | Alleles | Mutation | Chromosome aberrations
Bortezomib | TP53 gene | Plasma cell myeloma | Apoptosis | Necrosis | Polymorphism | MULTIPLE-MYELOMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | apoptosis | RISK | polymorphism | CELL BIOLOGY | plasma cell myeloma | LUNG-CANCER | P53 GENE | necrosis | PROTEASOME INHIBITOR BORTEZOMIB | PROGNOSTIC-SIGNIFICANCE | MUTATIONS | LEUKEMIA | bortezomib | CODON 72 POLYMORPHISM | HUMAN CANCER | Plasma | Cell culture | Biotechnology | p53 Protein | Multiple myeloma | Proline | Amino acids | Myeloma | Gene polymorphism | Cancer therapies | DNA repair | Subgroups | Proteins | Genotype & phenotype | Signal transduction | Cell growth | Arginine | Cell cycle | Gangrene | Bone marrow | Genotypes | Deoxyribonucleic acid--DNA | Sensitivity analysis | Hematology | Cultures | Gene expression | Patients | Blood donors | Medical prognosis | Alleles | Mutation | Chromosome aberrations
Journal Article
Journal of Clinical Epidemiology, ISSN 0895-4356, 2013, Volume 66, Issue 3, pp. 286 - 295
Abstract Objective To compare methodological characteristics of randomized controlled trials (RCTs) published in higher vs. lower impact Core Clinical...
Internal Medicine | Systematic review | Research design | Periodical | Randomized controlled trial | Humans | Data interpretation | BIAS | PUBLIC, ENVIRONMENTAL & OCCUPATIONAL HEALTH | CONCEALMENT | CLINICAL-TRIALS | HEALTH CARE SCIENCES & SERVICES | EMPIRICAL-EVIDENCE | SUBGROUP ANALYSIS | OUTCOMES | METHODOLOGICAL QUALITY | Data Interpretation, Statistical | Sample Size | Randomized Controlled Trials as Topic - methods | Bias | Journal Impact Factor | Research Design - standards | Periodicals as Topic - statistics & numerical data | Periodicals as Topic - standards | Randomized Controlled Trials as Topic - standards | Medical policy | Medical colleges | Analgesics | Analysis | Children's hospitals | Clinical trials | Children | Health aspects | Biometry | Studies | Medicine | Confidence intervals | Health sciences | Internal medicine | Sample size | Journals | Public health | Obstetrics
Internal Medicine | Systematic review | Research design | Periodical | Randomized controlled trial | Humans | Data interpretation | BIAS | PUBLIC, ENVIRONMENTAL & OCCUPATIONAL HEALTH | CONCEALMENT | CLINICAL-TRIALS | HEALTH CARE SCIENCES & SERVICES | EMPIRICAL-EVIDENCE | SUBGROUP ANALYSIS | OUTCOMES | METHODOLOGICAL QUALITY | Data Interpretation, Statistical | Sample Size | Randomized Controlled Trials as Topic - methods | Bias | Journal Impact Factor | Research Design - standards | Periodicals as Topic - statistics & numerical data | Periodicals as Topic - standards | Randomized Controlled Trials as Topic - standards | Medical policy | Medical colleges | Analgesics | Analysis | Children's hospitals | Clinical trials | Children | Health aspects | Biometry | Studies | Medicine | Confidence intervals | Health sciences | Internal medicine | Sample size | Journals | Public health | Obstetrics
Journal Article
Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology, ISSN 0893-133X, 07/2012, Volume 37, Issue 8, pp. 1856 - 1866
Recent in vitro results suggest that cocaine may exert direct and/or indirect allosteric enhancing actions at dopamine (DA) D(2) receptors (D(2)Rs). In the...
Microinjections | Rats, Wistar | Nucleus Accumbens - metabolism | Receptors, Dopamine D2 - agonists | Dopamine - pharmacology | Rats | Male | Receptors, Dopamine D2 - metabolism | Corpus Striatum - metabolism | Piperazines - pharmacology | Quinpirole - administration & dosage | Rats, Sprague-Dawley | Drug Synergism | Glutamic Acid | Cocaine - administration & dosage | Cocaine - pharmacology | Guanosine 5'-O-(3-Thiotriphosphate) - metabolism | Animals | Corpus Striatum - drug effects | Receptors, Dopamine D2 - physiology | Quinpirole - pharmacology | Nucleus Accumbens - drug effects | GTP-Binding Protein alpha Subunits, Gi-Go - metabolism | Locomotion - drug effects | [35S]GTPgammaS | Addiction & Substance Abuse | Molecular & Cellular Neurobiology | microdialysis | GBR 12783 | D2-like receptors | locomotor activity | nucleus accumbens | Neuropharmacology | Original | glutamate | [35S]GTPγS
Microinjections | Rats, Wistar | Nucleus Accumbens - metabolism | Receptors, Dopamine D2 - agonists | Dopamine - pharmacology | Rats | Male | Receptors, Dopamine D2 - metabolism | Corpus Striatum - metabolism | Piperazines - pharmacology | Quinpirole - administration & dosage | Rats, Sprague-Dawley | Drug Synergism | Glutamic Acid | Cocaine - administration & dosage | Cocaine - pharmacology | Guanosine 5'-O-(3-Thiotriphosphate) - metabolism | Animals | Corpus Striatum - drug effects | Receptors, Dopamine D2 - physiology | Quinpirole - pharmacology | Nucleus Accumbens - drug effects | GTP-Binding Protein alpha Subunits, Gi-Go - metabolism | Locomotion - drug effects | [35S]GTPgammaS | Addiction & Substance Abuse | Molecular & Cellular Neurobiology | microdialysis | GBR 12783 | D2-like receptors | locomotor activity | nucleus accumbens | Neuropharmacology | Original | glutamate | [35S]GTPγS
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 8/2018, Volume 55, Issue 8, pp. 7038 - 7048
Antagonistic allosteric A2AR-D2R receptor-receptor interactions in heteroreceptor complexes counteract cocaine self-administration and cocaine seeking in rats...
Substance use disorder | Neurology | Neurosciences | Biomedicine | Dopamine D2 receptor | Adenosine A2A receptor | Cocaine self-administration | Neurobiology | Interfering peptides | Heteroreceptor complexes | Dimerization | Cell Biology | A(2A) RECEPTORS | RECEPTOR-RECEPTOR INTERACTIONS | ADENOSINE A | USE DISORDER | NEUROSCIENCES | ADDICTION | RELEVANCE | DOPAMINE | SEEKING | PROXIMITY LIGATION ASSAY | EXTINCTION | Amino Acid Sequence | Peptides - chemistry | Humans | DNA, Complementary - genetics | Male | Receptor, Adenosine A2A - metabolism | Receptors, Dopamine D2 - metabolism | Rats, Sprague-Dawley | Peptides - administration & dosage | Peptides - pharmacology | Cocaine - administration & dosage | Self Administration | Animals | HEK293 Cells | Fluorescence Resonance Energy Transfer | Receptor, Adenosine A2A - chemistry | Animal experimentation | Physiological aspects | Substance abuse | Cocaine | Peptides | Nucleus accumbens | Drug self-administration | Allosteric properties | Bioluminescence | Rodents | Rats | Energy transfer | Biological Sciences | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Naturvetenskap | Biologiska vetenskaper | Medicinska och farmaceutiska grundvetenskaper | Natural Sciences | Neurovetenskaper
Substance use disorder | Neurology | Neurosciences | Biomedicine | Dopamine D2 receptor | Adenosine A2A receptor | Cocaine self-administration | Neurobiology | Interfering peptides | Heteroreceptor complexes | Dimerization | Cell Biology | A(2A) RECEPTORS | RECEPTOR-RECEPTOR INTERACTIONS | ADENOSINE A | USE DISORDER | NEUROSCIENCES | ADDICTION | RELEVANCE | DOPAMINE | SEEKING | PROXIMITY LIGATION ASSAY | EXTINCTION | Amino Acid Sequence | Peptides - chemistry | Humans | DNA, Complementary - genetics | Male | Receptor, Adenosine A2A - metabolism | Receptors, Dopamine D2 - metabolism | Rats, Sprague-Dawley | Peptides - administration & dosage | Peptides - pharmacology | Cocaine - administration & dosage | Self Administration | Animals | HEK293 Cells | Fluorescence Resonance Energy Transfer | Receptor, Adenosine A2A - chemistry | Animal experimentation | Physiological aspects | Substance abuse | Cocaine | Peptides | Nucleus accumbens | Drug self-administration | Allosteric properties | Bioluminescence | Rodents | Rats | Energy transfer | Biological Sciences | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Naturvetenskap | Biologiska vetenskaper | Medicinska och farmaceutiska grundvetenskaper | Natural Sciences | Neurovetenskaper
Journal Article
Pharmacological Reports, ISSN 1734-1140, 04/2014, Volume 66, Issue 2, pp. 334 - 334
Journal Article
Progress in Neuropsychopharmacology & Biological Psychiatry, ISSN 0278-5846, 04/2014, Volume 50, pp. 1 - 10
Preclinical investigations have demonstrated that drugs of abuse alter the levels of lipid-based signalling molecules, including endocannabinoids (eCBs) and...
N-acylethanolamine | Endocannabinoid | “Yoked” procedure | Cocaine self-administration | "Yoked" procedure | DRUG | INDUCED REINSTATEMENT | PSYCHIATRY | PROLIFERATOR-ACTIVATED RECEPTORS | CONDITIONED PLACE PREFERENCE | ANANDAMIDE | ENDOGENOUS CANNABINOID SYSTEM | NEUROSCIENCES | CLINICAL NEUROLOGY | NUCLEUS-ACCUMBENS | ACID AMIDE HYDROLASE | SEEKING BEHAVIOR | DOPAMINE | PHARMACOLOGY & PHARMACY | Endocannabinoids - metabolism | Reinforcement (Psychology) | Rats | Male | Glycerides - metabolism | Polyunsaturated Alkamides - metabolism | Brain - drug effects | Brain - metabolism | Oleic Acids - metabolism | Cocaine - administration & dosage | Cocaine - pharmacology | Arachidonic Acids - metabolism | Self Administration | Animals | Ethanolamines - metabolism | Extinction, Psychological | Conditioning, Operant | Palmitic Acids - metabolism | Brain | Cocaine | Index Medicus
N-acylethanolamine | Endocannabinoid | “Yoked” procedure | Cocaine self-administration | "Yoked" procedure | DRUG | INDUCED REINSTATEMENT | PSYCHIATRY | PROLIFERATOR-ACTIVATED RECEPTORS | CONDITIONED PLACE PREFERENCE | ANANDAMIDE | ENDOGENOUS CANNABINOID SYSTEM | NEUROSCIENCES | CLINICAL NEUROLOGY | NUCLEUS-ACCUMBENS | ACID AMIDE HYDROLASE | SEEKING BEHAVIOR | DOPAMINE | PHARMACOLOGY & PHARMACY | Endocannabinoids - metabolism | Reinforcement (Psychology) | Rats | Male | Glycerides - metabolism | Polyunsaturated Alkamides - metabolism | Brain - drug effects | Brain - metabolism | Oleic Acids - metabolism | Cocaine - administration & dosage | Cocaine - pharmacology | Arachidonic Acids - metabolism | Self Administration | Animals | Ethanolamines - metabolism | Extinction, Psychological | Conditioning, Operant | Palmitic Acids - metabolism | Brain | Cocaine | Index Medicus
Journal Article