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Biology of Blood and Marrow Transplantation, ISSN 1083-8791, 03/2019, Volume 25, Issue 3, p. S60
To reduce non-relapse mortality (NRM) due to graft versus host disease (GVHD) in matched related HSCT, we used cyclophosphamide (CY) T cell tolerization within... 
Relapse | Operating systems | Analysis | Mortality | Patient outcomes | Stem cells | Transplantation | T cells | Hematopoietic stem cells | Diseases
Journal Article
Cell Cycle, ISSN 1538-4101, 09/2010, Volume 9, Issue 17, pp. 3506 - 3514
Journal Article
Cell Cycle, ISSN 1538-4101, 12/2009, Volume 8, Issue 23, pp. 3984 - 4001
Journal Article
Cell Cycle, ISSN 1538-4101, 08/2011, Volume 10, Issue 15, pp. 2504 - 2520
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 05/2019, Volume 37, Issue 15_suppl, pp. 6632 - 6632
Journal Article
Cell Cycle, ISSN 1538-4101, 08/2010, Volume 9, Issue 16, pp. 3276 - 3296
Loss of stromal fibroblast caveolin-1 (Cav-1) is a powerful single independent predictor of poor prognosis in human breast cancer patients, and is associated... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Caveolin-1 | Oxidative stress | Mitochondrial dysfunction | DNA damage | Anti-oxidant cancer therapy | Reactive oxygen species (ROS) | Aneuploidy | In cancer biology | Cancer associated fibroblasts | Autophagy | Genomic instability | The "field effect" | Nitric oxide (NO) | the "field effect" in cancer biology | nitric oxide (NO) | autophagy | NITRIC-OXIDE SYNTHASE | ONCOGENICALLY TRANSFORMED-CELLS | DOWN-REGULATION | anti-oxidant cancer therapy | mitochondrial dysfunction | reactive oxygen species (ROS) | HUMAN BREAST-CANCER | cancer associated fibroblasts | ARGININE METHYL-ESTER | CELL BIOLOGY | caveolin-1 | N-ACETYL CYSTEINE | PROSTATE-CANCER | aneuploidy | GENE-EXPRESSION | genomic instability | SUPEROXIDE-DISMUTASE | oxidative stress | COMPLEX-I DEFICIENCY | Cell Line | Genomic Instability | Reactive Oxygen Species - metabolism | Oxidative Stress | Coculture Techniques | Down-Regulation | Humans | Caveolin 1 - genetics | Mitochondria - metabolism | Breast Neoplasms - metabolism | Biological Evolution | Caveolin 1 - metabolism | Breast Neoplasms - genetics | RNA Interference | Female | DNA Damage | Histones - metabolism | Nitric Oxide Synthase Type III - metabolism | Lactic Acid - pharmacology | Mitochondria - physiology | Nitric Oxide - metabolism | Fibroblasts - metabolism | RNA, Small Interfering - metabolism | the “field effect” in cancer biology | Report
Journal Article
Blood, ISSN 0006-4971, 11/2018, Volume 132, Issue Supplement 1, pp. 3372 - 3372
Abstract CMV reactivation post HSCT has been associated with an expansion of the CD3/8 T cell (CD8) compartment. We reported that in patients undergoing T... 
Journal Article
Cell Cycle, ISSN 1538-4101, 12/2011, Volume 10, Issue 23, pp. 4047 - 4064
We have recently proposed a new mechanism for explaining energy transfer in cancer metabolism.  In this scenario, cancer cells behave as metabolic parasites,... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Oxidative phosphorylation (OXPHOS) | NADH dehydrogenase | Complex I | Mitophagy | Warburg's respiratory enzyme | Autophagy | Complex IV | Mitochondria | Respiratory chain | Cancer metabolism | Cytochrome c oxidase (COX) | Aerobic glycolysis | Metformin | Electron transport | Warburg effect | RESPIRATORY-CHAIN | STROMAL CAVEOLIN-1 | CELL BIOLOGY | BREAST-CANCER | LACTATE | electron transport | STRESS | aerobic glycolysis | complex IV | autophagy | mitochondria | metformin | FIBROBLASTS | cancer metabolism | mitophagy | MICROENVIRONMENT | complex I | ENERGY-METABOLISM | cytochrome c oxidase (COX) | EXPRESSION | respiratory chain | oxidative phosphorylation (OXPHOS) | Oxidative Stress | Epithelial Cells - drug effects | Humans | Gene Expression Regulation, Neoplastic | Tumor Microenvironment | Cell Line, Tumor - drug effects | Malonates - pharmacology | Mitochondrial Proteins - genetics | Breast Neoplasms - metabolism | Electron Transport Complex IV - metabolism | Neoplasm Metastasis | Mitochondria - genetics | Membrane Transport Proteins - genetics | Mitochondrial Proteins - metabolism | Biomarkers, Tumor - metabolism | Female | Membrane Transport Proteins - metabolism | Metformin - pharmacology | Computational Biology | Enzyme Inhibitors - pharmacology | Oxidative Phosphorylation | Receptors, Cell Surface - metabolism | Mitochondria - metabolism | Mitochondria - drug effects | Energy Metabolism | Glycolysis | Enzyme Activation | Receptors, Cell Surface - genetics | Warburg respiratory enzyme | Report
Journal Article
Cell Cycle, ISSN 1538-4101, 09/2010, Volume 9, Issue 17, pp. 3515 - 3533
Recently, using a co-culture system, we demonstrated that MCF7 epithelial cancer cells induce oxidative stress in adjacent cancer-associated fibroblasts,... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Caveolin-1 | Oxidative stress | TIGAR | NFκB | BNIP3 | Tumor stroma | Cancer associated fibroblasts | Mitophagy | Autophagy | HIF1 | Hypoxia | LC3 | Predictive biomarker | hypoxia | autophagy | ONCOGENICALLY TRANSFORMED-CELLS | NITRIC-OXIDE SYNTHASE | DOWN-REGULATION | INDUCIBLE FACTOR INDUCTION | cancer associated fibroblasts | CELL BIOLOGY | BREAST-CANCER | caveolin-1 | mitophagy | predictive biomarker | tumor stroma | CAROTID-BODY | EXPRESSION | NF kappa B | oxidative stress | MITOCHONDRIAL COMPLEX-III | Up-Regulation | Oxidative Stress | Microtubule-Associated Proteins - metabolism | Coculture Techniques | Humans | Tumor Microenvironment | NF-kappa B - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Breast Neoplasms - metabolism | Cell Hypoxia | Chloroquine - pharmacology | RNA Interference | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Female | Glutathione Synthase - metabolism | Membrane Proteins - metabolism | Antirheumatic Agents - pharmacology | Glutathione Synthase - antagonists & inhibitors | Fibroblasts - metabolism | Proto-Oncogene Proteins - metabolism | Tumor Suppressor Proteins - metabolism | Paracrine Communication | Cell Survival | Stromal Cells - metabolism | Caveolin 1 - genetics | Mice, Knockout | Caveolin 1 - metabolism | Animals | Breast Neoplasms - pathology | Cell Line, Tumor | Mice | RNA, Small Interfering - metabolism | Report | cancer-associated fibroblasts
Journal Article
Cell Cycle, ISSN 1538-4101, 09/2010, Volume 9, Issue 17, pp. 3485 - 3505
Journal Article
Seminars in Oncology, ISSN 0093-7754, 12/2012, Volume 39, Issue 6, pp. 627 - 628
Journal Article
Seminars in Oncology, ISSN 0093-7754, 02/2012, Volume 39, Issue 6, pp. 694 - 706
Strategies that exploit natural killer (NK) cell alloreactivity or attenuate rather than deplete T cells have resulted in improved outcomes after... 
Journal Article
Cancer Biology & Therapy, ISSN 1538-4047, 11/2011, Volume 12, Issue 10, pp. 924 - 938
Here, we show that tamoxifen resistance is induced by cancer-associated fibroblasts (CAFs). Coculture of estrogen receptor positive (ER+) MCF7 cells with... 
Warburg Effect | TIGAR | metformin | reactive oxygen species (ROS) | cancer associated fibroblasts | microenvironment | glucose uptake | drug resistance | tumor stroma | mitochondrial oxidative phosphorylation | aerobic glycolysis | tamoxifen | oxidative stress | Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Oxidative stress | Reactive oxygen species (ROS) | Tumor stroma | Cancer associated fibroblasts | Microenvironment | Drug resistance | Tamoxifen | Glucose uptake | Aerobic glycolysis | Metformin | Mitochondrial oxidative phosphorylation | Warburg effect | CYCLIN D1 | TAMOXIFEN RESISTANCE | C-MYC | ENDOCRINE THERAPY | ESTROGEN-RECEPTOR | FIBROBLASTS | NEOADJUVANT CHEMOTHERAPY | CAVEOLIN-1 EXPRESSION | ONCOLOGY | NITRIC-OXIDE | Estradiol - analogs & derivatives | Apoptosis - drug effects | Coculture Techniques | Humans | Ketone Bodies - metabolism | Drug Resistance, Neoplasm | Intracellular Signaling Peptides and Proteins - metabolism | Breast Neoplasms - metabolism | Estrogen Receptor Modulators - therapeutic use | Female | Estradiol - pharmacology | Fibroblasts - metabolism | Tumor Microenvironment - drug effects | Estradiol - therapeutic use | Arsenicals - pharmacology | Lactic Acid - metabolism | Metformin - pharmacology | Mitochondria - metabolism | Mitochondria - drug effects | Breast Neoplasms - drug therapy | Poly(ADP-ribose) Polymerase Inhibitors | Estrogen Receptor Modulators - pharmacology | Hypoglycemic Agents - pharmacology | Oxides - pharmacology | Fibroblasts - drug effects | Tamoxifen - therapeutic use | Tamoxifen - pharmacology | Cell Line, Tumor | Doxorubicin - pharmacology | Index Medicus | Research Paper
Journal Article
Cell Cycle, ISSN 1538-4101, 06/2010, Volume 9, Issue 12, pp. 2423 - 2433
Loss of stromal caveolin 1 (Cav-1) is a novel biomarker for cancer-associated fibroblasts that predicts poor clinical outcome in breast cancer and DCIS... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Caveolin-1 | Lysosomal degradation | Chloroquine | Myofibroblast | Tumor stroma | TGFbeta signaling | Cancer associated fibroblasts | Autophagy | ACTIVATION | autophagy | TGF-BETA | lysosomal degradation | myofibroblast | SYSTEMIC-SCLEROSIS | cancer associated fibroblasts | CELL BIOLOGY | chloroquine | caveolin-1 | GROWTH | GENE-EXPRESSION | STROMAL FIBROBLASTS | VIMENTIN EXPRESSION | tumor stroma | TENASCIN | MMP-9 | PROGRESSION | Prognosis | Extracellular Matrix Proteins - biosynthesis | Coculture Techniques | Humans | Carcinoma, Intraductal, Noninfiltrating - metabolism | Transforming Growth Factor beta - biosynthesis | Autophagy - drug effects | Actins - genetics | Breast Neoplasms - metabolism | Chloroquine - pharmacology | Smad2 Protein - biosynthesis | Vimentin - genetics | Female | Microfilament Proteins - genetics | Fibroblasts - metabolism | Actins - biosynthesis | Calcium-Binding Proteins - biosynthesis | Vimentin - biosynthesis | Extracellular Matrix Proteins - genetics | Caveolin 1 - genetics | Caveolin 1 - metabolism | Microfilament Proteins - biosynthesis | Phenotype | Cell Line, Tumor | Biomarkers, Tumor - genetics | Transforming Growth Factor beta - metabolism | Biomarkers, Tumor - biosynthesis | Calcium-Binding Proteins - genetics
Journal Article