The Journal of Allergy and Clinical Immunology, ISSN 0091-6749, 2006, Volume 118, Issue 2, pp. 389 - 395
Epidemiologic data indicate an increased incidence of asthma in the obese. Because serum levels of the insulin-sensitizing and anti-inflammatory adipokine...
airway responsiveness | T-cadherin | adiponectin receptor 1 | adiponectin receptor 2 | Lung | IgE | adipocyte | IL-5 | IL-13 | eosinophil | WEIGHT-REDUCTION | NECROSIS-FACTOR-ALPHA | IMMUNOLOGY | lung | OBESE MICE | ASTHMA SEVERITY | ALLERGY | INSULIN-RESISTANCE | MONOCYTE-DERIVED MACROPHAGES | BODY-MASS INDEX | PLASMA-PROTEIN | E-DEFICIENT MICE | ADIPOSE-TISSUE | Bronchial Hyperreactivity - immunology | Cadherins - metabolism | Pneumonia - prevention & control | Bronchoalveolar Lavage Fluid - immunology | Male | Immunoglobulin E - blood | Adipose Tissue - metabolism | RNA, Messenger - biosynthesis | Receptors, Adiponectin | Adiponectin - genetics | Pneumonia - immunology | Female | Bronchoalveolar Lavage Fluid - cytology | Cadherins - genetics | Adiponectin - pharmacology | Receptors, Cell Surface - metabolism | Bronchial Hyperreactivity - prevention & control | Recombinant Proteins - pharmacology | Lung - physiology | Ovalbumin - pharmacology | Adiponectin - blood | Gene Expression Regulation - drug effects | Animals | Lung - drug effects | Leukocytes - drug effects | Allergens - pharmacology | Mice | Mice, Inbred BALB C | Adipose Tissue - drug effects | Receptors, Cell Surface - genetics | Albumin | Peptide hormones | Public health | Asthma | Human subjects | Obesity | Weight control | Lungs | Rodents | Protein expression | Adipocytes | Gene expression
airway responsiveness | T-cadherin | adiponectin receptor 1 | adiponectin receptor 2 | Lung | IgE | adipocyte | IL-5 | IL-13 | eosinophil | WEIGHT-REDUCTION | NECROSIS-FACTOR-ALPHA | IMMUNOLOGY | lung | OBESE MICE | ASTHMA SEVERITY | ALLERGY | INSULIN-RESISTANCE | MONOCYTE-DERIVED MACROPHAGES | BODY-MASS INDEX | PLASMA-PROTEIN | E-DEFICIENT MICE | ADIPOSE-TISSUE | Bronchial Hyperreactivity - immunology | Cadherins - metabolism | Pneumonia - prevention & control | Bronchoalveolar Lavage Fluid - immunology | Male | Immunoglobulin E - blood | Adipose Tissue - metabolism | RNA, Messenger - biosynthesis | Receptors, Adiponectin | Adiponectin - genetics | Pneumonia - immunology | Female | Bronchoalveolar Lavage Fluid - cytology | Cadherins - genetics | Adiponectin - pharmacology | Receptors, Cell Surface - metabolism | Bronchial Hyperreactivity - prevention & control | Recombinant Proteins - pharmacology | Lung - physiology | Ovalbumin - pharmacology | Adiponectin - blood | Gene Expression Regulation - drug effects | Animals | Lung - drug effects | Leukocytes - drug effects | Allergens - pharmacology | Mice | Mice, Inbred BALB C | Adipose Tissue - drug effects | Receptors, Cell Surface - genetics | Albumin | Peptide hormones | Public health | Asthma | Human subjects | Obesity | Weight control | Lungs | Rodents | Protein expression | Adipocytes | Gene expression
Journal Article
2.
Full Text
Nivolumab-Induced Subcutaneous Fat Necrosis: Another FDG-Avid Immune-Related Adverse Event
Clinical Nuclear Medicine, ISSN 0363-9762, 11/2019, p. 1
ABSTRACTA 59-year-old woman with history of metastatic melanoma, currently on nivolumab, presents for a restaging FDG PET/CT scan. New subcutaneous...
Journal Article
The Journal of Allergy and Clinical Immunology, ISSN 0091-6749, 2005, Volume 115, Issue 1, pp. 103 - 109
Epidemiologic data indicate that the incidence of asthma is increased in obese patients. Because the serum levels of the satiety hormone and proinflammatory...
Airway responsiveness | cytokines | bronchoalveolar lavage | leptin | IgE | ovalbumin | airway responsiveness | PROLIFERATION | IMMUNOLOGY | HYPERRESPONSIVENESS | MURINE ASTHMA MODEL | ALLERGY | CELL ACTIVATION | IMMUNE-RESPONSES | OBESE GENE | BODY-MASS INDEX | WEIGHT | EXPRESSION | GENE-PRODUCT | Allergens | Ovalbumin | Infusion Pumps, Implantable | Respiratory Hypersensitivity - etiology | Cytokines - analysis | Bronchoalveolar Lavage Fluid - immunology | RNA, Messenger - analysis | Male | Administration, Cutaneous | Respiratory Hypersensitivity - immunology | Immunoglobulin E - blood | Respiratory Hypersensitivity - blood | Animals | Leptin - blood | Female | Bronchoalveolar Lavage Fluid - cytology | Leptin - immunology | Leptin - pharmacology | Mice | Mice, Inbred BALB C | Methacholine Chloride | Lung - immunology | Disease Models, Animal | Phosphates | Leptin | Sugars | Monosaccharides | Asthma | Aerosols | Obesity | Rodents
Airway responsiveness | cytokines | bronchoalveolar lavage | leptin | IgE | ovalbumin | airway responsiveness | PROLIFERATION | IMMUNOLOGY | HYPERRESPONSIVENESS | MURINE ASTHMA MODEL | ALLERGY | CELL ACTIVATION | IMMUNE-RESPONSES | OBESE GENE | BODY-MASS INDEX | WEIGHT | EXPRESSION | GENE-PRODUCT | Allergens | Ovalbumin | Infusion Pumps, Implantable | Respiratory Hypersensitivity - etiology | Cytokines - analysis | Bronchoalveolar Lavage Fluid - immunology | RNA, Messenger - analysis | Male | Administration, Cutaneous | Respiratory Hypersensitivity - immunology | Immunoglobulin E - blood | Respiratory Hypersensitivity - blood | Animals | Leptin - blood | Female | Bronchoalveolar Lavage Fluid - cytology | Leptin - immunology | Leptin - pharmacology | Mice | Mice, Inbred BALB C | Methacholine Chloride | Lung - immunology | Disease Models, Animal | Phosphates | Leptin | Sugars | Monosaccharides | Asthma | Aerosols | Obesity | Rodents
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 05/2006, Volume 290, Issue 5, pp. 856 - 865
Epidemiological studies indicate the incidence of asthma is increased in obese and overweight humans. Responses to ozone (O3), an asthma trigger, are increased...
Macrophage inflammatory protein-2 | Airway responsiveness | Ventilation | Neutrophil | Interleukin-1β | Leptin | airway responsiveness | PHYSIOLOGY | WEIGHT-REDUCTION | ventilation | NECROSIS-FACTOR-ALPHA | leptin | neutrophil | SPLICE VARIANTS | ASTHMA SEVERITY | IN-VITRO | interleukin-1 beta | RESPIRATORY SYSTEM | INSULIN-RESISTANCE | macrophage inflammatory protein-2 | BODY-MASS INDEX | AIRWAY HYPERRESPONSIVENESS | LEPTIN RECEPTOR | TNF-ALPHA | Bronchoalveolar Lavage Fluid | Lung - pathology | Neutrophils - physiology | Obesity - physiopathology | Ozone - toxicity | Respiratory Physiological Phenomena - drug effects | Lung - physiopathology | DNA Primers | RNA - genetics | Animals | Mice, Mutant Strains | Base Sequence | Lung - drug effects | Interleukin-6 - physiology | Polymerase Chain Reaction | Mice | Leptin - deficiency
Macrophage inflammatory protein-2 | Airway responsiveness | Ventilation | Neutrophil | Interleukin-1β | Leptin | airway responsiveness | PHYSIOLOGY | WEIGHT-REDUCTION | ventilation | NECROSIS-FACTOR-ALPHA | leptin | neutrophil | SPLICE VARIANTS | ASTHMA SEVERITY | IN-VITRO | interleukin-1 beta | RESPIRATORY SYSTEM | INSULIN-RESISTANCE | macrophage inflammatory protein-2 | BODY-MASS INDEX | AIRWAY HYPERRESPONSIVENESS | LEPTIN RECEPTOR | TNF-ALPHA | Bronchoalveolar Lavage Fluid | Lung - pathology | Neutrophils - physiology | Obesity - physiopathology | Ozone - toxicity | Respiratory Physiological Phenomena - drug effects | Lung - physiopathology | DNA Primers | RNA - genetics | Animals | Mice, Mutant Strains | Base Sequence | Lung - drug effects | Interleukin-6 - physiology | Polymerase Chain Reaction | Mice | Leptin - deficiency
Journal Article
Journal of nuclear medicine technology, 06/2019
Waldenström's Macroglobulinemia (WM) is an indolent B-cell lymphoproliferative disorder. When there is involvement of the CNS, WM is known as Bing Neel...
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 03/2015, Volume 33, Issue 10, pp. 1216 - 1217
ONCOLOGY | RESPONSE ASSESSMENT | Hodgkin Disease - diagnostic imaging | Lymphoma - therapy | Lymphoma - diagnostic imaging | Lymphoma, Non-Hodgkin - therapy | Hodgkin Disease - therapy | Humans | Fluorodeoxyglucose F18 | Tomography, X-Ray Computed | Positron-Emission Tomography | Lymphoma, Non-Hodgkin - diagnostic imaging | Radiopharmaceuticals
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 07/2011, Volume 45, Issue 1, pp. 111 - 119
Airway smooth muscle (ASM) cells have been reported to contribute to the inflammation of asthma. Because the thiazolidinediones (TZDs) exert anti-inflammatory...
TNF-α | IL-1β | shRNA | Anti-inflammatory | PPARγ | ACTIVATED PROTEIN-KINASE | 5-AMINOIMIDAZOLE-4-CARBOXAMIDE RIBONUCLEOSIDE | TNF-alpha | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR-GAMMA AGONISTS | IL-1 beta | NECROSIS-FACTOR-ALPHA | PPAR gamma | HUMAN ADIPOCYTES | CELL BIOLOGY | MURINE MODEL | RESPIRATORY SYSTEM | anti-inflammatory | GENE-EXPRESSION | PPAR-GAMMA | BINDING-SITES | NF-KAPPA-B | Enzyme Activators - pharmacology | Respiratory System - pathology | AMP-Activated Protein Kinases - metabolism | Asthma - metabolism | Humans | Myocytes, Smooth Muscle - pathology | PPAR gamma - metabolism | Aminoimidazole Carboxamide - pharmacology | Gene Knockdown Techniques | Ribonucleotides - pharmacology | Chromans - pharmacology | Inflammation Mediators - metabolism | Thiazolidinediones - pharmacology | Cytokines - genetics | Myocytes, Smooth Muscle - metabolism | Antimetabolites - pharmacology | PPAR gamma - genetics | Pyrazoles - pharmacology | Anti-Inflammatory Agents - pharmacology | AMP-Activated Protein Kinases - antagonists & inhibitors | Metformin - pharmacology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Pyrimidines - pharmacology | Asthma - drug therapy | Asthma - genetics | Hypoglycemic Agents - pharmacology | Vidarabine - pharmacology | PPAR gamma - antagonists & inhibitors | Aminoimidazole Carboxamide - analogs & derivatives | Respiratory System - metabolism | Anilides - pharmacology | Rosiglitazone | Asthma - pathology | AMP-Activated Protein Kinases - genetics | Cytokines - biosynthesis | Troglitazone
TNF-α | IL-1β | shRNA | Anti-inflammatory | PPARγ | ACTIVATED PROTEIN-KINASE | 5-AMINOIMIDAZOLE-4-CARBOXAMIDE RIBONUCLEOSIDE | TNF-alpha | BIOCHEMISTRY & MOLECULAR BIOLOGY | RECEPTOR-GAMMA AGONISTS | IL-1 beta | NECROSIS-FACTOR-ALPHA | PPAR gamma | HUMAN ADIPOCYTES | CELL BIOLOGY | MURINE MODEL | RESPIRATORY SYSTEM | anti-inflammatory | GENE-EXPRESSION | PPAR-GAMMA | BINDING-SITES | NF-KAPPA-B | Enzyme Activators - pharmacology | Respiratory System - pathology | AMP-Activated Protein Kinases - metabolism | Asthma - metabolism | Humans | Myocytes, Smooth Muscle - pathology | PPAR gamma - metabolism | Aminoimidazole Carboxamide - pharmacology | Gene Knockdown Techniques | Ribonucleotides - pharmacology | Chromans - pharmacology | Inflammation Mediators - metabolism | Thiazolidinediones - pharmacology | Cytokines - genetics | Myocytes, Smooth Muscle - metabolism | Antimetabolites - pharmacology | PPAR gamma - genetics | Pyrazoles - pharmacology | Anti-Inflammatory Agents - pharmacology | AMP-Activated Protein Kinases - antagonists & inhibitors | Metformin - pharmacology | Cells, Cultured | Enzyme Inhibitors - pharmacology | Pyrimidines - pharmacology | Asthma - drug therapy | Asthma - genetics | Hypoglycemic Agents - pharmacology | Vidarabine - pharmacology | PPAR gamma - antagonists & inhibitors | Aminoimidazole Carboxamide - analogs & derivatives | Respiratory System - metabolism | Anilides - pharmacology | Rosiglitazone | Asthma - pathology | AMP-Activated Protein Kinases - genetics | Cytokines - biosynthesis | Troglitazone
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 02/2005, Volume 288, Issue 2, pp. 390 - 397
This study sought to examine the role of interleukin-6 (IL-6) in ozone (O3)-induced airway injury, inflammation, and hyperresponsiveness (AHR). Subacute (72 h)...
Airway responsiveness | Inflammation | Neutrophil | Injury | Soluble tumor necrosis factor receptor | airway responsiveness | NECROSIS-FACTOR RECEPTOR | PHYSIOLOGY | INDUCED NEUTROPHIL | UNRESTRAINED PLETHYSMOGRAPHY | ANTIINFLAMMATORY CYTOKINE | neutrophil | EMERGENCY-ROOM VISITS | BAROMETRIC PLETHYSMOGRAPHY | EPITHELIAL-CELLS | inflammation | INDUCED LUNG INFLAMMATION | RESPIRATORY SYSTEM | soluble tumor necrosis factor receptor | ALVEOLAR MACROPHAGES | injury | TRANSGENIC MICE | Receptors, Tumor Necrosis Factor, Type II - chemistry | Male | Ozone - administration & dosage | Receptors, Tumor Necrosis Factor, Type I - metabolism | RNA, Messenger - metabolism | Bronchi - drug effects | Receptors, Tumor Necrosis Factor, Type I - chemistry | Receptors, Tumor Necrosis Factor, Type II - metabolism | Bronchial Hyperreactivity - physiopathology | Interleukin-6 - physiology | Bronchi - metabolism | Female | Lung - metabolism | Neutrophils - pathology | Bronchial Hyperreactivity - chemically induced | Drug Administration Schedule | Interleukin-6 - genetics | Mice, Inbred C57BL | Solubility | Interleukin-6 - deficiency | Mice, Knockout | Bronchial Hyperreactivity - pathology | Animals | Ozone - pharmacology | Mice | Bronchoalveolar Lavage Fluid - chemistry
Airway responsiveness | Inflammation | Neutrophil | Injury | Soluble tumor necrosis factor receptor | airway responsiveness | NECROSIS-FACTOR RECEPTOR | PHYSIOLOGY | INDUCED NEUTROPHIL | UNRESTRAINED PLETHYSMOGRAPHY | ANTIINFLAMMATORY CYTOKINE | neutrophil | EMERGENCY-ROOM VISITS | BAROMETRIC PLETHYSMOGRAPHY | EPITHELIAL-CELLS | inflammation | INDUCED LUNG INFLAMMATION | RESPIRATORY SYSTEM | soluble tumor necrosis factor receptor | ALVEOLAR MACROPHAGES | injury | TRANSGENIC MICE | Receptors, Tumor Necrosis Factor, Type II - chemistry | Male | Ozone - administration & dosage | Receptors, Tumor Necrosis Factor, Type I - metabolism | RNA, Messenger - metabolism | Bronchi - drug effects | Receptors, Tumor Necrosis Factor, Type I - chemistry | Receptors, Tumor Necrosis Factor, Type II - metabolism | Bronchial Hyperreactivity - physiopathology | Interleukin-6 - physiology | Bronchi - metabolism | Female | Lung - metabolism | Neutrophils - pathology | Bronchial Hyperreactivity - chemically induced | Drug Administration Schedule | Interleukin-6 - genetics | Mice, Inbred C57BL | Solubility | Interleukin-6 - deficiency | Mice, Knockout | Bronchial Hyperreactivity - pathology | Animals | Ozone - pharmacology | Mice | Bronchoalveolar Lavage Fluid - chemistry
Journal Article
Journal of Nuclear Medicine Technology, ISSN 0091-4916, 2014, Volume 42, Issue 3, pp. 238 - 239
Stress injury is a common cause of exercise-induced anterior shin pain. It is important to distinguish between the various causes of stress injury in a timely...
Molecular imaging | Joint/muscle | Pediatrics | Bone | Medial tibial stress syndrome | Bone scan | Diagnosis, Differential | Technetium Tc 99m Medronate | Humans | Tibia - diagnostic imaging | Medial Tibial Stress Syndrome - diagnostic imaging | Tibia - injuries | Medial Tibial Stress Syndrome - complications | Pain - diagnostic imaging | Adolescent | Radionuclide Imaging | Pain - etiology | Running | Female | Radiopharmaceuticals
Molecular imaging | Joint/muscle | Pediatrics | Bone | Medial tibial stress syndrome | Bone scan | Diagnosis, Differential | Technetium Tc 99m Medronate | Humans | Tibia - diagnostic imaging | Medial Tibial Stress Syndrome - diagnostic imaging | Tibia - injuries | Medial Tibial Stress Syndrome - complications | Pain - diagnostic imaging | Adolescent | Radionuclide Imaging | Pain - etiology | Running | Female | Radiopharmaceuticals
Journal Article
American Journal of Respiratory and Critical Care Medicine, ISSN 1073-449X, 10/2007, Volume 176, Issue 7, pp. 650 - 658
Rationale: Epidemiologic data indicate an increased incidence of asthma in the obese. Objectives: To determine whether obese mice exhibit augmented pulmonary...
Immunoglobulin E | Pulmonary resistance | Airway responsiveness | Eosinophil | Interleukin-13 | airway responsiveness | C-REACTIVE PROTEIN | WEIGHT-REDUCTION | ACUTE OZONE EXPOSURE | NECROSIS-FACTOR-ALPHA | immunoglobulin E | interleukin-13 | pulmonary resistance | PULMONARY RESPONSES | RESPIRATORY SYSTEM | INSULIN-RESISTANCE | INFLAMMATION | BODY-MASS INDEX | ASTHMA | eosinophil | RESPIRATORY SYMPTOMS | CRITICAL CARE MEDICINE | Ovalbumin | Immunization | Lung - pathology | Receptors, Tumor Necrosis Factor, Type I - blood | Cytokines - metabolism | Obesity - complications | Mice, Inbred C57BL | Bronchial Provocation Tests | Respiratory Hypersensitivity - complications | Respiratory Hypersensitivity - metabolism | Obesity - physiopathology | Airway Resistance | Respiratory Hypersensitivity - pathology | Immunoglobulin E - blood | Animals | Receptors, Tumor Necrosis Factor, Type II - blood | Respiratory Hypersensitivity - physiopathology | Mice, Obese | Bronchoalveolar Lavage Fluid - cytology | Mice | Bronchoalveolar Lavage Fluid - chemistry | Bronchoconstrictor Agents | Methacholine Chloride | Th2 Cells - pathology | A. Asthma and Allergy
Immunoglobulin E | Pulmonary resistance | Airway responsiveness | Eosinophil | Interleukin-13 | airway responsiveness | C-REACTIVE PROTEIN | WEIGHT-REDUCTION | ACUTE OZONE EXPOSURE | NECROSIS-FACTOR-ALPHA | immunoglobulin E | interleukin-13 | pulmonary resistance | PULMONARY RESPONSES | RESPIRATORY SYSTEM | INSULIN-RESISTANCE | INFLAMMATION | BODY-MASS INDEX | ASTHMA | eosinophil | RESPIRATORY SYMPTOMS | CRITICAL CARE MEDICINE | Ovalbumin | Immunization | Lung - pathology | Receptors, Tumor Necrosis Factor, Type I - blood | Cytokines - metabolism | Obesity - complications | Mice, Inbred C57BL | Bronchial Provocation Tests | Respiratory Hypersensitivity - complications | Respiratory Hypersensitivity - metabolism | Obesity - physiopathology | Airway Resistance | Respiratory Hypersensitivity - pathology | Immunoglobulin E - blood | Animals | Receptors, Tumor Necrosis Factor, Type II - blood | Respiratory Hypersensitivity - physiopathology | Mice, Obese | Bronchoalveolar Lavage Fluid - cytology | Mice | Bronchoalveolar Lavage Fluid - chemistry | Bronchoconstrictor Agents | Methacholine Chloride | Th2 Cells - pathology | A. Asthma and Allergy
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 10/2007, Volume 37, Issue 4, pp. 477 - 484
Interleukin (IL)-1, a proinflammatory cytokine, is expressed in the lung after ozone (O-3) exposure. IL-1 mediates its effects through the type I IL-1...
Neutrophil | Bronchoalveolar lavage | Soluble tumor necrosis factor receptor | Interleukin-6 | Macrophage | BIOCHEMISTRY & MOLECULAR BIOLOGY | macrophage | NECROSIS-FACTOR-ALPHA | neutrophil | interleukin-6 | NEUTROPHIL RECRUITMENT | ESCHERICHIA-COLI PNEUMONIA | CELL BIOLOGY | EPITHELIAL-CELLS | RESPIRATORY SYSTEM | GENE-EXPRESSION | soluble tumor necrosis factor receptor | INFLAMMATORY CYTOKINES | bronchoalveolar lavage | INDUCED AIRWAY HYPERRESPONSIVENESS | NF-KAPPA-B | SMOOTH-MUSCLE CELLS | LUNG INFLAMMATION | Cell Count | Interleukin-1alpha - metabolism | Male | Interleukin-1beta - genetics | RNA, Messenger - metabolism | Receptors, Interleukin-1 Type I - metabolism | Interleukin-1beta - metabolism | Respiration - drug effects | Female | Bronchoalveolar Lavage Fluid - cytology | Interleukin-1alpha - genetics | Lung - metabolism | Interleukin-6 - metabolism | Receptors, Interleukin-1 Type I - deficiency | Lung - pathology | Interleukin-6 - genetics | Mice, Inbred C57BL | RNA, Messenger - genetics | Inflammation | Gene Expression Regulation - drug effects | Intercellular Adhesion Molecule-1 - metabolism | Animals | Ozone - pharmacology | Intercellular Adhesion Molecule-1 - genetics | Lung - drug effects | Mice
Neutrophil | Bronchoalveolar lavage | Soluble tumor necrosis factor receptor | Interleukin-6 | Macrophage | BIOCHEMISTRY & MOLECULAR BIOLOGY | macrophage | NECROSIS-FACTOR-ALPHA | neutrophil | interleukin-6 | NEUTROPHIL RECRUITMENT | ESCHERICHIA-COLI PNEUMONIA | CELL BIOLOGY | EPITHELIAL-CELLS | RESPIRATORY SYSTEM | GENE-EXPRESSION | soluble tumor necrosis factor receptor | INFLAMMATORY CYTOKINES | bronchoalveolar lavage | INDUCED AIRWAY HYPERRESPONSIVENESS | NF-KAPPA-B | SMOOTH-MUSCLE CELLS | LUNG INFLAMMATION | Cell Count | Interleukin-1alpha - metabolism | Male | Interleukin-1beta - genetics | RNA, Messenger - metabolism | Receptors, Interleukin-1 Type I - metabolism | Interleukin-1beta - metabolism | Respiration - drug effects | Female | Bronchoalveolar Lavage Fluid - cytology | Interleukin-1alpha - genetics | Lung - metabolism | Interleukin-6 - metabolism | Receptors, Interleukin-1 Type I - deficiency | Lung - pathology | Interleukin-6 - genetics | Mice, Inbred C57BL | RNA, Messenger - genetics | Inflammation | Gene Expression Regulation - drug effects | Intercellular Adhesion Molecule-1 - metabolism | Animals | Ozone - pharmacology | Intercellular Adhesion Molecule-1 - genetics | Lung - drug effects | Mice
Journal Article
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 02/2006, Volume 34, Issue 2, pp. 213 - 218
Th2 cytokines induce the release of vascular endothelial growth factor (VEGF) from cultured human airway smooth muscle cells. The objective of this study was...
TNFα | Asthma | IL-4Rα | mRNA stability | Polymorphism | LUNG | TNF alpha | EOTAXIN RELEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | VEGF GENE | asthma | IL-4R alpha | polymorphism | MESSENGER-RNA STABILITY | IL-13 | IDENTIFICATION | CELL BIOLOGY | RESPIRATORY SYSTEM | UP-REGULATION | EXPRESSION | FACTOR GENE | Humans | Cells, Cultured | Trachea - drug effects | Trachea - metabolism | Vascular Endothelial Growth Factor A - metabolism | Interleukin-13 - pharmacology | Vascular Endothelial Growth Factor A - genetics | Dose-Response Relationship, Drug | Genetic Variation | Haplotypes - genetics | Interleukin-4 - pharmacology | Receptors, Interleukin-4 - genetics | Polymorphism, Single Nucleotide | Vascular Endothelial Growth Factor A - drug effects | Myocytes, Smooth Muscle - drug effects | Dactinomycin - pharmacology | Myocytes, Smooth Muscle - metabolism
TNFα | Asthma | IL-4Rα | mRNA stability | Polymorphism | LUNG | TNF alpha | EOTAXIN RELEASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | VEGF GENE | asthma | IL-4R alpha | polymorphism | MESSENGER-RNA STABILITY | IL-13 | IDENTIFICATION | CELL BIOLOGY | RESPIRATORY SYSTEM | UP-REGULATION | EXPRESSION | FACTOR GENE | Humans | Cells, Cultured | Trachea - drug effects | Trachea - metabolism | Vascular Endothelial Growth Factor A - metabolism | Interleukin-13 - pharmacology | Vascular Endothelial Growth Factor A - genetics | Dose-Response Relationship, Drug | Genetic Variation | Haplotypes - genetics | Interleukin-4 - pharmacology | Receptors, Interleukin-4 - genetics | Polymorphism, Single Nucleotide | Vascular Endothelial Growth Factor A - drug effects | Myocytes, Smooth Muscle - drug effects | Dactinomycin - pharmacology | Myocytes, Smooth Muscle - metabolism
Journal Article
Clinical Nuclear Medicine, ISSN 0363-9762, 07/2015, Volume 40, Issue 7, pp. 592 - 595
ABSTRACTThe patient is a 44-year-old woman with Crohn’s disease, who presented to the ED with abdominal pain. Initial imaging by CT and US showed mild biliary...
hepatobiliary | false negative | biliary leak | scintigraphy | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | Organotechnetium Compounds | Multimodal Imaging | Humans | Tomography, Emission-Computed, Single-Photon | Adult | Female | Tomography, X-Ray Computed | Biliary Tract Diseases - diagnostic imaging | Imino Acids | Radiopharmaceuticals
hepatobiliary | false negative | biliary leak | scintigraphy | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | Organotechnetium Compounds | Multimodal Imaging | Humans | Tomography, Emission-Computed, Single-Photon | Adult | Female | Tomography, X-Ray Computed | Biliary Tract Diseases - diagnostic imaging | Imino Acids | Radiopharmaceuticals
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 10/2003, Volume 285, Issue 4, pp. 907 - 914
The chemokine thymus- and activation-regulated chemokine (TARC) induces selective migration of Th2, but not Th1, lymphocytes and is upregulated in the airways...
Macrophage-derived chemokine | Interferon-γ | Interleukin-1β | Tumor necrosis factor-α | Asthma | Polymorphism | Thymus- and activation-regulated chemokine | interleukin-beta | ILE50VAL VARIANT | PHYSIOLOGY | BRONCHIAL EPITHELIAL-CELLS | EOTAXIN RELEASE | thymus- and activation-regulated chemokine | asthma | polymorphism | INTERLEUKIN-4 RECEPTOR | ACTIVATION-REGULATED CHEMOKINE | tumor necrosis factor-alpha | SIGNAL-TRANSDUCTION | macrophage-derived chemokine | ALPHA-CHAIN | RESPIRATORY SYSTEM | TNF-ALPHA | interferon-gamma | NF-KAPPA-B | Chemokine CCL17 | Trachea - cytology | Humans | Cells, Cultured | Genotype | Trachea - drug effects | Trachea - metabolism | Interleukin-13 - pharmacology | RNA, Messenger - metabolism | Interleukin-4 - pharmacology | Receptors, Interleukin-4 - physiology | Receptors, Interleukin-4 - genetics | Chemokines, CC - genetics | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Chemokines, CC - metabolism
Macrophage-derived chemokine | Interferon-γ | Interleukin-1β | Tumor necrosis factor-α | Asthma | Polymorphism | Thymus- and activation-regulated chemokine | interleukin-beta | ILE50VAL VARIANT | PHYSIOLOGY | BRONCHIAL EPITHELIAL-CELLS | EOTAXIN RELEASE | thymus- and activation-regulated chemokine | asthma | polymorphism | INTERLEUKIN-4 RECEPTOR | ACTIVATION-REGULATED CHEMOKINE | tumor necrosis factor-alpha | SIGNAL-TRANSDUCTION | macrophage-derived chemokine | ALPHA-CHAIN | RESPIRATORY SYSTEM | TNF-ALPHA | interferon-gamma | NF-KAPPA-B | Chemokine CCL17 | Trachea - cytology | Humans | Cells, Cultured | Genotype | Trachea - drug effects | Trachea - metabolism | Interleukin-13 - pharmacology | RNA, Messenger - metabolism | Interleukin-4 - pharmacology | Receptors, Interleukin-4 - physiology | Receptors, Interleukin-4 - genetics | Chemokines, CC - genetics | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Chemokines, CC - metabolism
Journal Article
American Journal of Physiology - Lung Cellular and Molecular Physiology, ISSN 1040-0605, 06/2005, Volume 288, Issue 6, pp. L1040 - L1048
Vascular endothelial growth factor (VEGF), a potent angiogenesis factor, likely contributes to airway remodeling in asthma. We sought to examine the effects...
Monocyte chemoattractant protein-1 | Interleukin-1 receptor 1 | Vascular endothelial growth factor | Piceatannol | Signal transducer and activator of transcription 3 | Interleukin-6 | PHYSIOLOGY | interleukin-1 receptor 1 | piceatannol | TRANSCRIPTION 3 | CARDIAC MYOCYTES | monocyte chemoattractant protein-1 | FACTOR MESSENGER-RNA | signal transducer and activator of transcription 3 | ASTHMATIC SUBJECTS | FIBROBLASTS IN-VITRO | interleukin-6 | STAT3 ACTIVATION | RESPIRATORY SYSTEM | vascular endothelial growth factor | GENE-EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | SIGNAL TRANSDUCER
Monocyte chemoattractant protein-1 | Interleukin-1 receptor 1 | Vascular endothelial growth factor | Piceatannol | Signal transducer and activator of transcription 3 | Interleukin-6 | PHYSIOLOGY | interleukin-1 receptor 1 | piceatannol | TRANSCRIPTION 3 | CARDIAC MYOCYTES | monocyte chemoattractant protein-1 | FACTOR MESSENGER-RNA | signal transducer and activator of transcription 3 | ASTHMATIC SUBJECTS | FIBROBLASTS IN-VITRO | interleukin-6 | STAT3 ACTIVATION | RESPIRATORY SYSTEM | vascular endothelial growth factor | GENE-EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | SIGNAL TRANSDUCER
Journal Article
Journal of Clinical Imaging Science, ISSN 2156-7514, 01/2014, Volume 4, Issue 1, p. 39
Journal Article
Journal of Clinical Imaging Science, ISSN 2156-7514, 01/2014, Volume 4, Issue 1, p. 39
Objectives: To investigate the effects of ambient glucose on quantitative analysis of hepatic tumors on 2-deoxy-2-(18F)-fluoro-D-glucose (18FDG) positron...
fluorodeoxyglucose positron emission tomography | Background liver | tumor to background ratio | hepatic metastases | glucose sensitivity | standardized uptake value
fluorodeoxyglucose positron emission tomography | Background liver | tumor to background ratio | hepatic metastases | glucose sensitivity | standardized uptake value
Journal Article
The Journal of Allergy and Clinical Immunology, ISSN 0091-6749, 2005, Volume 115, Issue 3, pp. 514 - 520
Eotaxin is implicated in asthmatic eosinophilia. Oncostatin M (OSM) causes eotaxin release from fibroblasts. We sought to examine the effects and mechanism of...
c-Jun N-terminal kinase | extracellular signal-regulated kinase | vascular endothelial growth factor | Signal transducer and activator of transcription 3 | monocyte chemoattractant protein 1 | IL-4Rα | Extracellular signal-regulated kinase | Vascular endothelial growth factor | Monocyte chemoattractant protein 1 | SIGNALING PATHWAYS | MYELOID PROGENITOR CELLS | EOSINOPHILIC INFLAMMATION | signal transducer and activator of transcription 3 | IL-4R alpha | LEUKEMIA INHIBITORY FACTOR | IMMUNOLOGY | FIBROBLASTS IN-VITRO | EPITHELIAL-CELLS | CHEMOKINE RELEASE | INTERFERON-GAMMA | ALLERGY | M RECEPTOR | EXPRESSION | Phosphorylation | Chemokines, CC - secretion | Trans-Activators - drug effects | Humans | Interleukin-13 - immunology | RNA, Messenger - analysis | Muscle, Smooth - immunology | DNA-Binding Proteins - metabolism | Muscle, Smooth - drug effects | Flow Cytometry | Chemokines, CC - immunology | Lung - metabolism | Mitogen-Activated Protein Kinases - immunology | Growth Inhibitors - pharmacology | Trans-Activators - immunology | DNA-Binding Proteins - immunology | Enzyme-Linked Immunosorbent Assay | Interleukin-4 - metabolism | DNA-Binding Proteins - drug effects | Chemokine CCL11 | Muscle, Smooth - metabolism | Receptors, Interleukin-4 - immunology | Interleukin-13 - metabolism | Oncostatin M | Receptors, Interleukin-4 - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Peptides - pharmacology | Drug Synergism | Interleukin-4 - immunology | Lung - drug effects | Trans-Activators - metabolism | STAT3 Transcription Factor | Mitogen-Activated Protein Kinases - drug effects | Lung - immunology | Mitogen-Activated Protein Kinases - metabolism | Physiological aspects | Ciliary neurotrophic factor | Public health | Endothelium | Proteins | Cytokines | Rodents | Smooth muscle | Muscular system | Kinases | Experiments | Asthma
c-Jun N-terminal kinase | extracellular signal-regulated kinase | vascular endothelial growth factor | Signal transducer and activator of transcription 3 | monocyte chemoattractant protein 1 | IL-4Rα | Extracellular signal-regulated kinase | Vascular endothelial growth factor | Monocyte chemoattractant protein 1 | SIGNALING PATHWAYS | MYELOID PROGENITOR CELLS | EOSINOPHILIC INFLAMMATION | signal transducer and activator of transcription 3 | IL-4R alpha | LEUKEMIA INHIBITORY FACTOR | IMMUNOLOGY | FIBROBLASTS IN-VITRO | EPITHELIAL-CELLS | CHEMOKINE RELEASE | INTERFERON-GAMMA | ALLERGY | M RECEPTOR | EXPRESSION | Phosphorylation | Chemokines, CC - secretion | Trans-Activators - drug effects | Humans | Interleukin-13 - immunology | RNA, Messenger - analysis | Muscle, Smooth - immunology | DNA-Binding Proteins - metabolism | Muscle, Smooth - drug effects | Flow Cytometry | Chemokines, CC - immunology | Lung - metabolism | Mitogen-Activated Protein Kinases - immunology | Growth Inhibitors - pharmacology | Trans-Activators - immunology | DNA-Binding Proteins - immunology | Enzyme-Linked Immunosorbent Assay | Interleukin-4 - metabolism | DNA-Binding Proteins - drug effects | Chemokine CCL11 | Muscle, Smooth - metabolism | Receptors, Interleukin-4 - immunology | Interleukin-13 - metabolism | Oncostatin M | Receptors, Interleukin-4 - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Peptides - pharmacology | Drug Synergism | Interleukin-4 - immunology | Lung - drug effects | Trans-Activators - metabolism | STAT3 Transcription Factor | Mitogen-Activated Protein Kinases - drug effects | Lung - immunology | Mitogen-Activated Protein Kinases - metabolism | Physiological aspects | Ciliary neurotrophic factor | Public health | Endothelium | Proteins | Cytokines | Rodents | Smooth muscle | Muscular system | Kinases | Experiments | Asthma
Journal Article
Practical Radiation Oncology, ISSN 1879-8500, 03/2017, Volume 7, Issue 2, pp. 120 - 125
Evaluation of local tumor progression (LP) has typically been defined by contrast-enhanced computed tomography (CT) imaging after stereotactic body radiation...
EFFICACY | ONCOLOGY | PHASE-II | CHEMORADIOTHERAPY | RADIOSURGERY | RADIOTHERAPY | OUTCOMES | CANCER | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | GEMCITABINE | Adenocarcinoma | CT imaging | Care and treatment | PET imaging | Pancreatic cancer | Patient outcomes | Development and progression | Radiotherapy | Comparative analysis | Methods
EFFICACY | ONCOLOGY | PHASE-II | CHEMORADIOTHERAPY | RADIOSURGERY | RADIOTHERAPY | OUTCOMES | CANCER | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | GEMCITABINE | Adenocarcinoma | CT imaging | Care and treatment | PET imaging | Pancreatic cancer | Patient outcomes | Development and progression | Radiotherapy | Comparative analysis | Methods
Journal Article