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Journal of Counseling & Development, ISSN 0748-9633, 01/2018, Volume 96, Issue 1, pp. 86 - 96
Using consensual qualitative research, the authors examined the treatment planning process of experienced counselors (N = 9). The data analysis resulted in 4... 
treatment planning | clinical mental health counseling | REPLAN model | common factors | contextual approach | PSYCHOLOGY, APPLIED | PSYCHOTHERAPY | UPDATE | Qualitative research | Impressions | Mental health care | Counseling | Planning | Expertise | Clinical assessment
Journal Article
2003, 1, ISBN 0833032925, xxiv, 390
The collapse of the Soviet Union and the end of the Cold WarÑand then the terrorist attacks of September 11, 2001Ñtransformed the task of American foreign and... 
World politics | United States Defenses | United States Military policyDecision making | National security | 21st century | United States | Decision making | Defenses | Military policy | Political Science | National Security | Terrorism and Homeland Security | Global Security Environment | International Affairs
Book
American Journal of Physiology, ISSN 0193-1849, 01/2015, Volume 308, Issue 2, p. E144
  Skeletal muscle atrophy is a common and debilitating condition that remains poorly understood at the molecular level. To better understand the mechanisms of... 
Musculoskeletal system | Pathogenesis | Ribonucleic acid--RNA | Gene expression
Journal Article
American Journal of Physiology, ISSN 0193-1849, 08/2014, Volume 307, Issue 3, p. E245
  Immobilization causes skeletal muscle atrophy via complex signaling pathways that are not well understood. To better understand these pathways, we... 
Musculoskeletal system | Rodents | Genomes | Ribonucleic acid--RNA
Journal Article
Cell Metabolism, ISSN 1550-4131, 2011, Volume 13, Issue 6, pp. 627 - 638
Journal Article
American Journal of Physiology, ISSN 0193-1849, 10/2013, Volume 305, Issue 7, p. E907
  Skeletal muscle denervation causes muscle atrophy via complex molecular mechanisms that are not well understood. To better understand these mechanisms, we... 
Proteins | Musculoskeletal system | DNA damage | Rodents | Ribonucleic acid--RNA
Journal Article
Physical Chemistry Chemical Physics, ISSN 1463-9076, 10/2013, Volume 15, Issue 43, pp. 18992 - 19000
Alkaline anion-exchange membranes (AAEMs) containing cationic head-groups (e.g. involving quaternary ammonium and imidazolium groups) are of interest with... 
Bismaleimides | Electrodes | Reduction (electrolytic) | Platinum | Cationic | Tafel slopes | Electrolytes | Density
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2012, Volume 7, Issue 6, p. e39332
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 1782, Volume 289, Issue 21, pp. 14913 - 14924
Skeletal muscle atrophy is a common and debilitating condition that lacks an effective therapy. To address this problem, we used a systems-based discovery... 
HYPERTROPHY | ALPHA-TOMATINE | ACTIVATION | MANAGEMENT | BIOCHEMISTRY & MOLECULAR BIOLOGY | TOMATOES | SARCOPENIA | DEFICIENT MICE | EXPRESSION SIGNATURES | FAT MASS | PROTECTS | Gene Expression - drug effects | TOR Serine-Threonine Kinases - metabolism | Humans | Mitochondria, Muscle - metabolism | Drug Discovery - methods | Immunoblotting | Male | Multiprotein Complexes - genetics | Muscle, Skeletal - metabolism | Muscle Fibers, Skeletal - drug effects | Muscle Fibers, Skeletal - metabolism | Tomatine - pharmacology | Mechanistic Target of Rapamycin Complex 1 | Multiprotein Complexes - metabolism | Myoblasts, Skeletal - cytology | TOR Serine-Threonine Kinases - genetics | MCF-7 Cells | Muscle, Skeletal - drug effects | Muscle Proteins - metabolism | Muscular Atrophy - prevention & control | Mitochondria, Muscle - genetics | Myoblasts, Skeletal - metabolism | Cell Line | Muscular Atrophy - metabolism | Mice, Inbred C57BL | Cells, Cultured | Muscular Atrophy - genetics | Tomatine - analogs & derivatives | Reverse Transcriptase Polymerase Chain Reaction | Muscle Proteins - genetics | Animals | Signal Transduction - drug effects | Cell Line, Tumor | HL-60 Cells | Muscle Fibers, Skeletal - cytology | Mice | Mitochondria, Muscle - drug effects | Muscle, Skeletal - pathology | Myoblasts, Skeletal - drug effects | Skeletal Muscle | Skeletal Muscle Metabolism | Muscle Atrophy | Tomatidine | Metabolism | Systems Biology | mTOR complex (mTORC)
Journal Article
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 10/2013, Volume 305, Issue 7, pp. E907 - E915
Journal Article
PloS one, 06/2010, Volume 5, Issue 6, p. e11380
The tumor necrosis factor receptor (TNFR) superfamily mediates signals critical for regulation of the immune system. One family member, CD40, is important for... 
Cell Line | Animals | CD40 Antigens - metabolism | Carrier Proteins - metabolism | Immunoprecipitation | Signal Transduction | Electrophoresis, Polyacrylamide Gel | Mass Spectrometry | NF-kappa B - metabolism | Mice
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2012, Volume 287, Issue 33, pp. 27290 - 27301
Diverse stresses including starvation and muscle disuse cause skeletal muscle atrophy. However, the molecular mechanisms of muscle atrophy are complex and not... 
ACTIVATION | PROTEIN | PATHWAY | PGC-1-ALPHA | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENE-EXPRESSION | DEGRADATION | GENERATION | FOXO TRANSCRIPTION FACTORS | UBIQUITIN LIGASES | COACTIVATORS | Stress, Physiological | Muscle, Skeletal - metabolism | Proto-Oncogene Proteins c-akt - genetics | RNA, Messenger - biosynthesis | Cell Nucleus - metabolism | Cell Nucleus - pathology | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Muscle Proteins - metabolism | Trans-Activators - genetics | Cell Cycle Proteins - genetics | Protein Biosynthesis - genetics | Energy Metabolism - genetics | Nuclear Proteins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Cell Line | Muscular Atrophy - metabolism | Muscular Atrophy - pathology | RNA, Messenger - genetics | Cell Cycle Proteins - metabolism | Gene Expression Regulation | Activating Transcription Factor 4 - genetics | Muscular Atrophy - genetics | Nuclear Proteins - metabolism | Signal Transduction - genetics | Mice, Knockout | Muscle Proteins - genetics | Animals | Activating Transcription Factor 4 - metabolism | Cell Nucleus - genetics | Trans-Activators - metabolism | Mice | Transcription Factors | Muscle, Skeletal - pathology | Molecular Bases of Disease | Transcription Regulation | Skeletal Muscle | Skeletal Muscle Metabolism | Stress Response | Gadd45a | Muscle Atrophy | ATF4
Journal Article
Physical Chemistry Chemical Physics, ISSN 1463-9076, 11/2013, Volume 15, Issue 43, pp. 18992 - 19000
Journal Article
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 08/2014, Volume 307, Issue 3, pp. E245 - E261
Immobilization causes skeletal muscle atrophy via complex signaling pathways that are not well understood. To better understand these pathways, we investigated... 
Skeletal muscle atrophy | Immobilization | p21 | Skeletal muscle | Activating transcription factor 4 | p53 | ACTIVATION | PHYSIOLOGY | immobilization | PROTEIN | skeletal muscle atrophy | PHOSPHORYLATION | DNA-DAMAGE | INDUCTION | TRANSLATION | TOTAL KNEE ARTHROPLASTY | ENDOCRINOLOGY & METABOLISM | GENE-EXPRESSION | skeletal muscle | STRESS | activating transcription factor 4 | Up-Regulation | Male | Muscle, Skeletal - metabolism | Tumor Suppressor Protein p53 - genetics | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Muscular Atrophy - etiology | Recombinant Proteins - metabolism | Promoter Regions, Genetic | Genome-Wide Association Study | Disease Resistance | Muscular Atrophy - metabolism | Mice, Inbred C57BL | Muscular Atrophy - pathology | Tumor Suppressor Protein p53 - metabolism | Activating Transcription Factor 4 - genetics | Cyclin-Dependent Kinase Inhibitor p21 - antagonists & inhibitors | Mice, Transgenic | Mutant Proteins - metabolism | Immobilization - adverse effects | Hindlimb Suspension | Mice, Knockout | Animals | Activating Transcription Factor 4 - metabolism | MicroRNAs | Mice | Muscle, Skeletal - pathology | Amino Acid Substitution | Crosses, Genetic | Muscles | Genetic research | Genetic aspects | Research | Gene expression | Atrophy, Muscular | Health aspects
Journal Article